ライフサイエンスコーパス: dendritic cell

1 duced expression of IFN-I in macrophages and dendritic cells.

2 fferentiation, predominantly of plasmacytoid dendritic cells.

3 anslational profile of renal macrophages and dendritic cells.

4 itiate disease exacerbation via plasmacytoid dendritic cells.

5 pro-inflammatory responses in monocytes and dendritic cells.

6 ing leukocytes, including antigen-presenting dendritic cells.

7 diverse MPS populations, including classical dendritic cells.

8 acterized in vitro using bone marrow-derived dendritic cells.

9 inder initial activation of naive T-cells by dendritic cells.

10 s and an increased frequency of plasmacytoid dendritic cells.

11 s to regional lymph nodes through activating dendritic cells.

12 invades human gingival epithelial and blood-dendritic cells.

13 and CD11c(+) CD103(+) and CD11c(+) CD11b(+) dendritic cells.

14 hich is inducibly expressed on monocytes and dendritic cells.

15 mmation, primarily through the activation of dendritic cells.

16 ation of dead cell-derived antigens by mouse dendritic cells.

17 olated cf-mt-DNA capable of activating human dendritic cells.

18 as T follicular helper cells and follicular dendritic cells.

19 ion or functions of T cells, macrophages and dendritic cells.

20 -presented by professional APC, specifically dendritic cells.

21 mononuclear cells (PBMCs, 106,545 cells) and dendritic cells (19,806 cells) across all subjects and,

22 firmed increased frequencies of conventional dendritic cells (3.1% versus 2.1%; P = 0.023) and CD4 TN

23 nflammatory macrophage skewing and increased dendritic cell activation in the tumor microenvironment,

24 ostaglandin E(2) biosythesis favors CD103(+) dendritic cell activation that primes a Tc1-polarized CD

25 hat interfere with epithelial cell alarmins, dendritic cell activation, macrophage function and T-cel

26 s, CD8(+) T cells, B cells, macrophages, and dendritic cells after systemic delivery in mice.

27 filtrates CD3(+)/CD8(+) (T cells) and CD11c (dendritic cells) after 2 weeks of treatment, correspondi

28 is currently much interest in how different dendritic cell and macrophage populations contribute to

29 equired or contributed to BAFF expression in dendritic cell and MO subsets, respectively.

30 riasis tissues shared increases in levels of dendritic cell and T-cell markers (CD3, ITGAX/CD11c, and

31 ructures that contain T cells and follicular dendritic cells and are particularly rich in B cells.

32 y and detect couplings between monocytes and dendritic cells and between erythrocytes and basophils t

33 butyrate was mediated by GPR43 signaling in dendritic cells and by GPR43-independent signaling in T

34 subpopulations of ductal cells, macrophages, dendritic cells and cancer cells have spatially restrict

35 re statistically increased blood tolerogenic dendritic cells and cell phenotypes correlating with chr

36 HDM + DEP exposed bone marrow derived dendritic cells and IL33 pulsed BMDC promote a mixed Th2

37 n to be downstream of AhR in macrophages and dendritic cells and in livers of infected mice.

38 APCs, resulting in an 11.5-fold increase of dendritic cells and infiltration of lymphocytes througho

39 The interaction of dendritic cells and macrophages with a variety of rigid

40 ss both mMHCII antigen presentation in mouse dendritic cells and MHCII-dependent CD4(+) T cell activa

41 Antigen-presenting cells (APCs) including dendritic cells and monocytes instruct naive T cells to

42 d the frequencies and activation profiles of dendritic cells and monocytes present in the blood and l

43 erential contribution of specific subsets of dendritic cells and monocytes to ARDS is still poorly un

44 bers of the microbiota regulate plasmacytoid dendritic cells and monocytes via TLR7 and MyD88 signali

45 ions by recruiting FOXP3(+)T cells, CD11c(+) dendritic cells and natural killer cells.

46 ates the role of donor T cells in activating dendritic cells and positions GM-CSF-producing T cells a

47 umor cell-derived lactate activates GPR81 in dendritic cells and prevents presentation of tumor-speci

48 lycoprotein (MOG)(35-55) induced tolerogenic dendritic cells and suppressed the development of experi

49 thelial dysfunction, alter the phenotypes of dendritic cells and T and B lymphocytes, and lead to exa

50 esponses in the lungs that is independent of dendritic cells and T-cell priming in the draining lymph

51 a mechanism involving IL33 signaling in both dendritic cells and T-cells.

52 (-/-) mice led to its presentation by thymic dendritic cells and the deletion of Ag-specific thymocyt

53 ection in mice, with increased maturation of dendritic cells and the induction of protective CD8(+) T

54 ls have sustained contacts with intratumoral dendritic cells and tumor targets compared with low-avid

55 fessional' phagocytes (such as monocytes and dendritic cells) and 'non-professional' phagocytes, such

56 und that histologic grade, intratumoral S100 dendritic cells, and CD8 T lymphocytes and CD57 natural

57 mmunity and comprise monocytes, macrophages, dendritic cells, and granulocytes.

58 late both iNKT-mediated activation of thymic dendritic cells, and iNKT availability in extrathymic si

59 including basal keratinocytes, fibroblasts, dendritic cells, and lymphocytes.

60 l responses of human monocytes, macrophages, dendritic cells, and neutrophils in response to stimulat

61 to ovalbumin, levels of tolerogenic CD103(+) dendritic cells, and regulatory T (Treg) cells, as well

62 Dendritic cells are an important link between innate and

63 tively activated macrophages and tolerogenic dendritic cells are dominant in the TME.

64 Dendritic cells are efficient at capturing microbes when

65 d systemic lupus erythematosus, plasmacytoid dendritic cells are not effector cells, have lost capaci

66 itors, monocytes, macrophages, and classical dendritic cells) are controlled by signals from the M-CS

67 c cellular sources, rather than plasmacytoid dendritic cells, are responsible for interferon producti

68 killer cells, conventional and plasmacytoid dendritic cells, B cells and especially plasma cells.

69 nts and in their dependence on B7-expressing dendritic cells, B cells, and thymic epithelial cells.

70 e show that RSV-infected bone marrow-derived dendritic cells (BMDC) as well as pulmonary dendritic ce

71 aride (LPS)-primed mouse bone marrow-derived dendritic cells (BMDC).

72 ction, including IFN-alpha from plasmacytoid dendritic cells, but only in the presence of anti-Qbeta

73 ) directly reduces FcepsilonRI expression on dendritic cells by inhibiting transcription factor bindi

74 Thus, dendritic cells can control their migration patterns and

75 to surface receptors on conventional type 1 dendritic cells can enhance induction of Ab and T cell r

76 te-like cell subsets, including plasmacytoid dendritic cells; CD1c + myeloid DCs; neutrophils; macrop

77 Development and function of conventional dendritic cell (cDC) subsets, cDC1 and cDC2, depend on t

78 Classical dendritic cells (cDC) can be classified into two major s

79 F8(+) type 1 and IRF4(+) type 2 conventional dendritic cells (cDC1s and cDC2s, respectively) is well

80 Type 1 conventional dendritic cells (cDC1s) are typically thought to be dysr

81 Within the tumor, conventional type 1 dendritic cells (cDC1s) are uniquely positioned to respo

82 ndicates Batf3-dependent conventional type 1 dendritic cells (cDC1s) rarely found within the tumor my

83 the number of activated type 2 conventional dendritic cells (cDC2s) in draining lymph nodes.

84 1 was released by dermal conventional type 2 dendritic cells (cDC2s) recruited to wounds and increase

85 ther myeloid subsets, including conventional dendritic cells (cDCs) and monocyte (MO) subsets, expres

86 lood-borne pathogens, including conventional dendritic cells (cDCs) and MZ B cells.

87 Antigen-presenting conventional dendritic cells (cDCs) are broadly divided into type 1 a

88 Conventional dendritic cells (cDCs) are comprised of two major subset

89 We found that conventional dendritic cells (cDCs), defined in mice via expression o

90 are differences in infiltrating conventional dendritic cells (cDCs).

91 e most strongly enriched in AD, and we found dendritic cell (CLEC7A, amphiregulin/AREG, EREG) and mac

92 mulator of maturation of bone marrow-derived dendritic cells compared to E. coli LPS.

93 It is not known how the dendritic cells control these properties.

94 The duration of antigen presentation by dendritic cells controlled the magnitude and quality of

95 lopment, only one contributed to intrathymic dendritic cell (DC) differentiation, predominantly of pl

96 whether zoledronate (Zol) treatment impaired dendritic cell (DC) functions and increased bacterial lo

97 The most upregulated gene was the dendritic cell (DC) marker CD209a only in PVPV-Akt1KO th

98 Dendritic cell (DC) maturation is a prerequisite for the

99 ns and marks Langerhans cells (LC), the only dendritic cell (DC) population of the epidermis.

100 Generation of pDCs from bone marrow dendritic cell (DC) progenitors and their maintenance is

101 Although GM-CSF has been widely used in dendritic cell (DC) research, the mechanisms, factors, a

102 isease, which is attributed to a loss of the dendritic cell (DC) subset conventional DC2.

103 Recently, another dendritic cell (DC) subset, termed merocytic DCs (mcDCs)

104 Dendritic cell (DC) subsets are abundantly present in ge

105 mong the large variety of antigen-presenting dendritic cell (DC) subsets in the LN.

106 microenvironment diminishes the efficacy of dendritic cell (DC)-based cancer immunotherapy.

107 s have reported that, in tonsils, slan marks dendritic cell (DC)-like cells, as defined by morphologi

108 ty by acting as danger signals recognized by dendritic cells (DC) facilitating the establishment of a

109 dendritic cells (BMDC) as well as pulmonary dendritic cells (DC) from RSV-infected mice upregulated

110 Intestinal dendritic cells (DC) have the responsibility of establis

111 Dendritic cells (DC) play an essential role in innate im

112 After ischemic injury to the myocardium, dendritic cells (DC) respond to cardiomyocyte necrosis,

113 epidermal Langerhans cells (LC), and dermal dendritic cells (DC).

114 ends on IL-12 produced by CD8alpha(+) type 1 dendritic cells (DC1) for its differentiation.

115 Conventional type 1 dendritic cells (DC1s) control the response to checkpoin

116 re-associated DNA damages that are sensed by dendritic cells (DCs) and activates the host cytosolic D

117 receptor (TLR) recruitment to phagosomes in dendritic cells (DCs) and downstream TLR signaling are e

118 Dendritic cells (DCs) and other innate immune cells patr

119 We show that NVP was readily taken up by dendritic cells (DCs) and promoted DC maturation and ant

120 Dendritic cells (DCs) are a diverse group of specialized

121 Dendritic cells (DCs) are antigen-presenting cells contr

122 Dendritic cells (DCs) are antigen-presenting cells subdi

123 Dendritic cells (DCs) are critical for the differentiati

124 Dendritic cells (DCs) are highly susceptible to extrinsi

125 Dendritic cells (DCs) are one of the earliest targets of

126 Dendritic cells (DCs) are professional APCs, which sampl

127 developed a vaccine whereby patient-derived dendritic cells (DCs) are programmed to induce Th17 resp

128 Airway dendritic cells (DCs) are recognized as important factor

129 aive CD4 T cells were primed via antigen and dendritic cells (DCs) at fever temperatures, the Th2 swi

130 Dendritic cells (DCs) develop in the bone marrow from ha

131 hat accessible DNA elements in skin-resident dendritic cells (DCs) exhibit the highest enrichment of

132 For the purpose of studying functional human dendritic cells (DCs) in a humanized mouse model that mi

133 Conventional dendritic cells (DCs) in particular exist as two major s

134 ined.Objectives: To examine the role of lung dendritic cells (DCs) in T follicular helper (Tfh)-cell

135 of GM-CSF(+) CD4(+) T cells and inflammatory dendritic cells (DCs) in the CNS.

136 tion of a population of tolerogenic CD103(+) dendritic cells (DCs) in the spleen.

137 Migratory dendritic cells (DCs) in tumors transport antigens and s

138 Focusing on interactions between T cells and dendritic cells (DCs) in vitro and in vivo, we map T cel

139 Osteoclasts (OCs) and much less dendritic cells (DCs) induce significant expansion and f

140 T cell activation by dendritic cells (DCs) involves forces exerted by the T c

141 The formation of mammalian dendritic cells (DCs) is controlled by multiple hematopo

142 Dendritic cells (DCs) of the cDC2 lineage initiate aller

143 C903 model, and the depletion of Langerin(+) dendritic cells (DCs) or selective depletion of LCs dimi

144 Dendritic cells (DCs) participate in the pathogenesis of

145 Dendritic cells (DCs) play a central role in the early i

146 Dendritic cells (DCs) play a critical role in shaping ad

147 monocytes, resident tissue macrophages, and dendritic cells (DCs) present in every tissue in the bod

148 mast cells (MCs) were studied; in addition, dendritic cells (DCs) purified from the draining lymph n

149 -cell lung cancers, we identify a cluster of dendritic cells (DCs) that we name 'mature DCs enriched

150 ntima is thought to be populated by vascular dendritic cells (DCs) that, during hypercholesterolemia,

151 oDCA) increased Foxp3 induction by acting on dendritic cells (DCs) to diminish their immunostimulator

152 ergenic homologues on human monocyte-derived dendritic cells (DCs) to investigate how they polarize n

153 Cross-presentation allows dendritic cells (DCs) to present peptides derived from e

154 lar signal-related kinase (ERK) signaling in dendritic cells (DCs) versus macrophages.

155 ent upon NFkappaB-p65 and IL-6 expression in dendritic cells (DCs), as well as aryl hydrocarbon recep

156 mphocytes (CTLs) and CD103+ cross-presenting dendritic cells (DCs), as well as up-regulation of MHC c

157 e, we demonstrate that provision of OX40L by dendritic cells (DCs), but not T cells, B cells nor grou

158 osome antibodies, whereas Tlr9 deficiency in dendritic cells (DCs), plasmacytoid DCs, and neutrophils

159 al CD4+ T cells (Tconvs) and proinflammatory dendritic cells (DCs), which are both implicated in GVHD

160 study, we explore the functions of PD-L1 on dendritic cells (DCs), which highly express PD-L1.

161 absence of SIGN-R1(+) macrophages, DCIR2(+) dendritic cells (DCs), which play a key role in priming

162 herapy (PDT) and promoting the maturation of dendritic cells (DCs).

163 ormed to assess alum and PM effects in human dendritic cells (DCs).

164 immunity by recruiting regulatory T cells to dendritic cells (DCs).

165 -12 and elevated CD40 expression on CD11c(+) dendritic cells (DCs).

166 ptive immune responses via interactions with dendritic cells (DCs).

167 (IFN) responses in mouse bone marrow-derived dendritic cells (DCs).

168 others, including basophil and plasmacytoid dendritic cell depletion, correlate strongly with diseas

169 ipheral blood mononuclear cells, or immature dendritic cells derived from human peripheral blood mono

170 For dendritic cells derived from STING-deficient mice, no ac

171 Dendritic cells derived in vitro from MDS monocytes fail

172 T cell activation, B cell proliferation, and dendritic cell differentiation in vitro and suppresses i

173 Conventional dendritic cells driven by the transcription factor Batf3

174 d tumor cells efficiently activated immature dendritic cells, evidenced by enhanced expression of MHC

175 local immune-cell infiltration and activated dendritic cells, evoking a potent anti-AML response.

176 ed activation of primary bone marrow-derived dendritic cells ex vivo.

177 s can recognize and eliminate the follicular dendritic cell (FDC) reservoir of HIV-bound immune compl

178 of infectious HIV that exists on follicular dendritic cells (FDCs), persists in vivo during antiretr

179 Studies performed with bone marrow-derived dendritic cells from C-type lectin receptor-deficient mi

180 Finally, human monocyte-derived dendritic cells from IRF5 disease-associated genetic ris

181 ine secretion compared with monocyte-derived dendritic cells from low-expressing IRF5 genetic variant

182 ly reported that myeloid cells, particularly dendritic cells, from the thymic tumor microenvironment

183 CD38 as a critical regulator of plasmacytoid dendritic cell function in response to influenza virus.

184 by a dynamic mixture of macrophage subtypes, dendritic cells, granulocytes, and lymphocytes.

185 Furthermore, immature dendritic cells (iDC) responded to mCD40L with enhanced

186 and B. licheniformis Monocytes and immature dendritic cells (iDCs) responded differentially to the P

187 c mRNA levels, and the frequencies of mature dendritic cells, IFNgamma-, and IL-17- producing CD4(+)T

188 show in this study that conventional type 1 dendritic cell IL-27 production in the draining lymph no

189 otch signaling in CD8(+) T cell responses to dendritic cell immunization, Listeria infection, and vir

190 vorinostat (VOR) and AGS-004, an autologous dendritic cell immunotherapeutic, on the HIV reservoir.

191 of T cells, B cells, NK cells, monocytes and dendritic cells in 931 participants from the Health and

192 The role of dendritic cells in bone homeostasis, however, is not und

193 work redefines the role of monocyte-derived dendritic cells in melanoma and provides a novel strateg

194 activation of CD8(+) T, natural killer, and dendritic cells in relevant tissues; and facilitated ant

195 demonstrate that IFN-lambda produced by lung dendritic cells in response to a synthetic viral RNA ind

196 e factors associated with TNBC, whereas CD83 dendritic cells in the ALNs(-) were associated with the

197 cells, Tbet(+) /CD8(+) T cells, and lobular dendritic cells in the non-TOL group.

198 ted antigens then promotes the maturation of dendritic cells in the tumor-draining lymph nodes, subse

199 d monocytes rapidly replaced macrophages and dendritic cells in tumors following administration of an

200 is needed for optimal cross-presentation by dendritic cells in vitro and cross-priming of CD8 T cell

201 diated immune complex delivery to follicular dendritic cells in vivo.

202 G9Calpha(-/-)CD8(+) T-cell cytotoxicity and dendritic cell-induced proliferation, they failed to suf

203 nfects the mucosal epithelium and submucosal dendritic cells, inducing immune and inflammatory cell i

204 kin fibroblasts or mouse bone marrow-derived dendritic cells infected with the protozoan parasite Tox

205 Plasmacytoid dendritic cells influenced by microbes migrate from the

206 ssed on the surface of alveolar macrophages, dendritic cells, innate lymphoid type 2 cells, and subpo

207 ne expressing monocytes and monocyte-derived dendritic cells involved in innate immune activation, cy

208 ular patterns, metabolism of macrophages and dendritic cells is shifted from oxidative phosphorylatio

209 Bone marrow-derived macrophages and dendritic cells, lamina propria macrophages, and mesente

210 s with distinct T/B cell zones with adjacent dendritic cells, macrophages, plasma cells, high endothe

211 inetide lowered bone-marrow-derived-immature-dendritic cell maturation and subsequently reduced allog

212 Cibinetide's effects on dendritic cell maturation were investigated in vitro.

213 the miRNAs to regulate Th2 polarization and dendritic cell maturation.

214 mmune responses in mouse bone marrow-derived dendritic cells (mBMDCs) and a synergistic response in b

215 changes in gene expression in human myeloid dendritic cells (mDCs) and monocytes, identify signaling

216 CD11c(+) macrophages/dendritic cells (MDCs) are increased and display the cla

217 , CD8 T, B, and NK cells, monocytes, myeloid dendritic cells (mDCs), and plasmacytoid dendritic cells

218 at the ablation of Ctsk dramatically reduced dendritic cell-mediated inflammatory signaling.

219 d was able to 1) inhibit bone marrow-derived dendritic cell-mediated T cell functions and reduce seru

220 systemic increase of cf-mt-DNA that promotes dendritic cell-mediated, age-specific inflammatory respo

221 up by a population of IRF4+ dermal migratory dendritic cells (migDC2) that similarly upregulate surfa

222 re observed in most CD11c(+)CD103(+) myeloid dendritic cells migrating to mediastinal draining lymph

223 Here, we quantify dendritic cell migration under well-defined 2-dimensiona

224 allergy model and on human monocyte-derived dendritic cells (moDCs) and PBMCs.

225 activated upon injection of monocyte derived dendritic cells (moDCs) that presented Candida albicans.

226 ates: persistent migration, during which the dendritic cells move along curved paths, and diffusive m

227 ponses to cancer and virally-infected cells, dendritic cells must capture antigens present in tissues

228 We also find that monocytes, neutrophils, dendritic cells, natural killer cells, innate lymphoid c

229 h endothelial venules, supporting follicular dendritic cells network, and functional germinal centers

230 lted in increased pulmonary monocyte-derived dendritic cell numbers and increased IFN-gamma-dependent

231 lymphoid cells (ILC2s) and monocyte-derived dendritic cell numbers in the mediastinal lymph nodes, a

232 Dendritic cells obtained from wt mice and infected in vi

233 ease markers of maturation on cultured human dendritic cells or induce activation of T cells from pat

234 of six CBMPs containing regulatory T cells, dendritic cells, or macrophages; patient selection and i

235 Dendritic cells "patrol" the human body to detect pathog

236 alters TLR7-MyD88 signaling in plasmacytoid dendritic cells (pDCs) and blunts systemic production of

237 Plasmacytoid dendritic cells (pDCs) are a major source of type I inte

238 Plasmacytoid dendritic cells (pDCs) are potent producers of type I an

239 Plasmacytoid dendritic cells (pDCs) are robust producers of IFNalpha

240 Plasmacytoid dendritic cells (pDCs) express Toll like receptors (TLRs

241 oid dendritic cells (mDCs), and plasmacytoid dendritic cells (pDCs) from three individuals with ZIKV

242 Plasmacytoid dendritic cells (pDCs) produce abundant type I IFNs (IFN

243 Plasmacytoid dendritic cells (pDCs) produce type I interferon (IFN-I)

244 Plasmacytoid dendritic cells (pDCs) require a particularly tight regu

245 que lungs include the influx of plasmacytoid dendritic cells (pDCs), an Interferon (IFN)-responsive m

246 rences in number or function of plasmacytoid dendritic cells (pDCs), because these cells are potent i

247 Using isolated plasmacytoid dendritic cells (pDCs), we demonstrated that TLR7 is an

248 esponse in both mouse and human plasmacytoid dendritic cells (pDCs).

249 R7-mediated activation of human plasmacytoid dendritic cells (pDCs).

250 s autoreactivity by stimulating plasmacytoid dendritic cells-(pDCs)-Type I interferon (IFN-I) and act

251 In their search, dendritic cells perform a random walk by amoeboid migrat

252 in alternatively activated M2 macrophage and dendritic cell phenotypes.

253 ave previously reported both macrophages and dendritic cells play important role in the protection in

254 ctin polymerization waves that contribute to dendritic cell polarization and migration.

255 D8 T cells, as they have natural tropism for dendritic cells, preeminent inducers of CD8 T cell immun

256 onclassical monocytes and CD1c+ conventional dendritic cells preferentially migrate from blood to lun

257 reveal that dermal dendritic cells produce interleukin-31, which acts on ne

258 The dendritic cell receptor Clec9A facilitates processing of

259 s) significantly increases the activation of dendritic cells relative to their nonoxidized counterpar

260 remodeling of light and dark zone follicular dendritic cells required CXCL12-dependent crosstalk with

261 ically, RUBCN deficiency in CD301b(+) dermal dendritic cells results in their increased antigen prese

262 We show that ACKR4 inhibited CD103+ dendritic cell retention in tumors through regulation of

263 Acute lesions showed marked dendritic-cell signatures and a prominent enrichment of

264 e osteoclast marker genes TRAP, Cathepsin K, dendritic cell-specific transmembrane protein (DCSTAMP),

265 s, thymic fibroblast subtypes, and activated dendritic cell states.

266 nscriptome sequencing analysis of Ctsk (-/-) dendritic cells stimulated with lipopolysaccharide demon

267 oice of targeting receptor, even on the same dendritic cell subpopulation, may strongly influence the

268 ke by lymph node-resident and migratory skin dendritic cell subpopulations, including Langerhans cell

269 L is required by a Batf3-dependent classical dendritic cell subset (cDC1) for optimal CD8 T cell prim

270 Although conventional dendritic cell subsets have received much attention for

271 mal psoriatic features, including T-cell and dendritic cell subsets, were examined using immunohistoc

272 ive changes in vasculature and maturation of dendritic-cell subsets with chronicity, with FOXK1 actin

273 ynergies between TNF and IFNbeta controlling dendritic cell-T cell crosstalk.

274 d for the development of conventional type 1 dendritic cells that are essential for cross-presentatio

275 t for induction of CD11c(+) MHC class II(hi) dendritic cells that are implicated in Tfh cell developm

276 ulation is driven in part by macrophages and dendritic cells that are polarized to a mixed proinflamm

277 umors, accompanied by an increase in CD1C(+) dendritic cells, the tumor-associated macrophages (TAMs)

278 1 (LYVE-1) mediates the docking and entry of dendritic cells to lymphatic vessels through selective a

279 y APC and demonstrate the unusual ability of dendritic cells to process stable protein aggregates.

280 stimulation and increased migration of lung dendritic cells to the draining lymph nodes, resulting i

281 (-/-) mice lacking the CD103(+) conventional dendritic cell type 1 (cDC1) subpopulation important for

282 preceded by poor activation of conventional dendritic cells type 2 (cDC2) due to reduced type 1 inte

283 t the extent of activation in a plasmacytoid dendritic cell-type I IFN-T/B lymphocyte network.

284 ization efficacy by targeting and activating dendritic cells via C-type lectin receptors and reduce s

285 Activation of Gpr81 in dendritic cells was associated with decreased cAMP, IL-6

286 nt response, whereas priming by conventional dendritic cells was dispensable.

287 Expression of LACC1 by dendritic cells was required for increasing expression o

288 7 macrophages and murine bone marrow-derived dendritic cells; we now show that SLP-8321 and SLP-5818

289 opria macrophages, and mesenteric lymph node dendritic cells were examined.

290 T cells, monocytes-1 and dendritic cells were found to be crucial for the IL-17 s

291 D56(dim) natural killer cells, monocytes and dendritic cells were not reduced in number and hence rel

292 hritis), mainly inflammatory macrophages and dendritic cells, were characterized by co-expression of

293 Neutrophils and dendritic cells when migrating in confined environments

294 ing that IL-36 acts directly on plasmacytoid dendritic cells, where it potentiates toll-like receptor

295 edullary thymic epithelial cells (mTECs) and dendritic cells, whereas TRP1 expression was restricted

296 mplexes that upon cellular turnover activate dendritic cells, which amplify psoriasis inflammation.

297 n of Ly6C(hi) monocytes into macrophages and dendritic cells, which infiltrate the spleen and major b

298 ed by a distinct subset of CD301b(+)OX40L(+) dendritic cells, which migrate into the bladder epitheli

299 ily, is the prototypic endocytic receptor of dendritic cells, whose ligands include phosphorothioated

300 ifying a potential role for PD-L1 expressing dendritic cells within the lymph node in regulation of a