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1 2% to 42% of zOTUs in saliva; 2.5% to 38% in dental plaque).
2 es disease by surviving acidic conditions in dental plaque.
3 the mixed-species environment of subgingival dental plaque.
4 to the initiation of the oral biofilm called dental plaque.
5 mportant role in the ecology of S. mutans in dental plaque.
6 reptococcus cristatus, an early colonizer of dental plaque.
7 alence of mixed-species communities in early dental plaque.
8 ispecies bacterial biofilm commonly known as dental plaque.
9 and plays a pivotal role in the formation of dental plaque.
10  the inflammatory response of the gingiva to dental plaque.
11 e fluids reflect the cariogenic potential of dental plaque.
12 he complex multiple species biofilm known as dental plaque.
13 ly significant in the establishment of early dental plaque.
14  gordonii-A. naeslundii communities in early dental plaque.
15 onema socranskii, and Treponema vincentii in dental plaque.
16  formation of mixed-species biofilms such as dental plaque.
17 as each new bacterial cell binds to existing dental plaque.
18 m important interspecies interactions within dental plaque.
19 onsortium in the microbiome of supragingival dental plaque.
20  an oral commensal and an early coloniser of dental plaque.
21 ing are required for the periodic removal of dental plaque.
22 its a general inflammatory response to local dental plaque.
23 t of the complex oral biofilm referred to as dental plaque.
24 . gingivalis and S. cristatus in subgingival dental plaque.
25 cterial surfaces and to colonize subgingival dental plaque.
26  abundant species in the oral biofilm called dental plaque.
27 d-species colonies during formation of early dental plaque.
28  an inflammatory reaction to the bacteria in dental plaque.
29 ntitis in response to challenge by microbial dental plaque.
30  a substantial proportion of the bacteria in dental plaque (30 to 40%) bear PC antigen; this antigen
31                             The formation of dental plaque, a highly complex biofilm that causes ging
32 a natural microbial community, namely, human dental plaque, a microbial biofilm.
33                        Gingival bleeding and dental plaque accumulation are also more frequent among
34 in this association, our hypothesis was that dental plaque accumulation in healthy subjects would eli
35                         We hypothesized that dental plaque accumulation over 21 days in the experimen
36  differences in the inflammatory response to dental plaque accumulation.
37   Notably, models distinguishing saliva from dental plaques achieved superior performance compared to
38 nly from FBS, but also from human saliva and dental plaque after the incubation of 0.45-microm membra
39  salivary cytokine levels were measured, and dental plaque analyzed by 16S rRNA high throughput seque
40 ltispecies biofilm on tooth surfaces forming dental plaque and a potential agent of endocarditis.
41 llied food industries on enzymes to break up dental plaque and a vaccine against tooth decay with que
42 er the bacteria were isolated from saliva or dental plaque and appeared to be coordinately regulated.
43 anium disks were inoculated with subgingival dental plaque and cultured anaerobically for 21 days.
44 cer mortality was positively associated with dental plaque and gingival inflammation.
45 tococcus sanguinis are pioneer colonizers of dental plaque and important agents of bacterial infectiv
46 t the glutamine endoprotease is derived from dental plaque and likely microbial in origin.
47                                   Removal of dental plaque and local application of local chemical ad
48 dy reveals an intriguing resemblance between dental plaque and non-host environments indicated by the
49 this assumption, we analyzed patient-matched dental plaque and odontogenic abscess clinical specimens
50 o examine the prevalence of P. gingivalis in dental plaque and of serum immunoglobulin G (IgG) antibo
51                                              Dental plaque and prostatic secretion samples were used
52                    Although bacteremias from dental plaque and/or elevated circulating inflammatory c
53 . mutans to dominate the microenvironment of dental plaques and form dental caries.
54                        However, PCR of whole dental plaques and subsequent analysis of up to 130 indi
55                Bacterial levels, gingivitis, dental plaque, and caries experience were assessed.
56 ucation, income, smoking, diabetes mellitus, dental plaque, and presence of any of 8 subgingival micr
57 lis creates a dysbiosis between the host and dental plaque, and this may represent one mechanism by w
58 levels of Treponema denticola in subgingival dental plaque are associated with severe periodontal dis
59 y half of the bacteria in the saliva and the dental plaque are not cultivable.
60 ity and complexity of the microbial biota in dental plaque are significantly less in S-ECC children t
61       Porphyromonas gingivalis is present in dental plaque as early as 4 h after tooth cleaning, but
62  It is generally recognized that bacteria in dental plaque at sites of periodontal diseases are not c
63  have shown that a significant proportion of dental plaque bacteria contain PC as determined by react
64    Once thought to occur exclusively between dental plaque bacteria, there are increasing reports of
65 ard pertains to only a few of the acidogenic dental plaque bacteria.
66 lectin-like adhesins on other members of the dental plaque biofilm community.
67 on of these bacteria by other members of the dental plaque biofilm community.
68                                              Dental plaque biofilm formation proceeds through a devel
69                                              Dental plaque biofilm is a complex ecosystem.
70                                              Dental plaque biofilm is considered to be the underlying
71 atypica, two early colonizing members of the dental plaque biofilm, have been postulated to participa
72 atypica, two early-colonizing members of the dental plaque biofilm, participate in a relationship tha
73              In one such community-the human dental plaque biofilm-a network of filamentous Corynebac
74                   Acidogenic bacteria within dental plaque biofilms are the causative agents of carie
75 ogic agent of human dental caries that forms dental plaque biofilms containing functional amyloids.
76 and Streptococcus intermedius in subgingival dental plaque biofilms may contribute to forms of period
77  device for the in vivo generation of intact dental plaque biofilms on natural tooth surfaces in huma
78                      Streptococcus mutans in dental plaque biofilms play a role in caries development
79  is the anaerobic environment of subgingival dental plaque, but initial colonization of the oral cavi
80 ent by altering the microbial composition in dental plaque, but little is known regarding HSB-specifi
81 ary for initial or sustained colonization in dental plaque by this major dental pathogen.
82 from five specimens of Neanderthal calcified dental plaque (calculus) and the characterization of reg
83  oral microbial communities within calcified dental plaque (calculus) has revealed key insights into
84 lysis of urea by ureases of oral bacteria in dental plaque can cause a considerable increase in plaqu
85 port the hypothesis that the accumulation of dental plaque can result in a measurable systemic inflam
86  to determine if proteases produced by early dental plaque colonizers such as Streptococcus gordonii
87                              We propose that dental plaque communities originate as a result of intim
88 ve advantages over nonureolytic organisms in dental plaque, constituting an important determinant of
89 ommonly occurring bacterial species found in dental plaque contain PC antigen and that immunization w
90                           Historic calcified dental plaque (dental calculus) can provide a unique per
91                                    Calcified dental plaque (dental calculus) preserves for millennia
92 examination was performed in order to assess dental plaque, dental calculus and gingival inflammation
93  pH and H(2)O(2) concentration produced by a dental plaque-derived multispecies biofilm grown on hydr
94                                              Dental plaque-derived multispecies biofilms were culture
95 ay determine polymicrobial succession during dental plaque development, but the ecological constraint
96 d presence of P. gingivalis at all stages of dental plaque development.
97 streptococci and actinomyces, are central to dental plaque development.
98 al bacteria and individual species counts in dental plaque did not differ significantly between basel
99             The microbiomes of supragingival dental plaque differ substantially among tooth surfaces
100                                              Dental plaque DNA samples were amplified by targeting th
101        Newly formed biofilms and more mature dental plaque each have a level of spatial organization
102         Overall, these data demonstrate that dental plaque eDNA is potentially an important target fo
103  The predominant species group in developing dental plaque films during density-dependent growth was
104 saturation with respect to enamel mineral in dental plaque fluid following sucrose exposure.
105 e originating as a low-abundance organism in dental plaque, Fn. animalis typically outcompetes other
106 m disulfide bonds plays an important role in dental plaque formation and fitness for the bacteria.
107 pportunistic pathogen, is thought to promote dental plaque formation by serving as a bridge bacterium
108 al oral hygiene measures was used to compare dental plaque formation following use of chlorhexidine (
109                        The initial stages of dental plaque formation involve the adherence of early c
110 h surfaces colonization and contributions to dental plaque formation, as well as their potential role
111                               During initial dental plaque formation, the ability of a species to gro
112  characterized by gingivitis associated with dental plaque formation, which progresses to periodontit
113 nii, are pioneer oral bacteria that initiate dental plaque formation.
114 is interaction may play an important role in dental plaque formation.
115 aining chewing gum appears useful to control dental plaque formation.
116                 The microbial composition of dental plaque from 42 severe ECC children was compared w
117  in both prostatic secretion and subgingival dental plaque from the same individual.
118 e microbiomes of site-specific supragingival dental plaques from children with different caries statu
119 egrate multi-omics of supragingival biofilm (dental plaque) from 416 preschool-age children (208 male
120                        To define the role of dental plaque fructans and the enzymes involved in their
121    All-cause mortality risk were raised with dental plaque, gingival inflammation, >10 missing teeth
122                                              Dental plaque harbored a greater diversity of fusobacter
123 ty were also positively associated with high dental plaque (HR = 3.30, [95% CI: 1.76-6.17]), high gin
124 constituent of supragingival and subgingival dental plaque in children and adults.
125 gy or the physicochemical characteristics of dental plaque in such a way as to reduce its cariogenic
126 l flora and pH-lowering capacity of the same dental plaques in relation to caries.
127 -specific bacterial taxa after adjusting for dental plaque index, decayed missing filled teeth (DMFT)
128 al attachment loss, bleeding on probing, and dental plaque index.
129 nd the same DNA mixed with DNA isolated from dental plaque, indicating that P. gingivalis levels can
130 om the biofouling of ocean-going vessels, to dental plaque, infections of the urinary tract, and cont
131               These biofilms were grown from dental plaque inoculum (oral microcosms) and were obtain
132                                              Dental plaque is a complex biofilm that accretes in a se
133                                              Dental plaque is a complex multispecies biofilm, and is
134  It also suggests that the overall effect of dental plaque is a function of the balance between patho
135                                  Subgingival dental plaque is an ecosystem playing a key role in supp
136                   Secondary caries caused by dental plaque is one of the major reasons for the high f
137                   Applying the ZG16B-mGAP to dental plaque isolates revealed that ZG16B predominantly
138 nths, independent of age, smoking status, or dental plaque levels.
139 , micro-fossils preserved in the mineralized dental plaque, macrowear, and buccal microwear.
140 ilar effects on bacterial composition of the dental plaque may occur in vivo in patients on statins,
141 ilar effects on bacterial composition of the dental plaque may occur in vivo in patients on statins,
142                  Composition and activity of dental plaque microbiome from 115 participants was analy
143 med to characterize the healthy salivary and dental plaque microbiome in young children.
144 and characterized over time the salivary and dental plaque microbiome of children before clinical dia
145 ctors explained more of the variation in the dental plaque microbiome than in saliva.
146 gate the effects on dentin bond strength and dental plaque microcosm biofilms for the first time.
147  Streptococcus gordonii DL1, a nonureolytic, dental plaque microorganism.
148                   We propose that PC-bearing dental plaque microorganisms may induce an antibody resp
149   Future studies assessing a larger panel of dental plaque microorganisms, with shorter intervals bet
150 lammatory disease initiated and sustained by dental plaque microorganisms.
151 ajor direct cause of chronic inflammation is dental plaque, much of the new research is directed at m
152  Eikenella corrodens isolates recovered from dental plaque, mucosal surfaces, and saliva of 24 subjec
153 p(+) S. mutans and C. albicans in saliva and dental plaque of children with varying caries statuses,
154 ry pathogens have been shown to colonize the dental plaque of hospitalized intensive care and nursing
155 ative phase of treatment, and in subgingival dental plaque of periodontitis patients, indicating that
156  and Fusobacterium nucleatum, in subgingival dental plaque of pregnant women in the OPT Study and the
157 lms inhabit a range of environments, such as dental plaques or soil micropores, often characterized b
158  = 1.01, 95% CI [1.00, 1.02], p = 0.022) and dental plaque (OR = 1.04, 95% CI [1.01, 1.07], p = 0.013
159 = 3.89, 95% CI [1.28, 11.82], p = 0.017) and dental plaque (OR = 1.07, 95% CI [1.01, 1.13], p = 0.028
160 ssociation was observed for number of teeth, dental plaque, or detectable oral mucosal lesions and PD
161 gnificantly higher percentages of sites with dental plaque (P <0.0001), gingival bleeding (P <0.05),
162 sis did not reveal susceptibility of certain dental plaque pathogens to light, and it was not possibl
163                Polymicrobial interactions in dental plaque play a significant role in dysbiosis and h
164                                              Dental plaque, previously considered a polymicrobial com
165 and this ability has an effect on a range of dental plaque-related phenotypes.
166                       Pi was detected in all dental plaque samples but not in the prostatic secretion
167 ere, we assemble metagenomes from tongue and dental plaque samples from multiple individuals and reco
168  of cells of individual bacterial species in dental plaque samples is needed for understanding the ba
169                     An extensive analysis of dental plaque samples over the years has led to the iden
170                                       Pooled dental plaque samples were collected and analyzed using
171                                       Pooled dental plaque samples were collected from 20 children ag
172                                 Salivary and dental plaque samples were collected from the children a
173 tatus (smoker or non-smoker), and full-mouth dental plaque score.
174 lobacter concisus and Oribacterium sinus and dental plaque-specific Lautropia mirabilis, Rothia aeria
175 gation between two key initial colonizers of dental plaque, Streptococcus gordonii and Veillonella pa
176 e data suggest that some early colonizers of dental plaque, such as S. cristatus, may be beneficial t
177 s of bleeding on probing, root calculus, and dental plaque than adolescents without subclinical perio
178 ans is the principal acidogenic component of dental plaque that demineralizes tooth enamel, leading t
179 disease, is caused by the bacterial biofilm (dental plaque) that accumulates on teeth adjacent to the
180 dependent on tooth-borne microbial biofilms (dental plaque) that trigger host inflammation resulting
181 ispecies oral biofilm known as supragingival dental plaque; they grow by fermentation of sugars to or
182 ranspose these interactions from undisturbed dental plaque to an experimentally tractable in vitro bi
183 cies colonies of bacteria, e.g., biofilms or dental plaque, to behave as pseudomulticellular organism
184                                Supragingival dental plaque was collected from tooth surfaces that wer
185      From strains of S. mutans isolated from dental plaque, we discovered, in the present study, a po
186       Percentages of tooth surfaces carrying dental plaque were 41% and 36% for right and left sides,
187 veral of these species are commonly found in dental plaque, while N. meningitidis is primarily found
188 nce of an inflammatory infiltrate induced by dental plaque within the gingival tissue.
189 e result of infection by anaerobic bacteria; dental plaque would seem to be a logical source of these

 
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