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1 -based therapy for DCAs targeting the dorsal dentate nucleus.
2 rojections from the cerebellar cortex to the dentate nucleus.
3 ostnatally in the granule cell layer and the dentate nucleus.
4 ia and had abnormal signals in brainstem and dentate nucleus.
5 volvement of white matter and the cerebellar dentate nucleus.
6 iated with increased signal intensity in the dentate nucleus.
7 ated amino acid precursors injected into the dentate nucleus.
8 tDNA-encoded proteins only in neurons of the dentate nucleus.
9 f inferior olive nuclei and dysplasia of the dentate nucleus.
10  motor-cognitive dichotomy in the cerebellar dentate nucleus.
11  images relative to reference tissues in the dentate nucleus (0.53% signal intensity increase per inj
12 lesions in the substantia nigra, caudate and dentate nucleus, a reliable and repeatable 12-tiered gra
13  tract changes correlated with reductions in dentate nucleus activation recorded during task performa
14 coustic transmissions to click change in the dentate nucleus after conditioning, that changes in resp
15 present in a small number of neurones in the dentate nucleus and diffusely in cerebellar astrocytes.
16 rotopias, and simplified convolutions of the dentate nucleus and inferior olive.
17 s with Friedreich ataxia had lower volume of dentate nucleus and superior cerebellar peduncles; small
18 timulating electrodes targeted to the dorsal dentate nucleus and tested a spectrum of frequencies ran
19  these neurons were found in portions of the dentate nucleus and the internal segment of the globus p
20 mapping, we identified distinct areas in the dentate nucleus and the lateral cerebellum of both hemis
21 easured in regions of interest placed in the dentate nucleus and the pons, and we calculated the dent
22 rimotor cortex, the right cerebellum (dorsal dentate nucleus), and the right superior temporal gyrus
23 ation of alpha(2/3)-receptor subunits in the dentate nucleus, and correspondingly, we studied the evo
24 red nucleus, cerebellar peduncle, colliculi, dentate nucleus, and globus pallidus.
25  of these proteins in the cerebellar cortex, dentate nucleus, and inferior olivary nucleus from 2 aut
26 m, cerebrospinal fluid (CSF), basal ganglia, dentate nucleus, and kidney samples.
27 ites, neurons in internal granular layer and dentate nucleus, and neuronal elements in gut and kidney
28 , ventral intermediate thalamic nucleus, and dentate nucleus, and observed abnormal functional connec
29     The SI in the globus pallidus, thalamus, dentate nucleus, and pons was measured at unenhanced T1-
30 ior cingulate, inferior temporal lobule, the dentate nucleus, and the cerebellar lobules IV/V, VI, an
31 ections from caudal and ventral parts of the dentate nucleus appear to overlap oculomotor inputs to r
32  of deep brain stimulation to the cerebellar dentate nucleus as a promising tool for modulation of la
33                        Silencing activity in dentate nucleus by photoactivating inhibitory Purkinje c
34 decay times of the GABAergic currents to the dentate nucleus can facilitate sustained oscillatory act
35 The findings support the hypothesis that the dentate nucleus can play a significant role in short as
36 ls in the basal ganglia, midbrain, thalamus, dentate nucleus, cerebellar peduncles, cerebellar vermis
37 ry indices for frontal cortex, thalamus, and dentate nucleus combined as well as individually for fro
38 ied deep brain stimulation to the cerebellar dentate nucleus combined with renewed physical rehabilit
39  pallidus, putamen, substantia nigra and the dentate nucleus compared to PD and controls.
40 e results indicated that inactivation of the dentate nucleus disrupted effort-based decision making.
41 ighted signal intensity (SI) increase in the dentate nucleus (DN) and globus pallidus (GP) in relatio
42 ing deep brain stimulation of the cerebellar dentate nucleus (DN) for chronic, poststroke motor rehab
43 ention we recorded extracellular activity of dentate nucleus (DN) neurons in two non-human primates (
44 ght Crus I (RCrus I) Purkinje cells (PCs) or dentate nucleus (DN) neurons while synchronously imaging
45 eglumine on the signal intensity (SI) of the dentate nucleus (DN) of the pediatric brain on nonenhanc
46  of deep brain stimulation of the cerebellar dentate nucleus (DN) on chronic poststroke motor rehabil
47  (GBCAs) on the signal intensity (SI) of the dentate nucleus (DN) on unenhanced T1-weighted magnetic
48 est for measurable visual enhancement of the dentate nucleus (DN) on unenhanced T1-weighted magnetic
49                         Purpose To develop a dentate nucleus (DN) segmentation tool using deep learni
50 ) in the globus pallidus (GP), thalamus (T), dentate nucleus (DN), and pons (P) were measured on unen
51 d T1 signal intensities were measured in the dentate nucleus (DN), globus pallidus (GP), crus anterio
52 llowing five regions of interest (ROIs): the dentate nucleus (DN), pons, substantia nigra (SN), pulvi
53                   Stimulation of FN, but not dentate nucleus (DN), significantly reduced lesion volum
54  age, 49 years; age range, 25-73 years), the dentate nucleus (DN)-to-middle cerebral peduncle (MCP) S
55 is, and deep cerebellar nuclei including the dentate nucleus during absolute, duration-based timing a
56 ce imaging of the lateral cerebellar output (dentate) nucleus during passive and active sensory tasks
57 oning task, cerebellar output neurons in the dentate nucleus exhibit preparatory activity similar to
58 gest that mitochondrial abnormalities in the dentate nucleus in conjunction with loss of Purkinje cel
59  Purkinje cells to 46% of the neurons of the dentate nucleus in MERRF cerebellum.
60 uditory hallucinations were connected to the dentate nucleus in the cerebellum.
61 to determine whether signal intensity in the dentate nucleus is increased in unenhanced T1-weighted i
62 minations had increased SI ratios within the dentate nucleus (mean SI ratio +/- standard error of the
63 wed visually detectable T1 shortening in the dentate nucleus (n = 13), globus pallidus (n = 13), subs
64 la nucleus, select brainstem nuclei, and the dentate nucleus of cerebellum.
65    Recordings were made from 95 units of the dentate nucleus of naive cats to determine if patterns o
66 nts and controls, although R2* values in the dentate nucleus of patients were significantly higher, w
67 en, pallidum, thalamus, midbrain, and in the dentate nucleus of the cerebellum (t's > 2.7, P's < 0.02
68 e targets of disynaptic projections from the dentate nucleus of the cerebellum and from the internal
69                We previously showed that the dentate nucleus of the cerebellum has a disynaptic proje
70  were found in frontal cortex, thalamus, and dentate nucleus of the cerebellum in all 7 boys, but not
71 ined organization of the projection from the dentate nucleus of the cerebellum to the ventral lateral
72 rafascicular thalamus, precentral gyrus, and dentate nucleus of the cerebellum.
73 nd-order neurons in posterior regions of the dentate nucleus of the cerebellum.
74 hyperintensity of the globus pallidus and/or dentate nucleus on unenhanced T1-weighted magnetic reson
75 euronal transport of virus were found in the dentate nucleus only after injections into areas 46d, 9m
76  .026) and T2 of the whole brain (P = .004), dentate nucleus (P = .023), and thalamus (P = .002) show
77 rain (P < .001), globus pallidus (P = .002), dentate nucleus (P = .046), and thalamus (P = .026) and
78             T1 mapping showed no changes for dentate nucleus, pons, and thalamus.
79                 Globus pallidus-thalamus and dentate nucleus-pons SI ratios were calculated and compa
80                        The difference in the dentate nucleus-pons signal intensity ratio between the
81                        The difference in the dentate nucleus-pons signal intensity ratio between the
82  and spinal cord, including the hippocampus, dentate nucleus, pontine nuclei, locus coeruleus, and pa
83  neurons, the locus ceruleus, the cerebellar dentate nucleus, Purkinje cells, the basis pontis, numer
84                Stimulation of the FN but not dentate nucleus reduced the volume of the focal infarcti
85  putamen, subthalamic nucleus, midbrain, and dentate nucleus relative to controls and PD patients (vo
86  involved in sensorimotor processing (dorsal dentate nucleus, sensorimotor cortex).
87  [CI]: 0.03, 0.67; P = .03), but not between dentate nucleus SI and patient age (r = 0.23; 95% CI: -0
88  There was a significant correlation between dentate nucleus SI and total cumulative gadolinium dose
89 levated free-water in all regions except the dentate nucleus, subthalamic nucleus, and corpus callosu
90 terest were measured in the globus pallidus, dentate nucleus, thalamus, and pons.
91 olliculus, the Edinger-Westphal nucleus, the dentate nucleus, the raphes linearis and pontis, the dor
92 s pallidus, substantia nigra, and cerebellar dentate nucleus); this suggests a much more extensive ro
93 umine administrations and an increase in the dentate nucleus-to-middle cerebral peduncle signal inten
94 sity ratios (globus pallidus-to-thalamus and dentate nucleus-to-pons ratios), and T1 and T2 relaxatio
95  nucleus and the pons, and we calculated the dentate nucleus-to-pons signal intensity ratios and the
96 out Lewy bodies also had degeneration of the dentate nucleus (two cases).
97 ted serotonin synthesis in the contralateral dentate nucleus was observed.
98      Unenhanced T1 signal intensities of the dentate nucleus were measured from magnetic resonance (M
99 rior cerebellar peduncle and the ipsilateral dentate nucleus, which correspond to the ipsilateral por
100 n output stage of cerebellar processing, the dentate nucleus, with an input stage of basal ganglia pr