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1 a, which was abrogated by preincubation with deoxyribonuclease-I.
2  acute liver failure plasma with and without deoxyribonuclease-I.
3 selective serine 3 cofilin kinase binds to a deoxyribonuclease I affinity column, whereas the nonspec
4 odified 3'-phosphate of oligonucleotides and deoxyribonuclease I and ribonuclease H cleavages.
5 lease A) and six acidic proteins (myoglobin, deoxyribonuclease I, beta-lactoglobulin A, beta-lactoglo
6                                            A deoxyribonuclease I binding assay shows that the number
7      Detection of pointed ends in situ using deoxyribonuclease I binding demonstrates that this incre
8 reated wild-type MLEC were hypersensitive to deoxyribonuclease I compared with wild-type cells, demon
9                                    Employing deoxyribonuclease I (DNase I) as a model enzyme template
10                 However, NET disruption with deoxyribonuclease I (DNase I) efficiently inhibited airw
11 are concentrated in regulatory DNA marked by deoxyribonuclease I (DNase I) hypersensitive sites (DHSs
12          It was demonstrated previously that deoxyribonuclease I (DNase I) is a highly active renal e
13                            Bovine pancreatic deoxyribonuclease I (DNase I) is a nuclease of relativel
14                            Bovine pancreatic deoxyribonuclease I (DNase I) is a well characterised en
15                            Recombinant human deoxyribonuclease I (DNase I) is an important clinical a
16                                              Deoxyribonuclease I (DNase I) is an important enzyme tha
17  DNA (cf-mtDNA) levels in mice, and systemic Deoxyribonuclease I (DNase I) treatment attenuated RS-in
18                                     Although deoxyribonuclease I (DNase I) was used to probe the stru
19                                        Human deoxyribonuclease I (DNase I), an enzyme recently approv
20                                        Human deoxyribonuclease I (DNase I), an enzyme used to treat c
21 ty (71 and 63% similarity), respectively, to deoxyribonuclease I (DNase I).
22 nalised with dornase alfa (recombinant human deoxyribonuclease I, DNase), demonstrating DNA degradati
23                                              Deoxyribonuclease I (DNaseI) hypersensitivity analyses i
24      They belong to nine different proteins (deoxyribonuclease I, enolase, hen egg-white lysozyme, hu
25                                              Deoxyribonuclease I footprint analysis of the minimal pr
26 tituted in vitro transcription reactions and deoxyribonuclease I footprinting assays confirmed the ab
27                                              Deoxyribonuclease I footprinting identified a specific s
28                              We used genomic deoxyribonuclease I footprinting to map nucleotide resol
29                                              Deoxyribonuclease I footprinting was used to identify a
30                                Gel-shift and deoxyribonuclease-I footprinting assays revealed four DN
31                                              Deoxyribonuclease I forms a very tight complex with acti
32      The identification of a tissue-specific deoxyribonuclease I hypersensitive site approximately 3k
33 portant transcriptional regulatory elements, deoxyribonuclease I hypersensitive site mapping studies
34                               Colon-specific deoxyribonuclease I hypersensitive sites (DHS) have been
35 d single nucleotide polymorphisms (SNPs) and deoxyribonuclease I hypersensitive sites (DHSs) from 112
36  locus control region (LCR) composed of five deoxyribonuclease I hypersensitive sites (HSs).
37   Analysis of transcription factor motifs in deoxyribonuclease I hypersensitive sites at cell-type-sp
38    A 3.5-kb fragment containing one of these deoxyribonuclease I hypersensitive sites, located -14 kb
39           However, removal of SWI/SNF left a deoxyribonuclease I-hypersensitive site specifically at
40 cs of regulatory DNA, we mapped >1.3 million deoxyribonuclease I-hypersensitive sites (DHSs) in 45 mo
41                       Myeloid-cell-specific, deoxyribonuclease-I-hypersensitive sites localized to th
42 t mutation (Hg19) in a noncoding region of a deoxyribonuclease I hypersensitivity binding site was fo
43 ility as detected by histone acetylation and deoxyribonuclease I hypersensitivity.
44 i to digestion with micrococcal nuclease and deoxyribonuclease I, indicating that chromatin structure
45 l protein (CLC); carboxypeptidase A3 (CPA3); deoxyribonuclease I-like 3 (DNASE1L3); IL-1beta (IL1B);
46 ous ATP-actin structures from complexes with deoxyribonuclease I, profilin, and gelsolin, monomeric A
47                                              Deoxyribonuclease I protection assays confirmed the pres
48             Conjugation of recombinant human deoxyribonuclease I (rhDNase) to polyethylene glycol (PE
49 markers acetyl-H4 and H4K20m, and regions of deoxyribonuclease I-sensitive chromatin compared with co
50 iple histone marks and Pol II, as well as in deoxyribonuclease I sensitivity and nucleosome positioni