ライフサイエンスコーパス: depolarizing

1 -70 mV shifted to become barely net inward (depolarizing).

2 ective foci, and in 54.5% the foci were also depolarizing.

3 data suggest that GABA acquires a transient depolarizing action during recovery from UVN, which pote

4 ly suggesting that GABA acquires a transient depolarizing action in the VN during the recovery period

5 found that ethanol exposure potentiates the depolarizing action of GABA in GABAergic cortical intern

6 During development, GABA exerts depolarizing action on immature neurons and, acting in s

7 During development GABA exerts a depolarizing action on immature neurons.

8 le in cortical development through its early depolarizing action.

9 s importantly contribute to GDPs, due to the depolarizing actions of GABA early in development.

10 ene regulation to be tailored to the type of depolarizing activity along the somato-dendritic axis of

11 Thus, dysregulation in GABA(A) depolarizing activity delayed dendritic development and

12 The membrane-depolarizing activity is alleviated by coproduction of t

13 ow that in flies with reduced Arr1 prolonged depolarizing afterpotential can be triggered with fewer

14 sting induction and suppression of prolonged depolarizing afterpotential.

15 uning characteristics.SIGNIFICANCE STATEMENT Depolarizing afterpotentials (DAPs) are frequently obser

16 shoulder, or a depolarization reminiscent of depolarizing afterpotentials (DAPs) recorded in vitro in

17 locked the ability of GLP-1 agonists and the depolarizing agent KCl to potentiate this.

18 eatic islets and is upregulated by beta-cell depolarizing agents.

19 eatment of cultured cells with mitochondrial depolarizing agents.

20 ree cycles), occurred when PVs and PYRs were depolarizing and entrained their membrane potential dyna

21 s channels of interest, including dephasing, depolarizing and erasure channels.

22 transiently switched from hyperpolarizing to depolarizing and excitatory.

23 At these synapses, GABA is depolarizing and exerts a bimodal control on excitabilit

24 y of T4 found that the integration of offset depolarizing and hyperpolarizing inputs is critical for

25 ionally control the membrane potential using depolarizing and hyperpolarizing opsins; the ability to

26 We identified distinct roles of the depolarizing and hyperpolarizing phases of tACS in entra

27 ctance increased stepwise and gradually with depolarizing and hyperpolarizing pulses, respectively.

28 ctivated, closed states of the channel under depolarizing and hyperpolarizing transmembrane voltages

29 g voltage-gated ion channels responsible for depolarizing and repolarizing the action potential were

30 cantly by 50-150%, and the peak shifted by a depolarizing approximately 10 mV.

31 onships were shifted linearly by significant depolarizing approximately 9 and approximately 18 mV, re

32 n of light responses from hyperpolarizing to depolarizing before passing them on to ganglion cells.

33 elease by closing ATP-sensitive K+ channels, depolarizing beta cells, and opening voltage-dependent C

34 of the ERG b-wave, a component that reflects depolarizing bipolar cell (DBC) function.

35 ues molecular pathology at the photoreceptor-depolarizing bipolar cell (photoreceptor-DBC) synapse an

36 For normal function, ON or depolarizing bipolar cells (DBCs) require the G-protein-

37 rly in the pre-ictal period, and can display depolarizing block during the ictal event.

38 GABAergic current was depolarizing but inhibitory in 8.5 mM K(+), suggesting a

39 faced particle photothermally induces a cell-depolarizing capacitive current, and predicts that deliv

40 coded by the SCN5A gene, conducts the inward depolarizing cardiac Na(+) current (INa) and is vital fo

41 ffect mediated by the emergence of a rapidly depolarizing cell population, and the expression of hERG

42 We showed that depolarizing changes in the presynaptic holding potentia

43 o-threshold of p ~ 0.50 under a single qubit depolarizing channel applied to all qubits.

44 be searched, when decoherence is modelled by depolarizing channels' deleterious effects imposed on th

45 internal reflectivity, presence of internal depolarizing characteristics, and presence of overlying

46 leotide-gated (CNG) channel and stimulates a depolarizing chloride current by opening the olfactory C

47 er direct control, we used densely-expressed depolarizing (ChR2) or hyperpolarizing (eArch3.0, eNpHR3

48 Hence, despite the well known depolarizing Cl(-) equilibrium potential of neonatal hip

49 ropose that, even in the presence of a local depolarizing Cl(-) gradient, HCO3(-) efflux through GABA

50 Aergic-mediated feed-forward inhibition, the depolarizing component of spikelets transiently increase

51 consistent with the idea that under extreme depolarizing conditions, the biophysical difference betw

52 Independently, phasic DA activated a slow depolarizing conductance and the late burst through a D1

53 ogeneous medium internal reflectivity and no depolarizing contents (55.3% of drusen).

54 e-type releaser at DAT that evoked an inward depolarizing current and calcium influx, whereas other a

55 ts due to a severe reduction of a muscarinic depolarizing current and M1 receptor internalization.

56 activation might cause a sharp escalation in depolarizing current and underlie the steep initial rise

57 the number of action potentials evoked by a depolarizing current at 2X rheobase in neurons from CCD

58 s can fire single action potentials (APs) to depolarizing current commands, but are unable to dischar

59 es the action potential waveform by reducing depolarizing current during the plateau phase of the act

60 e sodium current (INa) that provides a rapid depolarizing current during the upstroke of the action p

61 dium current (I(Na)), which provides a rapid depolarizing current during the upstroke of the action p

62 ing sodium channel that conducts a transient depolarizing current in multidendritic sensory neurons o

63 Single pulses or a train of pulses of depolarizing current injected into an ET cell evoked sup

64 id not affect spike frequency in response to depolarizing current injection or hyperpolarization-indu

65 re quiescent or made to fire at low rates by depolarizing current injection, light-induced activation

66 the voltage-response to hyperpolarizing and depolarizing current injection.

67 The magnitude of [Ca(2+)]i reduction during depolarizing current injections correlated with the ampl

68 fying, and analyzing the firing responses to depolarizing current injections for every hippocampal ne

69 The maximum spike frequency evoked by depolarizing current injections in Scn8a(N1768D/+) CA1,

70 e majority of cells preset at -80 mV, 500 ms depolarizing current injections to cells led to a brief

71 by increased neuronal spiking in response to depolarizing current injections, whereas blockade of Kv7

72 ippocampal neurons in response to repetitive depolarizing current injections.

73 re performed and, following the injection of depolarizing current into the dendrites, layer 5 neurons

74 y quantifying the impact of an infinitesimal depolarizing current pulse on the time of occurrence of

75 the number of action potentials elicited by depolarizing current pulses was significantly increased

76 l Pv interneuron activation by intracellular depolarizing current pulses.

77 ion of TRN neurons, synaptic inputs or brief depolarizing current steps led to long-lasting plateau p

78 persistent firing that is induced by a brief depolarizing current stimulus in the presence of muscari

79 When driven by various forms of depolarizing current stimulus, Re neurons display consid

80 ted sodium channel Na(V)1.4 conducts the key depolarizing current that drives the upstroke of the ske

81 ion of duck TG neurons evokes high-amplitude depolarizing current with a low threshold of activation,

82 esponse increased indefinitely with injected depolarizing current, but reached saturation with chemic

83 on in vascular myocytes enables a persistent depolarizing current, leading to constriction of coronar

84 tion of an M1-coupled, pirenzepine-sensitive depolarizing current, which appeared to be, at least in

85 A receptor activation initiated a secondary, depolarizing current.

86 sponses to hyperpolarizing (P < 0.00007) and depolarizing currents (P < 0.001) in threshold electroto

87 ber of action potentials that were evoked by depolarizing currents as well as maximal firing rates we

88 e 2 setting through (1) providing additional depolarizing currents during action potential plateau ph

89 edictions and experimental results: net soma depolarizing currents increased choice hysteresis, while

90 In contrast, with stronger depolarizing currents, eliciting firing above approximat

91 s, PAG shifted the transwall gradient in the depolarizing direction.

92 NOS-KO mice, the gradient was shifted in the depolarizing direction.

93 channels seem to provide the major nonlinear depolarizing drive in thin dendrites, even allowing full

94 t (AIS) showed that axo-axonic synapses were depolarizing during this period.

95 citability, we mimicked the mutant channel's depolarizing effect by current injection to produce equi

96 patch-clamp recordings demonstrated that the depolarizing effect of 5-HT on PVINs was mediated by 5-H

97 The depolarizing effect of GLP-1 on electrical activity was

98 est that GABA controls STDP polarity through depolarizing effects at distal dendrites of striatal out

99 The depolarizing effects of GABA are strongest at the soma o

100 lopmentally regulated and cell-type-specific depolarizing effects on auditory brainstem neurons of Mo

101 no such effect was observed when placing the depolarizing electrode over lateral PFC.

102 We placed the soma depolarizing electrode over medial frontal PFC.

103 idification, crucial for ClC-5 activation by depolarizing endosomes.

104 ic ventral posterior medial neurons and with depolarizing events in the posterior nucleus neurons.

105 to OPA1 cleavage induced in CGNs by removing depolarizing extracellular potassium (5K apoptotic condi

106 ernal depolarizing structures and associated depolarizing foci.

107 s in adolescent and adult rat DGCs are still depolarizing from rest.

108 These include the depolarizing 'funny current' (If ) and the sodium-calciu

109 The neurosteroid estradiol potently augments depolarizing GABA action in the immature hypothalamus by

110 ur findings show that early post-SE abnormal depolarizing GABA and p75(NTR) signaling fosters a long-

111 evelopment, yet the mechanisms that regulate depolarizing GABA are not well understood.

112 ing the seizure significantly attenuated the depolarizing GABA(A)R responses and also reduced the ext

113 s of the K(+)/Cl(-) cotransporter KCC2 and a depolarizing GABAA receptor-mediated synaptic component

114 We found that depolarizing GABAergic and glutamatergic currents act in

115 A similar depolarizing GABAergic plexus exists in the developing h

116 increase in glutamatergic input and requires depolarizing GABAergic transmission and NMDA receptor ac

117 fuse intracerebrally a specific inhibitor of depolarizing GABAergic transmission as well as a functio

118 rewiring of excitatory circuits, an abnormal depolarizing gamma-aminobutyric acidergic (GABAergic) dr

119 Our simulations suggest that depolarizing GGN at its input branch can globally inhibi

120 ent individual principal cells from strongly depolarizing granule cells, which likely discharge in re

121 13R expression is uniquely associated with a depolarizing, HCO3(-) independent, Cl(-) -conductance in

122 Preconditioning the cell model with depolarizing/hyperpolarizing prepulses allowed us to hig

123 abilization could also be elicited by single depolarizing/hyperpolarizing pulses of very high field s

124 tracellular Na(+) (Na(i)) that decreases the depolarizing I(NCX) thereby suppressing the action poten

125 enerated in vitro from progenitors exhibited depolarizing, immature GABA responses, like those of ear

126 ointestinal physiology because they transmit depolarizing impulses in enteric neurons, thereby enabli

127 rpolarizing in CA1 principal cells (PCs) but depolarizing in dentate gyrus (DG) PCs.

128 ge from hyperpolarizing in intact neurons to depolarizing in injured neurons.

129 and miR-34c, which subsequently targeted key depolarizing (INa) and repolarizing (Ito) currents alter

130 easing activity during behavior and directly depolarizing inhibitory cells.

131 ounted for largely by granule cells, receive depolarizing input from M/T dendrites and in turn inhibi

132 athway provides on average 3.7-fold stronger depolarizing input to layer 2/3 inhibitory PV neurons th

133 li), persistent spiking in response to brief depolarizing inputs and the relationship between firing

134 In the FB(LM-->V1) pathway, depolarizing inputs to layer 2/3 PV neurons and Pyr cell

135 aptic activation of mGluR5 acts primarily by depolarizing interneurons and evoking widespread dendrit

136 g to a loss of ion selectivity and gain of a depolarizing inward cation conductance.

137 A depolarizing 'leak' current supports this firing pattern

138 atrial cardiomyocyte monolayers expressing a depolarizing light-gated ion channel (Ca(2+)-translocati

139 We demonstrate that the depolarizing M-current conducted by Kv7 channel is signi

140 al of 30.5% of the drusen exhibited internal depolarizing material; 0.3% presented overlying hyperref

141 Despite depolarizing MCs directly, the net effect of nAChR activ

142 rrent clamp, block of BKCa current increased depolarizing membrane potential excursions, raising the

143 amped horizontal cells, BKCa channels subdue depolarizing membrane potential excursions, reduce the a

144 Resulting depolarization conducts to SMCs, depolarizing membrane potential, activating L-type Ca(2+

145 In response to a depolarizing membrane potential, the S4 helix undergoes

146 It is triggered by depolarizing mitochondria or promoting PTP openings.

147 by potent inhalation anesthetics and/or the depolarizing muscle relaxant succinylcholine in malignan

148 In both cell types, depolarizing neurons increased the size of voltage fluct

149 bative: e.g., by intentionally polarizing or depolarizing one spin species while detecting the respon

150 ience research through their use as membrane-depolarizing optogenetic tools for targeted photoactivat

151 manipulated the activity of CI1 by injecting depolarizing or hyperpolarizing current or killing the c

152 post-trial activity decay through simulated depolarizing or hyperpolarizing network stimulation.

153 With mildly depolarizing or hyperpolarizing pulses just above thresh

154 oltage sensor domains, in response to either depolarizing or polarizing transmembrane voltages, to op

155 Depolarizing PCs initiated a long-lasting increase in GA

156 Depolarizing pFL neurons produced active expiration at r

157 shallow hyperpolarizing phase followed by a depolarizing phase of briefer duration.

158 odium (Nav) channels are responsible for the depolarizing phase of the action potential in most nerve

159 may hold even greater potential as tools for depolarizing political debates and resolving policy disp

160 At a depolarizing potential of -5 mV, the ICa,L current densi

161 Early network giant depolarizing potential oscillatory activity was compromi

162 l neurons between two SLEs correlated with a depolarizing potential.

163 ntaneous network events known as field giant depolarizing potentials (fGDPs) and of the unit activity

164 Two primary burst types were studied: giant depolarizing potentials (GDPs) and spontaneous intericta

165 this activity is observed in vitro as giant depolarizing potentials (GDPs) during the first postnata

166 ting in synergy with glutamate, drives giant depolarizing potentials (GDPs) in the hippocampal networ

167 taneous network events, referred to as giant depolarizing potentials (GDPs) in the hippocampus.

168 Spontaneous giant depolarizing potentials (GDPs) were first identified wit

169 y, SPAs are replaced by synapse-driven giant depolarizing potentials (GDPs).

170 ated nicotinic ACh receptor (nAChR)-mediated depolarizing potentials and muscarinic ACh receptor (mAC

171 Our analysis reveals that these giant depolarizing potentials are mediated by the activation o

172 rticocallosal neurons lacking mAChR-mediated depolarizing potentials did not show persistent firing.

173 These prolonged depolarizing potentials generated persistent firing in c

174 rons, ACh generated prolonged mAChR-mediated depolarizing potentials in corticocollicular neurons.

175 the conductance-voltage relationship to more depolarizing potentials with a half-maximal effective co

176 re hyperexcitable and generated long-lasting depolarizing potentials with bursts of action potentials

177 im, calcium transients associated with giant depolarizing potentials, a hallmark of developmental net

178 ous activities appeared in the form of giant depolarizing potentials.

179 s, ACh release also generated nAChR-mediated depolarizing potentials.

180 olonged critical period permissive for giant depolarizing potentials.

181 ous MN activity is driven by recurrent giant depolarizing potentials.

182 and voltage, so that opening is promoted by depolarizing potentials.

183 rectification because of polyamine block at depolarizing potentials.

184 tiated delayed rectifier Kv1 channels at low depolarizing potentials.

185 and slowing of deactivation in response to a depolarizing prepulse.

186 inhibition was voltage-dependent, and strong depolarizing prepulses attenuated Hm-3 activity.

187 ) APD(90) in response to hyperpolarizing and depolarizing prepulses, respectively, whereas other mode

188 time course of recovery from short and long depolarizing prepulses, which, under drug-free condition

189 on was voltage-independent and unaffected by depolarizing prepulses.

190 oltage-dependent and transiently relieved by depolarizing prepulses.

191 ChR2 elevating the probability of release by depolarizing presynaptic boutons.

192 entries using Grover's QSA at an aggressive depolarizing probability of 10(-3), the success probabil

193 Thalamic neurons respond to a brief depolarizing pulse with a burst of action potentials; ho

194 Using long (20 s) depolarizing pulses both gating modes were activated, an

195 The sADPs generated by repeated depolarizing pulses summed to promote a plateau potentia

196 e receptor agonist (4-chloro-meta-cresol) or depolarizing pulses were used.

197 -VSD deactivation kinetics were modulated by depolarizing pulses with durations in the intermediate t

198 Finally, during a train of depolarizing pulses, paired pulse plasticity was signifi

199 cells of Ca(2+) alternans produced by small depolarizing pulses.

200 peting attractor network models predict slow depolarizing ramps.

201 rent-induced firing threshold, and decreased depolarizing response to NMDA in deep-layer PL-PFC neuro

202 and OFF synaptic layers, but with a pure ON (depolarizing) response to light.

203 Depolarizing responses in VIP or PV BCs resulted in incr

204 n of GABAergic cortical neurons is driven by depolarizing responses to ambient GABA present in the co

205 d Go-opsin1 coexpressed with two r-opsins in depolarizing rhabdomeric photoreceptor cells in the pigm

206 e polarized than control cells, confirming a depolarizing role of TWIK1 in kidney and pancreatic cell

207 rked changes in the hyperpolarization-evoked depolarizing sag and rebound firing across these groups.

208 Ih was identified to be responsible for the depolarizing sag and was increased across OVX --> diestr

209 fterhyperpolarization, firing frequency, and depolarizing sag), whereas the differences in the first

210 rebound or post-stimulation recovery, and no depolarizing sag.

211 -frequency plateau bursts, associated with a depolarizing shift in action potential threshold.

212 Ser-561 and Ser-641/Thr-642 recapitulate the depolarizing shift in activation and reduction in curren

213 l mutations (W1775R and L1831X) exhibiting a depolarizing shift in channel activation.

214 Application of VU0463271 caused a reversible depolarizing shift in E(GABA) values and increased spiki

215 According to computational modeling, a depolarizing shift in GABA reversal potential (EGABA) an

216 uced membrane hyperpolarization and caused a depolarizing shift in GABA reversal potential of dorsal

217 ificant increase in the I(h) amplitude and a depolarizing shift in I(h) activation curve.

218 shold voltage for activation, and produced a depolarizing shift in inactivation in wild-type - but no

219 Whether the depolarizing shift in resting potential and enhanced spo

220 indicated that this increase is caused by a depolarizing shift in the activation curve of the native

221 physically distinct K(+) currents revealed a depolarizing shift in the activation of a rapidly inacti

222 aturating concentrations, SNX-482 produced a depolarizing shift in the voltage dependence of activati

223 istent currents by 72% and produces a 5.8-mV depolarizing shift in the voltage dependence of activati

224 of kinetics, and, most surprisingly, a 30 mV depolarizing shift in the voltage dependence of activati

225 CCt induced a depolarizing shift in the voltage dependence of both CaV

226 nction modulator that inhibits the canonical depolarizing shift in the voltage dependence of HCN4 in

227 rent, incomplete channel inactivation, and a depolarizing shift in the voltage dependence of steady-s

228 ts revealed that L203P at S4 induces a large depolarizing shift in voltage dependence of K(v)7.2 chan

229 SNAP caused a depolarizing shift in voltage-dependent N-type channel a

230 ming, and larger ramp currents, override the depolarizing shift of activation, to produce hyperexcita

231 Removal of the beta3-ECD abrogated both the depolarizing shift of steady-state inactivation and the

232 urrent amplitudes (3 pore mutations) or by a depolarizing shift of the activation curve (2 voltage se

233 fter spinal cord injury (SCI) resulting in a depolarizing shift of the chloride equilibrium potential

234 Maximal depolarizing shift of the If activation curve induced by

235 fect the previously observed beta3-dependent depolarizing shift of V (1/2) of steady-state inactivati

236 d with loss-of-function effects, including a depolarizing shift of voltage-dependent activation or a

237 )) of PVN presympathetic neurons undergoes a depolarizing shift that diminishes GABA inhibition in sp

238 isolated from ST3Gal4(-/-) mice demonstrated depolarizing shifts in activation gating of the transien

239 Non-depolarizing shunts of 3-10 nS converted cells from clas

240 Persistently depolarizing sodium (Na(+)) leak currents enhance electr

241 out preventing further prolongation by brief depolarizing somatic prepulses.

242 ulse potentiation," in which activation by a depolarizing step facilitates activation in a subsequent

243 reported that the K current in response to a depolarizing step to ENa was delayed if the step was pre

244 We found that application of brief depolarizing steps (<100 ms) to cones evoked exocytosis

245 age-dependence of channel activation so that depolarizing steps evoke larger sodium currents.

246 ion, rather than to the timing of subsequent depolarizing steps, suggesting that cholinergic signal t

247 During depolarizing stimulation, newly appearing vesicles appro

248 CO2/H+ changes), in the absence of external depolarizing stimulation, showed no signs of postinhibit

249 unced slowing of current inactivation during depolarizing stimuli (p.G407R).

250 ormally governs cortical neuron responses to depolarizing stimuli by opposing prolonged discharges an

251 porting persistent firing modes triggered by depolarizing stimuli following cholinergic receptor acti

252 T-channels switched burst firing with lower depolarizing stimuli to regular spiking, and fully aboli

253 entate granule neurons in response to strong depolarizing stimuli was also observed.

254 xhibited an inability to properly respond to depolarizing stimuli, demonstrating that Na(V)1.7 is a k

255 ted modest transient electrical responses to depolarizing stimuli, revealing the potential for circad

256 ted modest transient electrical responses to depolarizing stimuli.

257 granules and are differentially activated by depolarizing stimuli.

258 d secrete it in response to glucose or other depolarizing stimuli.

259 unds were able to block Ca(2+) entry after a depolarizing stimulus and showed an improved Cav1.3/Cav1

260 rotein densin and CaMKII and that outlasts a depolarizing stimulus by seconds.

261 il current of up to 8 s duration following a depolarizing stimulus in both tsA-201 cells and male rat

262 enings that can last for seconds following a depolarizing stimulus train.

263 d in relocation of the AIS of DGCs without a depolarizing stimulus.

264 utward tail current of up to 8 s following a depolarizing stimulus.

265 litation of channel activity that outlasts a depolarizing stimulus.

266 n scheme, including the presence of internal depolarizing structures and associated depolarizing foci

267 rk models in which neurons receive sustained depolarizing synaptic input during a field crossing, suc

268 peak spike rates, and were more sensitive to depolarizing synaptic input.

269 n contrast, PID ripples were associated with depolarizing synaptic inputs frequently reaching the thr

270 totic capacitance increases evoked by 200-ms depolarizing test steps, whereas FALI more strongly inhi

271 ide-gated (HCN) channels, and contributes to depolarizing the afferent to potentials where a single E

272 y of ENaCs, which augments synaptic drive by depolarizing the basal membrane potential close to the a

273 rast, IRAG causes a gain of HCN4 function by depolarizing the basal voltage dependence in the absence

274 to heat, which changes membrane capacitance, depolarizing the cell and eliciting action potentials.

275 ctivity of sarcolemmal Na(+)-Ca(2+) exchange depolarizing the cell membrane.

276 pulating the endogenous bioelectric state by depolarizing the injured tissue during the first 3 h of

277 ong the GnRH neuron projection is capable of depolarizing the membrane potential and initiating actio

278 are biased toward open/inactivated states by depolarizing the membrane potential under voltage-clamp

279 on potential firing to current injection and depolarizing the membrane potential.

280 intains viability during oxygen depletion by depolarizing the membrane.

281 hore that uncouples the mitochondria without depolarizing the plasma membrane, as a lead compound for

282 g the firing rate and regularity, as well as depolarizing the resting membrane potential in mHb ChNs

283 oss-linked 300 kDa increased excitability by depolarizing the resting membrane potential, and decreas

284 es IP3R up-regulation during hypertension by depolarizing the VSM cell membrane.

285 l enhancement is accomplished by selectively depolarizing the xenon within a cage molecule which, upo

286 cted selectively to increase their activity, depolarizing these neurons and increasing their firing r

287 An accompanying increase in depolarizing threshold electrotonus at 90 to 100 millise

288 alteration in recovery cycle parameters and depolarizing threshold electrotonus.

289 ts transiently switched their direction from depolarizing to hyperpolarizing as a result of neuronal

290 We found that the timing of the switch from depolarizing to hyperpolarizing GABA is delayed in the c

291 irectionally selective (DS) light responses, depolarizing to stimuli that move centrifugally away fro

292 itial membrane potential, the climbing fiber depolarizing transient activates two distinct sets of ch

293 onist during afferent stimulation trains and depolarizing voltage steps caused a significant, sustain

294 A 5 Hz, 1 min train of depolarizing voltage steps elicited voltage-gated Ca(2+)

295 ed that strong hyperpolarization preceding a depolarizing voltage-clamp pulse delayed the rise of the

296 Being activated by depolarizing voltages and increases in cytoplasmic Ca(2+

297 JZTx-27 was more efficacious at weaker depolarizing voltages and significantly slowed the activ

298 is not dependent on the level of maturation (depolarizing vs. hyperpolarizing) of postsynaptic GABAA

299 AP rise times and conduction velocity as the depolarizing wavefront approaches the epicardial surface

300 V1 on ganglion cells from hyperpolarizing to depolarizing, which was also transient.