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1 , which was brief but sufficient to activate dermal dendritic cells.
2 ers of lymph node-resident but not migratory dermal dendritic cells.
3 en by pMHCII presentation by local CD11b(hi) dermal dendritic cells.
4 ere revealed to be T-cells, macrophages, and dermal dendritic cells.
5 3 was found to reduce Treg interactions with dermal dendritic cells.
6 ic cells, Langerhans cells, and interstitial dermal dendritic cells.
7 mal injection by LECs was similar to that of dermal dendritic cells.
8                                     CD11c(+) dermal dendritic cells and CD163(+) macrophages expresse
9                                              Dermal dendritic cells and epidermal Langerhans cells ar
10 tifungal defenses through their influence on dermal dendritic cells and induction of IL-17A.
11 ells, infiltrating epidermal CD8(+) T cells, dermal dendritic cells and macrophages, and increased ex
12 sident memory T cells, Langerhans cells, and dermal dendritic cells, and there is growing interest in
13 ection of Langerhans cells, macrophages, and dermal dendritic cells, and these cells emigrated from s
14                                     Finally, dermal dendritic cells, but not LCs isolated from the dr
15 ection of Langerhans cells, macrophages, and dermal dendritic cells by 75-90% and reduced the overall
16                   In this study, we explored dermal dendritic cell (DC) homeostasis in mice and human
17 ocytes, epidermal Langerhans cells (LC), and dermal dendritic cells (DC).
18                                      Myeloid dermal dendritic cells (DCs) accumulate in chronically i
19 es occur via 'cooperation' between migratory dermal dendritic cells (DCs) and basophils positive for
20 ed by CD31(+) endothelial cells and CD11c(+) dermal dendritic cells (DCs) in lesional psoriatic skin.
21 sease, P. gingivalis associates in situ with dermal dendritic cells (DCs), many of which express DC-S
22 inducible elimination of LCs and Langerin(+) dermal dendritic cells (dDCs) after administration of di
23 harbours specialized immune cells, including dermal dendritic cells (DDCs) and interleukin (IL)-17-pr
24 avital two-photon microscopy, we report that dermal dendritic cells (DDCs) and Langerhans cells (LCs)
25 of human skin migratory CD1a+ LCs and CD11c+ dermal dendritic cells (DDCs) demonstrated significant d
26 capacity of human Langerhans cells (LCs) and dermal dendritic cells (DDCs) to induce antigen-specific
27 ans required IL-23 production from CD301b(+) dermal dendritic cells (dDCs).
28 l lipid antigens to specific T cells whereas dermal dendritic cells express much less CD1a molecules
29 rhans cells, expressing CD1a and HLA-DR, and dermal dendritic cells, expressing HLA-DR, are known to
30  difference in the migration of hapten-laden dermal dendritic cells (FITC+, CD11c+, Langerin-) into t
31 ss from skin and enables the accumulation of dermal dendritic cells in skin-draining LN.
32  of CD1 was detected on cortical thymocytes, dermal dendritic cells in the skin, follicular dendritic
33 onal and monocyte derived) but not migratory dermal dendritic cells in the skin-draining lymph nodes
34 y taken up by epidermal Langerhans cells and dermal dendritic cells in the vicinity of the MCs or tra
35 in the number of resident (but not migratory dermal) dendritic cells in the lymph node but showed no
36 ls, including Langerhans cells, macrophages, dermal dendritic cells, mast cells, fibroblasts, and lym
37 eration of contact hypersensitivity and that dermal dendritic cells may play a more important role.
38                                  reveal that dermal dendritic cells produce interleukin-31, which act
39 fection of Langerhans cells and interstitial dermal dendritic cells results in an impaired ability to
40  Specifically, RUBCN deficiency in CD301b(+) dermal dendritic cells results in their increased antige
41 lines in CD4 T cells, whereas Langerhans and dermal dendritic cells serve as viral reservoirs and poi
42 D103-mediated interactions between Tregs and dermal dendritic cells support regulation of skin inflam
43 A to the skin can target distinct subsets of dermal dendritic cells to confer a superior immune respo
44         While T cells, Langerhans cells, and dermal dendritic cells were infected abortively, keratin
45  Langerhans cells in the dermis and CD11c(+) dermal dendritic cells were observed, corresponding to t
46 yte-derived dendritic cells and interstitial dermal dendritic cells, yet the virus fully replicates o