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1 life-threatening fungal infection caused by dermatophytes.
2 nterlaboratory agreement when tested against dermatophytes.
3 ing method has been standardized for testing dermatophytes.
4 o determine the antifungal susceptibility of dermatophytes.
5 ool for antifungal susceptibility testing of dermatophytes.
6 ample, Eurotiomycetes, which includes common dermatophytes.
7 does not specifically address the testing of dermatophytes.
8 and, Ohio, for testing the susceptibility of dermatophytes.
9 i does not explicitly address the testing of dermatophytes.
10 nst various fungi species, such as molds and dermatophytes.
11 the NCCLS M38-A standard for the testing of dermatophytes.
12 usefulness for presumptive identification of dermatophytes.
13 y that discriminated T. tonsurans from other dermatophytes.
14 idation of the method with a large number of dermatophytes.
15 antifungal susceptibility testing method for dermatophytes.
16 ghest antifungal activity against all of the dermatophytes.
17 the susceptibility testing of ME1111 against dermatophytes according to M38-A2 methodology, which sti
19 ndula luisieri essential oils against yeast, dermatophyte and Aspergillus strains responsible for hum
20 an optimal medium for conidial formation by dermatophytes and (ii) validation of the method with a l
22 s, such as highly divergent ATG8 paralogs in dermatophytes and multiple ATG15 duplications in mushroo
25 quence, we questioned whether its binding to dermatophytes can induce tyrosine phosphorylation in den
29 DiversiLab system for identification of the dermatophytes commonly encountered in a clinical mycolog
30 the DiversiLab system for identification of dermatophytes commonly isolated in a clinical laboratory
31 t the in vitro activities of TDT 067 against dermatophytes, compared with those of the Transfersome v
32 archived dermatophyte isolates and 71 fresh dermatophyte cultures were evaluated using both librarie
33 spectrometer (MS) for the identification of dermatophytes from clinical cultures was compared to tha
35 btilase homologue, Tri r 2, derived from the dermatophyte fungus Trichophyton rubrum, exhibits unique
38 Only 25 of 77 dermatophytic isolates caused dermatophyte identification medium (DIM) to turn purple
39 om clinical cultures was compared to that of dermatophyte identification using 28S rRNA gene sequenci
40 tive to traditional or molecular methods for dermatophyte identification, provided that the reference
43 the introduction of this method for testing dermatophytes in the future version of the CLSI M38-A st
46 arotitis, polymicrobial bacteremia, invasive dermatophyte infection and Clostridium difficile-associa
47 ndition is different from common superficial dermatophyte infection and has been reported in patients
49 pes zoster (IR, 1.11; 95% CI, 0.88-1.39), 57 dermatophyte infections (IR, 0.88; 0.67-1.14), and 52 or
50 , are unable to clear superficial Candida or Dermatophyte infections and suffer with chronic mucocuta
53 of seven antifungal agents tested against 25 dermatophyte isolates (5 blinded pairs of five dermatoph
54 One hundred well-characterized, archived dermatophyte isolates and 71 fresh dermatophyte cultures
55 study, the voriconazole susceptibilities of dermatophyte isolates obtained from a worldwide tinea ca
61 ed the antifungal susceptibility profiles of dermatophytes received by our laboratory from institutio
63 ve identification of an unknown isolate as a dermatophyte required only the transfer of a portion of
65 rmatophyte isolates (5 blinded pairs of five dermatophyte species per site for a total of 300 tests),
66 tion of multiple genetic strains of a single dermatophyte species should not be unexpected in areas o
70 by anthropophilic, geophilic, and zoophilic dermatophyte strains and (ii) the keratinocyte signaling
71 067 demonstrated potent activity against the dermatophyte strains tested, with an MIC range of 0.0000
73 mation along with the optimal conditions for dermatophyte susceptibility testing proposed by Norris e
76 r a total of 300 tests), using the method of dermatophyte testing developed at the Center for Medical
77 has more potent antifungal activity against dermatophytes that cause nail infection than conventiona
78 mined the antifungal susceptibilities of 217 dermatophytes to fluconazole, griseofulvin, itraconazole
79 r determining antifungal susceptibilities of dermatophytes to terbinafine, ciclopirox, and voriconazo
82 efine two distinct antigens derived from the dermatophyte Trichophyton that serve as targets for dive
83 howed soluble recombinant DC-HIL to bind the dermatophytes Trichophyton rubrum and Microsporum audoui
84 ound have been tested against the pathogenic dermatophytes Trichophyton rubrum and Trichophyton menta
88 s demonstrate that terbinafine resistance in dermatophytes was relatively common over this 2-year per
90 voriconazole against 19 different species of dermatophytes were compared with those of terbinafine, i
93 ates, with the exception of 45 yeasts and 15 dermatophytes, were recovered from both storage temperat