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1  life-threatening fungal infection caused by dermatophytes.
2 nterlaboratory agreement when tested against dermatophytes.
3 ing method has been standardized for testing dermatophytes.
4 o determine the antifungal susceptibility of dermatophytes.
5 ool for antifungal susceptibility testing of dermatophytes.
6 ample, Eurotiomycetes, which includes common dermatophytes.
7 does not specifically address the testing of dermatophytes.
8 and, Ohio, for testing the susceptibility of dermatophytes.
9 i does not explicitly address the testing of dermatophytes.
10 nst various fungi species, such as molds and dermatophytes.
11  the NCCLS M38-A standard for the testing of dermatophytes.
12 usefulness for presumptive identification of dermatophytes.
13 y that discriminated T. tonsurans from other dermatophytes.
14 idation of the method with a large number of dermatophytes.
15 antifungal susceptibility testing method for dermatophytes.
16 ghest antifungal activity against all of the dermatophytes.
17 the susceptibility testing of ME1111 against dermatophytes according to M38-A2 methodology, which sti
18 to test the susceptibility of 47 isolates of dermatophytes against 8 antifungals.
19 ndula luisieri essential oils against yeast, dermatophyte and Aspergillus strains responsible for hum
20  an optimal medium for conidial formation by dermatophytes and (ii) validation of the method with a l
21           In studies of over 500 isolates of dermatophytes and common nondermatophyte molds, as well
22 s, such as highly divergent ATG8 paralogs in dermatophytes and multiple ATG15 duplications in mushroo
23                                              Dermatophytes are common causes of skin, hair, and nail
24                                              Dermatophyte binding was inhibited completely by the add
25 quence, we questioned whether its binding to dermatophytes can induce tyrosine phosphorylation in den
26                                              Dermatophytes cause superficial and cutaneous fungal inf
27 c fungi that were morphologically similar to dermatophytes caused false-positive results.
28 the isolates for the majority of tested drug-dermatophyte combinations.
29  DiversiLab system for identification of the dermatophytes commonly encountered in a clinical mycolog
30  the DiversiLab system for identification of dermatophytes commonly isolated in a clinical laboratory
31 t the in vitro activities of TDT 067 against dermatophytes, compared with those of the Transfersome v
32  archived dermatophyte isolates and 71 fresh dermatophyte cultures were evaluated using both librarie
33  spectrometer (MS) for the identification of dermatophytes from clinical cultures was compared to tha
34                                          The dermatophyte fungus Trichophyton exhibits unique immunol
35 btilase homologue, Tri r 2, derived from the dermatophyte fungus Trichophyton rubrum, exhibits unique
36        Skin testing with an extract from the dermatophyte fungus Trichophyton tonsurans can result in
37                                A new medium, Dermatophyte Identification Medium (DIM) (trade mark pen
38  Only 25 of 77 dermatophytic isolates caused dermatophyte identification medium (DIM) to turn purple
39 om clinical cultures was compared to that of dermatophyte identification using 28S rRNA gene sequenci
40 tive to traditional or molecular methods for dermatophyte identification, provided that the reference
41          A standardized reference method for dermatophyte in vitro susceptibility testing is lacking.
42 method for the presumptive identification of dermatophytes in the clinical mycology laboratory.
43  the introduction of this method for testing dermatophytes in the future version of the CLSI M38-A st
44                    Initially, he worked with dermatophytes in Winnipeg, where he was influenced by Bu
45 aptation of S. aureus to the colonization of dermatophyte-infected hedgehogs.
46 arotitis, polymicrobial bacteremia, invasive dermatophyte infection and Clostridium difficile-associa
47 ndition is different from common superficial dermatophyte infection and has been reported in patients
48      Over half of outpatient visits were for dermatophyte infections (4981444 visits, total cost $802
49 pes zoster (IR, 1.11; 95% CI, 0.88-1.39), 57 dermatophyte infections (IR, 0.88; 0.67-1.14), and 52 or
50 , are unable to clear superficial Candida or Dermatophyte infections and suffer with chronic mucocuta
51                Mucocutaneous candidiasis and dermatophyte infections occur either in isolation or alo
52                                              Dermatophytes initiate dermatophytosis, but susceptibili
53 of seven antifungal agents tested against 25 dermatophyte isolates (5 blinded pairs of five dermatoph
54     One hundred well-characterized, archived dermatophyte isolates and 71 fresh dermatophyte cultures
55  study, the voriconazole susceptibilities of dermatophyte isolates obtained from a worldwide tinea ca
56               During this 2-year period, 271 dermatophyte isolates were included, the majority of whi
57                                  Forty-eight dermatophyte isolates, including Trichophyton rubrum (n
58 f squalene epoxidase mutations among toenail dermatophyte isolates.
59                                              Dermatophytes of the genus Trichophyton cause infections
60 e fungal diseases caused by Candida species, dermatophytes, or Phialophora verrucosa.
61 ed the antifungal susceptibility profiles of dermatophytes received by our laboratory from institutio
62  understanding of cutaneous immunity against dermatophytes remains limited.
63 ve identification of an unknown isolate as a dermatophyte required only the transfer of a portion of
64                  Although various species of dermatophytes respond to common antifungal drugs, the re
65 rmatophyte isolates (5 blinded pairs of five dermatophyte species per site for a total of 300 tests),
66 tion of multiple genetic strains of a single dermatophyte species should not be unexpected in areas o
67                              Initially, five dermatophyte species were tested for their ability to in
68       Clinical information was reviewed, and dermatophyte species with or without squalene epoxidase
69 lemented library containing an additional 20 dermatophyte spectra (S-MBL).
70  by anthropophilic, geophilic, and zoophilic dermatophyte strains and (ii) the keratinocyte signaling
71 067 demonstrated potent activity against the dermatophyte strains tested, with an MIC range of 0.0000
72       Whereas superficial fungal diseases by dermatophytes such as athlete's foot and onychomycosis a
73 mation along with the optimal conditions for dermatophyte susceptibility testing proposed by Norris e
74 ed with commercially marketed media, such as dermatophyte test medium (DTM).
75                                          The dermatophytes tested included Trichophyton rubrum, Trich
76 r a total of 300 tests), using the method of dermatophyte testing developed at the Center for Medical
77  has more potent antifungal activity against dermatophytes that cause nail infection than conventiona
78 mined the antifungal susceptibilities of 217 dermatophytes to fluconazole, griseofulvin, itraconazole
79 r determining antifungal susceptibilities of dermatophytes to terbinafine, ciclopirox, and voriconazo
80        We also demonstrate that the hedgehog dermatophyte Trichophyton erinacei produces two beta-lac
81 s a nonpathogenic fungus which resembles the dermatophyte Trichophyton rubrum.
82 efine two distinct antigens derived from the dermatophyte Trichophyton that serve as targets for dive
83 howed soluble recombinant DC-HIL to bind the dermatophytes Trichophyton rubrum and Microsporum audoui
84 ound have been tested against the pathogenic dermatophytes Trichophyton rubrum and Trichophyton menta
85 matory responses induced by a representative dermatophyte, Trichophyton equinum.
86 cating that germination may be important for dermatophyte virulence and host immune activation.
87                             The frequency of dermatophytes was 37.6%; of isolates belonging to the Tr
88 s demonstrate that terbinafine resistance in dermatophytes was relatively common over this 2-year per
89         Using T. equinum as a representative dermatophyte, we found that the mitogen-activated protei
90 voriconazole against 19 different species of dermatophytes were compared with those of terbinafine, i
91  conidial growth, representative isolates of dermatophytes were grown on different agars.
92      Seven antifungals with activity against dermatophytes were tested, including ciclopirox, flucona
93 ates, with the exception of 45 yeasts and 15 dermatophytes, were recovered from both storage temperat