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1 effects on desensitisation or recovery from desensitisation.
2 nological changes corresponded with clinical desensitisation.
3 xpression and their relative contribution to desensitisation.
4 pha 7 nAChR activation and non-alpha 7 nAChR desensitisation.
5 M 5-hydroxyindole (5-OHi) to reduce receptor desensitisation.
6 id not increase the rate of glycine receptor desensitisation.
7 nce or placebo, despite effectively inducing desensitisation.
8 ive tool for rehabilitation involving visual desensitisation.
9 lving sequential re-challenge and rapid drug desensitisation.
10 ral immunotherapy and enables safe and rapid desensitisation.
11 whether peanut oral immunotherapy can induce desensitisation (an increased allergic reaction threshol
15 (threonine-1172-alanine) showed a defect in desensitisation and underwent a more sustained EGF-induc
16 en, 0.07 +/- 0.008; n = 15), little apparent desensitisation and were also activated by alpha,beta-me
17 emotherapy agents, monitor drug tolerance in desensitisation, and predict and address the safety of n
18 nsitisation and faster current recovery from desensitisation, and was mediated by residues facing the
20 We calculated the degree of bronchodilator desensitisation by comparing the dose-response (for maxi
21 37) or without (control, N = 39) the visual desensitisation component for up to 5-10 min, twice dail
26 e existing rehabilitation approach of visual desensitisation into an online game environment to enhan
30 med to establish the efficacy of OIT for the desensitisation of children with allergy to peanuts.
33 consequence of abl PTK activity and involves desensitisation of signal transduction events stimulated
34 superagonist behaviour was caused by reduced desensitisation of the extrasynaptic-type receptors.
36 ed from 1 mM to 100 microM, is mainly due to desensitisation of the SR Ca2+ release mechanism, which
37 st notably the early altered trafficking and desensitisation of Tregs induced by a single ultra-low d
41 eventual loss of the facial transplant, the desensitisation protocol used for this highly immunosens
42 received placebo met the primary outcome of desensitisation (risk difference [RD] 69%, 95% CI 59-79;
43 s significantly more prone to bronchodilator desensitisation than Arg 16, with the influence of Gly 1
47 used to compare the proportion of those with desensitisation to peanut after 6 months between the act
49 ssive ideation, compulsive repetition, rapid desensitisation to violence, diminished affective reacti
51 echanisms, namely sensing, sensitisation and desensitisation, which become important when plants in t
53 (p < 0.05) greater degree of bronchodilator desensitisation with homozygous Gly 16 than with homozyg
54 ponses to P2X agonists showed signs of rapid desensitisation with the peak frequency of discharge bei