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1 toantibodies (IgG) targeting desmoglein 3, a desmosomal cadherin.
2 alloproteinase-dependent proteolysis of this desmosomal cadherin.
3 f possible synergism between a classical and desmosomal cadherin.
4 , including both acetylcholine receptors and desmosomal cadherins.
5 not dependent on the equimolar expression of desmosomal cadherins.
6 ness depends on the organised arrangement of desmosomal cadherins.
7 nents, in which they link desmoplakin to the desmosomal cadherins.
8 nity to desmoglein 3, desmocollin 3, or both desmosomal cadherins.
9 ent (IF)-binding protein desmoplakin (DP) to desmosomal cadherins.
10 attention has been paid to the importance of desmosomal cadherins.
11 ensus sequence not conserved among the other desmosomal cadherins.
12 report a novel role of Gal3 in stabilizing a desmosomal cadherin and intercellular adhesion in intest
13 smocollin 2 increased 1.7-2.0-fold, and both desmosomal cadherin and plaque components were recruited
14        Using purified proteins, we show that desmosomal cadherins and alpha-catenin compete directly
15 these diseases, autoantibodies against other desmosomal cadherins and E-cadherin may also be present.
16 c areas possess autoantibodies against other desmosomal cadherins and E-cadherin.
17               When expressed in L-cells, the desmosomal cadherins and plakoglobin exhibited a diffuse
18                          To determine if the desmosomal cadherins and plakoglobin interact with the a
19 rminal desmoplakin polypeptide (DP-NTP), the desmosomal cadherins and plakoglobin were observed in pu
20  histone deacetylase inhibition up-regulates desmosomal cadherins and prevents the loss of adhesion i
21 ll adhesion by compromising the link between desmosomal cadherins and the intermediate filament cytos
22 ing list of human mutations that target both desmosomal cadherins and their associated cytoskeletal a
23 atable connection between both classical and desmosomal cadherins and their respective cytoskeletal l
24 he intermediate filament cytoskeleton to the desmosomal cadherins and thereby confers structural stab
25 ividing epidermal cells, DP and PG anchor to desmosomal cadherins and to each other, forming an order
26                                          The desmosomal cadherins are calcium-dependent transmembrane
27                   Characteristic patterns of desmosomal cadherins are tightly regulated and distinct
28                                              Desmosomal cadherins are transmembrane adhesion molecule
29 etween adjacent cells, this study implicates desmosomal cadherins as key components of a signaling ax
30 suggest that desmocollin-1 can function as a desmosomal cadherin both in basal and suprabasal cells.
31 igus is caused by IgG autoantibodies against desmosomal cadherins, but the precise mechanisms are in
32                Suprabasal layers upregulated desmosomal cadherins, but without classical cadherins, t
33 at is similar to that of adherens junctions, desmosomal cadherins - called desmogleins and desmocolli
34 dillo repeat region reduces the affinity for desmosomal cadherins, calorimetric measurements show no
35 g2 processing, supporting the idea that this desmosomal cadherin can be regulated by multiple ADAM fa
36   Modulation of the palmitoylation status of desmosomal cadherins can affect desmosome dynamics.
37  arm repeats exhibit distinct binding to the desmosomal cadherins comparable in strength to that of t
38 ferentiation via proteolytic cleavage of the desmosomal cadherin component desmoglein 1 (Dsg1).
39                                          The desmosomal cadherins, comprising the desmogleins and des
40                          The Dsg subclass of desmosomal cadherins contains a C-terminal unique region
41                                          The desmosomal cadherin desmocollin (Dsc)1 is expressed in u
42 wn that one of the two intestinal epithelial desmosomal cadherins, desmocollin-2 (Dsc2) loss promotes
43                                          The desmosomal cadherin desmoglein 2 (Dsg2) localizes to the
44 re the structure of the entire ectodomain of desmosomal cadherin desmoglein 2 (Dsg2), using a combina
45 lgaris (PV), autoantibodies (IgG) target the desmosomal cadherin desmoglein 3 (Dsg3) and compromise k
46 mporal dynamics of order and disorder of the desmosomal cadherin desmoglein 3 (Dsg3) in living cells.
47 ted a central role for downregulation of the desmosomal cadherin desmoglein 3 (DSG3) in the pathogene
48      IgG autoantibodies directed against the desmosomal cadherin desmoglein 3 (Dsg3), the major autoa
49 dies against the extracellular domain of the desmosomal cadherin desmoglein 3 cause potentially fatal
50 utoantibodies (IgG) are directed against the desmosomal cadherin desmoglein 3.
51 we have focused on the palmitoylation of the desmosomal cadherin desmoglein-2 (Dsg2) and characterize
52 n our study, we found that cells lacking the desmosomal cadherin Desmoglein-2 (Dsg2) displayed a sign
53 vulgaris (PV) pathogenic antibodies bind the desmosomal cadherin desmoglein-3 (dsg3), causing epiderm
54 er caused by antibodies directed against the desmosomal cadherin desmoglein-3 (Dsg3).
55      The autoantibodies generated target the desmosomal cadherin desmoglein-3 (Dsg3).
56 caused by autoantibodies primarily targeting desmosomal cadherins desmoglein 3 (DSG3) and DSG1, leadi
57 understand the reciprocal regulation between desmosomal cadherins (desmoglein and desmocollin) and pl
58      Dominant and recessive mutations in the desmosomal cadherin, desmoglein (DSG) 1, cause the skin
59 mosomes through depletion of the superficial desmosomal cadherin, desmoglein 1, impedes basal stratif
60 FR inhibition results in accumulation of the desmosomal cadherin, desmoglein 2 (Dsg2), at cell-cell i
61  recognizing several proteins, including the desmosomal cadherin, desmoglein 3.
62 both a classical cadherin, P-cadherin, and a desmosomal cadherin, desmoglein 3.
63                                          The desmosomal cadherin, desmoglein-1 (DSG1), promotes kerat
64 osomal trafficking component, VPS35, and the desmosomal cadherin, desmoglein-1 (Dsg1).
65 dy we show that loss of the other intestinal desmosomal cadherin, desmoglein-2 (Dsg2) that pairs with
66  filaments (IFs) by its interaction with the desmosomal cadherins, desmoglein (Dsg), and desmocollin
67        The intercellular interactions of the desmosomal cadherins, desmoglein and desmocollin, are re
68 ithelial cells (IEC) exclusively express the desmosomal cadherins, Desmoglein-2 and Desmocollin-2 (Ds
69                                              Desmosomal cadherins, desmogleins (Dsgs) and desmocollin
70                                          The desmosomal cadherins, desmogleins (Dsgs) and desmocollin
71 e it binds to the cytoplasmic domains of the desmosomal cadherins, desmogleins and desmocollins.
72                                          The desmosomal cadherins, desmogleins, and desmocollins medi
73 n desmosome assembly, interactions among the desmosomal cadherins, desmoplakin, and the armadillo fam
74 nstrating that the binding of plakoglobin to desmosomal cadherins does not require its soluble state
75            We show that Dsg2 but not another desmosomal cadherin, Dsc2, is cleaved by cysteine protea
76 in turn promotes differentiation through the desmosomal cadherin Dsg1.
77 ogether, these data demonstrate that partner desmosomal cadherins Dsg2 and Dsc2 play opposing roles i
78 -Dsc or -Dsg antibodies demonstrate that the desmosomal cadherins, Dsg2 and Dsc1a, are involved in a
79 ring disease in which antibodies against the desmosomal cadherin, DSG3 (desmoglein-3), cause acanthol
80 e potential role of differentiation-specific desmosomal cadherins during apoptosis has not been exami
81 lds but, with the exception of classical and desmosomal cadherin EC1 domains, most of them do not app
82 uggest that the extracellular domains of the desmosomal cadherins exhibit functional properties disti
83 n desmoglein 4 (DSG4), a novel member of the desmosomal cadherin family that is expressed in the hair
84 omes contain multiple representatives of the desmosomal cadherin family, which includes three desmogl
85    This fit suggests an arrangement in which desmosomal cadherins form trans interactions but are too
86 clusters on chromosomes 12q and 17q, and the desmosomal cadherin gene cluster on chromosome 18q.
87  clusters on chromosomes 12q and 17q and the desmosomal cadherin gene cluster on chromosome 18q.
88 d the cytoplasmic tails of the transmembrane desmosomal cadherins, has been proposed to link IF to th
89 uses a reduction in the levels of endogenous desmosomal cadherins in a dose-dependent manner, leading
90 : type 1 cadherins in adherens junctions and desmosomal cadherins in desmosomes.
91 othesized that the arrangement, or order, of desmosomal cadherins in the intercellular space is criti
92              In contrast, antibodies against desmosomal cadherins, including human and mouse pemphigu
93 els of 2 genes as the primary genes: DSG2, a desmosomal cadherin involved in Wnt/beta-catenin signali
94                Although the structure of the desmosomal cadherins is known, the desmosome architectur
95 c deletion of desmocollin 3, the other major desmosomal cadherin isoform expressed in the basal epide
96              Rules governing the assembly of desmosomal cadherin isoforms into desmosomes of differen
97 ous disease caused by autoantibodies against desmosomal cadherins known as desmogleins.
98 , which are characterized by the presence of desmosomal cadherins, known as desmogleins and desmocoll
99 an ex vivo human skin model, suggesting that desmosomal cadherins may have different roles during acq
100                                              Desmosomal cadherins mediate cell-cell adhesion in epith
101       Sporadic BCCs also overexpress Dsg2, a desmosomal cadherin normally found in the basal layer.
102 -terminal fragment of desmoglein 2 (Dsg2), a desmosomal cadherin often overexpressed in malignancies.
103 ), which is caused by autoantibodies against desmosomal cadherins, often have dry eye disease.
104 ve strength is the level and organization of desmosomal cadherins on the cell surface.
105 ify a role for desmoplakin in organizing the desmosomal cadherin-plakoglobin complex and provide new
106                  We previously showed that a desmosomal cadherin promotes keratinocyte differentiatio
107 and highlight a novel mechanism by which the desmosomal cadherins regulate beta-catenin signaling.
108 punctate structures made up of transmembrane desmosomal cadherins termed desmoglein-2 (Dsg2) and desm
109                     Desmoglein-2 (Dsg2) is a desmosomal cadherin that is aberrantly expressed in huma
110                     Desmoglein 1 (Dsg1) is a desmosomal cadherin that is essential to epidermal integ
111 ly upon the up-regulation of desmoglein 1, a desmosomal cadherin that maintains the integrity and dif
112                              Desmogleins are desmosomal cadherins that mediate cell-cell adhesion.
113 atively normal intercellular distribution of desmosomal cadherins, their cytoplasmic plaques are spar
114  suggest that a greater understanding of the desmosomal cadherin TMDs will further our understanding
115 mportant property, specific contributions of desmosomal cadherins to intestinal mucosal repair after
116          To directly test the ability of the desmosomal cadherins to mediate adhesion, desmoglein-1 (
117 cyte adhesion in linking the transmembranous desmosomal cadherins to the cytoplasmic keratin filament
118  integral part of desmosomes, where it links desmosomal cadherins to the intermediate filaments.
119                   Differential regulation of desmosomal cadherin transport could provide a mechanism
120 role for endocytic trafficking in regulating desmosomal cadherin turnover and function and raise the
121  Our observations illustrate a new mechanism desmosomal cadherins use to control their surface levels
122 olved in the interaction of plakoglobin with desmosomal cadherins, we have developed direct binding a
123                  When L cells expressing the desmosomal cadherins were tested for the ability to aggr
124 esmosomes mediate cell-cell adhesion through desmosomal cadherins, which differ from classical cadher
125             In contrast to the classical and desmosomal cadherins, which in general consist of 15-17
126 somes mediate intercellular adhesion through desmosomal cadherins, which interface with plakoglobin (
127 keleton, but only gamma-catenin binds to the desmosomal cadherins, which links them to intermediate f

 
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