ライフサイエンスコーパス: desynchronization

1 during baseline conditions (i.e. hippocampal desynchronization).

2 ctions can mediate either synchronization or desynchronization.

3 ation and inhibition of GoC firing and spike desynchronization.

4 amplitude of melatonin release under forced desynchronization.

5 significance of reducing forced ventricular desynchronization.

6 noting whether stimulation produced cortical desynchronization.

7 MS/DB neurons, thereby producing hippocampal desynchronization.

8 ation, whereas males showed stronger mu-beta desynchronization.

9 locks that drives the population to complete desynchronization.

10 between genes-predict symmetry breaking and desynchronization.

11 te on the mechanisms that drive its eventual desynchronization.

12 ompanied with an enhanced rate of cell cycle desynchronization.

13 ization, while acetylcholine caused complete desynchronization.

14 cantly higher intensity to achieve long-term desynchronization.

15 ion (cpLDF), specifically designed to induce desynchronization.

16 cell resolution through a chemically induced desynchronization.

17 l resources by facilitating movement-related desynchronization.

18 y, caused a delay of alpha parieto-occipital desynchronization.

19 inergic modulation, was sufficient to induce desynchronization.

20 f global cortical network synchronization or desynchronization.

21 tates are not equivalent to brief periods of desynchronization.

22 pup by removing littermates induced further desynchronization.

23 identify several mechanisms responsible for desynchronization: (1) shared inhibitory inputs in local

24 spection of individual profiles of localized desynchronization (10-18Hz) revealed left hemispheric do

25 heta-band responses but increased alpha/beta desynchronization (10-25 Hz) that correlated with behavi

26 tricular conduction, and prevent ventricular desynchronization (530 patients).

27 uld be mapped by lateralized beta (12-30 Hz) desynchronization, a widely used marker of action select

28 These FC changes were driven by brain desynchronization across spatial scales (areal, global),

29 lso found less efficient alpha event-related desynchronization (alpha-ERD) and alpha inter-trial phas

30 The degree and duration of desynchronization among SCN neurons depended on both the

31 ther associated with less task-related theta desynchronization, an electrophysiological signature of

32 Desynchronization and arousal distinctly influenced stim

33 ogenetic inhibition of SOM responses blocked desynchronization and decorrelation, demonstrating that

34 n LC neuronal discharge, and a transient EEG desynchronization and decrease in mitral cell discharge.

35 ated weaker post-stimulus beta event-related desynchronization and earlier and shorter event-related

36 Consistent with this link between desynchronization and engagement, rewards had a long-las

37 DAR activity in PV neurons triggered network desynchronization and enhanced broadband y power.

38 Here, we show that two competing hypotheses, desynchronization and entrainment in a population of mod

39 a model in which independent states of local desynchronization and global arousal jointly optimise se

40 ventricular pacing, which causes ventricular desynchronization and has been linked to an increased ri

41 ht to be supported by neocortical alpha/beta desynchronization and hippocampal theta/gamma synchroniz

42 n of cortical network disorganization (i.e., desynchronization and hypersynchronization) that affects

43 The precise behavioral correlates of desynchronization and its global organization are unclea

44 causal relationship between prefrontal beta desynchronization and memory formation.

45 al dual-chamber pacing, prevents ventricular desynchronization and moderately reduces the risk of per

46 This decrease was caused by a desynchronization and overall reduction in frequency of

47 ustom auto-similarity function to assess the desynchronization and quantify the convergence to an asy

48 ignificantly improve the stimulation-induced desynchronization and reduce the amount of the administe

49 y and severely impaired participants showing desynchronization and synchronization, respectively; and

50 that the strength of the peri-movement beta desynchronization and the post-movement beta rebound wer

51 owing they are involved in ACh-dependent EEG desynchronization, and others suggesting that this effec

52 apLDF, the extent of the stimulation-induced desynchronization, and the integral stimulation time req

53 e processing models suggesting neuro-cardiac desynchronization as a mechanism for "noisy" bottom-up i

54 eme deals explicitly with the problem of TEC desynchronization as transcript synthesis proceeds, and

55 ssociation between attention-modulated alpha desynchronization, associated with the enhancement of se

56 es at similar rates gives way to substantial desynchronization at larger firing rate differences.

57 I and Type II neurons, we observe clustered desynchronization at many pulsing frequencies.

58 rrhythmia at the single-cell level and phase desynchronization at the network level--can account for

59 and cortical desynchronization, with maximum desynchronization at ~130 Hz.

60 This triggers network desynchronization because heterogeneous coupling to surr

61 Although premovement beta-band event-related desynchronization (beta-ERD; 13-30 Hz) from sensorimotor

62 nt enrichment combined with warming dampened desynchronization between control (ambient) and treatmen

63 findings illustrated significant and unique desynchronization between EZ and the rest of the brain i

64 Desynchronization between physiological and behavioral r

65 ind that transcranially inducing oscillatory desynchronization between the frontopolar and -parietal

66 (dm) SCN, and a jetlag paradigm that induces desynchronization between these SCN subregions, we show

67 ing revealed that posterior cingulate cortex desynchronization can be explained by increased excitabi

68 Depending on the ictal frequency pattern, desynchronization can occur later, but a late and termin

69 ic spike failure contributes to the cortical desynchronization caused by DBS.

70 psilateral synchronization and contralateral desynchronization coding for high-confidence responses.

71 pected parieto-occipital low-alpha (8-10 Hz) desynchronization contralateral to the cued location.

72 ignment that results from circadian internal desynchronization could preserve the ability of light to

73 ork synchronization or trigger rapid network desynchronization depending on the synaptic input.

74 nal peptide-expressing neurons did not block desynchronization, despite these neurons being activated

75 c orthosis and turned sensorimotor beta-band desynchronization during motor imagery (MI) of finger ex

76 ovement in the GPi, which also had more beta desynchronization during movement.

77 We anticipated increased beta desynchronization during the transfer phase when cues pr

78 ent-related analysis showed time-locked beta desynchronization during WAKE movements.

79 -shaped relationship: States of intermediate desynchronization elicited minimal response bias and fas

80 cal responses: powerful cortical and VTA EEG desynchronization, EMG activation, a large brain tempera

81 One hypothesis holds that desynchronization enhances stimulus coding in the releva

82 Spectral power and event related desynchronization (ERD) analyses at the group level show

83 l conditions, significant beta event-related desynchronization (ERD) and gamma event-related synchron

84 processes were examined using event-related desynchronization (ERD) at lower (8-10 Hz) and upper (10

85 D showed attenuated alpha band event-related desynchronization (ERD) during encoding.

86 Event-related desynchronization (ERD) in alpha (8-12Hz) and low beta b

87 lp EEG studies have shown that event-related desynchronization (ERD) in the alpha (8-13 Hz) and beta

88 h much of it, in parallel with event-related desynchronization (ERD) in the alpha band.

89 MI led to event-related desynchronization (ERD) of oscillatory beta activity in

90 peed, while a decrease in beta event related desynchronization (ERD) predicted quicker movement initi

91 P had a weaker beta (18-24 Hz) event-related desynchronization (ERD), post-movement beta rebound (PMB

92 f brain oscillations is termed event-related desynchronization (ERD).

93 s: a decrease during movement (event-related desynchronization, ERD), followed by an increase (event-

94 earning) in the alpha and beta event-related desynchronizations (ERDs) associated with practising an

95 We extracted Event-Related Desynchronizations (ERDs) in the theta, alpha, and beta

96 analyzed alpha and beta bands Event-Related Desynchronizations (ERDs).

97 DN corresponded to a period of event-related desynchronization extending across a wide low-frequency

98 thy human subjects is a beta (13-30 Hz) band desynchronization followed by a postmovement event-relat

99 analysis showed that the amplitude of alpha-desynchronization followed the time course of temporal e

100 A rapid (~ 40 ms) EEG desynchronization following the LC stimulation suggested

101 e, synchronization along short distances and desynchronization for long distances, and the decrease o

102 The extent of the decreased event-related desynchronization for median nerve-innervated digits was

103 Furthermore, beta-band desynchronizations for antisaccades started earlier, wer

104 In contrast, the beta-band event-related desynchronization from the motor cortex was preserved.

105 tasks of efficient coherent synchronization/desynchronization guided by the collective influence of

106 is crucial for the fitness of organisms, and desynchronization has been linked to numerous physical a

107 chronized spontaneous activity, but cortical desynchronization has not generally been associated with

108 orienting was correlated with alpha/low-beta desynchronization (i.e., power suppression).

109 ng paradigm, we show that picrotoxin-induced desynchronization impairs the discrimination of molecula

110 Ventricular desynchronization imposed by ventricular pacing even whe

111 lations and triggered 15-30 Hz event-related desynchronization in ACs.

112 different scalp sites, ii) the event-related desynchronization in alpha and synchronization in theta,

113 of consciousness in the form of a sustained desynchronization in alpha/beta frequency and (ii) a rea

114 l covariance analysis suggests network-level desynchronization in brain volume in both male and femal

115 s evidenced by reduced RIF and stronger beta desynchronization in fronto-parietal brain regions durin

116 In each patient, there was alpha/beta band desynchronization in M1 for stop trials.

117 However, the degree of desynchronization in M1 was less for successfully than u

118 comes, and further examine the role of alpha desynchronization in mediating altered states of conscio

119 Thus, nocturia might occur as a result of desynchronization in one or more of these circadian regu

120 The effect of pacing-induced ventricular desynchronization in patients with normal baseline QRSd

121 nia have impaired movement-related beta band desynchronization in primary motor and sensory cortices.

122 activity, and cause surround inhibition and desynchronization in response to excitatory input.

123 , at the single-trial level in humans, local desynchronization in sensory cortex (expressed as time-s

124 Desynchronization in steady light lowers the sensitivity

125 that seizure-like events are associated with desynchronization in such networks is consistent with re

126 and sentence comprehension tasks revealed a desynchronization in the 10-18Hz range, localized to the

127 EEG sensorimotor power spectra (ie, stronger desynchronization in the alpha and beta bands) occurred

128 arked expressions are associated with larger desynchronization in the alpha band than expressions wit

129 We demonstrated that beta-band event-related desynchronization in the auditory cortex differentiates

130 e model, we uncovered perirhinal-hippocampal desynchronization in the MTL regions that is associated

131 otor affordance signatures: an event-related desynchronization in the mu frequency and an increased P

132 ST-ARM group had impaired beta and low gamma desynchronization in the primary motor cortex.

133 Beta-band desynchronization in the STN may reflect the additional

134 Previous studies have shown that beta-band desynchronization in the subthalamic nucleus (STN) is re

135 -13 Hz) and beta band (13-35 Hz) local field desynchronizations in sensorimotor and parietal cortex,

136 to alpha (8-13 Hz) and beta band (13-30 Hz) desynchronizations in the sensorimotor and posterior par

137 Alpha desynchronization increased rhythmically, peaking just b

138 This high level of desynchronization indicates that ALAN disrupted the circ

139 with vIPS, reduced the high-alpha (10-12 Hz) desynchronization induced by shifting attention into bot

140 or the first time that robust alpha and beta desynchronization is a shared feature of sensorimotor co

141 , it remains elusive whether prefrontal beta desynchronization is causally relevant for memory format

142 presenting the sensorimotor mu rhythm, whose desynchronization is indicative for the degree of engage

143 Network desynchronization is perhaps the most dramatic and abrup

144 Detailed network models predict that desynchronization is robust, local, and dependent on syn

145 Fluctuations in arousal and neural desynchronization likely pose perceptually relevant stat

146 The adverse effects of ventricular desynchronization may explain the association of RVA pac

147 ght ventricular stimulation, and ventricular desynchronization may result.

148 ramework that integrates synchronization and desynchronization mechanisms to explain how the two syst

149 z) and high (21-30 Hz) beta movement-related desynchronization (MRD) effectively distinguished betwee

150 Specifically, desynchronization observed in a population of periodical

151 n Parkinson's disease and the greatest alpha desynchronization occurring in essential tremor.

152 ssential tremor, with the greatest high-beta desynchronization occurring in Parkinson's disease and t

153 This neuro-cardiac desynchronization occurs due to the abnormal phase reset

154 The underlying cause is an Abeta-induced desynchronization of action potential generation in pyra

155 visual WM task revealed that the prestimulus desynchronization of alpha oscillations predicts the acc

156 ention is associated with spatially specific desynchronization of alpha-band activity over visual cor

157 sk were presented into states of high or low desynchronization of auditory cortex via a real-time clo

158 he circadian system also occur when there is desynchronization of clock phase with that of the outsid

159 We found that movement-related desynchronization of cortical activity in the upper alph

160 t high-gamma oscillations, consistent with a desynchronization of cortical populations.

161 Desynchronization of electrical stimuli have shown benef

162 ontaneous release and suppression as well as desynchronization of evoked release, recapitulating the

163 Syt2 to achieve a significant reduction and desynchronization of fast release.

164 cholinergic-like neocortical activation and desynchronization of functional networks in the mammalia

165 an raphe serotonergic neurons results in the desynchronization of hippocampal electroencephalographic

166 d in 17.8 +/- 1.4 days of training rewarding desynchronization of ipsilesional oscillatory sensorimot

167 processing and brain states, causing robust desynchronization of local field potentials and strong d

168 Specifically, desynchronization of local neuronal assemblies in the le

169 density decline, and c) synchronization and desynchronization of local population dynamics.

170 d states in the STDP-only model (i) with the desynchronization of models (iii) and (iv).

171 both an attenuation of spike frequency and a desynchronization of neighbors.

172 We argue that desynchronization of network activity is a fundamental s

173 development and progression of AD including desynchronization of neuronal action potentials, consequ

174 ective effects of psychedelics result from a desynchronization of ongoing oscillatory rhythms in the

175 ovements are associated with a dampening and desynchronization of oscillatory activity in STN neurons

176 to determine how spike failure affected the desynchronization of pathophysiological oscillatory acti

177 ative differences in the synchronization and desynchronization of responding neuronal populations.

178 This led to a desynchronization of rhythmic immune parameters, which m

179 elated behavior following UCMS and suggest a desynchronization of rhythms in the brain with an indepe

180 ms, such as VIP-VPAC2 signaling, can lead to desynchronization of SCN neuronal clocks and loss of beh

181 ask, hand-muscle activity and the associated desynchronization of sensorimotor oscillations were stro

182 that disruption of heminode positions causes desynchronization of SGN spikes leading to a loss of tem

183 noic acid (RA) in chicken embryos leads to a desynchronization of somite formation between the two em

184 ring of ON DS cell neighbors, resulting in a desynchronization of spike activity.

185 Therefore, developmental desynchronization of spontaneous neuronal activity is a

186 The selective desynchronization of stimulus-evoked oscillating neural

187 We compare the stimulation-induced desynchronization of synchronized states in the STDP-onl

188 Furthermore, we show that SOM activation and desynchronization of the BLA is PL-dependent and promote

189 Conversely, clinical relapse may reflect a desynchronization of the clock, indicative of a reactiva

190 A direct role for the MR in the desynchronization of the electroencephalographic activit

191 lysaccharide (LPS) were due to intercellular desynchronization of the molecular clock, a cell-intrins

192 upling of the two modules, in turn, causes a desynchronization of the populations that outlasts the c

193 fe is probably due, in part, to cold-induced desynchronization of the ripening program involving ethy

194 he pattern of melatonin release under forced desynchronization of these SCN subregions.

195 ings of the SCN from these mice showed rapid desynchronization of unit oscillators.

196 ors provide increasing evidence showing that desynchronization of ventricular electrical activation a

197 phase walk ascribable to weakened coupling (desynchronization) of two oscillators, AP-SND phase walk

198 This reduced desynchronization on successful stop trials could relate

199 response to VIP and that the transient phase desynchronization, or "phase tumbling", could arise from

200 Stronger alpha event-related desynchronization over occipital and frontocentral sites

201 ement initiation is accompanied by beta-band desynchronization over sensorimotor areas, whereas movem

202 ic cough group, the typical movement-related desynchronization over somatosensory-motor cortex during

203 opographic differentiation revealed stronger desynchronization over the (ipsilateral) right-hemispher

204 al brain damage.SIGNIFICANCE STATEMENT Alpha desynchronization over the hemisphere contralateral to t

205 e, MI, EF) and pacing promoters (ventricular desynchronization-paced QRSd and Cum%VP, and AV desynchr

206 ynchronization-paced QRSd and Cum%VP, and AV desynchronization-pacing mode).

207 g of location persists into brief periods of desynchronization prevalent in slow-wave sleep.

208 es and modeling studies indicated that this "desynchronization" process was dependent on presynaptic

209 eriments and simulations, we investigate the desynchronization properties in cervical cancer cells (H

210 hat transient periods of synchronization and desynchronization provide a mechanism for dynamically in

211 pulation of cells reveal that the cell cycle desynchronization rate is primarily sensitive to the var

212 Our results show that the desynchronization rate of artificially synchronized in-p

213 We posit that active desynchronization reduces summation of synaptic potentia

214 cally, we argue that the synchronization and desynchronization reflect a division of labor between a

215 Another hypothesis holds that desynchronization reflects global arousal, such as task

216 The rate and factors that control cellular desynchronization remain largely unknown.

217 nerve, can also cause cortical and thalamic desynchronization, resulting in a reduction of seizure a

218 Awareness of the problem of desynchronization should also lead to more regular monit

219 We also provide evidence on why a desynchronization-specific potentiation or depression of

220 er simulations and is explained by overdrive desynchronization: spikes desychronize when axons are st

221 was correlated with stronger EEG alpha-beta desynchronization, suggesting a common dependence on NE

222 Preservation of event-related desynchronization suggests that the cells of origin diff

223 20, and 20-30 Hz) by using the event-related desynchronization technique.

224 avioral one, with switch trials showing more desynchronization than non-switch trials across language

225 n ipsilesional premotor cortex event-related desynchronization that correlated with improvements in m

226 is stimulation produced electroencephalogram desynchronization that was blocked by systemic and corti

227 rast, auditory-induced alpha-beta (10-30 Hz) desynchronization (that is, decreased power), prevalent

228 d onset of phase synchronization and delayed desynchronization to the click train.

229 Spontaneous desynchronization under constant conditions was limited,

230 oscillations in the a band (8-14 Hz) undergo desynchronization under the control of prefrontal cortex

231 ulus combinations, the extent of oscillatory desynchronization varies with stimulus disparity.

232 No event-related desynchronization was detected; rather, there was a tran

233 l (ambient) and treatment mesocosms, whereas desynchronization was enhanced when simultaneously subje

234 Desynchronization was evident at multiple time scales, w

235 m imaging, we found that performance-related desynchronization was global and correlated better with

236 auditory tasks and found that in both cases desynchronization was global, including regions such as

237 tion of correlation accompanying brain state desynchronization was largely explained by a decrease in

238 esponses to visual stimuli, while alpha/beta desynchronization was preserved.

239 Phase errors induced by sweep-trigger desynchronization were effectively reduced by spectral p

240 Saccade-related beta-band desynchronizations were observed just before and during

241 and the duration of epilepsy intensify this desynchronization, which can be the outcome of abnormal

242 band power changes and broadband (4-150 Hz) desynchronization, which predicted significant reduction

243 rily exhibited a delayed and sustained alpha desynchronization, while ventrolateral extrastriatal are

244 f the disorder, with abnormal volatility and desynchronization with neocortical language nodes.

245 ationship between DBS frequency and cortical desynchronization, with maximum desynchronization at ~13

246 F was associated with a more pronounced beta desynchronization within the left dorsolateral prefronta

247 This led to a stronger beta desynchronization within the parietal cortex in a later