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1 he hypogastric (presumably inhibitory to the detrusor muscle).
2 matory markers were increased in CYP-treated detrusor muscles.
3 el mechanism involving interstitial cells in detrusor muscles.
4 ient contraction and prolonged relaxation of detrusor muscles.
5 -labeling, was up-regulated in fetal bladder detrusor muscle and lamina propria cells and that this w
6                   Three types existed in the detrusor muscle and one major type in the sub-urothelium
7  an explanation for purinergic relaxation in detrusor muscles and show that there are no discrete inh
8 eported a new class of interstitial cells in detrusor muscles and showed that these cells could be id
9 e pelvic nerve (presumably excitatory to the detrusor muscle); and a pathway involving the pelvic ner
10 ereas PAR-1 and PAR-2 are predominant in the detrusor muscle, and PAR-4 is expressed in peripheral ne
11 re was reduced birefringent collagen between detrusor muscle bundles, and atomic force microscopy sho
12  of inflammatory and pro-fibrotic markers in detrusor muscle cells.
13 d PDGFRalpha and SK3 protein were reduced in detrusor muscle extracts.
14 ammation, increased mast cell numbers in the detrusor muscle have been reported in a subset of IC pat
15 eflexively increases activity of the bladder detrusor muscle in anesthetized rats.
16             Treatment for overactive bladder detrusor muscle, including anticholinergics (eg, trospiu
17         The strong expression of PARs in the detrusor muscle indicates the need for studies on the ro
18 rative histopathological findings to confirm detrusor muscle infiltration.
19 ccount of this evidence, we propose that the detrusor muscle is arranged into modules, which are circ
20             Our results demonstrate that the detrusor muscle is hyperactive, voiding volume is decrea
21 early-stage cancer without invasion into the detrusor muscle layer.
22 ooth muscle cells (SMCs) in vitro and in the detrusor muscle of a mechanically overloaded bladder in
23 c component of neurotransmission in isolated detrusor muscle of guinea pig urinary bladder.
24 s assumption by studying E-C coupling in the detrusor muscle of wild type and Homer1(-/-) mice and by
25 mooth muscle cells (SMCs) were isolated from detrusor muscles of PDGFRalpha(+)/eGFP and smMHC/Cre/eGF
26 al recording from smooth muscle cells of the detrusor muscle revealed spontaneous depolarizations, di
27 omplished by suppressing contractions of the detrusor muscle that lines the bladder wall.
28 ls subjacent to the epithelium and a thinner detrusor muscle that was not attributable to disruption
29 as associated with higher sensitivity of the detrusor muscle to muscarinic stimulation and membrane d
30                   S1P-induced contraction of detrusor muscle was dependent on stretch and intracellul
31           Most nerve endings were located in detrusor muscle where the three types could be identifie
32 loss of coordinated neural control among the detrusor muscle, which increases bladder pressure to fac