ライフサイエンスコーパス: diastolic pressure

1 = 0.037 for systolic pressure; P = 0.019 for diastolic pressure).

2 ant change in heart rate or left ventricular diastolic pressure.

3 lobal longitudinal strain (LVGLS) and LV end-diastolic pressure.

4 ge occurrence of a sympathetic burst against diastolic pressure.

5 ction, and an increased left ventricular end-diastolic pressure.

6 peak pressure and a dramatic increase in end diastolic pressure.

7 ystolic pressure conferred greater risk than diastolic pressure.

8 ure and -0.2 (95% CI: -4.1, 3.7) mmHg/kg for diastolic pressure.

9 more rapid increase in left ventricular end-diastolic pressure.

10 Relationships were smaller for diastolic pressure.

11 dge pressure (PCWP) and left ventricular end-diastolic pressure.

12 s include LA dP/dt, LA relaxation and LV end-diastolic pressure.

13 kg/m2 for body mass index, and 0.48 mmHg for diastolic pressure.

14 the cuff technique in predicting the aortic diastolic pressure.

15 e systolic pressure without greatly reducing diastolic pressure.

16 intracellular calcium, but did not decrease diastolic pressure.

17 left ventricular developed pressure and end diastolic pressure.

18 fibrosis, and decreased left ventricular end-diastolic pressure.

19 reduced rate pressure product and increased diastolic pressure.

20 nd maximum LV dP/dt, as well as lower LV end-diastolic pressure.

21 mean arterial pressure than for systolic or diastolic pressure.

22 tion at rest and on exercise, and higher end-diastolic pressures.

23 r estimates of pulmonary artery systolic and diastolic pressures.

24 of pulse pressure above that of systolic and diastolic pressures.

25 ic LV diameter was smaller at matched LV end-diastolic pressures.

26 sure, -0.7 mm Hg (CI, -0.6 to -0.9 mm Hg) in diastolic pressure, -0.17 mmol/L (CI, -0.09 to -0.25 mmo

27 . 28+/-10 mmHg, P<0.001), and lowered LV end diastolic pressure (10+/-1 vs. 86+/-13 mmHg, P<0.001) fo

28 y artery pressure (25+/-10 mm Hg) and LV end-diastolic pressure (11+/-5 mm Hg; P<0.001 for both compa

29 RV dysfunction was indicated by elevated end-diastolic pressure (11.3+/-2.5 versus 5.7+/-2.0 mm Hg; P

30 ss (70 +/- 2 vs. 63 +/- 1%), enhanced LV end-diastolic pressure (14 +/- 2 vs. 8 +/- 1 mmHg) and these

31 -80 versus 165+/-71 mL, P<0.0001) and LV end-diastolic pressure (14.3+/-10.2 versus 9.9+/-9.3 mm Hg,

32 8+/-5 mm Hg, P=0.04), lower pulmonary artery diastolic pressures (14+/-9 versus 21+/-9 mm Hg, P=0.01)

33 an pressure, 39+/-12 mm Hg), elevated LV end-diastolic pressure (19+/-5 mm Hg), and reduced stroke vo

34 P-induced cardiac function (left ventricular diastolic pressure, 20 +/- 5.9%; +/- dP/dt, 5.2 +/- 4.5%

35 ents revealed similar reductions in systolic/diastolic pressure (-22/12 versus -21/13 mm Hg) and LV m

36 diastolic function (eg, left ventricular end-diastolic pressure 23+/-9 in WT and 51+/-5 mm Hg in GAMT

37 ities, including higher left ventricular end-diastolic pressures (24.3+/-4.6 versus 12.9+/-5.5 mm Hg)

38 n reduced cardiac function (left ventricular diastolic pressure, 39 +/- 3.8%; +/- dP/dt, 32 +/- 4.4%,

39 artery conduit group having a higher aortic diastolic pressure (55 versus 42 mm Hg), a narrowed syst

40 NTg (53 versus 38%, P<0.01), whereas LV end-diastolic pressure (6 versus 12 mm Hg, P<0.05) and lung

41 traction 24%, and mean right ventricular end-diastolic pressure 9 mm Hg.

42 patients with elevated left ventricular end-diastolic pressure, a finding largely attributed to the

43 End diastolic pressure, a negative index of myocardial perfo

44 he combination of a high systolic and normal diastolic pressure-a widened pulse pressure-seems to be

45 gridAND correlated with left ventricular end-diastolic pressure across both groups (average R(2) = 0.

46 Systolic and diastolic pressures among control and VVS were the same,

47 s >35% had an increased left ventricular end-diastolic pressure and a marked increase in heart weight

48 y artery pressure, high left ventricular end-diastolic pressure and a normal ejection fraction, some

49 An inverse relationship between diastolic pressure and adverse cardiac ischemic events (

50 End-diastolic pressure and chamber diastolic stiffness did n

51 Indeed, a J-shaped relationship between diastolic pressure and coronary events was documented in

52 Partial to complete recovery of diastolic pressure and developed pressure was seen irres

53 fractional shortening, and increased LV end-diastolic pressure and fibrosis (P<0.05 versus control w

54 An interaction between decreased diastolic pressure and history of revascularization was

55 cardiospheres decreased left ventricular end-diastolic pressure and increased cardiac output.

56 olic function, lowering left ventricular end-diastolic pressure and increasing the filling rate.

57 evidenced by increased left ventricular end-diastolic pressure and left ventricular volume indexes.

58 between the plasma DPPIV activity and LV end-diastolic pressure and lung congestion.

59 ed HF, CXL-1020 reduced left ventricular end-diastolic pressure and myocardial oxygen consumption whi

60 the J-shaped relationship persisted between diastolic pressure and primary outcome.

61 <.01 for difference between pulmonary artery diastolic pressure and pulmonary artery occlusion pressu

62 son, the regression between pulmonary artery diastolic pressure and pulmonary artery occlusion pressu

63 Improvement in postischemic recovery of end-diastolic pressure and reduction in infarct size was obs

64 beat-to-beat changes in left ventricular end-diastolic pressure and SV was used as an index of the dy

65 output and decreases in left ventricular end-diastolic pressure and systemic vascular resistance.

66 offset the pacing-induced increase in LV end-diastolic pressure and the time constant of isovolumic r

67 dysfunction became manifest as increased end-diastolic pressure and time to 90% relaxation.

68 of early pregnancy in humans despite reduced diastolic pressure and total peripheral resistance.

69 oconstriction from ET-1 led to higher aortic diastolic pressure and very narrow pulse pressure after

70 ding heart rate, peak-systolic pressure, end-diastolic pressure and volume, end-systolic pressure and

71 myocardium, as demonstrated by elevated end-diastolic pressures and decreased percent recovery of de

72 Similarly, left ventricular diastolic pressures and diastolic stiffness were elevate

73 ts, the pathophysiological cause of elevated diastolic pressures and heart failure is abnormal diasto

74 n Fontan, manifested by high ventricular end-diastolic pressures and pulmonary arterial wedge pressur

75 n, which subsequently led to elevated LV end-diastolic pressures and pulmonary hypertension.

76 remained in failure with similarly elevated diastolic pressures and reduced dP/dtmax.

77 , its association with increasing left heart diastolic pressures and systemic vascular stiffening, an

78 tolic pressure, 1.27 (95% CI, 1.01-1.60) for diastolic pressure, and 1.30 (95% CI, 1.05-1.61) for mea

79 e increase at 40 mm Hg, left ventricular end-diastolic pressure, and cardiac index, was significantly

80 de, left ventricular developed pressure, end-diastolic pressure, and coronary flow were significantly

81 aturation, elevated systemic ventricular end-diastolic pressure, and elevated main pulmonary artery p

82 lated with PH severity, left ventricular end-diastolic pressure, and left ventricular dilatation.

83 , mean arterial pressure, systolic pressure, diastolic pressure, and left ventricular systolic pressu

84 inephrine levels, lower left ventricular end-diastolic pressure, and lower right ventricle/body weigh

85 Left ventricular volumes, diastolic pressures, and diastolic stiffness were higher

86 vels, baseline and in-treatment systolic and diastolic pressures, and for diuretic therapy, losartan-

87 ed LV mass, posterior wall thickness and end diastolic pressures, and increased fractional shortening

88 d albuminuria, similar baseline systolic and diastolic pressures, and reductions in diastolic pressur

89 Elevation in right ventricular end-diastolic pressure appeared to predict poor vasodilatory

90 At similar left ventricular volumes, their diastolic pressures are more than twice as high as in co

91 Additionally, the end-diastolic pressure-area curves shifted to the left in al

92 Compliance was assessed by end-diastolic pressure-area curves.

93 l hearts had similar cardiac outputs and end-diastolic pressures as WT or transgenic hearts.

94 sure from randomization showed a benefit for diastolic pressure at month 12 (P = 0.039) but not at mo

95 the 63 patients; thus, 92% had elevated end-diastolic pressure (average, 24+/-8 mm Hg).

96 a function of elevated left ventricular end diastolic pressure but was associated with increased per

97 rd, decreasing cardiac output at matched end-diastolic pressure by 44%.

98 e (75 to 100 mmol/d) will, on average, lower diastolic pressure by approximately 1 mmHg and systolic

99 n resistance (homeostasis model assessment), diastolic pressure, C-reactive protein, and the number o

100 ng diastole; hence, an excessive decrease in diastolic pressure can significantly hamper perfusion.

101 trolled delivery group (left ventricular end-diastolic pressure, cardiac index, +dP/dt, -dP/dt, and t

102 Left ventricular diastolic pressure, cardiac index, dP/dt40 and negative

103 nt parameters, including RV systolic and end-diastolic pressures, cardiac output, RV size, and morbid

104 was proportional to the magnitude of the end-diastolic pressure change.

105 of relaxation and lower left ventricular end diastolic pressure compared with controls.

106 volume in IPAH (+7%; P < 0.05), whereas end-diastolic pressure continuously dropped.

107 End-diastolic pressure correlated significantly with tissue

108 Baseline noninvasive LV end-diastolic pressure correlated with invasive pulmonary ar

109 Although left ventricular end-diastolic pressure decreased in 45/10, it increased in 4

110 r time constant of isovolumic relaxation and diastolic pressure (Delta +29+/-9% and +38+/-12%) and a

111 CPA produced improvement in postischemic end-diastolic pressure, developed pressure, and rate-pressur

112 In contrast, changes in end-diastolic pressure did induce vasodilatation that, altho

113 ed (n=49) subgroups according to whether the diastolic pressure difference (diastolic pulmonary arter

114 The diastolic pressure difference (DPD) is recommended to di

115 trol for 15 variables plus proline, systolic/diastolic pressure differences were -2.7/-2.0 mm Hg (z s

116 emic O(2) delivery, and (4) elevated cardiac diastolic pressures do not represent systolic dysfunctio

117 ive ethnic differences in either systolic or diastolic pressure during childhood and adolescence.

118 e of the linear correlation between MSNA and diastolic pressure during spontaneous breathing.

119 velop increases in left ventricular (LV) end-diastolic pressures during exercise that contribute to d

120 astolic volume (EDV) and Doppler-derived end-diastolic pressure (EDP) were used to derive the diastol

121 t ventricular developed pressure (LVDP), end diastolic pressure (EDP), and ATP were measured througho

122 q, even with marked reduction of volume (end-diastolic pressure [EDP], 1 to 2 mm Hg), whereas in cont

123 -derived baseline estimated pulmonary artery diastolic pressure (ePAD) and change from baseline ePAD

124 +/-10 mm, estimated resting pulmonary artery diastolic pressure (ePAD) of 16+/-9 mm Hg, and diastolic

125 Median estimated pulmonary artery diastolic pressures (ePAD) were calculated every 24 hour

126 The pulmonary artery diastolic pressure estimate of pulmonary artery occlusio

127 ion coefficient between the pulmonary artery diastolic pressure estimate of pulmonary artery occlusio

128 contrast, the bias for the pulmonary artery diastolic pressure estimate significantly increased with

129 be at increased risk for coronary events if diastolic pressure falls below critical levels.

130 stolic pressure declined in both groups, but diastolic pressure fell less with ALT-711 (P=0.056).

131 dP/dt max, -dP/dt min, and left ventricular diastolic pressure followed injection of MSCs before ind

132 index, LV dP/dt40, LV negative dP/dt, and LV diastolic pressure followed injection of MSCs, regardles

133 d protocol based on the left ventricular end-diastolic pressure for the prevention of contrast-induce

134 unloading manifested by a decrease in LV end-diastolic pressure from 11.4 +/- 9.0 mm Hg to 8.8 +/- 5.

135 to 1.35; p = 0.15) when defined according to diastolic pressure gradient.

136 ng systolic blood pressure > 140 mmHg and/or diastolic pressure > 90 mmHg; incident hypertension used

137 tension (systolic pressure > or =140 mmHg or diastolic pressure > or = 90 mmHg) across quintiles of B

138 a systolic blood pressure > or =140 mm Hg or diastolic pressure > or =90 mm Hg at any FHS cycle exami

139 IV) more than doubled chamber stiffness (end-diastolic pressure >25 mm Hg, P<0.001), whereas stiffnes

140 ure into postcapillary (left ventricular end-diastolic pressure, >15 mm Hg; n=269) and precapillary g

141 Left ventricular end-diastolic pressure-guided fluid administration seems to

142 ntly in patients in the left ventricular end-diastolic pressure-guided group (6.7% [12/178]) than in

143 cated in a 1:1 ratio to left ventricular end-diastolic pressure-guided volume expansion (n=196) or th

144 Interventions were performed after LV end-diastolic pressure had increased approximately 7 mm Hg.

145 pressure, without associated changes in end-diastolic pressure, had no significant effect on vascula

146 re (PP), the difference between systolic and diastolic pressure, has been associated with cardiovascu

147 demonstrated significant increases in LV end-diastolic pressure, heart and body weight, and LV chambe

148 ut (57%) and significant decreases in LV end-diastolic pressure, heart rate, and systemic vascular re

149 n, in-treatment use of digoxin, systolic and diastolic pressure, heart rate, QRS duration, Cornell vo

150 Right ventricular systolic and diastolic pressures, heart rate, and pressure derivative

151 The isovolumic end-diastolic pressure, however, remained elevated throughou

152 =0.02) and more likely to have higher RV end-diastolic pressure (HR, 1.07; 95% CI, 1.00-1.15; P=0.057

153 n of I79N hearts significantly worsened (end-diastolic pressure: I79N 20 +/- 4 mmHg versus CON 13 +/-

154 ic measures of diastolic function and LV end-diastolic pressure improve in most patients.

155 Left ventricular (LV) end-diastolic pressure in AAV-VEGF-B and AAV-control was, re

156 eased tau (P<0.001) and left ventricular end-diastolic pressure in both old and young hearts.

157 all subjects with a significant reduction in diastolic pressure in the HYL group (P = 0.008) but not

158 Diastolic pressure increased by 6+/-2 mm Hg (P:<0.05), a

159 Although central diastolic pressure increased significantly with pacing,

160 Diastolic pressures increased dramatically in response t

161 further dichotomized by left ventricular end-diastolic pressure into postcapillary (left ventricular

162 N(+)HFpEF had increased left ventricular end-diastolic pressure, left atrial volume, N-terminal prope

163 pertension (systolic pressure 160-200 mm Hg; diastolic pressure <110 mm Hg) who were aged 80 years or

164 nd precapillary groups (left ventricular end-diastolic pressure, </=15 mm Hg; n=56).

165 fraction) and hemodynamic variables (LV end-diastolic pressure, LV dP/dtmax, preload adjusted maxima

166 of a noninvasive measure of left ventricular diastolic pressure (LVDP) early after acute myocardial i

167 paradoxically decreased left ventricular end-diastolic pressure (LVEDP) and left ventricular end-dias

168 t led to an increase of left ventricular end-diastolic pressure (LVEDP) by > or =20 mm Hg (ie, cardia

169 astolic volumes with little effect on LV end-diastolic pressure (LVEDP) or the end-diastolic P-V rela

170 lood pressure (DBP) and left ventricular end-diastolic pressure (LVEDP) to systolic blood pressure (S

171 ary enalaprilat reduced left ventricular end-diastolic pressure (LVEDP), but not left ventricular end

172 asurements, including cardiac output, LV end-diastolic pressure (LVEDP), rate of pressure rise at LV

173 onal marker of elevated left ventricular end-diastolic pressure (LVEDP), which adds prognostic value

174 In healthy and CHF (left ventricular end diastolic pressure (LVEDP): 6 +/- 1 versus 14 +/- 1 mmHg

175 creased DCS (isovolumic left ventricular end-diastolic pressure [LVEDP] increased 10 mm Hg, P<0.001,

176 omanometer-tipped catheters, and the mean LV diastolic pressure (M-LVDP) was used as a surrogate for

177 es, SMV activation increased the mean aortic diastolic pressure (MADP) by 26.5+/-3.5%, the mean diast

178 ew noninvasive methods to predict the aortic diastolic pressure may help in the future.

179 Ventricular end-diastolic pressure, mean PA pressure, and ventricular fu

180 ore defibrillation: 1) right atrial systolic/diastolic pressures (mm Hg) were lower (85 +/- 19, 4 +/-

181 stolic and diastolic function, higher LV end-diastolic pressure, more cardiomyocyte hypertrophy, and

182 )HFpEF had no change in left ventricular end-diastolic pressure, myocardial passive stiffness, collag

183 rformance with elevated left ventricular end-diastolic pressure, not seen in the wild-type.

184 early rapid filling and equalization of end-diastolic pressures obtained by cardiac catheterization

185 n, cardiac systolic pressure generation at a diastolic pressure of 15 mm Hg averaged 110 +/- 11, 66 +

186 end-diastolic volume at an idealized LV end-diastolic pressure of 20 mm Hg (EDV20), and RV remodelin

187 sure (systolic pressure of 130 to 139 mm Hg, diastolic pressure of 85 to 89 mm Hg, or both).

188 systolic pressure of 139 mm Hg or lower and diastolic pressure of 85 to 89 mm Hg, were randomly assi

189 of systolic pressure of 130 to 139 mm Hg and diastolic pressure of 89 mm Hg or lower, or systolic pre

190 vation in the systolic blood pressure with a diastolic pressure of less than 90 mm Hg.

191 pedance threshold valve results in sustained diastolic pressures of >55 mm Hg in patients in cardiac

192 r systolic pressures of 110 to 130 mm Hg and diastolic pressures of 70 to 90 mm Hg, which suggests th

193 r systolic pressures of 110 to 130 mm Hg and diastolic pressures of 70 to 90 mm Hg.

194 This adverse effect of too low a diastolic pressure on coronary heart disease leaves the

195 es, the end-diastolic volume at a common end-diastolic pressure on the sequential end-diastolic press

196 , increasing cardiac work while reducing the diastolic pressure, on which coronary flow is dependent.

197 th respect to time but did not change LV end-diastolic pressure or improve LV regional function.

198 function with elevated left ventricular (LV) diastolic pressure or may be merely a manifestation of a

199 tolic pressure without an increase in LV end-diastolic pressure, or decrease in LV dP/dt or LV wall t

200 temperature (P = .31), heart rate (P = .92), diastolic pressure (P = .31), or systolic pressure (P =

201 was seen for stiffness with ventricular end-diastolic pressure (P = 0.001) and pulmonary artery wedg

202 gement (p < 0.0001) and marginally increased diastolic pressure (p = 0.09).

203 d ejection fraction and left ventricular end-diastolic pressure (P<0.05).

204 ular pressure by micromanometer provided end-diastolic pressure (P) area (A) relations during initiat

205 e in the change in office based systolic and diastolic pressures (p = 0.18; p = 0.14); 24 hr.

206 systolic and diastolic pressures (p = 0.20; p = 0.18); and eGFR (p =

207 f renal-cell cancer (P for trend, <0.001 for diastolic pressure; P for trend, 0.007 for systolic pres

208 n recording of beat-by-beat pulmonary artery diastolic pressure (PAD), stroke volume index (SV index)

209 Her systolic/diastolic pressure peaked at 23 min to reach 123/70 mmHg

210 Her systolic/diastolic pressure peaked at 7 min to reach 144/78 mmHg

211 th SHF and those with DHF; however, elevated diastolic pressures play a pivotal role in the underlyin

212 s in blood pressure and left ventricular end-diastolic pressure produced by GTN in vivo.

213 0.01) but an inverse correlation with LV end-diastolic pressure (r = -0.53, p = 0.01).

214 the time required for reaching the lowest LV diastolic pressure (r = 0.70, p = 0.04).

215 versely associated with left ventricular end-diastolic pressure (r=-0.728; P<0.001), resulting in a b

216 as SR(E) was significantly related to LV end-diastolic pressure (r=0.52, P=0.005) in the experimental

217 tolic velocity (E/E ) correlated with LV end-diastolic pressure (r=0.52, P=0.007).

218 The delay related well with LV end-diastolic pressure (r=0.76) and volume (r=-0.73), and VO

219 olow-Lyon voltage, in-treatment systolic and diastolic pressure, randomized treatment, and standard c

220 sympathetic response divided by the maximum diastolic pressure reduction during straining, was the s

221 and sympathetic baroreflex sensitivity (MSNA-diastolic pressure relation) were measured.

222 p but has no effect on the stroke volume/end-diastolic pressure relationship.

223 ft ventricular (LV) function measured by end-diastolic pressure response to preload augmentation, con

224 effect on heart rate, LV relaxation, LV end-diastolic pressure, right atrial pressure, or pulmonary

225 section again blunted the increase in LV end-diastolic pressure secondary to volume expansion (+4+/-3

226 Excessive reduction in diastolic pressure should be avoided in patients with CA

227 ; however, there was no increase in ischemic diastolic pressure, slowing of the calcium transient dec

228 HF, in-treatment differences in systolic and diastolic pressures, Sokolow-Lyon voltage, and Cornell p

229 osis and left atrium diameter (marker of end-diastolic pressure), suggesting an improvement in diasto

230 there was no alteration in left ventricular diastolic pressure, suggesting that ischemic diastolic d

231 elated cardiac dysfunction, including LV end-diastolic pressure, systolic performance, and chamber st

232 The peak diastolic pressure tended to be higher post- than pre-fl

233 e LV contractile function, and higher LV end-diastolic pressure than Ptges(+/+) mice after myocardial

234 , Whites have more hypertension (systolic or diastolic pressure) than do Blacks.

235 proach would mitigate the increase in LV end-diastolic pressure that develops during volume loading i

236 However, it is a normal diastolic pressure that separates patients with isolated

237 cardiac ischemic events (i.e., the lower the diastolic pressure the greater the risk of coronary hear

238 citation cardiac index, left ventricular end-diastolic pressure, the rate of left ventricular pressur

239 iastolic dysfunction or left ventricular end-diastolic pressure thresholds.

240 e relations of total and LDL cholesterol and diastolic pressure to distensibility had been present at

241 relaxation and lowering left ventricular end diastolic pressure to facilitate ventricular filling, th

242 Resting diastolic pressure trended lower (P = 0.09), heart rate

243 mia, cardiac output decreased by 41% and end diastolic pressure tripled for CD36-null hearts, with no

244 omitantly lowered cardiac preload volume and diastolic pressure (venodilation) without a change in ar

245 fraction or stroke volume and decreased end diastolic pressure versus controls.

246 n fraction, end-systolic volume, and the end-diastolic pressure volume relationship by Ang-(1-9) trea

247 revealed significant improvements in the end-diastolic pressure volume relationship, relaxation kinet

248 .9+/-5.5 mm Hg), caused by an upward-shifted diastolic pressure-volume curve.

249 LV diastolic pressure-volume curves confirmed a substantial

250 A left shift of the diastolic pressure-volume curves without changes in thei

251 nction and prompt leftward shifts of the end-diastolic pressure-volume curves.

252 oke work relation were measured from the end-diastolic pressure-volume relation before and during del

253 -volume curve and a reduced slope of the end-diastolic pressure-volume relation in the myoblast-trans

254 The diastolic pressure-volume relation was shifted up and to

255 ntricle, and induced a downward shift of the diastolic pressure-volume relation.

256 /leftward shift in the left ventricular (LV) diastolic pressure-volume relationship (P = 0.001), a gr

257 diac hypertrophy, diastolic dysfunction (end diastolic pressure-volume relationship =0.051+/-0.009 in

258 tion, exacerbated diastolic dysfunction (end diastolic pressure-volume relationship =0.11+/-0.004 in

259 d against cardiac diastolic dysfunction (end diastolic pressure-volume relationship =0.110+/-0.009 in

260 n and exacerbated diastolic dysfunction (end diastolic pressure-volume relationship =0.124+/-0.005 in

261 normal dogs underwent measurement of the end-diastolic pressure-volume relationship during caval occl

262 on (p = 0.014) and improvement of the RV end-diastolic pressure-volume relationship in PH pigs treate

263 The LV end-diastolic pressure-volume relationship measured by the i

264 The end-diastolic pressure-volume relationship slope was elevate

265 in left ventricular chamber compliance (end-diastolic pressure-volume relationship; P<0.01) and cont

266 In both models, isolated, perfused heart end-diastolic pressure-volume relationships and passive left

267 d impaired relaxation and upward-shifted end-diastolic pressure-volume relationships despite preserve

268 end-diastolic pressure on the sequential end-diastolic pressure-volume relationships was measured (le

269 ers assessed changes in end-systolic and end-diastolic pressure-volume relationships, and microsphere

270 Stiffness coefficient of exponential diastolic pressure-wall thickness relation was higher fo

271 Myocardial stiffness was estimated from diastolic pressure-wall thickness relationship obtained

272 The LV end-diastolic pressure was >16 mm Hg in 58 of the 63 patient

273 on hemodynamics over time, mean right atrial diastolic pressure was 9 +/- 0.6 mm Hg with no lean, 10

274 t; 95% CI, 1.04-1.25; P = .006); however, if diastolic pressure was added, model fit improved and the

275 ral venous pressure nor left ventricular end diastolic pressure was altered by thapsigargin.

276 story of revascularization was observed; low diastolic pressure was associated with a relatively lowe

277 LV end-diastolic pressure was elevated (>/=15 mm Hg) in 72% of

278 Left ventricular diastolic pressure was estimated with Doppler echocardio

279 LV end-diastolic pressure was increased with CIH (CIH, 13.7+/-5

280 LV end-diastolic pressure was measured by micromanometer.

281 Diastolic pressure was reduced in young old participants

282 In the treated hearts, the increase in end-diastolic pressure was significantly attenuated at the e

283 versus 0.05+/-0.03 after MI, P=0.06), LV end-diastolic pressure was unchanged as MR resolved.

284 ction fraction [EF] and left ventricular end-diastolic pressure) was assessed at days 28 and 56.

285 lmonary artery pressure-left ventricular end-diastolic pressure) was normal (<7 mm Hg) or elevated (>

286 dP/dt min (-dP/dt min), and left ventricular diastolic pressure, was measured before inducing VF and

287 ht ventricular mass and left ventricular end diastolic pressure were increased and left ventricular s

288 tricle/body weight, and left ventricular end-diastolic pressure were increased and maximal left ventr

289 index, LV dP/dt40, LV negative dP/dt, and LV diastolic pressure were measured 4 wks after administeri

290 entricle weight/body weight ratio and LV end-diastolic pressure were significantly higher in hearts w

291 ulmonary capillary wedge pressure and LV end-diastolic pressure were significantly increased after th

292 LV diastolic pressures were lower during CHF exercise after

293 Left ventricular end-diastolic pressures were significantly elevated 4 weeks

294 Baseline mean systolic and diastolic pressures were similar in both groups.

295 ure and -0.4 (95% CI: -2.9, 2.2) mmHg/kg for diastolic pressure, while in within-pair analysis the va

296 The potential harm of further reducing the diastolic pressure with antihypertensive therapy, especi

297 ericardiotomy blunted the increase in LV end-diastolic pressure with saline infusion, while enhancing

298 c and diastolic pressures, and reductions in diastolic pressure with treatment but greater reductions

299 and total MI was J-shaped, particularly for diastolic pressure, with a nadir at 119/84 mm Hg.

300 measurements at 6 months showed lower LV end-diastolic pressures, with enhanced LV function (contract