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1 anin in conjunction with adjuvant intranasal diesel exhaust particles.
2  species and detoxify xenobiotics present in diesel exhaust particles.
3  nasal allergic responses in the presence of diesel exhaust particles.
4  higher levels of carbon dioxide, ozone, and diesel exhaust particles.
5 /L [-0.2-19.6], p=0.01) after challenge with diesel exhaust particles and allergens.
6                         The model pollutant, diesel exhaust particles, can participate with allergens
7      Further work investigated how pollutant diesel exhaust particles could modify these transcriptio
8 studies have reported an association between diesel exhaust particle (DEP) exposure, allergic sensiti
9 ent for established asthma in the context of diesel exhaust particle (DEP) exposure.
10 in the pathogenesis of lung injury caused by diesel exhaust particles (DEP) and bacterial lipopolysac
11                                              Diesel exhaust particles (DEP) and their organic constit
12                        The redox activity of diesel exhaust particles (DEP) collected from a light-du
13                                              Diesel exhaust particles (DEP) contain organic chemicals
14                               Ambient PM and diesel exhaust particles (DEP) contain redox cycling org
15 of the mice were co-exposed to mycotoxins or diesel exhaust particles (DEP) during pregnancy.
16                                      Inhaled diesel exhaust particles (DEP) exert proinflammatory eff
17 Increased exposure to air pollutants such as diesel exhaust particles (DEP) has been proposed as one
18                                              Diesel exhaust particles (DEP) have strong, selective Th
19 cies are involved in the adjuvant effects of diesel exhaust particles (DEP) in a murine OVA sensitiza
20 demonstrate that methanol extracts made from diesel exhaust particles (DEP) induce apoptosis and reac
21 usly shown that in vivo nasal challenge with diesel exhaust particles (DEP) induces both quantitative
22 el, here we showed that maternal exposure to diesel exhaust particles (DEP) predisposed offspring to
23 and either urban particulate matter (UPM) or diesel exhaust particles (DEP) via the airways and asses
24 dies have correlated inflammatory effects of diesel exhaust particles (DEP) with its organic constitu
25             We hypothesized that exposure to diesel exhaust particles (DEP), a major component of urb
26 effects of particulate pollutants, including diesel exhaust particles (DEP), are related to their con
27       Exposure to traffic pollution, notably diesel exhaust particles (DEP), increases risk for asthm
28 nce that particulate air pollutants, such as diesel exhaust particles (DEP), potentiate chronic infla
29        Ambient particulate matter, including diesel exhaust particles (DEP), promotes the development
30 ncluding ambient particulate matter (PM) and diesel exhaust particles (DEP).
31 encephalic neuron-glia cultures treated with diesel exhaust particles (DEP; 0.22 microM) (5-50 microg
32 ntly exposed via oropharyngeal aspiration to diesel exhaust particles (DEP; 50 mug x 6 doses) or vehi
33 combined prenatal exposure to air pollution (diesel exhaust particles, DEP) and maternal stress (MS)
34                                              Diesel exhaust particles (DEPs) and SHS can interact wit
35                                              Diesel exhaust particles (DEPs) are a major component of
36                                              Diesel exhaust particles (DEPs) are a major component of
37                                              Diesel exhaust particles (DEPs) are major air pollutants
38                                              Diesel exhaust particles (DEPs) can deposit onto the res
39                   Oxidant pollutants such as diesel exhaust particles (DEPs) can initiate and exacerb
40                            The inhalation of diesel exhaust particles (DEPs) is associated with incre
41 re given repeated intranasal applications of diesel exhaust particles (DEPs) or PBS.
42                         Here, we report that diesel exhaust particles (DEPs), a major constituent of
43  pollution particulate matter, predominantly diesel exhaust particles (DEPs), increases the risk of a
44                                  Exposure to diesel exhaust particles (DEPs), which are major constit
45 igh levels of airborne particulates, such as diesel exhaust particles (DEPs).
46 xposure to environmental pollutants, such as diesel exhaust particles (DEPs).
47 of traffic-related particulate matter (e.g., diesel exhaust particles [DEPs]) is associated with acut
48                                          The diesel exhaust particles enhancement was largest in pati
49                   5-Aza-2'-deoxycytidine and diesel exhaust particle exposure in human bronchial epit
50 ell-established allergic adjuvant effects of diesel exhaust particle exposure.
51 mon lipophilic pollutants benzo[a]pyrene and diesel exhaust particles impact on the activation of lip
52 y with allergen alone and with allergen plus diesel exhaust particles in a randomised order at separa
53                    Insights into the role of diesel exhaust particles in patients with severe asthma
54 f temperature on the number concentration of diesel exhaust particles in the nucleation mode in a pre
55 STM1 and GSTP1 modify the adjuvant effect of diesel exhaust particles on allergic inflammation.
56 re key regulators of the adjuvant effects of diesel exhaust particles on allergic responses.
57 t has been described that exposure to ozone, diesel exhaust particles, or tobacco smoke exacerbates a
58 4.7] vs 7.4 nmol/L [1.2-12.3], p=0.02) after diesel exhaust particles plus allergen challenge.
59                         Examples of UFPs are diesel exhaust particles, products of cooking, heating,
60                                   Similarly, diesel exhaust particles showed a marginal inhibitory ef