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1 T-induced inhibition of ENaC similar to high dietary potassium.
2                                              Dietary potassium, an indicator of NEAP and fruit and ve
3  stroke, and an inverse relationship between dietary potassium and CVD.
4 dance of kidney ARH protein was modulated by dietary potassium and inversely correlated with changes
5         We investigated the relation between dietary potassium and protein, NEAP (with an algorithm i
6 fruit and vegetable intake may help increase dietary potassium and reduce hypertension-related diseas
7                     Observational studies on dietary potassium and risk of cardiovascular disease (CV
8                                              Dietary potassium and the molar ratio of dietary sodium
9 enoprotective, but the effects of increasing dietary potassium and the risk of hyperkalemia are unkno
10                               Moreover, high dietary potassium and valsartan each augmented the inhib
11                                              Dietary potassium can bypass mTORC2 to regulate sodium-g
12                           Consumption of low dietary potassium, common with ultraprocessed foods, act
13                                           In dietary potassium deficiency there is a decrease in the
14                      Both dietary sodium and dietary potassium depletion substantially increase activ
15  increase in potassium absorption induced by dietary potassium depletion.
16                          In conclusion, high dietary potassium enhances the inhibitory effect of AA a
17                                         High dietary potassium increased 11,12-EET in the isolated co
18 ith 109 +/- 35 mmol/d at 4 wk, P = 0.01) and dietary potassium intake (3775 +/- 1189 mg/d at baseline
19  Observational studies suggest that adequate dietary potassium intake (90-120 mmol/day) may be renopr
20                                         When dietary potassium intake and plasma potassium are low, t
21 ortantly, in experimental animal models, low dietary potassium intake induces kidney injury through s
22  understand one important mechanism by which dietary potassium intake influences sodium excretion and
23                            In contrast, when dietary potassium intake is high, aldosterone is stimula
24                    In healthy adults, higher dietary potassium intake is recommended given that potas
25  (encoded by Kcnj16) in mediating effects of dietary potassium intake on the NCC and renal potassium
26  plays a key role in mediating the effect of dietary potassium intake on the thiazide-sensitive NaCl
27       We examined associations between daily dietary potassium intake scaled to energy intake, ascert
28 In patients with CKD stage G3b-4, increasing dietary potassium intake to recommended levels with pota
29 recommendations, we examined associations of dietary potassium intake with mortality in a nationally
30 volume], the gold standard for assessment of dietary potassium intake, represents a risk factor for g
31                         It is concluded that dietary potassium is a potent determinant of hypertensio
32 iated with incident type 2 diabetes, and low dietary potassium is more common in African Americans th
33      It was demonstrated previously that low dietary potassium (K) intake stimulates Src family prote
34 is model stems from the combination of fixed dietary potassium load in the face of reduced filtration
35 secretion in this model is a function of the dietary potassium load.
36 t in the distal tubules of mice subjected to dietary potassium loading and restriction, KS-WNK1 knock
37                                        Acute dietary potassium loading increases the relative abundan
38 nts contributing to an alkaline environment (dietary potassium, magnesium, and fruit and vegetables)
39  cortical collecting duct, we tested whether dietary potassium modulates ENaC function.
40    Future studies that measure the effect of dietary potassium on blood pressure will need to evaluat
41                    The beneficial effects of dietary potassium on both systolic and diastolic blood p
42 study was to assess the effect of increasing dietary potassium on serum potassium concentration in hy
43  This study demonstrates that an increase in dietary potassium over a 4-wk period is safe in hyperten
44 ced kidney dysfunction, guidelines recommend dietary potassium restriction given concerns about hyper
45          Lowering plasma potassium levels by dietary potassium restriction increased NCC activity in
46 however, followed a reverse pattern in which dietary potassium restriction resulted in higher levels
47               This hypothesis predicted that dietary potassium restriction would lower aldosterone an
48 out mice maintained plasma [K(+) ] following dietary potassium restriction, but DKO mice developed se
49 d PTK-dependent pathways under conditions of dietary potassium restriction.
50                      Kir5.1 is essential for dietary potassium's effect on NCC and for maintaining po
51 from protein metabolism and bicarbonate from dietary potassium salts of organic acids are the major v
52 ormal kidney function, the lowest tertile of dietary potassium scaled to energy intake was associated
53                                        Lower dietary potassium scaled to energy intake was associated
54 examined the impact of the interplay between dietary potassium, source of potassium intake (animal- c
55                                         High dietary potassium stimulates the renal expression of cyt
56                                              Dietary potassium stimulates the surface expression of R
57                                              Dietary potassium was not associated with albuminuria.
58 c/Latino adults, high dietary sodium and low dietary potassium were independently associated with an
59 vity, NCC could be stimulated by restricting dietary potassium, which also returned BP to control lev
60          We hypothesized that an increase in dietary potassium would not provoke hyperkalemia in this