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1 e and generalized by the study of the larger diethylamine.
2 ile with very electron-rich species, such as diethylamine.
3 r, which leads to the elimination of neutral diethylamine.
4                Each complex was reacted with diethylamine.
5 amine functionalities of 2,2'-(ethylenedioxy)diethylamine.
6 s similarly to 1 for allylic aminations with diethylamine.
7        The most potent compounds such as the diethylamine 44 (K(i) = 60 nM) contain a basic group at
8   Oxime carbamates derived from the volatile diethylamine afforded aryliminyl radicals that proved co
9 -order rate constants for reaction of 5 with diethylamine and 1,3-cyclohexadiene were determined to b
10 ts 2-lithio-N,N-dimethylbenzylamine (1), the diethylamine and diisopropylamino analogues (2, 3), and
11 mine derivative 2 (anion 1, where R = Et) to diethylamine and NO; (b) results for the zwitterionic pi
12 ric detection and quantification of ammonia, diethylamine and trimethylamine, which is further suppor
13 is considered here to be a rigid analogue of diethylamine, and thus, the target compounds are all con
14 ucts originated from the photolysis of 8a in diethylamine are characterized by analytical techniques,
15 paration of the flavonoids, based on a polar diethylamine-bonded silica stationary phase (ACQUITY Tor
16 rated on a weak convective interaction media diethylamine (CIM DEAE) monolithic chromatographic colum
17 nitroso-1-propylhydrazine (PAPA) NONOate and diethylamine (DEA) NONOate) that do not target thiol res
18   A methanol-to-water gradient containing 2% diethylamine (DEA) promotes separation based on differen
19                                              Diethylamine (DEA) was chosen as the amine source due to
20 using a mild high-solid process with aqueous diethylamine (DEA).
21 t systolic depression induced by an NO donor diethylamine(DEA)NO or nitroglycerin (NTG).
22 ), amines (including dimethylamine, DMA, and diethylamine, DEA), alkyl nitrates (RONO(2)) and nitrous
23  indicate a transition in structure, and for diethylamine (DeltaGB = 40 kJ/mol), the dominant structu
24 ssociation of the otherwise unfunctionalized diethylamine derivative 2 (anion 1, where R = Et) to die
25 ssing sGC resulted in a 30-50% inhibition of diethylamine diazeniumdiolate-NO-stimulated sGC activity
26 koff to Markovnikoff addition occurs without diethylamine dissociation.
27 enyl)[1-Me2Si((t)BuN)TiCl2]-3-C2H4-[N,N-bis((diethylamine)ethyl)-amine ]CrCl3 (Ti-C2-Cr(NNN)), are sy
28 > triethylamine > ethylamine > methylamine > diethylamine > tert-butylamine > ammonia.
29 apped stereoselectively by acrylonitrile and diethylamine in high yields.
30 r, a primary site of action of lysergic acid diethylamine (LSD), appears to dominate efavirenz's beha
31  and Boc-Tyr-OMe), an amine (propargylamine, diethylamine, morpholine, 3,5-dimethylaniline, and isopr
32           Consistent with this, an NO donor, diethylamine nitric oxide (DEANO), induced TGF-beta1 gen
33 NO or NO released from NO donors such as the diethylamine/nitric oxide complex (DEA/NO) and SNAP.
34                                The NO donor, diethylamine NO complex sodium salt (10-100 microM), act
35 icrovascular endothelial cells, the NO donor diethylamine-NO (DETA-NO, 100 microM) reduced VCAM-1 gen
36 rified DbetaH was inhibited by the NO donor, diethylamine/NO (DEA/NO), with an IC(50) of 1 mm.
37  of human neuroblastoma cells (SK-N-MC) with diethylamine/NO decreased cellular DbetaH activity witho
38 These data contrasted with the pure NO donor diethylamine/NO, which induced a negligible inotropic re
39                            NO generated from diethylamine NONOate (DEA-NONOate) or spermine NONOate (
40                               In this study, diethylamine NONOate (DEA/NO) was used to characterize t
41 taneous treatment with LPS and the NO donor, diethylamine NONOate (DEA/NO), enhanced and prolonged JN
42  that various nitric oxide donors, including diethylamine NONOate (DEA/NO), stimulated large increase
43                                 The NO donor diethylamine NONOate (DEA/NO, 10 microM) significantly i
44 and determined the abilities of an NO donor, diethylamine NONOate (DEANO), and a single dose of HU to
45 hen the pathway was stimulated by NO donors (diethylamine NONOate or sodium nitroprusside) or the cyc
46 on of either ANGII (500 fmol) or a NO donor (diethylamine nonoate, 500 pmol) both depressed barorefle
47                             Three NO donors, diethylamine NONOate, spermine NONOate, and S-nitrosoglu
48 tors S-nitroso-N-acetyl-D,L-pencillamine and diethylamine NONOate, the absorbance bands of the [2Fe-2
49         In the presence of the NO.-generator diethylamine nonoate, the electron spin resonance spectr
50 s prevented by coperfusion with the NO-donor diethylamine NONOate.
51 inoma cells exposed to the chemical NO donor diethylamine-NONOate showed increased immunoreactivity o
52 nd intracavernosal injection of the NO donor diethylamine-NONOate were augmented in eNOS-/- and nNOS-
53 elated peptide, whereas neither the NO donor diethylamine/NONOate nor the nitrovasodilator nitroglyce
54 ly, plasma cGMP was increased by infusion of diethylamine/NONOate or nitroglycerin but was unaffected
55                 For the smaller alkenes, the diethylamine nucleophilically attacks the coordinated al
56 presentative Re-BFPCA 8a in a model solvent, diethylamine, proceeded to give a high yield of intermol
57 xide (NO) donors sodium nitroprusside (SNP), diethylamine sodium (DEA), 3-morpholinosydnonimine (SIN-
58 compounds undergo nucleophilic addition with diethylamine to form a strong sigma-donor acyclic diamin
59 oreover, regeneration of the LPG surface via diethylamine treatment resulted in approximately 80% rem
60 ared to other commonly used basic additives (diethylamine, triethylamine, and isobutylamine) showing
61 icities and nucleophilicities: n-butylamine, diethylamine, triethylamine, N-methylpyrrolidine, and tr
62 nitrile, methanol, and water containing 0.1% diethylamine (v/v), at a flow rate of 2.5 ml/min, a colu
63 nitrile, methanol, and water containing 0.1% diethylamine (v/v).
64         Vascular smooth-muscle relaxation to diethylamine was enhanced in endothelium-denuded HC vess
65 l (HNO), a product of the reaction of 5 with diethylamine, was also observed.
66  mixing (baseline noise 0.8 nS/cm for 27 muM diethylamine) with low band dispersion (as small as 30 m