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1  on the spectrum of organizing pneumonia and diffuse alveolar damage.
2 ain histopathologic finding in the lungs was diffuse alveolar damage.
3 ostmortem biopsy for 3 patients showed acute diffuse alveolar damage.
4  22) demonstrated a preponderance of UIP and diffuse alveolar damage.
5  airspaces with protein-rich edema fluid and diffuse alveolar damage.
6  demonstrated particle-laden macrophages and diffuse alveolar damage.
7 tivities for MMPs and TIMPs were stronger in diffuse alveolar damage.
8       A postmortem examination showed severe diffuse alveolar damage.
9 data stratifying histopathological stages of diffuse alveolar damage.
10  of pulmonary oxygen toxicity in the form of diffuse alveolar damage.
11 DI) cells occur following SARS-CoV-2-induced diffuse alveolar damage.
12 te lung damage with organizing pneumonia and diffuse alveolar damage.
13 pathologic observations were consistent with diffuse alveolar damage (70/70) and capillary dilatation
14 e respiratory tract disease characterized by diffuse alveolar damage and hyaline membrane formation.
15                                              Diffuse alveolar damage and interstitial pneumonitis wer
16 ents leading to death, being responsible for diffuse alveolar damage and mononuclear T-cell inflammat
17  histologic features of acute and organizing diffuse alveolar damage and prominent interstitial and a
18 ns from patients with ARDS frequently reveal diffuse alveolar damage, and laboratory studies have dem
19 latation and congestion, interstitial edema, diffuse alveolar damage, and microthrombosis.
20 ization of viral antigen in association with diffuse alveolar damage are prominent features of infect
21 n, loss of lung volume, increased shunt, and diffuse alveolar damage-are also present in several crit
22  correlated with the pathologic processes of diffuse alveolar damage, capillary dilatation and conges
23 vere SARS CoV-2 infection reveal presence of diffuse alveolar damage consistent with ARDS but with a
24 IP) in 18 of 22 patients (81.8%), organizing diffuse alveolar damage (DAD) in 2, bronchiolitis oblite
25 es of acute respiratory insufficiency due to diffuse alveolar damage (DAD) should suggest the diagnos
26                                              Diffuse alveolar damage (DAD) was the second most common
27 lity of this approach by 3D visualization of diffuse alveolar damage (DAD) with its prominent hyaline
28          Both lungs showed various stages of diffuse alveolar damage (DAD), including edema, hyaline
29 f type II pneumocytes has been identified in diffuse alveolar damage (DAD), is associated with p53 an
30  regulatory proteins such as p53 and WAF1 in diffuse alveolar damage (DAD).
31 ute respiratory distress syndrome (ARDS) and diffuse alveolar damage (DAD).
32  4 per etiology) were used for comparison of diffuse alveolar damage, E-cadherin, and molecular biolo
33 ) is a devastating syndrome characterized by diffuse alveolar damage, elevated airspace levels of pro
34 oxicity, including one patient who died from diffuse alveolar damage following a dose of 50 x 10(6) C
35 eous improvement, some cases presenting with diffuse alveolar damage have also been reported.
36        In contrast, severe clinical disease, diffuse alveolar damage, hyaline membrane formation, alv
37              The major pulmonary finding was diffuse alveolar damage in the acute or organising phase
38 inent histopathological feature observed was diffuse alveolar damage in the lung in all case-patients
39 espiratory tract infection, that can lead to diffuse alveolar damage, interstitial and airspace infla
40 , the disease is limited to the lungs, where diffuse alveolar damage is accompanied by a disproportio
41                                              Diffuse alveolar damage is the histopathological hallmar
42                                        This "diffuse alveolar damage" is actually patchy in many pati
43                 Coronavirus infection causes diffuse alveolar damage leading to acute respiratory dis
44 ome, variable ventilation 1) decreased total diffuse alveolar damage (median [interquartile range]: v
45 nts with normal pulmonary histology (n = 3), diffuse alveolar damage (n = 14), and idiopathic pulmona
46 pendent lung regions, reduced the cumulative diffuse alveolar damage score across lungs (median [inte
47 owed focal areas of pulmonary hemorrhage and diffuse alveolar damage, splenic infarct, adrenal necros
48 ar progenitor cells, and temporally with the diffuse alveolar damage stage.
49 nt was higher in human lung biopsies showing diffuse alveolar damage than in normal human lung tissue
50       This suggests that the pathogenesis of diffuse alveolar damage that ultimately leads to the chr
51  consolidation, whereas histomorphologically diffuse alveolar damage was seen in 8 patients.
52 ctivates proinflammatory substances, causing diffuse alveolar damage, which is the histopathological
53 res in this case series included progressive diffuse alveolar damage with excessive thrombosis and la
54 ped fatal lung inflammation characterized by diffuse alveolar damage with marked peribronchial fibros
55 istologic pattern in the peripheral lung was diffuse alveolar damage with perivascular T-cell infiltr
56 cause tracheitis, bronchitis, bronchiolitis, diffuse alveolar damage with pulmonary edema and hemorrh
57                 Pathologic findings included diffuse alveolar damage with pulmonary edema and hyaline
58 ological examination of the autopsies showed diffuse alveolar damage with significant mononuclear inf
59 primary pathology observed in our cohort was diffuse alveolar damage, with virus located in the pneum