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1 as well as for the lateral segregation of a diglyceride.
2 lly active lipids phosphatidic acid (PA) and diglyceride.
3 wn products inositol 1,4,5-trisphosphate and diglyceride.
4 effect on phosphatidic acid, but not that on diglyceride.
5 his lethal process is potently suppressed by diglyceride.
6 des and in 2-3-fold accumulation of label in diglycerides.
7 al correlate for IL-1-generated ether-linked diglycerides.
11 Decreasing TG synthesis by deleting neuronal diglyceride acyltransferases (DGATs) and enhancing PL sy
12 lase, and three out of five putative type II diglyceride acyltransferases (DGATs), the enzymes that c
14 ine (ara-C) stimulates the formation of both diglyceride and ceramide in the acute myelogenous leukem
19 of natural ceramides with those of saturated diglycerides and an unsaturated ceramide demonstrates th
20 e fatty acid (FA) profile of monoglycerides, diglycerides and cholesteryl esters from retroperitoneal
21 R and attributed to partial incorporation of diglycerides and free fatty acids into gum bilayers afte
22 ers, dimers, oxidized triglyceride monomers, diglycerides and free fatty acids, and induction period
23 tty acids, although small proportions of 1,3-diglycerides and of 1-monoglycerides were also found.
26 centrations of two phosphatidylcholines, two diglycerides and two acyl-carnitines were significantly
29 vine cardiolipin, chloroplast monogalactosyl-diglyceride, and dioleoyl phosphatidylethanolamine as a
31 such as phosphatidylcholine, sphingomyelin, diglycerides, and triglycerides were detected and identi
33 cates cytokine receptor-induced ether-linked diglycerides as second messengers that inhibit the bioac
34 pases are differentially activated such that diglyceride breakdown is approximately four times faster
35 gment ions corresponding to loss of the full diglyceride chain as well as the remaining headgroup bou
38 ificant changes in inflammation, ceramide or diglyceride contents, endoplasmic reticulum stress, or a
39 , diacetyl tartaric acid esters of mono- and diglycerides (DATEM), and/or modified DATEMs with differ
43 estigation of de novo triglycerides (TG) and diglycerides (DG) during the ovarian previtellogenic (PV
44 t age-related changes in triglycerides (TG), diglycerides (DG), phosphatidylcholine, phosphatidyletha
46 t of sea urchin membrane vesicle PtdIns into diglycerides enriched in long chain, polyunsaturated spe
47 in addition to increasing triglycerides and diglycerides, especially enriched with unsaturated fatty
48 n in 1-monoglycerides and an increase in 1,2-diglycerides, especially in the margarines with higher w
49 aracterized by high levels of triglycerides, diglycerides, fatty acids, ceramides, and oxidized fatty
50 atty acids, together with triacylglycerides, diglycerides, free fatty acids and ergosterol in salmon
51 rolysis, mobilization of intracellular Ca2+, diglyceride generation, and redistribution of protein ki
54 te that production of membrane-destabilizing diglycerides in membranes enriched in PtdIns may facilit
55 ation and higher levels of triglycerides and diglycerides in the myocardium, aggravating oxidative st
56 and the rate of palmitate incorporation into diglycerides in white muscle was 93% higher in obese rat
57 ut affecting the induced increase in nuclear diglyceride, indicating that the increase in nuclear PLD
58 nterleukin-1 receptor-generated ether-linked diglycerides inhibit immunoprecipitated protein kinase C
59 ramide because, in contrast to the bacterial diglyceride kinase, ceramide is not phosphorylated under
60 with nonprogressors, with simultaneous lower diglycerides, lysophosphatidylcholines, triglycerides, a
63 characterized by increased triglycerides and diglycerides, n3 polyunsaturated fatty acid depletion, a
64 ent score [NES], 2.68), PEs (NES, 2.48), and diglycerides (NES, 1.65) were positively associated, whe
65 in pre-emulsions containing triglyceride- or diglyceride-oil on the emulsifying and gelling propertie
66 ol, treating human fibroblasts with octanol, diglyceride, or ceramide stimulated the rapid inactivati
67 complex of poly(ethylene argininylaspartate diglyceride) (PEAD) polycation, heparin, and cargo insul
68 olism, including decreased triglycerides and diglycerides, phosphatidylcholine/phosphatidylserine rat
71 < 0.0001) without increased accumulation of diglycerides, resulting in significant improvement of th
72 dothelin, selectively generates ether-linked diglyceride species (alkyl, acyl- and alkenyl, acylglyce
74 itory profile of these ether-linked glucosyl diglycerides strengthens the hypothesis that such glycol
75 of sterols, fatty acids, monoglycerides, and diglycerides that are not detected in IR-MALDI MSI exper
76 ctive peptides stimulate distinct species of diglycerides that differentially regulate protein kinase
79 ed defects in FA uptake and in conversion of diglycerides to triglycerides that are similar to those
80 sity lipoprotein cholesterol, triglycerides, diglycerides, total and monounsaturated fatty acids, fat
81 phosphatidic acids but reduced abundance of diglycerides, triglycerides, and phosphatidylglycerol li
84 r ones, such as 1- and 2-monoglycerides, 1,2-diglycerides, vegetable stanols and sterols, and sorbic
85 ol (DPG) vesicles (but not with digalactosyl diglyceride vesicles) as demonstrated by flotation in su
86 PLD2 and PAP2b act sequentially to generate diglyceride within this specialized membrane compartment