ライフサイエンスコーパス: dim

1 The accumulation of Gr-1(dim)CD11b(+) cells is accompanied by the disappearance o

2 The Gr-1(dim)CD11b(+) cells suppress T cell proliferation and IFN

3 cells exhibit unsegmented nuclei, have Gr-1(dim)Ly-6G(dim)CD11b(+) phenotype, and express F4/80, CD4

4 e provide examples of both kinds in 4- and 6-dim Hilbert spaces.

5 ibit unsegmented nuclei, have Gr-1(dim)Ly-6G(dim)CD11b(+) phenotype, and express F4/80, CD49d, Ly-6C,

6 e-, fruit-, and soy-rich (VFS) pattern and a dim sum- and meat-rich (DSM) dietary pattern.

7 l and temporal frequencies was measured at a dim luminance level (2.6 .

8 oupling, but the signal-to-noise ratio for a dim (multiphoton) light response is increased at night b

9 Maintaining diabetic animals in a dim-adapting light did not slow the progression of these

10 evices, suggests that V. parvulus lived in a dim-light setting.

11 anipulated expectations about the onset of a dim visual target using a temporal cueing paradigm, and

12 he birds fly to their goal perch with only a dim point light source as a beacon, showing that they do

13 participants (aged 18-30) were exposed to a dim control (<1 lux) and a range of experimental light l

14 onstrated with transition from the dark to a dim light background.

15 is study investigated whether, compared to a dim light condition (the control), exposure to long-wave

16 ly dichromatic) color vision attributed to a dim-light lifestyle of early snakes [2-4].

17 Vertebrates acquired dim-light vision when an ancestral cone evolved into the

18 8.87 [2.83] at baseline vs 7.33 [3.52] after dim-red LT) and the Parkinson's Disease Sleep Scale (97.

19 1 [19.86] at baseline vs 99.28 [16.94] after dim-red LT).

20 Mb output potentiation selectively amplifies dim retinal inputs at Mb --> ganglion cell synapses.

21 photoreceptors that function in daylight and dim light, respectively.

22 t, when given a choice among blue, green and dim light, fruit flies exhibit an unexpectedly complex p

23 Both bright LT and dim-red LT were associated with improvements in sleep qu

24 dual cone central core reflectances appeared dim, suggesting loss of photoreceptor outer segments.

25 t image motion produces the same problems as dim light: photon noise and low signal-to-noise ratio.

26 We assessed dim-flash-stimulated OP delays using a novel handheld ER

27 d bipolar (RB) cell pathway that operates at dim backgrounds and a rod --> cone --> cone bipolar cell

28 ng the loss of visual information carried by dim scotopic signals.

29 s were derived, at least in part, from CD11c(dim)CD11b(int)Gr1(-) lung-resident monocytic cells trans

30 es over time in vivo even in initially CD123(dim) populations, and that human CD123-redirected T cell

31 the migration of purified CD4(+)CD25(+)CD127(dim) T regulatory cells.

32 CD14(dim)CD16(+) and CD14(+)CD16(+) monocytes showed a prefer

33 ases on whole-mount histology, although CD14(dim) cells disappeared from blood.

34 tes, was observed in CD14(+)CD16(-) and CD14(dim)CD16(+) monocytes, but not in CD14(+)CD16(+) monocyt

35 + of T-cell subsets and %CD14+CD16+ and%CD14(dim)CD16+ monocyte subsets.

36 dothelial migration was not observed in CD14(dim)CD16(+) monocytes during the 30-min observation peri

37 Long-range crawling behavior in CD14(dim)CD16(+) monocytes was abrogated by blockade of ICAM1

38 ate (CD14(+)CD16(+)), and nonclassical (CD14(dim)CD16(+)) monocytes.

39 ate (CD14(+)CD16(+)), and nonclassical (CD14(dim)CD16(+)) monocytes.

40 iate [CD14(+)CD16(+)] and nonclassical [CD14(dim)CD16(+)]) monocytes was increased in the peripheral

41 bsets, CD14(+)CD16(-) (classical) and CD14(+/dim)CD16(+) (nonclassical/intermediate), have been descr

42 ples had significantly higher levels of CD16(dim) and CD16(-) neutrophils and CD16(+) 'intermediate'

43 In particular, CD56(bright)CD16(-/dim) NK cells are the focus of interest.

44 weight, including 5 of 5 patients with CD19(dim) or CD19(-) B-ALL.

45 MS patients, increased levels of CD3(+)CD20(dim) T cells are effectively depleted by RTX.

46 ally demonstrate the existence of CD3(+)CD20(dim) T cells.

47 activated CD8(+) effector T cells with a CD4(dim) CD8(+) phenotype, both exhibiting exquisite specifi

48 ingle positive, CD8 single positive, and CD4(dim)CD8(bright)) were found in NSG-huPBMC mouse brain wi

49 This population is known as CD4(dim)CD8(bright) T cells.

50 Brain CD4(dim)CD8(bright) T cells from HIV-infected mice exhibited

51 ositive T cells into mouse brain induced CD4(dim)CD8(bright) T cells by 10-fold, which were prolifera

52 Wnts secreted from astrocytes induced CD4(dim)CD8(bright) T cells by 2-fold in vitro.

53 We evaluated the role of CD4(dim)CD8(bright) and CD8 single positive T cells in HIV-i

54 Further, higher frequency of CD4(dim)CD8(bright) T cells (R = -0.62; p </= 0.001), but no

55 Thus, CD4(dim)CD8(bright) T cells are capable of HIV control in th

56 NS during HIV infection can give rise to CD4(dim)CD8(bright) T cells, likely through a Wnt signaling-

57 for the first time that MHC class II(+)CD40(dim)CD86(dim)IL-10(+) microglia are potent inducers of A

58 se results indicate that MHC class II(+)CD40(dim)CD86(dim)IL-10(+) microglia have regulatory properti

59 odstream for a longer time (CXCR4(bright)CD5(dim) cells).

60 Rs and BCRs induced differentiation into CD5(dim) (B-1b) cells in MyD88-dependent and CNI-resistant m

61 responsive to BCR ligation than isolated CD5(dim)CXCR4(bright) cells of the same patient.

62 ng immunophenotypes: CD1a(-), CD8(-), CD5(-)(dim), and positivity for 1 or more stem cell or myeloid

63 CD56(dim) cells are generally considered more cytotoxic, wher

64 CD56(dim)CD16(+) NK cells made up the vast majority of NK cel

65 CD56(dim)CD57(+) NK cells are less responsive to IL-2 and pro

66 was most prominent within the adaptive CD56(dim) NK-cell population lacking PLZF expression.

67 ptor CD16 is present on essentially all CD56(dim) peripheral blood natural killer (NK) cells.

68 Also, CD56(dim) NK cells cocultured with M2 displayed lower degranu

69 a and degranulation by CD56(bright) and CD56(dim) NK cells following NKG2D stimulation were dependent

70 marily on conventional CD56(bright) and CD56(dim) NK cells from blood.

71 nal population between CD56(bright) and CD56(dim) NK cells.

72 +) NK cells, including CD56(bright) and CD56(dim) subsets, exhibit impaired cell activation and IFN-g

73 major NK cell subsets (CD56(bright) and CD56(dim)) exist in humans and have distinct anatomical local

74 ets, namely the CD56(bright)CD16(+) and CD56(dim)CD16(-) subsets, were increased in the peripheral bl

75 im)CD57(-)KIR(-)NKG2A(+) (NKG2A(+)) and CD56(dim)CD57(-)KIR(-)NKG2A(-) (lacking inhibitory receptors;

76 Peripheral blood CD56(dim)CD16(+) and CD56(bright)CD16(-) NK cells expressed s

77 PD-1 is expressed by CD56(dim) but not CD56(bright) NK cells and is confined to fu

78 CD57 was mostly expressed by CD56(dim)CD16(+) NK cells.

79 3(-) CD56(+) total NK cells and CD16(+) CD56(dim) NK cells were inversely correlated with HIV-1 DNA l

80 CD3(-) CD56(bright) CD16(-) and CD3(-) CD56(dim) CD16(+) subsets.

81 CD3(+)CD56(+) NKT-like cells and CD3(-)CD56(dim) and CD3(-)CD56(hi) NK cells at baseline, BCG revacc

82 6(bright), canonical, or adaptive CD3(-)CD56(dim) NK cells.

83 granzyme B but less perforin than CD3(-)CD56(dim) NK cells.

84 predominantly to differentiated CD57(+)CD56(dim) NK cells.

85 enescent CD8 + T cells, CD56 + T cells, CD56(dim) natural killer cells, monocytes and dendritic cells

86 c CD56(bright) NK cell to the cytolytic CD56(dim) NK cells.

87 D16(-) NK cells and less differentiated CD56(dim)CD16(+) NK cells.

88 pansion of a novel subset of FcRgamma(-)CD56(dim) NK cells with an altered activation receptor repert

89 Functionally, CD56(dim) NK cells responded poorly to target cells at the ti

90 nal, whereas CD107a(+) and IFN-gamma(+) CD56(dim) NK cells presented a different pattern of HLA class

91 of mouse NK cells are enriched in human CD56(dim) cells.

92 r the IgG Fc portion expressed on human CD56(dim) NK cells and involved in Ab-dependent cell cytotoxi

93 Human CD56(dim) NK cells were costimulated specifically by HVEM but

94 und that in addition to the known human CD56(dim)CD16(+), CD56(bright)CD16(-), and CD56(-)CD16(+) NK

95 ed for the generation of hyporesponsive CD56(dim) NK cells with limited degranulation and cytotoxic c

96 with STAT1 GOF mutations have immature CD56(dim) NK cells with decreased expression of CD16, perfori

97 P1), and RORA mRNA levels are higher in CD56(dim) cells.

98 in vivo restored perforin expression in CD56(dim) NK cells and partially restored NK cell cytotoxic f

99 s and are progressively demethylated in CD56(dim) NK cells as they mature and acquire KIR.

100 s higher in CD56(bright)CD16(-) than in CD56(dim)CD16(+) NK cells.

101 human peripheral blood, the more mature CD56(dim) NK cell efficiently kills malignant targets at rest

102 nduced an increased frequency of mature CD56(dim)NKG2A(+)CD57(+) NK cells in the blood that persisted

103 ) CD161(+) Siglec-7(+) subpopulation of CD56(dim) CD16(+) NK cells are more abundant in EC and HIV-ne

104 ) CD161(+) Siglec-7(+) subpopulation of CD56(dim) CD16(+) NK cells that differentiates HIV controller

105 with degranulation when a wide range of CD56(dim) NK cell activating receptors were stimulated, where

106 s the percentage and absolute number of CD56(dim) NK cells decreased.

107 NKp30 and NKp46 to approximately 90% of CD56(dim) NK cells in some VS HIV(+) individuals may influenc

108 RBV pretreatment, both the frequency of CD56(dim) NK cells with cytotoxic effector functions and the

109 8(+) T cells plus a milder expansion of CD56(dim) NKG2A(+) KIR(-) natural killer (NK) cells.

110 mediate CXCR3 levels on the majority of CD56(dim)CD16(+) pNK cells.

111 We show an expansion of CD56(dim)CD57(dim)CD69 + CCR7 +KIR+ NK cells in TILN.

112 tion, characterized by the expansion of CD56(dim)NKG2A(-)KIR(+) cells, even in the absence of NKG2C e

113 rs CD319 and CD229 on pDCs and CD319 on CD56(dim) NK cells was selectively increased.

114 xpression of CD319 on pDCs and CD229 on CD56(dim) NK cells, but RNA-IC stimulation increased CD319 an

115 cells and inhibitory receptor NKG2A on CD56(dim) NK cells, compared with nonresponders.

116 ated activation than do CD56(bright) or CD56(dim)CD57(-) NK cells.

117 n maintained independently from GPI(pos)CD56(dim).

118 ve compared the behavior of FACS-sorted CD56(dim)CD57(-)KIR(-)NKG2A(+) (NKG2A(+)) and CD56(dim)CD57(-

119 ls to be more metabolically active than CD56(dim) cells, which supports their production of large amo

120 The CD56(dim) NK cell subset had decreased expression of perforin

121 action of GPI-negative cells within the CD56(dim) NK cells was markedly lower than that of neutrophil

122 ge preceding terminal maturation to the CD56(dim) stage, considered the most enabled for cytotoxicity

123 K cells and downregulated CX3CR1 in the CD56(dim) subset.

124 degranulation, is maintained across the CD56(dim) subsets.

125 infections, drives the expansion of the CD56(dim)CD57(+)NKG2C(+) NK cell population, skewing the NK c

126 forin(low) NK cells, with others by the CD56(dim)perforin(high) cytotoxic counterpart.

127 oduce IFN-gamma at levels comparable to CD56(dim) NK cells.

128 ma(-) NK cells as a proportion of total CD56(dim) NK cells increased in cART-naive viremic HIV-infect

129 r rates of glucose uptake compared with CD56(dim) cells.

130 more metabolically active compared with CD56(dim) cells.

131 me 10 (PTEN) protein when compared with CD56(dim) NK cells.

132 We show an expansion of CD56(dim)CD57(dim)CD69 + CCR7 +KIR+ NK cells in TILN.

133 profound nuclear hypersegmentation, a CD62L(dim), CD16(bright), CD11b(bright), CD66b(bright), CD63(b

134 The differences between CD62L(dim) and mature neutrophils are unlikely to have been a

135 red much earlier in blood than labeled CD62L(dim) and segmented neutrophils, which shared similar lab

136 remely low transcriptional capacity of CD62L(dim) neutrophils and the fact that neutrophils do not di

137 Therefore, we propose CD62L(dim) neutrophils are a truly separate neutrophil subset

138 banded nucleus, and T-cell-suppressing CD62L(dim) neutrophils with a high number of nuclear lobes.

139 omes by cluster analysis revealed that CD62L(dim) neutrophils were clearly separate from conventional

140 a very immature thymic, CD34(+)/CD1a(-)/CD7(+dim) stage, before Ddelta2(Ddelta3)-Jdelta1 rearrangemen

141 responses occur within the CD38(+)CD27(-)CD8(dim)T cell population, the minority populations of CD8(b

142 Furthermore, the frequency of CD8(dim)T cells directly correlates with viral load and clin

143 ytokines coincides with the emergence of CD8(dim)T cells, and the size of this population inversely c

144 the low expression of the CD8 receptor (CD8(dim)).

145 of a functionally impaired HIV-specific CD8(dim)T cell population less efficient in controlling HIV

146 first time that MHC class II(+)CD40(dim)CD86(dim)IL-10(+) microglia are potent inducers of Ag-specifi

147 s indicate that MHC class II(+)CD40(dim)CD86(dim)IL-10(+) microglia have regulatory properties potent

148 ibility complex class II-restricted CD8alpha(dim) T cells that are generated through CD4 downregulati

149 ession-like phenotype observed under chronic dim LAN.

150 continuous bright light (CBL) or continuous dim (~1 lux) light (VDL) for 6.5 h during the biological

151 pulation of CLL cells that migrate are CXCR4(dim)CD5(bright) with higher CD49d, CD80, CD86, and HLA-D

152 t and moved into the peripheral blood (CXCR4(dim)CD5(bright) subpopulation) have higher cell surface

153 Also, isolated CD5(bright)CXCR4(dim) cells, representing CLL that had been newly release

154 ed CLL cells manifest a proliferative, CXCR4(dim)CD5(bright) phenotype compared with those in the PB

155 metry as CD63(bright)CD203c(+)CD123(+)HLA-DR(dim/-)CD41a(-)lineage(-).

156 re alleviated by imipramine treatment during dim night-time light.

157 hesized that nocturnal light exposure (i.e., dim LAN) would induce depressive responses and alter neu

158 igh-energy intermediate, capable of emitting dim light by itself, formed from the reaction between gu

159 declining percentages of the T-bet(hi) Eomes(dim) CD8 T-cell population.

160 of co-expressed bfloGFPc1 showing extremely-dim brightness due to low (0.1%) quantum efficiency.

161 phids possess several visual adaptations for dim-light conditions, including enlarged eyes, an aphaki

162 g insects are hypothesized to compensate for dim conditions by integrating light over longer times.

163 tude is only slightly perturbed, optimal for dim light and for small shifts 2) another class of sched

164 nd high photosensitivity in rod pigments for dim-light vision.

165 orming the transduction channel required for dim light vision and the ON pathway.

166 the vertebrate retina and is responsible for dim light vision.

167 fth cone for achromatic tasks, and a rod for dim-light vision.

168 r cells with differing sensitivity: rods for dim light and cones for bright light and colour detectio

169 tes have a duplex retina containing rods for dim light vision and cones for bright lights and color d

170 Most vertebrate eyes have rods for dim-light vision and cones for brighter light and higher

171 alia was dominated by positive selection for dim-light vision, supporting the predominate nocturnalit

172 ght vision and relatively weak selection for dim-light vision.

173 gle-QD properties, principally emitting from dim gray states but having high two-exciton (biexciton)

174 over a wide range of light intensities, from dim starlight to bright sunshine.

175 We identified two subpopulations of GEC (dim(tdT) and bright(tdT)) based on the fluorescence inte

176 eme lighting, peripheral vision, and general dim lighting.

177 GR(dim)-LPS mice also exhibited sustained locomotor impairm

178 GR(dim)-LPS mice exhibited elevated and prolonged levels of

179 However, dim fluorescence and the inability to make stably-expres

180 creased populations of B-1 cells (B220(+)IgD(dim)IgM(+)CD43(+)CD24(+)CD5(+)), and higher numbers of i

181 and higher numbers of immature B cells (IgD(dim)IgM(dim)CD21(neg)) at the expense of mature B cells

182 layed a shift from IgD(+)IgM(dim) toward IgD(dim)IgM(+) B cell populations in spleen, peritoneum and

183 her numbers of immature B cells (IgD(dim)IgM(dim)CD21(neg)) at the expense of mature B cells (IgD(+)I

184 taIItg mice displayed a shift from IgD(+)IgM(dim) toward IgD(dim)IgM(+) B cell populations in spleen,

185 We also provide directions on how to image dim signals such as those of radioluminescence (1-1.5 h)

186 In dim light conditions, AHA2 is found in intracellular com

187 In dim light, rod-photoreceptors are active, but colour vis

188 In dim light, vertebrates generally rely on a single rod op

189 In dim lights, rods relay relatively slow temporal variatio

190 six types of photoreceptors: rods, active in dim light, double cones that are thought to mediate achr

191 g brain organization among insects active in dim-light conditions.

192 can explain certain experiences of colour in dim lights, such as a 'blue shift' in twilight.

193 ion cell performs a different computation in dim light--averaging contrast within its receptive field

194 tant bright light and a control condition in dim light.

195 erin potentiates the sensitivity of cones in dim light conditions but does not contribute to their ca

196 hifts were calculated from the difference in dim light melatonin onset (DLMO) between CRs.

197 tion detection capabilities up to 10-fold in dim environments.

198 se development preserves retinal function in dim light.

199 ily apparent when the seedlings are grown in dim white light, were attenuated by treatment with eithe

200 ort, during a limited time of fast growth in dim white light beginning 2.5 days after germination.

201 mmalian retina encodes visual information in dim light using rod photoreceptors and a specialized cir

202 mmalian retina encodes visual information in dim light using rod photoreceptors and a specialized cir

203 responses of SbC-RGCs, which are observed in dim and bright light conditions.

204 sluggish, highly sensitive, and operates in dim or scotopic lights, whereas cone-driven vision is br

205 (AMD) have vision problems, particularly in dim light conditions.

206 Fluctuations result in dim lights at best and power outages and broken applianc

207 sms in the inner retina limit sensitivity in dim (scotopic) lights.

208 mpact of diabetes on contrast sensitivity in dim light is unknown.

209 To access reliable visual signals in dim-light environments, insects have evolved several vis

210 at the moth's visual processing does slow in dim light.

211 in bright and self-selected lighting than in dim light for both chronotypes, whereas visual comfort w

212 in rod photoreceptors that provide vision in dim ambient light.

213 od and cone photoreceptors mediate vision in dim and bright light, respectively, by transducing absor

214 asmin and experienced darkening of vision in dim illumination for 4 months, despite improvement in vi

215 ncreases photon capture, enhancing vision in dim light [1-5].

216 e specialized neurons that mediate vision in dim light and are the predominant photoreceptor type in

217 tion of ocular specializations and vision in dim light are discussed.

218 Vision in dim light depends on synapses between rods and rod bipol

219 communication is indispensible for vision in dim light.

220 e visual signal, thereby improving vision in dim light.

221 -photon responses are critical for vision in dim light.

222 oise and limits the sensitivity of vision in dim light.

223 receptors, the cells that initiate vision in dim light.

224 mes these limitations and supports vision in dim light.

225 n is for imaging signals that are inherently dim and undetectable using standard microscopy technique

226 ro measurements of rhodopsins to investigate dim-light adaptation.

227 of spatial details on bright days and large dim objects on moonless nights.

228 better sleep efficiency and exhibited later dim light melatonin onset phase than females, whereas fe

229 es exposed to 12 h light:12 h darkness (LD), dim light-at-night (DLAN) or constant bright light (LLbr

230 al exposures were abnormal, including light (dim), nutritional intake (reduced or absent and mistimed

231 ittermates were housed in a 12:12 hour light-dim light photocycle (30 lux during the day and 3 lux at

232 ither 7,000-lux bright white light or 50-lux dim red placebo light (N=23 for each group).

233 ce photoreceptor degeneration or maintenance dim light (25 lux).

234 Rhodopsin, the mammalian dim-light receptor, is a unique test case for understand

235 on where rod and cone photoreceptors mediate dim- and bright-light vision, respectively.

236 mperature-sensitive visual pigment mediating dim-light vision, offers an opportunity to enhance our u

237 In AS mice, dim(tdT) and bright(tdT) cells had different expression

238 Finally, neither dim light alone nor a shortened night is sufficient for

239 Interestingly, we also observed dim ORF1p immunoreactivity in histologically NE of all p

240 reduced sucrose preference, in weeks 2-3 of dim light at night, whereas WT mice did not.

241 ming than WT mice during weeks 1, 2 and 4 of dim light at night exposure.

242 Continuation of dim light is unnecessary for T15/30 behavioral entrainme

243 We conclude that detection of dim-flash OP delays could provide early detection of DR

244 y reduction in green preference in favour of dim light depends on the transient receptor potential (T

245 vels (41 min) and delayed circadian phase of dim-light melatonin offset (1.37 h), partially mediated

246 rly defined cell subsets than those based on dim markers and for rare populations.

247 2 hr bright lightratiodark (1000lux, BLD) or dim lightratiodark (50lux, DLD) condition.

248 ffect dCRY phototransduction under bright or dim light in vivo as measured by light-induced proteolys

249 rences, such as optimization for computer or dim-light working, or night driving, could be useful too

250 were randomized 1:1 to receive bright LT or dim-red LT (controlled condition) twice daily in 1-hour

251 e primarily exposed to intense (>300 lux) or dim (<30 lux) environmental light-stimuli at extreme end

252 rerequisite for rod cells, which mediate our dim-light vision.

253 The struggle to perceive dim downwelling light and bioluminescent sources and the

254 All candidate signatures appear radio-dim and do not have the X-ray to radio flux ratios requi

255 brate retinas are generally composed of rod (dim-light) and cone (bright-light) photoreceptors with d

256 light exposure were assessed using salivary dim-light melatonin onset (DLMO) and wrist-worn photomet

257 nificant for all parameters (except scotopic dim-flash b-wave implicit time), ranging from 0.34 to 0.

258 ponses of rod photoreceptors, which subserve dim light vision, are carried through the retina by thre

259 The dim population showed similar trends, though velocities

260 the camera was unlikely to have detected the dim outer edges of the flashes, given its weak response

261 in the red light mask condition than in the dim light condition.

262 he self-selected and the bright, but not the dim lighting condition, the onset of melatonin secretion

263 mechanism optimizing the performance of the dim-light channel of vision, which consists of sensitizi

264 ontinued activation of the photocycle of the dim-light receptor rhodopsin leads to the accumulation o

265 ey are nearer to ancestral pigments than the dim-light rod photoreceptor rhodopsin.

266 Scale score comparing the bright LT with the dim-red LT.

267 ry 6-sulfatoxymelatonin (aMT6s)) and timing (dim light melatonin onset (DLMO)), and evening salivary

268 nt was also agravitropic but when adapted to dim red light it displayed a reversed gravitropic respon

269 ionts on its carapace, usually associated to dim-light paleoenvironments and/or rock crevices, sugges

270 xposed 5XFAD mice and littermate controls to dim-light vs. bright-light photophases to investigate th

271 Exposure to dim light at night (dLAN) disrupts natural light/dark cy

272 Bdnf expression after 3 weeks of exposure to dim light at night, but only mice deficient for the PERI

273 Firstly, exposure to dim nocturnal illumination (<0.1 lux), rather than compl

274 potential (OP) implicit times in response to dim-flash stimuli (<-1.8 log cd . s/m(2)) occur prior to

275 action of both regions sustains responses to dim light, allows for the integration of light over time

276 Rod-driven responses to dim scotopic single-flash stimuli were normal in 7 patie

277 ny Muller cells suggests high sensitivity to dim conditions and high retinal regeneration.

278 es reduces retinal and visual sensitivity to dim light flashes.

279 t light should not compromise sensitivity to dim light.

280 s, implying strong selective pressure toward dim-light vision in Cambrian ecosystems.

281 visited by the LMCs differed between the two dim-light species, their dendritic extents were very sim

282 Rods, active under dim illumination, are thought to saturate at higher (pho

283 ght sunlight to single-photon counting under dim starlight.

284 paired in rh7 mutant flies, especially under dim light.

285 than with a spherical IOL, especially under dim light.

286 ina but not in the retina of mice kept under dim lighting.

287 ng 3 balanced crossover segments, once under dim light (DL: 8 lx), and once under either white light

288 efficiency that allows them to operate under dim light conditions.

289 tina, which triggers a visual response under dim light conditions.

290 ntrols the threshold responses of RGCs under dim ambient light.

291 such responses to be mediated by rods under dim lighting conditions, rods/M-cones/melanopsin under i

292 But very dim-light exposure quickly translocates them to the oute

293 For animals active in very dim light the visual system is challenged by several sou

294 Arch fluorescence, however, is very dim and is not optimal for applications in live-cell ima

295 ATR fluorescence is very dim, yet, using the retinal analog dimethylaminoretinal,

296 through associated limitations with the very dim-fluorescent acceptor ShadowG for mTFP1 and the red-s

297 robust physiological effects, even with very dim and/or brief photic exposure.

298 fects on heterochromatin compaction, whereas dim-3 caused more drastic changes, specifically decreasi

299 idual trajectories and can be used also with dim and dense molecules.

300 onstrate how altering light/dark cycles with dim LEN (dLEN) speed the development of breast tumors, i