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1  treating primary oviduct cell cultures with dimethyl sulfate.
2  as more classical reagents, such as Pb2+ or dimethyl sulfate.
3 lation of the respective diamidoxime 5a with dimethyl sulfate.
4  accelerated by replacing methyl iodide with dimethyl sulfate.
5 mple, diagnostic alkylation of guanine N7 by dimethyl sulfate.
6 nhibition is observed for DNA methylation by dimethyl sulfate.
7  and chemical modification interference with dimethyl sulfate, 1-cyclohexyl-3-(2-morpholinoethyl)carb
8 ins generally have higher reactivity to DMS (dimethyl sulfate), a chemical that covalently modifies a
9 proof of concept, we present high-throughput dimethyl sulfate accessibility data for a chimeric DNA/R
10 m riboswitch with N-methylisatoic anhydride, dimethyl sulfate and 1-cyclohexyl-3-(2-morpholinoethyl)c
11 robing of the P4-P6 tertiary structure using dimethyl sulfate and CMCT confirms that these TGGE exper
12 by electrophoretic mobility shift assays and dimethyl sulfate and diethyl pyrocarbonate interference
13 ing, chemical modification experiments using dimethyl sulfate and hydrazine were performed on both th
14 his study, we used a combination of DNase I, dimethyl sulfate and hydroxyl radical footprinting analy
15 edly demonstrated an average reactivity with dimethyl sulfate and minimal reactivity with RNase T1, t
16          This prediction was tested by using dimethyl sulfate and potassium permanganate footprinting
17 uch lower level by the SN2 methylating agent dimethyl sulfate and repaired much faster than 7MeGs in
18   For the procedure, gramine is treated with dimethyl sulfate and sodium in ethanol at room temperatu
19          We probed the structure of 13:22 by dimethyl sulfate and tested its partner in a base-triple
20 hyl) carbodiimide metho-p-toluene sulfonate, dimethyl sulfate, and kethoxal.
21    Chemical probing with chloroacetaldehyde, dimethyl sulfate, and potassium permanganate is consiste
22 groove-specific interactions, as detected by dimethyl sulfate, are diminished.
23 ctivity results in increased mutagenicity of dimethyl sulfate as evidenced by a 2-fold increase in La
24                     Previously, we developed Dimethyl-Sulfate-based Mutational Profiling and Sequenci
25 on, surrounding C2394, a base protected from dimethyl sulfate by E site tRNA, and in the phylogenetic
26 thylpurine-DNA glycosylase (MPG protein) and dimethyl sulfate-damaged DNA manifested sequence context
27 on nuclear protein-DNA interactions by using dimethyl sulfate (DMS) and DNase I ligation-mediated PCR
28 lation analyzed by primer extension (SHAPE), dimethyl sulfate (DMS) chemical probing, and nuclear mag
29  in infected cells by in vivo treatment with dimethyl sulfate (DMS) followed by visualization through
30 d nuclei, the rmm3 rDNA lacked the wild-type dimethyl sulfate (DMS) footprint in the promoter region
31 is, we developed BASH MaP, a single-molecule dimethyl sulfate (DMS) footprinting method and DAGGER, a
32 is tract both in vitro and in vivo using the dimethyl sulfate (DMS) footprinting technique and nucleo
33  analysis of the G-rich strand combined with dimethyl sulfate (DMS) footprinting, a polymerase stop a
34 lar dichroism (CD), thermal denaturation and dimethyl sulfate (DMS) footprinting, we found that a sin
35 h strand DNA of NHE was identified by CD and dimethyl sulfate (DMS) footprinting.
36 e of comparing this method with the standard dimethyl sulfate (DMS) in vivo method and previously rep
37 mobility shift assays, DNase I footprinting, dimethyl sulfate (DMS) interference assays, and DMS prot
38                                 For decades, dimethyl sulfate (DMS) mapping has informed manual model
39              We describe a protocol in which dimethyl sulfate (DMS) modification of the base-pairing
40                                      In vivo dimethyl sulfate (DMS) mutational profiling with sequenc
41                              Here we present dimethyl sulfate (DMS) mutational profiling with sequenc
42  Mg(2+) using both the traditional method of dimethyl sulfate (DMS) N1 methylation and a new approach
43 forms a Py.Pu.Py triplex as detected by both dimethyl sulfate (DMS) probing and a gel-shift assay; in
44 otides and then detects these events through dimethyl sulfate (DMS) probing and mutational profiling.
45                                              Dimethyl sulfate (DMS) probing of the cross-linked d[GCC
46 etermine aptamer sensitivity/selectivity and dimethyl sulfate (DMS) probing to explore aptamer bindin
47                        The workflow combines dimethyl sulfate (DMS) probing, ultra-processive RT, and
48                          Using time-resolved dimethyl sulfate (DMS) probing, we have analyzed time-de
49 ng the gel shift assay, chemical probing and dimethyl sulfate (DMS) protection studies, we determined
50 nd the same intracellular eukaryotic mRNA by dimethyl sulfate (DMS) structure probing.
51        We show through chemical probing with dimethyl sulfate (DMS) that conformational changes occur
52 sceptibility of adenine-N1 to methylation by dimethyl sulfate (DMS) when in an A-T Watson-Crick versu
53      Solvent accessibility reagents, such as dimethyl sulfate (DMS), 1-cyclohexyl-3-(2-morpholinoethy
54 and A1493 are protected from reactivity with dimethyl sulfate (DMS).
55 thylpyrocarbonate (DEPC; to probe A at N-7), dimethyl sulfate (DMS; to probe A at N-1, and C at N-3),
56 evelop DM-DMS-MaPseq, which utilizes in vivo dimethyl-sulfate (DMS) chemical probing and mutational p
57 ween the two guanosines protected by in vivo dimethyl sulfate DNA footprinting (GAAGAGTG) in a lucife
58  ligation-mediated PCR combined with in vivo dimethyl sulfate, DNase I, or UV treatment-of ICR sequen
59                                              Dimethyl sulfate footprint analysis confirmed that the i
60                                      In vivo dimethyl sulfate footprint analysis confirmed the existe
61                                      In vivo dimethyl sulfate footprinting analysis of the CUP1 promo
62              We found here, through in vitro dimethyl sulfate footprinting and gel mobility shift ass
63                                   We show by dimethyl sulfate footprinting and RNA polymerase arrest
64 ques including DNase I, hydroxyl radical and dimethyl sulfate footprinting and the circular permutati
65           Electrophoretic mobility shift and dimethyl sulfate footprinting assays demonstrated that a
66                              We used in vivo dimethyl sulfate footprinting by ligation-mediated PCR t
67                             However, in vivo dimethyl sulfate footprinting demonstrated that protein-
68 an beta-globin locus, we analyzed by in vivo dimethyl sulfate footprinting erythroid cells expressing
69                                              Dimethyl sulfate footprinting further revealed how slow
70                                      In vivo dimethyl sulfate footprinting of the cyclin E promoter r
71                                      In vivo dimethyl sulfate footprinting of the HS-2 region reveale
72                               First, in vivo dimethyl sulfate footprinting of the human LDL receptor
73 quence using a Taq polymerase stop assay and dimethyl sulfate footprinting revealed the formation of
74                         Hydroxyl radical and dimethyl sulfate footprinting show that both I(trap) and
75                                              Dimethyl sulfate footprinting showed that the major groo
76                                     By using dimethyl sulfate footprinting, we recently identified tw
77 ng crudely similar complexes, as revealed by dimethyl sulfate footprinting.
78                  A comparison of DNase I and dimethyl sulfate footprints in vivo and in vitro strongl
79 method reported here demonstrates the use of dimethyl sulfate for conversion of enaminoketones to bet
80 footprinting showed similar accessibility to dimethyl sulfate for PU.1/DNA and Ets-1/DNA complexes, i
81 se T1, RNAse V1, RNAse U2, lead acetate, and dimethyl sulfate has led to the generation of the first
82 ractions in this promoter were identified by dimethyl sulfate in vivo footprinting analysis of NG108-
83                                              Dimethyl sulfate in vivo footprinting identified 10 puta
84 se III mediated in vivo DNA footprinting and dimethyl sulfate in vivo footprinting revealed DNA prote
85 reased methylation of the N7 of guanines (by dimethyl sulfate) in the zinc finger contacts of the ICR
86 gle-stranded DNA-specific reagents KMnO4 and dimethyl sulfate indicated that RecBCD opened, in a Mg(2
87                      Base excision repair of dimethyl sulfate induced N-methylpurines (NMPs) was meas
88                Base excision repair rates of dimethyl sulfate-induced 3-methyladenine and 7-methylgua
89 issing-nucleoside interference experiment, a dimethyl sulfate interference experiment, and an examina
90                   Potassium permanganate and dimethyl sulfate interference experiments showed that RX
91                   Potassium permanganate and dimethyl sulfate interference experiments using the AIIA
92           Here we convert the common reagent dimethyl sulfate into a useful probe of all 4 RNA nucleo
93                        Induction of 7MeGs by dimethyl sulfate is affected by nearest-neighbor nucleot
94    The versatility of CAFA is illustrated by dimethyl sulfate mapping of RNA secondary structure and
95 ll occupied, are less protected from in vivo dimethyl sulfate methylation in a deltaGRF2 strain.
96                                              Dimethyl sulfate methylation interference assays indicat
97                                    Moreover, dimethyl sulfate methylation protection assays demonstra
98 ppears to form a stem-loop in vivo, based on dimethyl sulfate modification and the sequences of intra
99                                      In vivo dimethyl sulfate modification of ribosomal RNA and the d
100                                      We used dimethyl sulfate modification protection studies to show
101                         Using time-dependent dimethyl sulfate modification, we have determined that a
102 ppears to bind proteins that protect it from dimethyl sulfate modification.
103 le-cycle reverse transcription of long RNAs, dimethyl sulfate mutational profiling (DMS-MaP), selecti
104 rize secondary and tertiary structures using dimethyl sulfate mutational profiling and cryo-electron
105 ion and mutational profiling (SHAPE-MaP) and dimethyl sulfate mutational profiling and sequencing (DM
106                    We additionally performed dimethyl sulfate mutational profiling with sequencing (D
107 we probe hTR structure in living cells using dimethyl sulfate mutational profiling with sequencing (D
108                                  Here we use dimethyl sulfate mutational profiling with sequencing (D
109   In this work, we devised the mitochondrial dimethyl sulfate mutational profiling with sequencing (m
110 on factor binding sites identified by either dimethyl sulfate or DNase I in vivo footprinting of the
111 ethylation by treatment of cells or DNA with dimethyl sulfate or from reaction of DNA with DB[a,l]P i
112  was accomplished by treatment of cells with dimethyl sulfate or ultraviolet light, followed by ligat
113 ferential mutational profiling (PD-MaP) with dimethyl sulfate probing for high-throughput detection o
114                                              Dimethyl sulfate probing results suggest that the compac
115                                Surprisingly, dimethyl sulfate probing reveals that individual 3' isof
116 n living yeast (Saccharomyces cerevisiae) by dimethyl sulfate probing with or without Rev.
117                                              Dimethyl sulfate protection assays revealed limited cont
118 several areas of protein binding as shown by dimethyl sulfate protection or enhancement.
119 complex with 1, 10-phenanthroline-copper and dimethyl sulfate protection reveal that both the heptame
120   We observed good agreement between in vivo dimethyl sulfate reactivity and ribosome structure.
121 CoV-2 secondary structure and its underlying dimethyl sulfate reactivity data.
122 aracterize these conformational states using dimethyl sulfate reactivity studies and Bal 31 nuclease
123 ctural properties that may influence in vivo dimethyl sulfate reactivity, an orthogonal chemical appr
124 of unpaired adenine and cytosine residues by dimethyl sulfate, result in a stop in reverse transcript
125 ease-sensitive sites in vitro and by in vivo dimethyl sulfate RNA modification.
126         Using native gel electrophoresis and dimethyl sulfate structural probing, we monitored Mss-11
127  this, we used in vivo chemical probing with dimethyl sulfate to detect changes in pre-rRNA structure
128  fold independently, we used Fe(II)-EDTA and dimethyl sulfate to probe the solvent accessibility of s
129       Using ligation-mediated PCR on in vivo dimethyl sulfate-treated HTLV-1-infected cell lines MT-2
130  an in vivo footprinting method that couples dimethyl sulfate treatment and ligation-mediated PCR was
131 tifs from DNase I digestion or reaction with dimethyl sulfate was observed and phenobarbital treatmen
132 obtain a map of potential interaction sites, dimethyl sulfate was used to footprint regions of the in
133                   Using both Fe(II).EDTA and dimethyl sulfate, we have identified a strong footprint

 
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