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1 ethyltransferases (PRMTs) and is degraded by dimethylarginine dimethylaminohydrolase.
2                                              Dimethylarginine dimethylaminohydrolase 1 (DDAH1) degrad
3                                              Dimethylarginine dimethylaminohydrolase 1 (DDAH1) protec
4                       Neuropilin-1 maintains dimethylarginine dimethylaminohydrolase 1 expression in
5 ed higher amounts of ADMA-degradation enzyme dimethylarginine dimethylaminohydrolase-1 (but similar a
6 h the FXR agonist GW4064, we have identified dimethylarginine dimethylaminohydrolase-1 (DDAH1) as an
7                                              Dimethylarginine dimethylaminohydrolase-1 (DDAH1) is a m
8 verexpression of the ADMA-hydrolyzing enzyme dimethylarginine dimethylaminohydrolase-1 (DDAH1), we te
9                                              Dimethylarginine dimethylaminohydrolase-1 transgenic (TG
10     We further observed myocardial levels of dimethylarginine dimethylaminohydrolase-1 were increased
11  treatment led to proteolytic degradation of dimethylarginine dimethylaminohydrolase 2, which catabol
12                      Reports have shown that dimethylarginine dimethylaminohydrolase activity is down
13 ic dimethylarginine [ADMA, via inhibition of dimethylarginine dimethylaminohydrolase (DDAH) activity]
14 minated largely through active metabolism by dimethylarginine dimethylaminohydrolase (DDAH) and thus
15 termine whether overexpression of the enzyme dimethylarginine dimethylaminohydrolase (DDAH) could enh
16                                              Dimethylarginine dimethylaminohydrolase (DDAH) enzymes a
17                                   The enzyme dimethylarginine dimethylaminohydrolase (DDAH) hydrolyse
18                       In mammals, the enzyme dimethylarginine dimethylaminohydrolase (DDAH) is implic
19 hesized that lowering ADMA concentrations by dimethylarginine dimethylaminohydrolase (DDAH) overexpre
20                                              Dimethylarginine dimethylaminohydrolase (DDAH) regulates
21            To clarify this issue, we crossed dimethylarginine dimethylaminohydrolase (DDAH) transgeni
22     The activity, but not the expression, of dimethylarginine dimethylaminohydrolase (DDAH) was reduc
23 ntitumor therapeutics, as have inhibitors of dimethylarginine dimethylaminohydrolase (DDAH), an enzym
24  a corresponding increase in the activity of dimethylarginine dimethylaminohydrolase (DDAH), an enzym
25 bstituted arginine-modifying enzymes such as dimethylarginine dimethylaminohydrolase (DDAH), arginine
26 sed to inhibit the catabolic enzyme of ADMA, dimethylarginine dimethylaminohydrolase (DDAH), but the
27            ADMA is metabolized by the enzyme dimethylarginine dimethylaminohydrolase (DDAH), the acti
28 lled by two isoforms of its catabolic enzyme dimethylarginine dimethylaminohydrolase (DDAH), the dysr
29  was associated with the reduced activity of dimethylarginine dimethylaminohydrolase (DDAH), the enzy
30 vely metabolized by the intracellular enzyme dimethylarginine dimethylaminohydrolase (DDAH), which ca
31 endothelial cells (ECV304) and on the enzyme dimethylarginine dimethylaminohydrolase (DDAH), which de
32 inine is eliminated largely by the action of dimethylarginine dimethylaminohydrolase (DDAH), which ex
33 abel for the nitric oxide controlling enzyme dimethylarginine dimethylaminohydrolase (DDAH).
34 its NO synthase, is inactivated by N(G),N(G)-dimethylarginine dimethylaminohydrolase (DDAH).
35 methylarginine are metabolized by the enzyme dimethylarginine dimethylaminohydrolase (DDAH).
36              The enzymes dimethylargininase [dimethylarginine dimethylaminohydrolase (DDAH); EC 3.5.3
37 sociated with a reduction in the activity of dimethylarginine dimethylaminohydrolase (DDAH, the enzym
38 le of ADMA, and the enzymes metabolizing it, dimethylarginine dimethylaminohydrolases (DDAH) in the r
39 itric oxide synthase [eNOS], Rho-kinase, and dimethylarginine dimethylaminohydrolase [DDAH]) were ana
40                   ADMA can be metabolized by dimethylarginine dimethylaminohydrolases (DDAHs) and by
41                                              Dimethylarginine dimethylaminohydrolases (DDAHs) are kno
42                ADMA levels are controlled by dimethylarginine dimethylaminohydrolases (DDAHs), cytoso
43 ysregulation of the ADMA-metabolizing enzyme dimethylarginine dimethylaminohydrolase I (DDAH I) plays
44 The enzyme dimethylargininase (also known as dimethylarginine dimethylaminohydrolase or DDAH; EC 3.5.
45 ne deiminase (PaAgDI), and N(omega),N(omega)-dimethylarginine dimethylaminohydrolase (PaDDAH) indicat
46  lowering of ADMA by recombinant recombinant dimethylarginine dimethylaminohydrolase (rDDAH)-1 improv
47                       Effects of recombinant dimethylarginine dimethylaminohydrolase (rDDAH)-1 on I/R
48 crease ADMA because they bind to and inhibit dimethylarginine dimethylaminohydrolase, the enzyme that
49  co-workers to operate in the related enzyme dimethylarginine dimethylaminohydrolase, was further exp