戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ne are clearly protected from methylation by dimethylsulfate.
2  triad was not protected from methylation by dimethylsulfate.
3 lation of the respective diamidoxime 5a with dimethylsulfate.
4 melting, circular dichroism spectroscopy and dimethylsulfate alkylation experiments.
5 expression of H19, we have conducted in vivo dimethylsulfate and DNase I footprinting of regions upst
6 ved the presence of maternal-allele-specific dimethylsulfate and DNase I footprints at the promoter i
7 dification of P RNA-substrate complexes with dimethylsulfate and kethoxal was performed to determine
8 lly reactive toward chemical modification by dimethylsulfate and that methylation of this nucleoside
9       DNA-protein complexes were probed with dimethylsulfate (DMS) and N -ethylnitrosourea (EtNU) to
10                                              Dimethylsulfate (DMS) methylation-protection footprintin
11 e adjacent A2451 to become hyper-reactive to dimethylsulfate (DMS) modification in a pH-independent m
12                     By using high-throughput dimethylsulfate (DMS) probing, we identified hundreds of
13 onventional in vivo reactivity of the U with dimethylsulfate (DMS).
14 ed with either ultraviolet (UV) radiation or dimethylsulfate (DMS).
15 r dichroism, nuclear magnetic resonance, and dimethylsulfate footprinting analyses.
16                                      In vivo dimethylsulfate footprinting confirmed that DNA-protein
17 alysis of this 5' flanking region by in vivo dimethylsulfate footprinting in cultured endothelial cel
18                                Concordantly, dimethylsulfate footprinting in vivo revealed methylatio
19                         We performed in vivo dimethylsulfate footprinting of the 220 bp mouse proxima
20 tic analyses of duplex and bubble templates, dimethylsulfate footprinting, and zero-Angstrom crosslin
21 we find that only in nonexpressing cells are dimethylsulfate footprints and UV photofootprints affect
22                                     Based on dimethylsulfate genomic footprinting experiments, there
23 howed that two distal kappa B sites, a novel dimethylsulfate-hypersensitive sequence, and a promoter
24                        Ultraviolet light and dimethylsulfate in vivo genomic footprinting analyses re
25                           The combination of dimethylsulfate, kethoxal, and 1-cyclohexyl-3-(2-morphol
26 e show that classic chemical probes, such as dimethylsulfate, kethoxal, and 1-cyclohexyl-3-(2-morphol
27                                        Using dimethylsulfate/LMPCR, no detectable proteins bind the T
28       In structural probing experiments with dimethylsulfate, m(1)A is routinely detected by RT-arres
29 acid constant) based on the pH dependence of dimethylsulfate modification.
30                    Here, in a combination of dimethylsulfate mutational profiling with sequencing (DM
31 int, we observed a number of strand-specific dimethylsulfate reactivity differences specific to the m
32                                              Dimethylsulfate reactivity of the dG residues indicates
33 ion of a triplex having the 1-C-G triad with dimethylsulfate resulted in a 50% reduction of methylati
34            Ribosome probing experiments with dimethylsulfate revealed that specific base accessibilit
35 er congener is protected from methylation by dimethylsulfate when the oligomer is 3'-phosphorylated.