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1 ncluding superoxide dismutase, catalase, and dimethylthiourea.
2 8, n = 30), DMSO (0.64 +/- 0.03, n = 10), or dimethylthiourea (0.51 +/- 0.08, n = 10) did not alter t
3 ontaining 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea (1), [AuL(1)](n+) (where L is Cl(-), Br
4 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea, 1, en = ethane-1,2-diamine} is the pro
5 rivative, 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea (ACRAMTU) to various self-complementary
6 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea, acridinium cation, 1), the prototype o
7 uccess of 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea as a carrier group in cytotoxic platinu
8                                              Dimethylthiourea but not mannitol inhibited IL-8 inducti
9 ed > 75% by the urea transport inhibitor 1,3-dimethylthiourea, but not inhibited by the water transpo
10 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea)), derived from prototype 1 (with R = e
11 ase and the intracellular hydroxyl scavenger dimethylthiourea (DMTU) abrogated the thalidomide-induce
12 gulated in the presence of the ROS scavenger dimethylthiourea (DMTU) as compared with ABA alone, sugg
13  were treated with the synthetic antioxidant dimethylthiourea (DMTU) before or after the onset of lig
14 me rats were given the synthetic antioxidant dimethylthiourea (DMTU) by intraperitoneal injection and
15                   We tested UT inhibition by dimethylthiourea (DMTU) in 5/6 nephrectomy mice.
16 me rats were given the synthetic antioxidant dimethylthiourea (DMTU).
17 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea, en = ethylenediamine), a bifunctional
18 ers superoxide dismutase, catalase, DMSO, or dimethylthiourea for 12 min at 37 degrees C, and P(album
19 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea] have been investigated using restricti
20 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea) is a cytotoxic platinum-acridine conju
21 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea) is a dual metalating/intercalating DNA
22 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea), is a dual platinating/intercalating D
23  Neither superoxide dismutase, catalase, nor dimethylthiourea nor depletion of the media of O2 to pre
24                 Treatment with ROS scavenger dimethylthiourea or antioxidant N-acetylcysteine did not
25 ith the antioxidants, N-acetyl-L-cysteine or dimethylthiourea, prior to sensitization, challenge, or
26                    The H2O2 scavenger N,N(1)-dimethylthiourea reversed the MeJA-induced early protoxy
27 ype, this event being counteracted by N,N(1)-dimethylthiourea treatment.
28 en exclusively during sensitization, whereas dimethylthiourea was inhibitory even when administered w
29 ACRAMTU = 1-[2-(acridin-9-ylamino)ethyl]-1,3-dimethylthiourea) with adenine in DNA have been studied