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1 e-nucleotide polymorphisms while maintaining diploidy.
2 samples is challenging because of chromosome diploidy.
3  followed by massive gene loss that restored diploidy.
4 n is not followed by nuclear fusion and true diploidy.
5 n' (CSD), uses heterozygosity as a proxy for diploidy.
6 l hypotheses on the functional importance of diploidy.
7 ex; disomies X, Y, or 21; or meiosis I or II diploidies.
8 inomas begin as tetraploid then descend into diploidy accompanied by genome-wide LOH.
9 ely 5-fold increases in sperm with disomies, diploidies and complex genotypes involving chromosome X,
10                                        Thus, diploidy and alternative splicing each increased toleran
11  an insect with social behavior and/or haplo-diploidy and are an indication of the unique nature of t
12  males in an age-structured population under diploidy and autosomal inheritance, the total reproducti
13 tion, including evidence of urothelial hyper-diploidy and cell cycle skewing in wild-type but not Il4
14 olutionary forces associated with vegetative diploidy and provide a foundation for the use of Auxenoc
15 ion following the meiotic divisions restores diploidy and thereby mimics fertilization.
16 es of Bremia lactucae were inconsistent with diploidy and therefore likely heterokaryotic.
17 XY, disomy X, disomy Y, disomy 21, and sperm diploidy, and (b) examine the association between the fr
18 e, imprinted genes discard the advantages of diploidy, and for this reason the rationale for the evol
19 h as inbreeding depression, the evolution of diploidy, and levels of natural genetic variation.
20  positively correlated with low tumor grade, diploidy, and low S-phase fraction, all biological param
21 ercondensation, chromosome breakage, loss of diploidy, and premature sister chromatid separation.
22  crops due to its short seed-to-fruit cycle, diploidy, and sequenced genome.
23    Given that high-frequency MSI (MSI-H) and diploidy are correlated, we determined whether they are
24 ich the bacterium Bacillus subtilis enforces diploidy as it differentiates into a dormant spore.
25 S after adjustment for MYCN amplification or diploidy but had no significant effect on OS.
26  a model in which induction of SirA enforces diploidy by inhibiting replication initiation as B. subt
27 moting S phase and M phase, while preserving diploidy by suppressing endoreduplication.
28 ase in proliferating cells, while preserving diploidy by suppressing endoreduplication.
29 [DI] > 1) was clearly superior to those with diploidy (DI < or = 1): younger than 12 months, 83.7% +/
30 oid potato (Solanum tuberosum), reduction to diploidy (dihaploidy) allows for hybridization to diploi
31  sperm with immature chromatin, aneuploidies/diploidies, FGFR2 mutations (Apert syndrome), or sex rat
32 es, and no general evolutionary advantage of diploidy has been demonstrated.
33                         Cells deviating from diploidy have the ability to communicate with their micr
34 essary to promote proliferation and maintain diploidy in breast cancer cells.
35     It also demonstrates that the effects of diploidy in gene networks can have counter-intuitive con
36 to evolve: selection is more likely to favor diploidy in host species and haploidy in parasite specie
37                                              Diploidy is a fundamental genetic feature in mammals, in
38 ertilisation, when two haploid gametes fuse, diploidy is restored in the zygote.
39    By doubling the copy number of each gene, diploidy may increase the rate at which adaptive mutatio
40 nostic biologic findings included tumor-cell diploidy (n = 2) and unfavorable Shimada histopathology
41                        This is the result of diploidy, not mating type regulation.
42 or arbitrary modes of inheritance, including diploidy, polyploidy, sex linkage, cytoplasmic inheritan
43 patocytes proliferate to regenerate it, with diploidy providing a growth advantage over polyploidy.
44                                As predicted, diploidy slowed adaptation by large populations but not
45 nary timescales, inhibiting the reversion to diploidy that is typically seen in laboratory evolution
46    Little is known about the transition from diploidy to polyploidy but in some species, triploids ar
47 are predominantly diploid, the prominence of diploidy varies greatly among eukaryote life cycles, and
48                                              Diploidy was associated with better survival in MSI-H an
49 able (MSS)/low-frequency MSI (MSI-L) tumors, diploidy was associated with better survival.
50       Thus, for most genes the resolution to diploidy was lineage-specific.
51 lear genome was destroyed by irradiation and diploidy was restored by blocking the first embryonic cl
52 usion of two meiotic products to restore egg diploidy), whereas workers of other honeybee subspecies
53 crepancies occur mainly when BACDAC predicts diploidy with subclones rather than high-ploidy.