1 identify areas that remain understudied, and
discuss how a better understanding of these processes ca
2 We then
discuss how a spatially segregated state of the genome c
3 Within this paradigm, we
discuss how a turbulently mixed state depends on the flo
4 This Commentary
discusses how a parallel effort to address fungal pathog
5 In this review, we
discuss how active research in molecular deep learning c
6 We
discuss how advancing technologies have refined lineage-
7 We
discuss how ageing affects the development of myeloid ce
8 is to use examples from recent literature to
discuss how AI and ML are changing clinical practice and
9 r, morphological and environmental data, and
discuss how an ecosystems biology approach has expanded
10 We also
discuss how an imbalance in vascular activation by leuko
11 Finally, we
discuss how an understanding of the epidemiology, biolog
12 Here, we
discuss how application of this information in routine c
13 We also
discuss how bacteria achieve robust cell wall growth und
14 In this review we
discuss how bacteria are able to sense surfaces and how
15 s of the organismal proteostasis network and
discuss how both are linked in cancer.
16 We
discuss how both the permittivity and permeability need
17 tific evidence supporting this approach, and
discuss how both types of processing can lead to fitness
18 We also
discuss how,
by applying these powerful approaches, stud
19 In this review, we
discuss how calcium signaling pathways in endothelial ce
20 tial link between genotype and phenotype and
discuss how cells and biomimetic compartments can be app
21 In addition, we
discuss how central nervous system (CNS) degeneration mi
22 Here, we
discuss how centrosome defects promote invasive behavior
23 This review
discusses how changes in endothelial N-glycosylation may
24 In this commentary, we
discuss how choice of conference location-choosing less
25 We briefly
discuss how circulating factors either directly or indir
26 We
discuss how CIROAS may enhance abilities for molecular s
27 The authors
discuss how clinicians have a clear role to play in coun
28 Here, we
discuss how cobamides affect microbes at each of these t
29 We
discuss how combining human and machine intelligence can
30 We also
discuss how complex interactions between cancer cells an
31 We
discuss how coordinated evolution and trade-offs among f
32 In this review, we
discuss how CRISPR-KO/CRISPRa screening has contributed
33 We
discuss how CTSs help to inform target product profiles.
34 in genome-wide DNA methylation analysis and
discuss how cyst nematodes induce extensive and dynamic
35 Here we
discuss how cytoprotective and cytotoxic signaling modul
36 e different methodologies used, this article
discusses how data from anatomic studies can help inform
37 and challenges that our community faces, and
discuss how developmental biologists can make unique and
38 Here we
discuss how developmental psychology affords a unique an
39 erspective reviews this current evidence and
discusses how differences in drug response could inform
40 We
discuss how different lifestyles and environmental facto
41 We also
discuss how different therapies, including checkpoint in
42 eview, focusing mostly on metazoan cells, we
discuss how different types of local and global shape ch
43 n native and exotic species of Osmia, and we
discuss how disease spillover and enemy release in this
44 We further
discuss how distinct appetite and satiation systems for
45 We then
discuss how DNMTs are recruited to or excluded from cert
46 In addition, we
discuss how dysregulation of this system through genetic
47 We
discuss how each genetic screening platform can provide
48 We
discuss how early and intermediate endpoints, including
49 exotic, as they are sometimes portrayed, we
discuss how effects of language on perception naturally
50 from their sensory properties, we will also
discuss how electrochemical techniques can be used to st
51 c behaviors such as respiratory control, and
discuss how electrophysiological tools combined with new
52 g, including these alternative pathways, and
discusses how ethylene signaling has been manipulated fo
53 In this inaugural Voices essay, we
discuss how exclusion and inclusion events have contribu
54 We
discuss how existing methods in the field of movement ec
55 d to identify and characterize cells, and we
discuss how experimental and pathological perturbations
56 In addition, we
discuss how exposure to variability may influence specie
57 We briefly
discuss how failures in DNA repair produce a permissive
58 We
discuss how features of chromosome axis/loop architectur
59 In addition, we
discuss how following the wrong breadcrumb trail of tran
60 Here, we
discuss how food may alter the microbiota as well as muc
61 We
discuss how functional diversification of pleiotropic ge
62 This review
discusses how GDF15 regulates appetite and metabolism, t
63 We
discuss how gene regulation analyses have advanced our u
64 We
discuss how genetic variants and transcriptomic profiles
65 In this Review, we
discuss how genomics approaches have advanced our unders
66 Here, we
discuss how glial cell adhesion molecules and the extrac
67 Here, we
discuss how glycosylation contributes to extracellular r
68 In this Review, we
discuss how host epigenetic and epitranscriptomic proces
69 Then, we
discuss how HRL can advance our understanding of regulat
70 In addition, we will
discuss how humanized mouse models such as the human ski
71 cross-linkers, and functional additives, and
discuss how hydrogels can be employed as precursors and
72 Here, we
discuss how imbalanced interferon responses may contribu
73 We assess the plausibility of our method and
discuss how improved data and models can generate better
74 We
discuss how inactivation of IFNAR1 in CAFs acts to stimu
75 Here we
discuss how inclusive speaker selection fosters better s
76 We also
discuss how increased ROS generation by HIF-1 contribute
77 Specifically, we
discuss how individual miRNAs function in the regulation
78 We further
discuss how innate immune responses can be players of th
79 the loss of sensitivity due to dilution and
discuss how it may be mitigated by the frequent re-testi
80 escribe the molecular structure of TRPV6 and
discuss how its structural elements define its unique fu
81 Finally, we
discuss how knowledge of the p53 pro-survival and pro-de
82 umerating abortions in the United States and
discuss how lack of access to high-quality data limits o
83 This Review
discusses how language builds meaning and lays out direc
84 l studies to potential cellular examples and
discuss how layered topologies and self-regulation may i
85 We
discuss how lipid ligands on OMVs engage these pattern r
86 Specifically, we
discuss how long noncoding RNAs (lncRNAs) and three-dime
87 In this Review, we
discuss how machine learning can aid early diagnosis and
88 We
discuss how machine learning-derived elements complement
89 In this review we
discuss how MAIT cells display antimicrobial, homeostati
90 We then
discuss how mechanical cues are converted to biochemical
91 We
discuss how microbes and their products contribute to li
92 We further
discuss how mimicking the bulge immune privilege may be
93 We also
discuss how misregulation of the RAD51 gene family membe
94 We then
discuss how motor expertise results from establishing ta
95 This Review
discusses how mucosal IgA responses occur in an increasi
96 Here, we
discuss how nanomedicine-based treatment strategies are
97 recent evidence linking NCL genes to AD, and
discuss how NCL, AD, and PD converge on a shared molecul
98 We will also
discuss how new knowledge about secondary consequences o
99 We
discuss how new pharmacological, behavioural, and electr
100 We
discuss how NF-kappaB activation and control of cell dea
101 In this review, we
discuss how nitrogen is utilized in biosynthetic pathway
102 In this review, we
discuss how NKDs that affect NK cell cytotoxicity can be
103 We
discuss how novel genomic resources can identify candida
104 We
discuss how nucleolar function is linked to its organiza
105 In this review, we
discuss how OCM impacts the translation apparatus (compo
106 We also
discuss how oncogenic alterations in lymphoma cells may
107 In this Primer, we
discuss how one of the most versatile bioorthogonal reac
108 kept pace with the growth of UniProtKB, and
discuss how our evaluation of residue coverage may help
109 We
discuss how our findings may lead to technological and f
110 Here, we
discuss how our understanding of anthelmintic resistance
111 Here, I revisit these studies and
discuss how our understanding of the hippocampus has evo
112 We
discuss how PDNET can be used to predict contacts, dista
113 conceptualise multi-attribute choice and we
discuss how phenomena such as innate preferences, flower
114 Here, we
discuss how physical activity can prevent or ameliorate
115 We also
discuss how policies and social ties can promote their h
116 Here, we
discuss how post-transcriptional mechanisms enable stem
117 We also
discuss how previous studies may direct current lines of
118 We
discuss how probiotics, prebiotics, synbiotic, fecal mic
119 em, explain the critical role of theory, and
discuss how psychologists can add historical depth and n
120 In this review, we
discuss how PTMs, including phosphorylation, S-acylation
121 We then
discuss how quantitative descriptions of behavior can be
122 We also
discuss how recent advances in our understanding of neur
123 and localised delivery to brain tumours and
discuss how recent engineering advances may be used to e
124 In this Review, we
discuss how recent microbiological, structural, genetic
125 In this Review, we
discuss how recent mRNA profiles of mouse and human inte
126 Furthermore, we
discuss how recent studies have begun to unravel how the
127 Specifically, we
discuss how red blood cells (RBCs), platelets, neutrophi
128 It will also
discuss how regulating N-glycoforms on the endothelial s
129 In this Review, we
discuss how replication forks are actively stalled, remo
130 Reflecting on this transitional state, we
discuss how researchers will struggle to replicate the e
131 In this article, we
discuss how results from different systems shed light on
132 In addition, we
discuss how rising atmospheric CO(2) concentration has c
133 We
discuss how sensory neurons utilize TLRs and other PRR p
134 e observed sex differences in depression, we
discuss how sex might alter brain function through hormo
135 We highlight and
discuss how single-cell technologies, human-induced plur
136 We also
discuss how single-molecule analysis of engineered motor
137 nding to these categories beyond infancy and
discuss how social biases favoring own-race and female f
138 Ultimately, we
discuss how some highly conserved DNA MTases may emerge
139 We also
discuss how some observations, such as near-instantaneou
140 resulting from oxidative processes, and also
discusses how some oxidative lesions are probably direct
141 We
discuss how specific developmental mechanisms, which red
142 We review the existing knowledge and
discuss how state-of-the-art immune monitoring strategie
143 This month's Under the Lens
discusses how structural studies of the SARS-CoV-2 spike
144 Here, we
discuss how studies using genetic and biochemical manipu
145 We
discuss how such a data-driven approach might be used ge
146 We
discuss how such an interplay between appetitive and ave
147 We further
discuss how such DNA nanostructures can be rationally de
148 We
discuss how such molecular dynamics influences synaptic
149 We
discuss how T. gondii infection suppresses noradrenergic
150 ion, propagation, and therapy resistance and
discuss how targeting these pathways may be of therapeut
151 d archaeology on human tooth development and
discuss how teeth preserve a time-resolved record of our
152 Here we
discuss how telomere dysfunction can initiate genomic co
153 r the stabilization of a skyrmion phase, and
discuss how the absence of these transitions in the y =
154 In this review, we
discuss how the acidity of the tumor microenvironment in
155 We also
discuss how the cerebral circulation of giraffes is esta
156 Finally, we
discuss how the combination of multiple immune parameter
157 We first
discuss how the conceptualization of health equity and h
158 Also, we
discuss how the current situation offers a unique window
159 We
discuss how the deficits we observed in patients with fu
160 We
discuss how the development of some symptoms in allergic
161 We also
discuss how the diversity of T(RM) communities within sp
162 We
discuss how the early maturation of the caudal entorhina
163 In addition, we
discuss how the ECM helps to regulate tissue morphology
164 Here, we
discuss how the field can work together to further expan
165 The editorialists
discuss how the findings add to what we know about mask
166 We also
discuss how the framework can help government agencies t
167 I
discuss how the framework goes beyond prior work, and ho
168 ing and shaping the microbiota community and
discuss how the functional axes interconnecting gut micr
169 Here, we
discuss how the functional characteristics of the liver
170 Finally, we
discuss how the GLN method can be applied to study entan
171 We also
discuss how the Hsp90 modification state affects cellula
172 We
discuss how the increasing knowledge regarding the selec
173 Finally, we
discuss how the integration of computational psychiatry
174 Here, we
discuss how the integration of proteomics and activity-b
175 Finally, we
discuss how the knowledge gained from past domestication
176 We
discuss how the length scales of molecular gradients dic
177 In this review we
discuss how the metabolic changes that occur within the
178 In this opinion article, I
discuss how the nature of such repositories can subject
179 We
discuss how the neural mechanisms underlying STM are ine
180 We also
discuss how the neuropeptide oxytocin impacts these circ
181 We
discuss how the new, site-based studies complement the t
182 se next-generation physiology approaches and
discuss how the non-destructive analysis of cellular phe
183 More generally, we
discuss how the observed phenomenon of transient exposur
184 In this review, we
discuss how the Organ-on-a-Chip technology can have crit
185 We also
discuss how the outcomes associated with different timin
186 We also
discuss how the pathogenesis of these viruses influences
187 We also
discuss how the PROMISE framework reshapes dialogue arou
188 In this review, we
discuss how the recently defined functions of these AD r
189 We then
discuss how the regulation of intracellular Ca(2+) in tu
190 rinciples behind the selective autophagy and
discuss how the SARs may be involved in the pathogenesis
191 We
discuss how the single nucleotide polymorphism-based est
192 We
discuss how the synthesis and function of components of
193 We
discuss how the temporal niche occupied by each species
194 ize is affected by the ubiquitin system, and
discuss how the ubiquitin system affects proteins involv
195 nd psychostimulant drug exposure; we further
discuss how the understanding of these pathways may lead
196 We
discuss how the unique biology of different TEs influenc
197 structures and selected calculations, it is
discussed how the different interpretations of the equil
198 This critical review
discusses how the need for reduced clinical turnaround t
199 The review
discusses how the study of unfairness and radicalization
200 ovement-related signals in sensory areas and
discuss how their study, in the context of natural freel
201 Here, we
discuss how these advances are reshaping the fundamental
202 We
discuss how these approaches are advancing our mechanist
203 In addition, we
discuss how these assays should be used in high-throughp
204 Here, we
discuss how these basic questions have surprisingly rema
205 We
discuss how these c-di-AMP molecules are bound to the pr
206 in the current state of the science, and we
discuss how these can be overcome to maximize the potent
207 e the relevant light-matter interactions and
discuss how these can be utilized to classify nanomotors
208 advances in deep reinforcement learning and
discuss how these can inform the implementation of learn
209 We
discuss how these cells may be used to predict patient o
210 We
discuss how these communication networks break down duri
211 ime inconsistency and social normalization),
discuss how these concepts have been addressed in other
212 in neuronal plasticity in adult life and we
discuss how these contribute to structural and functiona
213 nce structures as two major barriers, and we
discuss how these could be overcome.
214 g larger genome-wide association studies, we
discuss how these could generate new lines of enquiry in
215 We also
discuss how these diabolic domains may be subject to "su
216 We
discuss how these differing patterns relate to their inv
217 We also
discuss how these discoveries shape our understanding of
218 lation can be modulated or regulated, and we
discuss how these effects are utilised by the cell to op
219 using both online and offline processes, and
discuss how these effects of emotion may contribute to m
220 We
discuss how these enhanced rainfall maps can be used to
221 We
discuss how these errors have affected downstream analys
222 In this review, we will
discuss how these experimental models are carving a way
223 rsity as a driver of anti-tumor immunity and
discuss how these factors can be probed using genomic ap
224 We
discuss how these findings can inform the interpretation
225 Here we
discuss how these findings may be combined to improve th
226 In this Review, we
discuss how these genomes, together with long-term enric
227 Here, we
discuss how these guardian pathways function, how their
228 We
discuss how these heterointeractions can contribute to t
229 nal makeup of stem and progenitor niches and
discuss how these high-resolution analyses fundamentally
230 tic methodologies used in these studies, and
discuss how these insights have aided our general unders
231 neuroinflammation and neurodegeneration and
discuss how these interactions shape pathological outcom
232 or the ligands that have been identified and
discuss how these ligands may activate AGPCRs in physiol
233 the different types of linkers published and
discuss how these linkers are designed to obtain the lea
234 We
discuss how these novel findings impact our understandin
235 We further
discuss how these properties contribute to intestinal de
236 In this paper, we
discuss how these questions can be approached and examin
237 emphasis on the role of C-type lectins, and
discuss how these receptors modulate these responses at
238 on by multi-tiered regulatory mechanisms and
discuss how these regulations couple metabolic inputs wi
239 apies beyond anti-CTLA-4 and anti-PD(L)1 and
discuss how these results are providing new insights int
240 We
discuss how these results can be reconciled with the pre
241 We further
discuss how these signaling properties might potentially
242 We next
discuss how these studies can advance human health in th
243 We
discuss how these studies have illustrated the roles of
244 ural and molecular understanding of ApoE and
discuss how these studies on ApoE have illuminated the p
245 approximately 100 nm-lateral resolution and
discusses how these analyses have already begun to trans
246 The editorialist
discusses how these findings help to further inform our
247 sms by which Tregs regulate angiogenesis and
discusses how these mechanisms differ in homeostatic and
248 We conclude by
discussing how these design characteristics might affect
249 of 15 other national immunology societies to
discuss how they and their members responded following t
250 ome of these transformative technologies and
discuss how they have been used to identify new modes of
251 related potential abnormalities in CHR-P and
discuss how they inform our understanding of information
252 ental predictability and animal movement and
discuss how they may react to anthropogenic disruption,
253 y common classes of alleles used in mice and
discuss how they might be engineered using different met
254 ces in the communications between clocks and
discuss how they relate to circadian physiology and meta
255 major points covered during this debate and
discuss how they tie into current thinking in the field
256 xploitation of MEG and EEG data, and we also
discuss how this 'living' set of guidelines will evolve
257 Here, we
discuss how this apparent conflict can be resolved by di
258 ure of morphogen gradient interpretation and
discuss how this can impact on cell-fate decisions.
259 We
discuss how this categorization may impact the choice of
260 We
discuss how this circuitry might be modulated by current
261 In this perspective, we
discuss how this conceptual transformation could potenti
262 n exhibited a worse clinical outcome, and we
discuss how this could, at least partially, be related t
263 life and death decision-making process, and
discuss how this decision is relevant to immune system r
264 We
discuss how this finding challenges and constrains theor
265 pdate to the work of the IMEx Consortium, we
discuss how this initiative has been working in practice
266 Finally, we
discuss how this knowledge informs therapeutic approache
267 We
discuss how this lateral flow test device can be an effe
268 organization of the lymphocyte membrane and
discuss how this new information influences our view of
269 Here we
discuss how this pathway contributes to the continuous s
270 resistance against antitumor immunity and we
discuss how this resistance could directly influence imm
271 Furthermore, we
discuss how this signalling network can affect therapeut
272 between human and mice immune dysbiosis and
discussed how this affects the host response to similar
273 We also
discuss how those interactions shape the assembly of pla
274 In this Review, we
discuss how tissue-clearing methods could provide an unb
275 In this Perspective, we first
discuss how to achieve atomically precise NCs and determ
276 We also
discuss how to achieve the atmospheric relevance for dif
277 alth and Social Care) convened 2 meetings to
discuss how to address research evidence gaps in order t
278 Here we
discuss how to balance clarity and thoroughness when rep
279 Here, we
discuss how to bridge transcriptional states to specific
280 Further, we
discuss how to configure an ERG system without a patch-c
281 We
discuss how to deal with sources of unwanted variation,
282 d their interactions with nanoparticles, and
discuss how to design and develop optimal HA-nanoparticl
283 rize the whole process of the experiment and
discuss how to expand our experiment in the future.
284 Finally, we
discuss how to interpret an NBDA model's output and prov
285 assay to determine pore-membrane binding and
discuss how to use single-channel current recordings and
286 Herein, it is
discussed how to navigate the multiple-choice adventure
287 This opinion article
discusses how to anticipate and detect those editing eve
288 This commentary
discusses how to balance a delay in cancer diagnosis or
289 see it with a positive spin on it." She also
discusses how to found fields (social cognition, health
290 Here, we
discuss how Toxoplasma strain type and parasite effector
291 ression at the three translational steps and
discuss how translation is used to detect and respond to
292 Finally, we
discuss how uHTS can inspire novel functional metagenomi
293 In this review, we will
discuss how various spatiotemporal scenarios of antigen
294 Here, we
discuss how varying methodologies for the experimental d
295 We
discuss how visual circuit development leads to precise
296 We
discuss how we have reached our current view of nitrate
297 We further
discuss how we met logistical challenges associated with
298 We
discuss how we now have enough scaffold for conceptual b
299 current understanding of early-onset CRC and
discuss how we should strategize future research to impr
300 t apply to both cancer and wound healing and
discuss how wounding, as in biopsy and surgery, might po