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1 d-Turcotte-Pugh or Model for End-Stage Liver Disease scores).
2 rdioembolic Event, (female) Sex, or Vascular Disease] score).
3 s had similar age, model for end-stage liver disease score.
4 of transplant, and model for end-stage liver disease score.
5 g age, gender, and Model for End-Stage Liver Disease score.
6 o normal or gld recipients gave a diminished disease score.
7 lity together with Model for End-Stage Liver Disease score.
8 ensation, and high Model for End-Stage Liver Disease score.
9 egardless of their Model for End-Stage Liver Disease score.
10 , adjusted for age, sex, and CT small vessel disease score.
11 rb2 in vivo eliminates bacteria and improves disease score.
12 ents with relapsing-remitting MS with a high disease score.
13 y associated with the total MRI small vessel disease score.
14 s who had the same Model for End-Stage Liver Disease score.
15 ory cytokines, and model for end-stage-liver disease scores.
16 malized ratio, and Model for End-Stage Liver Disease scores.
17 ogenic immunization reproducibly ameliorated disease scores.
18 ration of clinical signs, and increased peak disease scores.
19 ents of mutant bone marrow, developed normal disease scores.
20 -Turcotte-Pugh and Model for End-stage Liver Disease scores.
21 tients with higher model for end-stage liver disease scores.
22 alth independent of improvements in physical disease scores.
23 three expression modules correlated with GLS disease scores.
24 oth Child-Pugh and Model for End-Stage Liver Disease scores.
25 l disease stage and model of end-stage liver disease scoring.
26 tium to Establish a Registry for Alzheimer's Disease score, 1.2 [0.5] vs 1.4 [0.8], respectively; P =
30 tage Liver Disease/Pediatric End-Stage Liver Disease score (19 vs 11, respectively; P = .48), and don
34 ing DC had greater Model for End-stage Liver Disease scores (33 vs 27; P < .001); more frequent pretr
36 e-Pugh score 7 and Model for End-Stage Liver Disease score 6-29) were enrolled, 26 had hepatocellular
37 Pugh score >=7 and Model for End-Stage Liver Disease score 6-29) were enrolled, 26 had hepatocellular
38 cirrhosis (median Model for End-Stage Liver Disease score, 9; interquartile range, 7-13), and their
39 in group A and had a higher mean concomitant disease score, a higher proportion of men and a greater
41 neurologic deficits as indicated by clinical disease score, activity monitoring, and footprint analys
42 lementation of the model for end-stage liver disease score and a concomitant increase on the rate of
44 d increased median model for end-stage liver disease score and creatinine at the time of waitlisting.
45 tion was evidenced primarily by increases in disease score and degree of inflammation in the CNS.
48 T to LT, and lower model for end-stage liver disease score and maximum tumor diameter (C-statistic 0.
51 tium to Establish a Registry for Alzheimer's Disease scores and Braak and Braak stage), and hippocamp
52 er two strains, based on significantly lower disease scores and observably smaller and fewer lesions
53 ciated with higher model for end-stage liver disease scores and receiving a LT from a living donor.
54 luding with Porphyromonas that correlated to disease scores and salivary levels of interleukin (IL)-1
55 istologic sections were used to evaluate EAU disease scores and to assess outer blood retina barrier
57 c vascular disease and cerebral small vessel disease scores and, in the multiple sclerosis group, the
58 tis C virus (HCV), model for end-stage liver disease score, and acute rejection; and donor age and ra
60 by a higher median Model for End-Stage Liver Disease score, and associated with increased 90-day mort
61 s, calibrating the pediatric end-stage liver disease score, and deciding whether to accept an offered
62 x, race/ethnicity, Model for End-Stage Liver Disease score, and liver transplantation (adjusted hazar
63 h of stay, greater Model for End-stage Liver Disease score, and longer warm and cold ischemia times (
64 omorbidity burden, Model for End-Stage Liver Disease score, and serum level of albumin that predicts
65 younger age, lower Model for End-stage Liver Disease score, and shorter time from listing to first do
68 e weighting of the model for end-stage liver disease score, and the increased prevalence of nonalcoho
69 admissions during the previous year, chronic disease score, and use of non-statin lipid-lowering drug
70 age, coinfection, Model for End-Stage Liver Disease scores, and other potential confounders in a pro
71 ase-expressing virus), significantly reduced disease scores, and resulted in greater survival (P < 0.
72 icant correlations between interstitial lung disease score at CT and age or percentage predicted FVC,
73 igher median (IQR) Model for End-stage Liver Disease score at listing (35.0 [29.0-39.0] vs 20.0 [13.0
74 3 cm (P=0.02) and model for end-stage liver disease score at listing more than 11 (P=0.04) were inde
75 Disease/Pediatric Model for End-Stage Liver Disease score at LT significantly decreased across all a
76 ansplanted, median model for end-stage liver disease score at LT was 7 ((interquartile range [IQR]: 6
77 0.041), and lower Model for End-Stage Liver Disease score at the time of LT (23 vs. 29, P = 0.007).
78 , body weight, and pediatric end-stage liver disease score at the time of transplant were 13 months,
79 cantly higher mean Model for End-Stage Liver Disease score at time of waitlist registration than othe
80 owever, the median model for end-stage liver disease score at transplant (MMAT) is calculated based o
81 ased on the median model for end-stage liver disease score at transplant minus 3 (MMAT-3) of recipien
83 ee groups based on Model for End-Stage Liver Disease score at transplant: lower-score (regions 3, 10,
84 nger age and lower Model for End-Stage Liver Disease scores at listing were associated with an increa
85 rk so that patient model for end-stage liver disease scores at transplant is more uniform across regi
87 classical exposure measurement error using a disease score-based approach to standardization to the e
89 impairment and high model of end-stage liver disease scores before liver transplantation (LT) are inc
90 , the dichotomized model for end-stage liver disease score below and above the median, and the presen
91 ied recipient age, Model for End-Stage Liver Disease score, body mass index, diabetes, and dialysis b
93 tium to Establish a Registry for Alzheimer's Disease score, Braak stage score and clinical dementia r
94 ammonia levels or model for end-stage-liver disease scores, but patients in the rifaximin group had
95 on Comorbidity Index (c = 0.653) and Chronic Disease Score (c = 0.608) were similar discriminators of
97 nd mathematical models of prognosis in liver disease scores calculated at day 0, day 3, and day 7.
100 adjusting for age, model for end-stage liver disease score, cardiovascular risk index, history of cor
101 Stratified by model for end-stage liver disease score categories 6 to 14, 15 to 24, 25 to 34, an
102 45, c = 0.745) than medication-based Chronic Disease Score (CDS)-1 and CDS-2 (c = 0.738, c = 0.718).
104 EDI, size of HCC, model for end-stage liver disease score, Child-Pugh score were not statistically s
105 g for age, gender, Model for End-Stage Liver Disease score, Child-Pugh score, serum sodium, previous
106 disease including Model for End-Stage Liver Disease score compared to middle aged and older adults (
108 se activity, with the IRAMS showing enhanced disease scoring compared to standard EAE scoring methods
109 refore, the effect of the routine use of the disease score could result in fewer patients with severe
112 ntation at a lower model for end-stage liver disease score, decreased death on waitlist, and excellen
113 efficacy was evidenced by a reduced clinical disease score, decreased expression of various inflammat
114 e immunization in such mice produced similar disease scores, demonstrating that Fas/FasL interactions
115 , body mass index, model for end-stage liver disease score, diabetes, functional status at transplant
118 onist treatment also resulted in exacerbated disease scores, elevated proinflammatory mediators, and
119 tium to establish a registry for Alzheimer's disease score for neuritic plaques between, but Lewy bod
120 tium to Establish a Registry for Alzheimer's Disease score for neuritic plaques, p = 6.8 x 10-6) and
121 2 points on their Model for End Stage Liver Disease score for signing a donor card, 0.1 points for a
122 ys (OR 10.23), and Model for End-Stage Liver Disease score greater than 21 (OR 2.5) were significant.
123 ter than 55 years, Model for End-Stage Liver Disease score greater than 27, history of prior OLT grea
124 on of CRE post-LT, Model for End-Stage Liver Disease score greater than 32, combined transplantation,
125 coholic hepatitis (Model for End-Stage Liver Disease score > or = 15) were enrolled and randomized to
126 s mice showing the severe disease phenotype (disease score > or = 3)and 68 backcross mice of the resi
129 gen >27] + 0.4574 [Model for End-Stage Liver Disease score >21] + 1.1625 [intensive care unit days >3
131 et drug use (nonaspirin), albumin <3.0 g/dL, disease score >=2 (according to the Charlson Comorbidity
132 0.03-0.99), and a model for end-stage liver disease score >=25 (HR, 0.26; 95% CI, 0.08-0.92).
134 creatinine in the model for end-stage liver disease score has significantly increased the incidence
135 in recipients with model of end-stage liver disease score higher than 27 (13.2% vs. 23.0%, P < 0.001
136 nd inflammation were quantified by gross gut disease scoring, histologic scoring, type I collagen, an
137 01] and laboratory Model for End-stage Liver Disease score (HR = 1.01; 95% CI, 1.00-1.01) as independ
139 is also identified Model for End-Stage Liver Disease score, hypovolemic shock, and bacterial infectio
140 oid insufficiency, Model for End-Stage Liver Disease score, hypovolemic shock, hepatocellular carcino
141 -Turcotte-Pugh and model for end-stage liver disease scores improved significantly from baseline to 6
142 trials by addressing issues of standardizing disease scoring, improving the sensitivity and precision
143 integrated with the model of end-stage liver disease score in a donor-recipient (D-R) matching system
147 ticeable for lower model for end-stage liver disease scores in LD recipients (P<0.001) and younger do
148 dy to CLEC12A that significantly ameliorated disease scores in MOG35-55-induced progressive, as well
150 0; however, median Model for End-Stage Liver Disease scores in this BMI group were higher than those
151 e Charlson Comorbidity Index and the Chronic Disease Score, in assessing the comorbidity-attributable
153 (age of recipient, model for end-stage liver disease score, indication for LT, platelet count, and re
156 otics, exhibiting improved survival, reduced disease score, lower levels of serum cytokines, and impr
160 recipients with low model of end-stage liver disease scores (<27), without hepatitis C, not hospitali
162 8%), and had lower model for end-stage liver disease scores (median 9 versus 10) (all P < 0.05).
163 ege Criteria (KCC), Model of End Stage Liver Disease score (MELD), and serum sodium based modificatio
165 l [CI], 2.1-13.8), Model for End-Stage Liver Disease score (odds ratio, 1.11; 95% CI, 1.04-1.17), and
166 D angiographic score of 0), mild-to-moderate disease (score of 1 to 3), and severe disease (score of
167 derate disease (score of 1 to 3), and severe disease (score of 4 to 6) had median F11R plasma levels
168 ntation included a Model for End-stage Liver Disease score of <=25, specific anatomical characteristi
174 ated with having a model for end-stage liver disease score of 20 or greater (hazards ratio, 2.90; 95%
179 ugh a good correlation was found between the disease score of individual mice and some readout parame
181 acute rise in the Model for End-Stage Liver Disease score of more than 5 within 4 weeks before trans
183 ovided by studies of C3 knockout mice, where disease scores of gC-null virus were significantly highe
184 obal rating of the change in the severity of disease, scored on a scale of -3.0 to 3.0 at one year, w
185 sis and with worse Model for End-Stage Liver Disease score or diabetes, those taking prophylactic ant
186 post-ECP were compared with changes in skin disease scores or global organ involvement, or the abili
188 (P < 0.001), worse Model for End-Stage Liver Disease score (P < 0.001), more portosystemic collateral
190 gistic regression model showed that only the disease score (P <0.0005) was significantly associated w
193 : Child-Pugh class, model of end-stage liver disease score, pre- and post-DIPS PSGs, pre-DIPS liver f
194 tium to Establish a Registry for Alzheimer's Disease scores, presence of alpha-synuclein and TAR DNA-
195 y lower laboratory model for end-stage liver disease scores, pretransplant alpha fetoprotein, and cum
199 ificantly with the Model for End-Stage Liver Disease score (r = -0.39, P < 0.05), fasting venous ammo
200 lure as assessed by model for endstage liver disease score (r = 0.41, P = 0.006) and Maddrey's discri
201 ally and summed to give a total small vessel disease score (range 1-4) in each cohort separately, the
202 ces (a measure of day of onset and sustained disease scores) ranging from 367 to 663 with central ner
205 PN-dependent, the Pediatric End-Stage Liver Disease score remained normal throughout the follow-up p
206 considered if the Model for End-Stage Liver Disease score remains greater than 17 after 3 months of
207 outcomes, as measured by ordinal severity of disease scores, requiring longer time to improvement (ad
208 ors associated with disease persistence were disease score (SCORAD), involvement of the limbs, flexur
209 ice on average had lower cumulative clinical disease scores, shorter duration of clinical signs, and
210 or equal to 32 or Model for End-Stage Liver Disease score (starts at 6 and capped at 40; worst = 40)
211 in recipient age, model for end-stage liver disease score, steatosis, and ischemia times for the pea
212 cted C57BL/6 mice with this drug reduced the disease score substantially with a 93% decrease in the v
213 counted for by the model for end-stage liver disease scoring system and may benefit from the increase
216 milar earlier onset and more severe clinical disease score than WT mice engrafted with WT bone marrow
217 sed to obtain a global cerebral small vessel disease score that captured the presence and/or severity
218 with BM only, BM+T mice had higher systemic disease scores that correlated with tear fluid loss and
219 opensity score and Model for End-Stage Liver Disease score, the NSBB adjusted odds ratio for 6-week m
221 P = 0.03) and United Kingdom endstage liver disease score (UKELD) score (59 versus 57, P = 0.01) sig
222 the United Kingdom Model of End Stage Liver Disease score (UKELD) were calculated and area under the
223 ve prostate cancer phenotype with linkage-of-disease scores up to 2.16 and nonparametric linkage scor
224 ication method allows for timepoint-specific disease scores using morphological and physiological mea
225 erimental autoimmune encephalomyelitis (EAE) disease scores via the ligand-activated transcription fa
241 5%-21.3%); the mean nonalcoholic fatty liver disease score was reduced from 5 (IQR, 4-5) to 1 (IQR, 1
244 proposed MELD and Pediatric End-stage Liver Disease scores was assessed using Harrell's concordance
245 and lower baseline Model for End-Stage Liver Disease score were associated with better survival.
246 oints added to the Model for End Stage Liver Disease score were associated with lower waitlist mortal
247 ille model and the Model for End-Stage Liver Disease score were independently associated with 6-month
251 seline (P = .001); Model for End Stage Liver Disease scores were reduced by 40.4% and 33%, respective
252 During the development of colitis, clinical disease scores were reduced by 50% (P < 0.001), and hist
254 were depleted of Vgamma4(+) cells, clinical disease scores were significantly reduced and the incide
255 ly correlated with Model for End-Stage Liver Disease score, whereas CFBa and CFD were positively asso
256 tium to Establish a Registry for Alzheimer's Disease score, which describes the density of neuritic p
257 tween social disadvantage and CMR or chronic disease scores while adjusting for childhood covariates
259 isk Index with the model for end-stage liver disease score yields an AUC-ROC of 0.764 (95% CI, 0.756-