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1 uns and at higher magnitudes than homozygote disequilibria.
2 commonly linked to the occurrence of genetic disequilibria.
3 a, with D. magna showing the least amount of disequilibria.
4 co-metabolic interactions to maximize energy disequilibria.
5 e and genetic drift on cytonuclear genotypic disequilibria.
6 ing of inferences from marker Hardy-Weinberg disequilibria.
7 en the sexes on cytonuclear polymorphism and disequilibria.
8 mates; however, the additional dicytonuclear disequilibria allow more accurate estimates of both form
9           We further demonstrate that subtle disequilibria also affect Delta(47) in biogenic calcite.
10 ctions of nucleotide variability and linkage disequilibria among conversion-mediated sites in hsp70Ab
11 versity observed within populations, linkage disequilibria analyses and association indices calculate
12 are fundamental to our understanding of such disequilibria and ascent dynamics.
13 al selection regimes more likely to generate disequilibria and maintain cytonuclear polymorphism and
14 ons provide evidence for substantial genetic disequilibria and nonadditive genetic effects underlying
15    Here we use 210Pb-226Ra-230Th radioactive disequilibria and other geochemical attributes in oceani
16 s substantively lag climate forcing, causing disequilibria and reduced fitness, and abrupt responses
17 r's exact test for the genotypic cytonuclear disequilibria and some approximations of the exact test.
18 s of genetic variation (for example, linkage disequilibria) and tests of hypotheses (using simulation
19 ance constraints reveal evidence of chemical disequilibria, and insights into the planetary mass-meta
20 tensive genetic diversity, a lack of linkage disequilibria, and little phylogenetic structure, demons
21 mmetry, both nuclear-nuclear and cytonuclear disequilibria are equivalent.
22                                  Cytonuclear disequilibria are generated de novo in both sexes when b
23 onfirms the prediction that pairwise linkage disequilibria are predominantly generated by migration.
24                                     Di-genic disequilibria are significant for four of 74 di-locus pa
25  that disease susceptibilityweighted linkage disequilibria are zero, given disease heterogeneity, it
26 btain the dynamics of the sample cytonuclear disequilibria assuming random drift alone as the source
27 a simultaneously that are (1) Hardy-Weinberg disequilibria at each locus, (2) gametic disequilibrium
28 imentally, this study shows that cytonuclear disequilibria at life stages with sex differences can be
29                                    Where the disequilibria attained by seed and pollen flow are signi
30                                    Where the disequilibria between cytoplasmic and nuclear genes are
31 nal or paternal parents, and zygotic linkage disequilibria between different loci after the mutation
32 encies, recombination fractions, and linkage disequilibria between different markers.
33               If selection promotes coupling disequilibria between genes of similar effect, recombina
34  but we also derive some results for linkage disequilibria between neutral sites.
35 ate the existence of an agrifood debt (i.e., disequilibria between regions in the natural resources c
36                                      Linkage disequilibria between TNF and HLA alleles were calculate
37                             However, linkage disequilibria built up by correlational selection are ex
38 A mantle-melting model can simulate observed disequilibria but preservation requires a subsequent mec
39                    Additionally, three-locus disequilibria can be generated by higher-order intermigr
40        We show analytically that cytonuclear disequilibria can be generated de novo if the cytoplasmi
41 ial levels of permanent two- and three-locus disequilibria can be generated in adults by (i) nonzero
42 ure type individuals and nonzero cytonuclear disequilibria can be maintained within a hybrid zone if
43 and small in magnitude, measurable permanent disequilibria can result from selective differences both
44                                              Disequilibria cannot be created or maintained, and heter
45 w patterns of allele frequencies and linkage disequilibria change over time.
46          Assuming no interactions, the redox disequilibria could have been generated by interactions
47 ining timescales derived from uranium-series disequilibria, crystal sizes and trace-element zoning in
48 iii) readily creates permanent host-symbiont disequilibria de novo, whereas uniform transmission can
49 missense variant (S1370A), but these linkage disequilibria did not differ between the NIDDM and contr
50 icates that stochastically generated linkage disequilibria do select for increased recombination, a r
51 expected values of the cytonuclear genotypic disequilibria for both the homozygotes and heterozygotes
52 wever, consequences of the resulting climate disequilibria for ecosystem functioning are rarely consi
53              The large (238)U-(234)U-(230)Th disequilibria for some of the glasses, along with the wi
54 ate between loci associations due to linkage disequilibria from those caused in other ways can render
55     Although previous work suggests that the disequilibria generated by cytonuclear selection may be
56 age are also taken into account, the gametic disequilibria generated by the Bulmer and Hill-Robertson
57                  Past changes in ocean (14)C disequilibria have been suggested to reflect the Souther
58        We also find that patterns of linkage-disequilibria in admixed Hispanic/Latino populations are
59 shing, controls on MeHg solubility, or redox disequilibria in fens.
60 and seeds coupled with nuclear-mitochondrial disequilibria in migrant seeds, or different nuclear fre
61 seeds coupled with mitochondrial-chloroplast disequilibria in migrant seeds.
62 utline an exact test for allelic cytonuclear disequilibria in multiallelic systems.
63 rate allele frequency changes or cytonuclear disequilibria in populations with constant viability sel
64 ids interacted with the sediments, the redox disequilibria in secondary minerals suggest infiltration
65 ia can be generated in adults by (i) nonzero disequilibria in the migrant pools or (ii) intermigrant
66 of the statistical properties of cytonuclear disequilibria in two major ways.
67  The full bounds are derived for cytonuclear disequilibria in two-locus systems with an arbitrary num
68 ification of phenotypic traits suggests that disequilibria inherent to viral populations may provide
69                             Such significant disequilibria involving the paternally inherited organel
70                              This pattern of disequilibria is consistent with the action of genetic d
71 rstanding of processes that regulate climate disequilibria is essential for improving long-term proje
72                      Few significant linkage disequilibria (LDs) occur between the genome-wide CNV lo
73 uclear equilibrium structure, including when disequilibria may be indicators of gene flow.
74                 Approximate normality of the disequilibria measures are also demonstrated by Monte Ca
75  double heterozygote, gametic and nongametic disequilibria need to be combined into a composite digen
76                   The likelihood of magmatic disequilibria occurring before melt enters shallow crust
77     Second, our method can detect marker-QTL disequilibria of different orders and QTL epistatic inte
78  fitness interactions in males generate male disequilibria only.
79 selfing, or migrant pollen and seeds lacking disequilibria or intermigrant admixture effects.
80 simulations demonstrate that the majority of disequilibria produced by random selection are transient
81               Mutations promoting rotational disequilibria showed catalytic, biochemical and translat
82 quilibrium specifies five different types of disequilibria simultaneously that are (1) Hardy-Weinberg
83 n of DNA marker heterozygosities and linkage disequilibria that are likely to resemble those expected
84 ent, the virus exhibited internal population disequilibria that did not decline with increased adapta
85 ncrease recombination eliminate the negative disequilibria that impede selection and consequently inc
86 zygosities and low within-population linkage disequilibria) that differs qualitatively from the highl
87  investigate the potential magnitude of such disequilibria, their qualitative dynamics, the expected
88 nitude and temporal dynamics of cyto-nuclear disequilibria through time.
89        Our analysis introduces interspecific disequilibria to quantify nonrandom associations between
90                         We use host-symbiont disequilibria to quantify the role played by non-random
91             In this paper we use cytonuclear disequilibria to test the neutrality of mtDNA markers.
92 namics, the expected frequency of detectable disequilibria under particular patterns or strengths of
93       The dynamics of the variances of these disequilibria under the random drift model are studied b
94 rns of nuclear and cytonuclear associations (disequilibria) under various models of migration.
95 when nonadditive genetic effects and genetic disequilibria underlie a genetic system, genetic slippag
96 e develop a test statistic using cytonuclear disequilibria via the theory of generalized least square
97                                   At extreme disequilibria, we observed the onset of reaction front i
98 quilibrium systems initiates parent-daughter disequilibria, which re-equilibrate by the shorter half-
99 to define normalized measures of cytonuclear disequilibria, whose practical utility is illustrated th
100  or reflected their tight linkage respecting disequilibria with other class I variants.
101 lic frequencies of markers and their linkage disequilibria with QTL, because the probabilities of QTL
102 requencies of putative QTL and their linkage disequilibria with the markers are estimated by solving
103  genotypic diversity and evidence of genetic disequilibria, with D. magna showing the least amount of
104                          Significant linkage disequilibria within and between these genes were also d

 
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