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1 t timing abnormalities could lead to spatial disorientation.
2 in disA-1 cells display aberrant spacing and disorientation.
3 oscope from different perspectives to induce disorientation.
4 nteract during complex movements and spatial disorientation.
5 duct injuries occur as a result of operator disorientation.
6 ng to decrease the morbidity associated with disorientation.
7 to locate objects in a square chamber after disorientation.
8 a novel environment, and slow rotation after disorientation.
9 ic targets to treat diseases causing spatial disorientation.
10 f betrayal, detachment, self-alienation, and disorientation.
11 he anatomy and networks associated with time disorientation.
12 ts are key indicators of social and temporal disorientation.
13 ed to underlie patients experiencing spatial disorientation.
15 "unable to assess," while inattention (36%), disorientation (27%), and disorganized thinking (18%) we
16 % CI=3.67-5.16); for delirium, confusion, or disorientation, 5.14 (95% CI=4.54-5.82); and for panic d
17 auma with loss of consciousness, amnesia, or disorientation and a Glasgow Coma Scale score of 9-15).
18 ntext of the observed forms of topographical disorientation and are found to be in good agreement wit
20 mature tangles in the entorhinal cortex and disorientation and confusion when navigating familiar pl
21 p (+0.26; P=.04), depression (+0.25; P=.05), disorientation and detachment (+0.23; P= .05), and vital
22 rrent study investigates the anatomy of time disorientation and its network correlates in patients wi
23 en were at higher risk of delirium/confusion/disorientation and mania, while younger patients were at
25 Simulations revealed a high probability of disorientation and subsequent attraction for nearby bird
26 d review of the literature of "topographical disorientation" and describes several functional MRI stu
29 isual processing, disorders of topographical disorientation, and the influence of environmental condi
32 n of key perceptual-motor factors leading to disorientation, assessment of their relative impact, and
33 a," characterized by problems with thinking, disorientation, balance disturbances, vertigo, and impot
34 l dynamics are common in organogenesis, cell disorientation caused by loss of mechanosensation could
35 delirium, including cognitive impairment or disorientation, dehydration or constipation, hypoxia, in
36 hallucinations, prosopagnosia, topographical disorientation, disturbance of perception of velocity of
37 ea, fatigue, somnolence, euphoria, vomiting, disorientation, drowsiness, confusion, loss of balance,
38 Individuals with developmental topographic disorientation (DTD) have a life-long impairment in spat
43 osed that several varieties of topographical disorientation exist, resulting from damage to distinct
48 The maximum frequency that could cause such disorientation has been predicted to lie between 120 and
49 panic disorder, and delirium, confusion, or disorientation) have been reported to occur in 15.7 per
55 predict patients at a high risk for spatial disorientation in the community based on their VR naviga
56 irments predict a patient's risk for spatial disorientation in the real world is still poorly underst
58 levated TGF-beta expression, mitotic spindle disorientation, increased lumenization, disruption of RO
61 ge in traumatic brain injury leads to bundle disorientation, loss of axonal viability, and cognitive
63 ne causes swelling of unmyelinated axons and disorientation of axonal microtubules at a time when it
65 py showed an altered lamellar structure with disorientation of elastin fibers from the circumferentia
66 eginnings of an explanation for the magnetic disorientation of migratory birds exposed to anthropogen
67 an increase of second-order vasa vasorum and disorientation of normal vasa vasorum spatial pattern.
68 In addition, clemastine treatment leads to disorientation of Plasmodium mitotic spindles during the
69 display substantial cell morphology defects, disorientation of septum formation and a significantly p
70 ace to regain their sense of direction after disorientation, often ignoring nongeometric cues even wh
71 ephalopathy (HE) that does not cause obvious disorientation or asterixis (minimal HE [MHE]/grade 1 HE
74 g duration significantly predicted composite disorientation score on the DNT (beta = 0.422, p = 0.034
75 to 116 MHz and that birds' sensitivity to RF disorientation should fall by about two orders of magnit
77 lesions involving the PPA cause topographic disorientation, there is little causal evidence linking
78 hy, defined as a main diagnosis of delirium, disorientation, transient alteration of awareness, trans
79 ort temporary neurological symptoms, such as disorientation, visual disturbances, and motor issues, p
80 ons correlated with search performance under disorientation, whereas consistent marking of ground inf
81 ehavioral and anatomical association of time disorientation with memory impairment, such that the 2 p