ライフサイエンスコーパス: disruptant

1 e-GlcCer content, reduced by 50% in the smtB disruptant.

2 thetic pathway, was undetectable in the relA disruptant.

3 used unmodified to obtain the desired single disruptant.

4 The frog MAPK gene also suppressed the pmk1 disruptant.

5 s known to be essential, did not give viable disruptants.

6 In fau1 disruptants, 5-CHO-THF was elevated 4-fold over wild-typ

7 orlA disruptants accumulate trehalose-6-phosphate and have re

8 or the efficient construction of stable gene disruptants and of gene deletions in Streptomyces.

9 not occur in the presence of the aggregation disruptants ANS and CR.

10 at 42 degrees C, the conidia and hyphae from disruptants are chitin deficient, swell excessively, and

11 Yeast SPP381 disruptants are severely growth impaired and accumulate

12 lition of Gpi11p or Pig-Fp function in GPI11 disruptants blocks GPI anchoring and formation of comple

13 -related proteins and tested for the desired disruptants by PCR screening of 100 colonies.

14 enting canavanine hypersensitivity of scrheb disruptant cells.

15 tron microscopy studies showed that the alb1 disruptant exhibited a smooth conidial surface, whereas

16 us role in nocardicin biosynthesis, the nocL disruptant failed to generate the oxime-containing metab

17 FAT1 disruptants (fat1Delta) fail to accumulate the fluoresce

18 ype STE11alpha strain with a ste11alphaDelta disruptant for virulence using the mouse model showed th

19 e of 30 degrees C, gpi1 cells and gpi1::URA3 disruptants form large, round, multiply budded cells wit

20 ure, we examine acute effects of the osmotic disruptant glycyl-L-phenylalanine 2-naphthylamide (GPN).

21 When grown on agar, the disruptants grew more slowly than a control lysogen made

22 Importantly, the alb1 disruptant had a statistically significant loss of virul

23 Disruptants have a nearly-wild-type morphology at 32 deg

24 The slm9 disruptant is delayed at the G(2)-M transition as indica

25 er observed following membrane feeds of CTRP disruptant lines to Anopheline mosquitoes, despite the d

26 he mutant phenotypes observed in both a nog1 disruptant mutant (nog1dis) and snog1a can be attributed

27 t importantly, the virulence of a ste12alpha disruptant of serotype D strain was significantly reduce

28 Growth of the disruptant on low-phosphate medium did not restore actin

29 f E.coli DExH/D-box protein and DNA helicase disruptants should be useful for analyzing the function

30 Furthermore, both rad14 and mag1 disruptants show elevated levels of nitrogen mustard-ind

31 The resulting wdpks1Delta disruptants showed no morphological defects other than a

32 In an aep3-disruptant strain, the shorter ATP8/6 mRNA was undetecta

33 , mismatch repair-defective, rad27 and mre11 disruptant strains.

34 Pfs230 disruptants successfully emerge from RBCs and male gamet

35 In contrast, in the Pfs230 disruptants the expression and localization of other kno

36 in production medium than the wild type or a disruptant to which the intact relA gene was restored.

37 tion by allelic exchange in the case of each disruptant was confirmed by PCR and Southern blot analys

38 The sterol profile of an ERG25 disruptant was consistent with the erg25 allele obtained

39 Moreover, the disruptant was unable to accumulate ppGpp to the levels

40 nidial surfaces, and the conidia of the alb1 disruptant were ingested by human neutrophils at a highe

41 l of acute infection, wdchs2Deltawdchs3Delta disruptants were considerably less virulent in the same

42 Furthermore, ste12alpha disruptants were fertile when mated with MATa strains, a

43 diates in stationary-phase cells, since rev3 disruptants were more sensitive in this phase than in th

44 out selection, and in six other cases, where disruptants were not identified in the initial PCR scree

45 tion of the infection droplets of the double disruptant with either purified enzyme, PLA, or PLD, cau

46 Complementation of the double disruptant with pelD gene, or supplementation of the inf