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1 in relief, rehydration, and encouragement of diuresis.
2  diuretic therapy but are often resistant to diuresis.
3  water absorption, promoting natriuresis and diuresis.
4 seen in 15% of patients and was treated with diuresis.
5 imal tubule, leading to polyuria and osmotic diuresis.
6 de concentrations and causes natriuresis and diuresis.
7 l to show any benefit of dopamine other than diuresis.
8 sport process, which is upregulated by water diuresis.
9  mannitol in an attempt to induce an osmotic diuresis.
10 mmol/L) respectively in hyponatremia rats by diuresis.
11 sistent with the peak and postpeak phases of diuresis.
12 bbits (1 +/- 3 mL/kg; p < 0.01) due to lower diuresis.
13 centration) and lower blood pressure limited diuresis.
14  angiotensin, while inducing natriuresis and diuresis.
15 ary congestion via vasodilation and enhanced diuresis.
16 medullary interstitium, resulting in osmotic diuresis.
17 hrotoxic agents from the kidney by promoting diuresis.
18 sm is the prevention of urea-induced osmotic diuresis.
19 ffective renal plasma flow, natriuresis, and diuresis.
20 cantly, resulting in natriuresis and osmotic diuresis.
21 nal suppression and enhanced natriuresis and diuresis.
22  1.52; P<0.001), with a significantly higher diuresis (5776 versus 4381 mL; adjusted mean ratio, 1.33
23 ro rats, which manifest severe chronic water diuresis, a 117-kD band was seen, in addition to the pre
24 ons of both peptides include natriuresis and diuresis, a decrease in systemic blood pressure, and inh
25                                              Diuresis, a marker of improved kidney function, occurred
26 istration of furosemide alone caused a large diuresis, addition of BG9719 to furosemide increased diu
27 d points included cumulative natriuresis and diuresis after 2 days of treatment, length of stay, and
28 atinine was related to the degree of induced diuresis after controlling for baseline renal function,
29 ized selective kappa agonist U50,488-induced diuresis after sc administration in rats.
30 ied hourly during 4 hours with water-induced diuresis, after the ingestion of 5 g of creatinine and i
31          Ip6k1/2-/- mice also show increased diuresis, albuminuria, and hypercalciuria, although the
32  inner medulla, we tested whether furosemide diuresis alters active urea transport.
33 ut was associated with evidence for enhanced diuresis among patients with AHF.
34 ther the level of hydration after furosemide diuresis and 22 hr of sodium depletion affects the amoun
35 n of proximal tubule sodium reabsorption and diuresis and abolishes Na,K-ATPase inhibition and NHE-3
36 er, concerns that SGLT2i may provoke osmotic diuresis and activate vasopressin led to systematic excl
37            ECD animals exhibited marked anti-diuresis and anti-natriuresis (40 and 47%), which peaked
38 trolyte transporters in the kidney to induce diuresis and are often used in chronic kidney disease (C
39 d for Drosophila kinin-mediated induction of diuresis and chloride shunt conductance, evidenced by ch
40 signed a clinical trial to determine whether diuresis and colloid replacement in hypoproteinemic pati
41 ctly targets renal GPR39 to induce increased diuresis and consequently stimulate drinking behavior.
42  persisted after adjusting for parameters of diuresis and decongestion (odds ratio, 2.0; 95% confiden
43                                              Diuresis and decongestion were examined across a range o
44 eart failure treatment negatively influences diuresis and decongestion.
45 pithelial chloride conductance, resulting in diuresis and depolarization of the transepithelial poten
46 oration of the temporal relationship between diuresis and dyspnea relief and a possible clinical role
47                                          The diuresis and glomerular filtration rate were low and dec
48                                              Diuresis and glucose excretion were significantly increa
49  that in isolation, glucose-elicited osmotic diuresis and glucose transporter upregulation raise albu
50                                   Additional diuresis and hydration are recommended to improve the vi
51 pression with increased infection risk, cold diuresis and hypovolemia, electrolyte disorders, insulin
52 us population of KOR underlying KOR-mediated diuresis and illustrates a unique pharmacological profil
53 gh-dose strategy was associated with greater diuresis and more favorable outcomes in some secondary m
54 partly protected GFR, and reduced the marked diuresis and mTAL necrosis in radiocontrast-induced neph
55 enal delivery of BNP resulted in significant diuresis and natriuresis and an increase in GFR.
56 retic response measured by higher cumulative diuresis and natriuresis and shortened length of stay wi
57 eliminary study found that the ETBR-mediated diuresis and natriuresis are impaired in hypertension wi
58                  We further propose that the diuresis and natriuresis seen during air breathing were
59                        F+V and F+T increased diuresis and natriuresis to a similar extent during drug
60 human volunteers reveal that dopamine causes diuresis and natriuresis, as well as some degree of rena
61 ough acute caffeine intake produces moderate diuresis and natriuresis, caffeine increases the blood p
62              TMAO-treated rats showed higher diuresis and natriuresis, lower arterial pressure and pl
63 heir receptor (NPR1) to affect vasodilation, diuresis and natriuresis, lowering venous pressures and
64  failure (CHF) are associated with increased diuresis and natriuresis, preserved glomerular filtratio
65 lthough both CA and Ucn2 infusion produced a diuresis and natriuresis, responses with Ucn2 and Ucn+CA
66 te, increased renal blood flow, and promoted diuresis and natriuresis.
67 venous pressures, without obvious changes in diuresis and natriuresis.
68 at wCle elimination significantly slows both diuresis and protein catabolism.
69 effects of adenosine A1-receptor blockade on diuresis and renal function in patients with acute decom
70       Such combination therapy might enhance diuresis and salt excretion for treatment of hypertensio
71 vity was positively associated with residual diuresis and serum albumin and inversely associated with
72 onal switch in renal K handling during water diuresis and the relevance to renal K homeostasis in cys
73                                   Furosemide diuresis and water diuresis for 1 or 7 d had no effect o
74 ow UNa patients presented a lower cumulative diuresis and weight loss and presented more often with p
75                                              Diuresis and weight loss over 5 days (5.3 kg more in the
76              Dramatic reduction was found in diuresis, and albuminuria was evident after administrati
77  other adverse effects, including dysphoria, diuresis, and constipation.
78 n, decreases Na+-K+-ATPase activity, induces diuresis, and exerts a vasodilator effect.
79 ing growth, development, ecdysis, digestion, diuresis, and many more physiological processes.
80 C8D cause proximal tubular injury, increased diuresis, and mild Fanconi-like symptoms.
81 ve cardiac output, stimulate natriuresis and diuresis, and rapidly induce symptomatic relief.
82 ry promptly restored cardiac index, promoted diuresis, and reduced fluid requirements compared with t
83 ter reabsorption, limits natriuretic osmotic diuresis, and results in concurrent extracellular volume
84 , sex, comorbidities, diagnosis, creatinine, diuresis, and study.
85 d ability to concentrate urine, exhibit mild diuresis, and tend to be blood volume depleted.
86 c-derived hormones that promote natriuresis, diuresis, and vasodilation.
87 of cGMP decrease BP by inducing natriuresis, diuresis, and vasodilation.
88 n includes control of hypertension, cautious diuresis, and, if necessary, ventilatory support.
89 an arterial pressure, lactate clearance, and diuresis are maintained at acceptable levels.
90 safe, and resulted in higher natriuresis and diuresis, as well as a shorter length of stay.
91 e in proximal tubule sodium reabsorption and diuresis associated with inhibition of renal cortex Na,K
92 istence of a mechanism that protects against diuresis at a low T(B).
93 duces BP without sympathetic activation, but diuresis augments sympathetic activity in elderly hypert
94 ventional therapy, which included aggressive diuresis, blood transfusions to optimize oxygen-carrying
95       We conclude that use of LDD results in diuresis but does not improve other markers of renal or
96 I infusion normally promotes natriuresis and diuresis, but COX2 deficiency blocked this effect.
97 reater hypotension and a loss of the initial diuresis, but no significant change in creatinine cleara
98                                              Diuresis by either protocol was feasible, safe, and effe
99  pathway more strongly, and further enhanced diuresis by exacerbating the downregulation of the Na(+)
100 ockade may improve glomerular filtration and diuresis by exerting a direct beneficial effect on glome
101 conclude that the activation of GPR39 causes diuresis by opposing AVP-induced Na(+) and Cl(-) reabsor
102                           To prevent osmotic diuresis by the large quantity of urea generated by prot
103                                The increased diuresis can be accomplished without increased risk if d
104  OPC 31260 induced a significant increase in diuresis, decrease in urinary osmolality, and rise in pl
105 t the highest infusion rates, they exhibited diuresis, dehydration, and both decreased weight gain an
106 elevated solute excretion, long-term osmotic diuresis does not occur in humans with SGLT2 inhibition.
107                                        Rapid diuresis early in the hospitalization should similarly i
108 e, including increased plasma urea, impaired diuresis, elevated biomarkers, and changes in brain weig
109                This decoupling of thirst and diuresis enables water retention by the kidney while sup
110 n three digestive processes in the bed bug - diuresis, erythrocyte (red blood cell) lysis, and protei
111                Furosemide diuresis and water diuresis for 1 or 7 d had no effect on either basal or A
112 /kg)- and bremazocine (0.0032 mg/kg)-induced diuresis for 20 weeks.
113                Protective mechanisms include diuresis, further acceleration of lysine conjugate forma
114                                     Two-hour diuresis greatly increased.
115                          Additional study of diuresis guided by the NRPE is warranted.
116 d 6-10 hours after the fourth BNZ dose, when diuresis had subsided.
117 senting a time point after the late phase of diuresis has concluded.
118 .3 +/- 6 days) with sodium nitroprusside and diuresis, hemodynamics improved, eNO concentrations fell
119 ed glomerular filtration rate (GFR), osmotic diuresis, hypertrophy, and megalin and cubilin downregul
120                  A new finding is that water diuresis improves medullary oxygenation (delta R2* = 6.4
121 However, whether or not nesiritide increases diuresis in ADHF patients is unknown.
122  treatment was associated with an equivalent diuresis in both WT and CF mice.
123  sought to determine if nesiritide increases diuresis in congestive heart failure patients.
124 ffective adjunct to furosemide in increasing diuresis in critically ill children with fluid overload.
125 additional imaging with hydration and forced diuresis in individuals undergoing additional late scann
126                                   The marked diuresis in lopamidol treated rats (630 +/- 125.1%) was
127 y findings do not support a dominant role of diuresis in mediating the physiological changes or clini
128 that central KOR mediate KOR agonist-induced diuresis in monkeys.
129          Although potentially able to induce diuresis in patients otherwise resistant to high doses o
130 pronounced effects regarding natriuresis and diuresis in patients with a lower eGFR.
131 dium transporters, decreased natriuresis and diuresis in response to l-dihydroxyphenylalanine, and de
132 n and restored ETBR-mediated natriuresis and diuresis in SHRs.
133 tive renal vasodilation and a marked osmotic diuresis in the innervated kidney.
134 onists was actually associated with improved diuresis in this cohort.
135  statistically significant adverse event was diuresis in treated subjects (48% vs 13%, p = 0.02).
136  data show that activated LK cells stimulate diuresis in vivo, and that LK and IPC signaling affect f
137 f ANP results in substantial natriuresis and diuresis in wild-type mice but fails to cause significan
138 ning 4% BSA resulted in a marked natriuresis/diuresis in wild-type mice but no response in GC-A null
139                 Initial changes included net diuresis, increased ACE inhibitor doses, and frequent ad
140                                              Diuresis induced by frusemide (10 mg) is reported to imp
141 PCs but not ICs was stimulated by short-term diuresis (injections of furosemide) and reduced by antid
142  is no conclusive evidence that one means of diuresis is better than another.
143            (3) Either mesna or forced saline diuresis is recommended to decrease the incidence of uro
144                            Overly aggressive diuresis leading to intravascular volume depletion has b
145  inhibition promotes natriuresis and osmotic diuresis, leading to plasma volume contraction and reduc
146 SNA through suppressed renin activity, while diuresis may evoke sympathetic activation via the upregu
147 ptor) with improved oral efficacy in the rat diuresis model (ED50=0.01 mg/kg) as well as high oral bi
148 cal properties and in vivo activity in a rat diuresis model (ED50=0.3 mg/kg po).
149 monstrated efficacy in the volume-loaded rat diuresis model as well as promising in vitro and in vivo
150            When orally administered in a rat diuresis model, compound 14 promoted sodium excretion (E
151 ed along with their in vivo effects in a rat diuresis model.
152 K compound, 2 (MK-7145), demonstrated robust diuresis, natriuresis, and blood pressure lowering in pr
153          The main endpoints of decongestion, diuresis, natriuresis, and clinical outcomes are assesse
154                                              Diuresis, natriuresis, renal excretion, and tissue level
155                                           If diuresis occurs at a rate greater than extravascular flu
156 anifested the expected early natriuresis and diuresis of vasopressin escape.
157            This study tested whether chronic diuresis or antidiuresis regulates facilitated urea tran
158 ver, SBP reduction did not negatively affect diuresis or decongestion (P >/= 0.25 for all parameters)
159 duction was not associated with worsening of diuresis or decongestion.
160 y arteriolar pathology and was responsive to diuresis or dialysis but not to nitric oxide therapy.
161                                              Diuresis or fluid restriction may improve lung function
162 s, indicating that the effect was not due to diuresis or fluid shifts.
163 on is an absence of a large acute or chronic diuresis or natriuresis with these agents, either when g
164 ndicating a deconditioned state or excessive diuresis, or both.
165 er before diuretic administration or at peak diuresis (P 0.11 for all).
166                  The SAA-rich diet increased diuresis paralleled by downregulation of aquaporin-2 (AQ
167 model predicts that glucose-elicited osmotic diuresis per se raises albumin excretion only slightly.
168 sed basal Purea in terminal IMCD in all five diuresis protocols.
169                However, ETx stimulated early diuresis,reduced serum sodium levels, and had more prono
170 canagliflozin doses vs 22 [5%]), and osmotic diuresis-related events (pollakiuria: 12 [3%] for both d
171      The purpose of the study was to compare diuresis renography scan interpretation generated by a r
172                                   The forced diuresis resulted in higher urine flow rate (163.26+/-54
173                                              Diuresis significantly increased basal Purea in terminal
174 ogy, urine and serum parameters in different diuresis states, and metabolomics.
175 sporter are major factors in the natriuresis/diuresis that is one of the hallmarks of ischemic acute
176 e refers to the spontaneous and compensatory diuresis that occurs in primary aldosteronism to correct
177 ormone-mediated inhibition of MTs countering diuresis that provides a deeper understanding of this cr
178 ch as mannitol, a safe FDA approved drug for diuresis) through particle size measurement and forster
179 eks later), including vigorous hydration and diuresis to minimize radiation exposure to the fetus.
180 NCC does not cause salt wasting or excessive diuresis under basal conditions, raising the possibility
181 intravenously to saline-loaded rats, induced diuresis via antagonism of renal A1-adenosine receptors.
182 T2) inhibitor empagliflozin promotes osmotic diuresis via urinary glucose excretion and therefore, mi
183 T2) inhibitor empagliflozin promotes osmotic diuresis via urinary glucose excretion, likely leading t
184 meters, and caused a 50% increase in 24-hour diuresis volume (7.4 +/- 0.9 mL/100g in BDL vehicle vers
185 ffects and produced significant increases in diuresis volume and 24-hour natriuresis (0.08 +/- 0.02 m
186                                         This diuresis was compensated for by a drop in urine volume a
187 edema or fluid overload for which aggressive diuresis was intended.
188                                       Hourly diuresis was measured in the first 6 h of the study, and
189               However, furosemide-associated diuresis was reduced only in male, not female, mPGES1-de
190                                          The diuresis was reversible and showed urea > salt excretion
191 ion furosemide, or dual placebo, targeted to diuresis, weight loss, and serum total protein.
192                     Overall, natriuresis and diuresis were higher with acetazolamide, with a higher t
193 phorylated and ETBR-mediated natriuresis and diuresis were not evident.
194 ological/physiological maneuvers that induce diuresis were studied.
195  result of the use of diuretics or of solute diuresis, whereas only 50% of patients who were hypernat
196 , addition of BG9719 to furosemide increased diuresis, which was significant at the 0.75-microg/mL co
197 c is effective in maximizing natriuresis and diuresis while preserving renal function and inhibiting
198 reserved furosemide-mediated natriuresis and diuresis, while reducing cardiac preload and afterload.
199 rst, we aimed to assess the impact of forced diuresis with early furosemide injection on the detectio
200                             Metal chelation, diuresis with furosemide or chlorothiazide, and competit
201                                       Forced diuresis with intravenous crystalloid, furosemide, and m
202 re 98 participants were randomized to forced diuresis with intravenous crystalloid, furosemide, manni
203                We investigated the impact of diuresis with intravenous furosemide and oral hydration
204 as done to test the hypothesis that a forced diuresis with maintenance of intravascular volume after
205 ned by changes in serum creatinine (SCr) and diuresis with risk/injury/failure/loss/end stage (RIFLE)
206                                        Water diuresis (with or without food) for 3 to 5 d had no effe
207 lexible diuretic regimen after 4.2-liter net diuresis, with counseling also regarding diet and progre

 
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