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1 h the basic mechanism of the eukaryotic cell division cycle.
2 t reversible phosphorylation during the cell division cycle.
3 of interacting cells in a model of the cell division cycle.
4 ch other and with progression through a cell-division cycle.
5 ge in a characteristic manner throughout the division cycle.
6 g periodic gene expression in the human cell division cycle.
7 e for cytoplasmic territory and insulate the division cycle.
8 on in cytoskeletal organization and the cell division cycle.
9 nt and, therefore, able to re-enter the cell-division cycle.
10 microtubule cytoskeleton throughout the cell division cycle.
11 matin need to be duplicated during each cell division cycle.
12 growth genes to promote entry into the cell division cycle.
13 ch cell growth is controlled during the cell division cycle.
14 nd controlled through the course of the cell-division cycle.
15 e transient interactions throughout the cell-division cycle.
16 coupled to a single complete eukaryotic cell division cycle.
17 centrioles and centrosomes in a single cell division cycle.
18 olume fluctuations that occur over each cell division cycle.
19 icant loss of protocell contents during each division cycle.
20 is not influenced by the status of the cell division cycle.
21 cation, export, and assembly during the cell division cycle.
22 icting DNA replication to once in every cell division cycle.
23 ome replication and does not follow the cell division cycle.
24 companied by a withdrawal from the bacterial division cycle.
25 eract with, but are independent of, the cell division cycle.
26 of cell division starting at the second cell division cycle.
27 on appears to be independent of the cellular division cycle.
28 s important roles in the control of the cell division cycle.
29 t on variation of protein content during the division cycle.
30 of the S phase program in the mammalian cell division cycle.
31 on the pattern of DNA replication during the division cycle.
32 ately copies billions of DNA bases each cell division cycle.
33 ntus that controls an early step in the cell division cycle.
34 fferent patterns of cleavage in their second division cycle.
35 , G1 to S phase progression within the first division cycle.
36 is the last critical decision during a cell-division cycle.
37 is responsible for re-initiation of the cell division cycle.
38 ogression of the eukaryotic cell through its division cycle.
39 lex on chromatin during G1 phase of the cell division cycle.
40 most kinases that are known to regulate the division cycle.
41 ed that assumes expanding volumes and a cell-division cycle.
42 ome synthesis and specific steps in the cell division cycle.
43 d not affect the G1/S transition of the cell division cycle.
44 erial is accurately passed through each cell division cycle.
45 art, the irreversible commitment to the cell division cycle.
46 prevents entry into the S-phase of the cell division cycle.
47 r link between the cilia life cycle and cell-division cycle.
48 IDR controls entry into S phase of the cell division cycle.
49 of cells in the committed phase of the cell division cycle.
50 her continue to proliferate or exit the cell division cycle.
51 olites change significantly through the cell division cycle.
52 tabolic fluxes are coordinated with the cell division cycle.
53 al activity per DNA copy throughout the cell division cycle.
54 of CDK1 expression had an impact on the cell division cycle.
55 into the regulation of CDK1 during the cell division cycle.
56 ing orthogonal planes over three consecutive division cycles.
57 tiple feedback loops to yield two successive division cycles.
58 ach other, resulting in circadian-gated cell division cycles.
59 ect chromosome ends from erosion during cell division cycles.
60 e stably propagates through at least 14 cell division cycles.
61 stably transmitted through consecutive cell division cycles.
62 the morula stage after only two to four cell division cycles.
63 omatin is gradual and requires multiple cell division cycles.
64 ave traced these defects back to the nuclear division cycles.
65 ws to dynamically polarize during asymmetric division cycles.
66 times that emerge as 12-hr synchronized cell division cycles.
67 circadian clock-dependent synchronized cell division cycles.
68 [RIF1], Replication Factor A 3 [RFA3], Cell Division Cycle 13 [CDC13], Pbp1p Binding Protein [PBP2])
69 rved dual-specificity phosphatase human cell-division cycle 14A (hCDC14A) associates with the actin c
70 cted a possible phosphorylation site by cell division cycle 2 (Cdc2), which directly phosphorylated r
71 ee genes, two nuclear (beta-tubulin and cell division cycle 2) and a gene from the plastid genome (th
77 wed that induction of expression of the cell division cycle 20 gene (Cdc20), a key regulator of the m
78 MAK associates with CDH1 (FZR1, fizzy/cell division cycle 20 related 1) and phosphorylates CDH1 at
80 over the E3 ubiquitin ligase Cdc20-APC (cell division cycle 20-anaphase promoting complex) as a centr
84 ype that is linked to overexpression of cell division cycle 25 (Cdc25)A phosphatase and cell-cycle de
86 EM (rat sarcoma exchange motif), CDC25 (cell division cycle 25), and PR (proline-rich) tail domains.
87 lation of p27 at Thr187 was mediated by cell division cycle 25A (Cdc25A), confirmed using Cdc25A inhi
91 also decreased the levels of Cdc25B and cell division cycle 25C (Cdc25C) phosphatases with an increas
93 the let-7 target cell cycle regulators cell division cycle 34 (Cdc34) and E2F transcription factor 5
94 ynthetase domain containing 1 (Aarsd1), cell division cycle 37 (Cdc37), and stress induced phosphopro
98 ock protein 90) and its cofactor Cdc37 (cell division cycle 37 protein) are crucial to prevent the ce
99 eater proportion of the two Rho GTPases cell division cycle 42 (CDC42) and Rac family small GTPase 1
101 strate that T cell-specific deletion of cell-division cycle 42 (Cdc42) GTPase causes a profound loss
102 ulated by SRC-like adaptor 2 (Sla2) and cell division cycle 42 (Cdc42) independently of Sla2's role i
108 ses have suggested that reduced Duo and cell division cycle 42 (Cdc42) transcript expression is invol
109 dea, altered expression of genes in the cell division cycle 42 (CDC42)-CDC42 effector protein (CDC42E
111 bition of Rac family small GTPase 1 and cell division cycle 42 activation, as well as downstream intr
112 nascent axon, and the Rho GTPase Cdc42 (cell division cycle 42) activates the mPar6alpha/Par3 (Par fo
113 ere, we investigated the role of CDC42 (cell division cycle 42) during vascular morphogenesis and its
118 the Rho family, including Rho, Rac, and cell division cycle 42, regulate the actin cytoskeleton.
120 sulting in the activation of downstream cell division cycle 42/Rac family small GTPase 1 signaling, i
121 eplication, PP2A exists in complex with cell division cycle 45 (CDC45) and that increased PP2A activi
122 ing S phase in yeast, and Sld3 recruits cell division cycle 45 (Cdc45) to minichromosome maintenance
123 the replicative helicase containing the cell division cycle 45 (Cdc45)/minichromosome maintenance 2-7
126 he origin-recognition complex (ORC) and cell-division cycle 6 (Cdc6) proteins recognize and encircle
127 xplore the effect of phosphorylation of cell division cycle 6 (Cdc6), a DNA replication initiation fa
128 t cancer cell cycle, is associated with Cell Division Cycle 6 (CDC6), Cyclin-dependent kinase 2 (CDK2
131 f CHK1i 18 h after gemcitabine elicited cell division cycle 7 (CDC7)- and cyclin-dependent kinase 2 (
134 HPT-JT is caused by mutations of the cell division cycle 73 (CDC73) gene, located on chromosome 1q
137 ersistence is characterized by a halt in the division cycle, aberrant morphology, and, in the case of
138 cells cease proliferation within one or two division cycles after infection by HTLV-1 or transductio
140 e progression through each stage of the cell division cycle and as such are major targets for deregul
141 s with Cul4A/DDB1 during an unperturbed cell division cycle and both Chk1 phosphorylation and replica
145 Here we show that, when cells leave the division cycle and enter quiescence, telomeres gather in
146 it leads to a reversible exit from the cell division cycle and entry into G0, a cell cycle state cal
147 ission of the midbody at the end of the cell division cycle and for phosphorylation and activation of
148 d Scc2 chromatin association during the cell division cycle and found that the affinity of Scc2 for c
149 RNase enzymes, different phases of the cell division cycle and growth rates, and the existence of no
150 is deposited at centromeres during the cell division cycle and identify an evolutionally conserved p
151 he genome is completely duplicated each cell division cycle and in how the division of cells is spati
153 ormal centriole numbers within a single cell-division cycle and provide insights into the regulation
154 od to examine how cell size impacts the cell division cycle and reaffirm that there is a negative cor
155 s of 53 conditional lethal mutations in cell division cycle and RNA synthesis related genes, revealin
157 ing yeast Saccharomyces cerevisiae, the cell division cycle and sporulation are mutually exclusive ce
158 destruction of Dup is necessary for the cell division cycle and suggest that Geminin inhibition can r
159 to explore the relationship between the cell division cycle and the yeast respiratory oscillation, in
160 ls first proliferate via a canonical mitotic division cycle and then enter an endocycle, resulting in
162 Embryos lacking MEK exhibit faster and extra division cycles and fail to undergo timely midblastula t
164 oordination with other processes during cell division cycles and response to environmental cues.
165 that metabolic cycling does not require cell division cycling and that metabolic synchrony does not r
166 ey cellular system coordinated with the cell division cycle, and major efforts in systems biology cur
169 parasites have a life cycle with unique cell-division cycles, and a repertoire of divergent CDKs and
170 rn division of Drosophila syncytial cortical division cycles, and conventional spindle-directed furro
173 We conclude that mRNA variations during the division cycle, as measured by microarrays, cannot by th
174 kinesin family member 18B (KIF18B) and cell division cycle associated 3 (CDCA3) were of confirmed re
176 cycling occurs during the phases of the cell division cycle associated with mass accumulation in thes
177 ust be able to block progression through the division cycle at key transition points (called "checkpo
178 produces >6,000 nuclei that, during the next division cycle, become encased in plasma membrane in the
180 ow-activity state during an unperturbed cell division cycle but at the same time keeps Chk1 primed to
181 ates the serial events required for the cell division cycle, but no Cdk1 substrate has been identifie
182 Ai treatments does not affect the numbers of division cycles, but the gametophytes exhibit anomalous
183 primarily modulates the duration of the cell-division cycle by controlling the G1/S transition known
184 1 and p27, which regulate the mammalian cell division cycle by inhibiting cyclin-dependent kinases (C
186 ce DNA damage, but also during aberrant cell-division cycles caused by activated oncogenes and inacti
187 ecrease in protein levels of cyclin B1, cell division cycle (Cdc) 25B, and Cdc25C, leading to accumul
189 the interaction of two oscillators, the cell division cycle (CDC) and the yeast metabolic cycle (YMC)
191 n of immunohistochemical markers of the cell division cycle (CDC) in 5 of the 16 neurogenic niches of
194 n the cell cycle, notably cyclin, E2F1, cell division cycle (CDC), and minichromosome maintenance (MC
195 other hand, cyclins A1, A2, B1 and B2, cell division cycle (CDC)2 and its kinase, CDC25 A and B, bud
196 at it segregated at the very end of the cell division cycle: cells showed a single fluorescent focus
197 yeasts displayed highly unconventional cell division cycles compared to those of traditional model y
198 by completing and coordinating two cycles, a division cycle controlling cell size and a DNA replicati
201 ell growth during the G(1) phase of the cell division cycle dilutes the cell cycle inhibitor Retinobl
202 at neighboring nuclei are highly variable in division-cycle duration and that neighbors repel one ano
203 es of oscillations are coupled with the cell division cycle, exhibit period determination by CK1 and
204 on by DDK to form an active CMG [Cdc45 (cell division cycle gene 45), Mcm2-7, GINS (Go, Ichi, Ni, and
205 olved in cellular processes such as the cell division cycle, gene transcription, the DNA damage respo
206 ot show a change in stiffness throughout the division cycle, implying that enzymatic cell wall remode
207 m measurements of DNA replication during the division cycle in cells growing at different, and more r
209 cycling was detected in the G2 phase of the division cycle in fission yeast, consistent with the ide
210 orm essential mitotic functions during every division cycle in mammalian cells, they are required in
211 ukaryotic chromosomes occurs once every cell division cycle in normal cells and is a tightly controll
212 remain relatively unchanged during the cell division cycle in primary human T lymphocytes and in mon
213 couples centrosome duplication from the cell division cycle in prostate cancer cells through CEP57, a
214 gents of malaria, have evolved a unique cell division cycle in the clinically relevant asexual blood
215 iardial flagella undergo a multigenerational division cycle in which the parental eight flagella migr
216 es such as cytokinesis and syncytial nuclear division cycles in Drosophila Pseudocleavage furrow memb
220 ubiquitin ligase during an unperturbed cell division cycle, in response to replicative stress and on
221 ssive cascade known to underlie the parasite division cycle indicating that the unique relationship b
222 is transcribed periodically during the cell division cycle, indicating that properly timed gene expr
223 e transcription is a noisy process, and cell division cycle is an important source of gene transcript
228 The orderly progression through the cell division cycle is of paramount importance to all organis
229 rganisms, divergence from the canonical cell division cycle is often necessary to ensure the proper g
232 frame and critique hypotheses about how the division cycle is regulated in wild-type and mutant cell
233 n such a state, progression through the cell division cycle is reversibly arrested in an orderly mann
237 ll fates; glucose, which stimulates the cell division cycle, is a potent inhibitor of sporulation.
238 rive photosynthetic cell growth and the cell division cycle; it also exhibits a highly choreographed
239 lpha is phosphorylated on Ser123 by the cell division cycle kinase Cdk2 beginning early in S phase an
244 individual cells were followed over multiple division cycles, no direct correlation was observed betw
247 nt work demonstrating similarity between the division cycle of C. crescentus and that of A. tumefacie
250 sitions of microtubule arrays throughout the division cycle of cells lacking a defined centrosome.
254 associated with the G0/G1 phase of the cell division cycle of slowly growing budding yeast, transcri
255 oposed pattern of DNA replication during the division cycle of the K12 cells analysed is not consiste
257 ication, dMyc is dispensable for the mitotic division cycles of both germline and somatic components
258 pact the coordination of the replication and division cycles of Escherichia coli by monitoring the lo
259 at this "consistency test" prevents repeated division cycles of normal cells but might become defecti
264 Large T stabilization domain region to cell division cycle protein 20 (Cdc20) and, possibly, cdc20 h
265 hase-promoting complex (APC/C) bound to Cell division cycle protein 20 (CDC20), and ends upon mitotic
273 uivocally that the roles of CycE/Cdk2 in GSC division cycle regulation and GSC maintenance are separa
275 Cytokinesis, the final stage of the cell division cycle, requires the proper placement, assembly
278 oincide exactly with the S phase of the cell division cycle, suggesting that oxidative metabolism and
279 found that, during the G2 phase of the cell division cycle, TFAP4 is targeted for proteasome-depende
282 embryos display defects in the rapid nuclear division cycles that precede gastrulation in nuclear mig
283 easts reveal alternative mechanisms for cell division cycles that seem likely to expand the repertoir
284 of nutrients triggers an exit from the cell division cycle, the induction of autophagy, and eventual
285 one and only one daughter centriole per cell division cycle, the prevailing view is that centriole ov
288 DNA checkpoint kinase that couples the cell division cycle to the circadian cycle abolishes synchron
289 by coupling transcription kinetics with cell division cycles to delineate how they are combined to re
290 amily A) and the AAA-type ATPase Cdc48 (cell division cycle), Ubr1 directs the substrate to proteasom
291 /DNA synthesis)-phase transition of the cell division cycle, ultimately resulting in decreased cell p
292 impacts of the cht7 mutation during the cell division cycle under nutrient deficiency in light-dark s
295 Cytokinesis is the last step of the cell-division cycle, which requires precise spatial and tempo
296 tes transition between G1 and entry into the division cycle, while CDKB is essential specifically for
297 nd is essential for coordinating the nuclear division cycle with cytokinesis through the cytokinesis