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1 nd the primary motor cortex (M1) of the left-dominant hemisphere.
2 iability in language localization within the dominant hemisphere.
3 in the effect of cTBS on the dominant or non-dominant hemisphere.
4 pread and marked cortical decreases over the dominant hemisphere.
5 erall accelerated degradative process in the dominant hemisphere.
6  with computations normally performed by the dominant hemisphere.
7 d medial temporal lobe in the left, language-dominant hemisphere.
8 ization, as they were evoked only in the non-dominant hemisphere.
9 rocessing is strongly lateralized to the non-dominant hemisphere.
10 and asymmetric predilection for the language-dominant hemisphere?
11  by perisylvian cortex in the left (language-dominant) hemisphere, and thus, left anterior temporal l
12  suggest that networked brain regions in the dominant hemisphere are critical for maintaining an adap
13 that PPA symptoms may arise from a "non-left-dominant"-hemisphere degenerative process.
14  cortical metabolism in a pilot study of the dominant hemisphere in 9 control subjects and provide es
15 vated in both hemispheres but less so in the dominant hemisphere in all HD patients.
16 es showed lower values of NAA and Glx in the dominant hemisphere in all three groups but values of cr
17             Grip aperture deficits following dominant hemisphere lesions were bilateral and were unil
18 oth and efficient movements, whereas the non-dominant hemisphere mediates impedance control, importan
19                                          The dominant hemisphere mediates optimal control of limb dyn
20 es of creatine were selectively lower in the dominant hemisphere of only the presymptomatic patients.
21 ere, we show that resections in the language dominant hemisphere of patients with epilepsy reduce lym
22        Broca's region, which in the language-dominant hemisphere of the human brain plays a major rol
23 rofacial and vocal acts and, in the language dominant hemisphere of the human brain, has been adapted
24 ion of the perisylvian areas of the language-dominant hemisphere) offers novel insights into the rela
25 y outcomes following surgery in the language-dominant hemisphere (p<0.05; eta(p) (2)=0.30).
26 acquisition of language functions by the non-dominant hemisphere speech regions.
27                  We compared subcortical non-dominant hemisphere stroke patients in BCI-FES and Rando
28  of TMEM106B on functions lateralized to the dominant hemisphere, such as language.
29 adult patient with low grade glioma (LGG) in dominant hemisphere suggest brain plasticity process wit
30 y pathology was more intense in the language-dominant hemisphere, the Braak pattern of hippocampo-ent
31 specific from all the uCP population: in the dominant hemisphere, the contralateral CST group showed
32 ated with asymmetric atrophy in the language-dominant hemisphere (usually left), and differs from typ
33 ent polarities of cTBS over dominant and non-dominant hemisphere we delivered AP-PA and postero-anter
34 mporal cortex, these encoding changes in the dominant hemisphere were more likely to be polymodal, wh
35  middle temporal gyrus (MTG) in the language-dominant hemisphere, yet whether there are distinct cont