ライフサイエンスコーパス: don

1 dissolved organic carbon (DOC) and nitrogen (DON).

2 arly 50% of cyanobacterially derived high-MW DON.

3 n the "black box" of uncharacterized high-MW DON.

4 mbrane potential induced by T-2 toxin and/or DON.

5 d cerebrospinal fluid to plasma ratio versus DON.

6 samples according to the EU legal limit for DON.

7 entailing an even more pronounced effect on DON.

8 ctors also play a role in the development of DON.

9 h TaFROG, an orphan wheat protein induced by DON.

10 ents indicate that isoDON is less toxic than DON.

11 he biosensor chip to confirm the identity of DON.

12 and certification requirements for NHAs and DONs.

13 cation were assessed separately for NHAs and DONs.

14 lutamine analog (6-diazo-5-oxo-l-norleucine [DON]).

15 amine antagonist 6-diazo-5-oxo-l-norleucine (DON, 1) has shown robust anticancer efficacy in preclini

16 amine antagonist 6-diazo-5-oxo-l-norleucine (DON, 14) attenuates glutamate synthesis in HIV-infected

17 monisins, beauvericin (BEA), deoxynivalenol (DON), 15-acetyl-deoxynivalenol (15-ADON), 3-acetyl-deoxy

18 The limits of detection were 15microg/kg for DON, 24microg/kg for ZEA and 12microg/kg for T-2 toxin.

19 06, 0.57 and 0.3mug/kgbw/day for the sum of (DON+3-ADON+15-ADON), fumonisins (FB1+FB2+FB3), the sum o

20 Interestingly, the DON-3-glucoside concentrations increased (>300%) under m

21 DON-3-glucoside levels decreased at the end of fermentat

22 resulted in bread with greater quantities of DON-3-glucoside when fermentation occurred at 30 degrees

23 alenol (DON) and deoxynivalenol-3-glucoside (DON-3-glucoside) during the breadmaking process was stud

24 nivalenol (DON), deoxynivalenol-3-glucoside (DON-3-glucoside), 3-acetyldeoxynivalenol (3-ADON), 15-ac

25 An improved UPLC-MS/MS method to detect DON, DON-3-glucoside, 3-ADON, 15-ADON and DOM-1 in wheat bake

26 xamined to evaluate their effects on DON and DON-3-glucoside.

27 toxin) or at one third of the EC level (for DON: 400microg/kg).

28 ested the effect of the glutamine antagonist DON (6-diazo-5-oxo-l-norleucine) on the outcome of NSV i

29 Signal was amplified nearly 25-fold for DON, 90-fold for ZEA and 12-fold for T-2 toxin assay usi

30 Twelve subjects (6 females, age: 24 +/- 2 y) donned a water-perfused suit circulating 34 degrees C wa

31 Deoxynivalenol (DON), a cosmopolitan mycotoxin found in agricultural com

32 DON, a glutamine antagonist, inhibited the proliferation

33 ntally determine the effects of simultaneous DON additions and warming on planktonic community metabo

34 DON additions from wastewater treatment plant effluents

35 GN demonstrates remarkable discrimination of DON against similar mycotoxins (zearalenone and ochratox

36 minase inhibitor 6-diazo-5-oxo-L-norleucine (DON) ameliorates social avoidance and anhedonia-like beh

37 As a proof-of-principle, deoxynivalenol (DON), an important mycotoxin, was captured using an SPR

38 in at concentrations of 100ng/mL for OTA and DON and 3ng/mL for AFB1, spiked in a sample under analys

39 vels for recovery experiments were 89.6% for DON and 93.3% for FB1.

40 y were examined to evaluate their effects on DON and DON-3-glucoside.

41 )-NIR spectroscopy to assess the presence of DON and ergosterol in wheat samples through prediction a

42 aFROG contributes to host resistance to both DON and F. graminearum.

43 samples analyzed for mycotoxins occurrence, DON and FBs were present in 18% and 12%, respectively.

44 ed tumor cell-to-plasma ratio versus that of DON and resulting in a dose-dependent inhibition of cell

45 n Bradi5g03300 increases root sensitivity to DON and the susceptibility of spikes to F. graminearum,

46 31 and 9% higher concentrations of T-2/HT-2, DON and ZEA respectively, but concentrations of the thre

47 tion protocol is essential, including proper donning and doffing of personal protective equipment.

48 semble was standardized, HCP were trained on donning and doffing PPE, and a system of trained observe

49 system of trained observers supervising PPE donning and doffing was implemented.

50 ssolved organic carbon and nitrogen (DOC and DON) and aliphatic DOM contribution for five decades.

51 The stability of deoxynivalenol (DON) and deoxynivalenol-3-glucoside (DON-3-glucoside) du

52 incidence of the mycotoxins deoxynivalenol (DON) and fumonisin B1 (FB1) in industrial beers.

53 oflora and the occurrence of deoxynivalenol (DON) and fumonisins (FBs) in malting barley.

54 onship between T-2 toxin and deoxynivalenol (DON) and the risk of Kashin-Beck disease is still contro

55 acia crassicarpa monoculture had the highest DON, and 10-mixed species plantation had the highest DOC

56 TA), aflatoxin B1 (AFB1) and deoxynivalenol (DON), are subject to strict regulations and recommendati

57 These results suggest DON as a strong candidate for an effective adjunctive th

58 s selective and sensitive devices to analyse DON as the conventional methods are complex and time-con

59 ction by stimulating TRI gene expression and DON-associated cellular differentiation in F. graminearu

60 that TRI6 is essential for the regulation of DON biosynthesis by cAMP signalling but elevated PKA act

61 AMP phosphodiesterase to negatively regulate DON biosynthesis in F. graminearum.

62 uired for its normal functions in growth and DON biosynthesis.

63 ally recovered the defect of tri10 mutant in DON biosynthesis.

64 ergosterol and another that was analysed for DON by HPLC.

65 Dissolved organic nitrogen (DON) can be a significant part of the reactive N in aqua

66 technique for ergosterol quantification and DON classification of samples according to the EU legal

67 of nitrogen-rich dissolved organic nitrogen (DON) compounds that are ubiquitously distributed in mari

68 Dissolved organic nitrogen (DON) comprises the largest pool of fixed N in the surfac

69 FAC components were positively correlated to DON concentration.

70 The lowest DOC and DON concentrations were both observed in Eucalyptus urop

71 that agriculture promotes increased DOC and DON concentrations, entailing an even more pronounced ef

72 Dissolved organic nitrogen (DON) concentrations frequently exceed those of dissolved

73 ssolved organic carbon and nitrogen (DOC and DON) concentrations in supra-permafrost groundwater (SPG

74 on and conjugation suggest that the speed of DON conjugation rather than the increase of D3G per se i

75 These enzymes reduced the DON content by 10-14% at 45 degrees C.

76 at 30 degrees C, these enzymes increased the DON content by 13-23%.

77 The effect of xylanase and alpha-amylase on DON content depended on the fermentation temperature.

78 chis rate, Fusarium-diseased kernel rate and DON content in harvested kernels, while silencing WFhb1-

79 Further, treatment with DON decreased hyaluronan and collagen in the tumor micro

80 T-2 toxin or DON decreased proliferation in a time- and concentration

81 Our results showed that DON decreased the self-renewal potential and metastatic

82 A DON degradation of 6% (crackers), 5% (biscuits) and 2% (

83 sence of 4 out of 12 studied trichothecenes: DON (deoxynivalenol), 15AcDON (15-acetyl-deoxynivalenol)

84 The stability of deoxynivalenol (DON), deoxynivalenol-3-glucoside (DON-3-glucoside), 3-ac

85 In vivo study showed that T-2 toxin and DON did not accumulate preferentially in the knee joint

86 Frustration with donning/doffing was especially apparent when needing sup

87 An improved UPLC-MS/MS method to detect DON, DON-3-glucoside, 3-ADON, 15-ADON and DOM-1 in wheat

88 We have elucidated the fate of DON during baking of crackers, biscuits and bread, which

89 administrator (NHA) and director of nursing (DON) education and certification is related to resident

90 ggested that high-molecular-weight (high-MW) DON exists only in two closely related forms, the second

91 administration of 6 delivered 11-fold higher DON exposure to tumor (target tissue; AUC(0- t) = 5.1 nm

92 counts for an important proportion of marine DON flux.

93 mL) in the presence or absence of 0.5 mug/mL DON for indicated time points.

94 DOM, as well as dissolved organic nitrogen (DON) forms in headwater streams, including temporal vari

95 The mycotoxin deoxynivalenol (DON) frequently contaminates cereals crops worldwide, an

96 d both the photolabile and the photoproduced DON from both natural and anthropogenic sources.

97 teins, amino sugars, lignins, and tannins in DON from runoff and pond outflow water.

98 the turnover of dissolved organic nitrogen (DON) from both natural and anthropogenic sources at the

99 In ICUs where healthcare workers donned gloves and gowns for all patient contact, patient

100 OTA is more stable than DON in a baking treatment.

101 The major part of DOC and DON in agricultural and forest reference streams is expo

102 The level of DON in breads without added enzymes was reduced (17-21%)

103 graminearum, highlighting the involvement of DON in early steps of infection.

104 and cost-effective alternative for detecting DON in food and feed samples.

105 tinal homogenates yet was readily cleaved to DON in P493B lymphoma cells, exhibiting a 55-fold enhanc

106 determined the distribution of T-2 toxin and DON in Sprague-Dawley (SD) rats after a single dose expo

107 iodegradability and molecular composition of DON in the urban stormwater runoff and outflow water fro

108 t in plasma and preferentially activate over DON in tumor.

109 Few methods exist to track the sources of DON in watersheds.

110 he biosynthesis of mycotoxin deoxynivalenol (DON) in Fusarium graminearum is regulated by two pathway

111 The biodegradability of DON increased from 10% in the stormwater runoff to 40% i

112 Furthermore, Sch A lowered DON-induced catalase, superoxide dismutase and glutathio

113 In addition, Sch A decreased the DON-induced cyclooxygenase-2 expression and prostaglandi

114 Sch A appeared to protect against DON-induced cytotoxicity in HT-29 cells, and significant

115 ne), was investigated in HT-29 cells against DON-induced cytotoxicity, oxidative stress and inflammat

116 hypothesis that EPA and DHA could alleviate DON-induced injury to intestinal porcine epithelial cell

117 ese results suggest that EPA and DHA prevent DON-induced intestinal cell injury and enhance barrier f

118 chanistic studies suggest that Sch A reduced DON-induced oxidative stress by down-regulating heme oxy

119 that Sch A exerted a cytoprotective role in DON-induced toxicity in vitro, and it would be valuable

120 This may be mediated by preventing DON-induced translocation of nuclear factor-kappaB, as w

121 dwide, and are a public health concern since DON ingestion may result in persistent intestinal inflam

122 These results suggest that T-2 toxin and/or DON inhibit proliferation and induce apoptosis through a

123 In vitro studies showed that DON inhibited stimulus-induced proliferation of lymphocy

124 However, because DON inhibited the immune response to infection, clearanc

125 Herein we demonstrate that DON inhibits glutamine metabolism and provides antitumor

126 has been related to the ability to conjugate DON into a glucosylated form, deoxynivalenol-3-O-glucose

127 ut introduce larger quantities of potent LMW-DON into coastal systems.

128 , we show that the Bradi5g03300 UGT converts DON into D3G in planta.

129 Don is an endemic species of the dune systems of the Atl

130 Unlike inorganic N (nitrate and ammonium) DON is comprised of many different molecules of variable

131 So far it is not clear i) to which extent DON is degraded during baking and ii) if a degradation r

132 ly and efficient UGT-mediated conjugation of DON is necessary and sufficient to establish resistance

133 We found that in predenitrification BNR, LMW-DON is released during the post-aerobic step following t

134 6-Diazo-5-oxo-l-norleucine (DON) is a glutamine antagonist with robust anticancer ef

135 Deoxynivalenol (DON) is considered to be one of the most important conta

136 Deoxynivalenol (DON) is one of the most common mycotoxins that contamina

137 Dysthyroid optic neuropathy (DON) is the commonest cause of blindness in thyroid asso

138 Dissolved organic nitrogen (DON) is the nitrogen (N)-containing component of dissolv

139 The most stable compound (5c, methyl-POM-DON-isopropyl-ester) was evaluated in monkeys, where it

140 Don) Lemmon is less well documented than subgenus Pinus

141 rley, while still efficiently eliminated the DON levels and toxicogenic fungal biomass as quantified

142 t, leading to increased dissolved organic N (DON) loading.

143 e advantage in vivo over strains lacking the don locus.

144 rate that the chemical structures of high-MW DON may be much more diverse than previously thought.

145 sis of the effects of wild-type fungus and a DON minus mutant derivative.

146 ave good performance in increasing soil DOC, DON, N mineralization and plant biodiversity, we recomme

147 ms, the contribution of DOC and particularly DON occurring in the form of nonhumic high-molecular-wei

148 to compare the cytotoxicity of T-2 toxin and DON on cell lines representative of cell types encounter

149 nase inhibitors, 6-Diazo-5-oxo-L-norleucine (DON) or CB-839, hypersensitize cancer cells to alkylatin

150 al details of the previously uncharacterized DON pool and outlines the means to further advance our u

151 Many components of the DON pool have been shown to supply nitrogen nutrition to

152 fraction of the dissolved organic nitrogen (DON) pool, can form dichloroacetamide (DCAcAm) during ch

153 hich contribute to the greatest identifiable DON portion in natural waters, is limited.

154 Furthermore, cross-reacting conjugates of DON present in a naturally contaminated beer could be su

155 y, we synthesized a series of tumor-targeted DON prodrugs designed to circulate inert in plasma and p

156 Deoxynivalenol (DON) produced by the pathogen is an important mycotoxins

157 rium head blight (FHB) and a deoxynivalenol (DON) producer.

158 Don, produces hundreds of biologically active monoterpen

159 ile cultures, and repression of TRI genes in DON-producing cultures.

160 In contrast, Fgskn7 mutants were reduced in DON production and defective in H2 O2 -induced TRI gene

161 ys and further regulates fungal development, DON production and plant infection in F. graminearum.

162 osine monophosphate (cAMP) treatment induced DON production by stimulating TRI gene expression and DO

163 Cellular differentiation associated with DON production was stimulated by exogenous cAMP or delet

164 irement of TRI10 for TRI gene-expression and DON production, and Pde2 is the major cAMP phosphodieste

165 se and reactive oxygen species (ROS) induces DON production, in this study, we characterized function

166 GK3 is important for growth, conidiogenesis, DON production, pathogenicity, and stress responses in F

167 It was significantly reduced in DON production, TRI gene expression, and virulence.

168 d intracellular PKA activities and increased DON production.

169 athogenic in infection assays and blocked in DON production.

170 FgAP1 or FgATF1 had no significant effect on DON production.

171 l/sexual differentiation and deoxynivalenol (DON) production, respectively.

172 eased toxisome formation and deoxynivalenol (DON) production, which was largely depended on cAMP sign

173 duced ascospore ejection and deoxynivalenol (DON) production.

174 is one of the main processes that shapes the DON quality in aquatic systems regardless of its origina

175 ersal gelator should be left in the hands of Don Quixote and as researchers we must focus on identify

176 molar DOS versus dissolved organic nitrogen (DON) ratios of 0.11 +/- 0.024 in Atlantic surface water

177 ted, yet the types and parameters useful for DON remain inconclusive.

178 glutamine analog 6-diazo-5-oxo-L-norleucine (DON) rescues mice from CM when administered late in the

179 of<40, 0.1-0.2 and<10ng/mL for OTA, AFB1 and DON, respectively, fall within the majority of currently

180 1.17 mg/kg and 501 ug/kg for ergosterol and DON, respectively.

181 (CERC), Leading Edge Endowment Fund (LEEF), Don Rix BC Leadership Chair in Genetic Medicine, Nationa

182 However, masking both DON's amine and carboxylate functionalities imparted suf

183 To enhance DON's therapeutic index, we utilized a prodrug strategy

184 To circumvent DON's toxicity, we synthesized a series of tumor-targete

185 Additionally, treatment with DON sensitized pancreatic tumors to anti-PD1 therapy, re

186 Eyes of TAO patients with DON showed delayed P100 latencies, decreased P100 amplit

187 ural soils as important DOC and particularly DON sources to headwater streams.

188 Overall, the predicted fraction of nonpoint DON sources was consistent with previous reports of incr

189 matrix (EEM) fluorescence of eight discrete DON sources was measured and modeled with parallel facto

190 HT-29 cells, and significantly lessened the DON-stimulated intracellular reactive oxygen species and

191 Our results suggest that PAs may be common DON substrates for marine bacterioplankton, in line with

192 cle enlargement also occur in orbits without DON suggests that other factors also play a role in the

193 Data analysis workflows typically don't access the full breadth of information about instr

194 oblem, we find robustly that inferences that don't account for post-death transmission tend to undere

195 Current methods don't adequately address this issue since they rely on e

196 While simple resource-competition models don't allow for coexistence of a large number of species

197 bout having to initiate discussions ("If you don't ask, nobody is going to tell you anything") and th

198 notropic glutamate receptor family, yet they don't bind glutamate and are considered orphan.

199 However, existing measures don't capture whether persons who inject drugs (PWIDs) s

200 s modifies AD risk for those individuals who don't carry the epsilon4 variant of APOE.

201 Moreover, (3) social rewarders don't condition on efficacy or other difficult-to-observ

202 ts that superficially reassemble CRISPRs but don't contain diverse spacers and have no companion cas

203 But we don't do justice to that view -- how could we?

204 But we don't do justice to that view-how could we, older mammal

205 But we don't do justice to that view-how could we?

206 the noncanonical up-regulation of so-called don't eat me molecules on inflamed phagocytes, which red

207 deficiency preserved the expression of CD47 (don't eat me signal) on cardiac MSCs after both TLR4 sti

208 specific immune checkpoint that engages the "don't eat me" signal CD47 expressed on tumors and normal

209 CD47, a cell-surface protein that acts as a "don't eat me" signal co-opted by cancers to evade innate

210 lexity has limited understanding of how the "don't eat me" signal is transduced biochemically.

211 alpha (SIRPalpha) on macrophages to CD47, a "don't eat me" signal on cancer cells, prevents macrophag

212 investigated the impact of eliminating the "don't eat me" signal on tumor growth and tumor-TME inter

213 CD47 acts as a "don't eat me" signal that protects cells from phagocytos

214 cells despite their expression of the CD47 "don't eat me" signal.

215 egulated gene in all human cancers and is a "don't eat me" signal; blocking it with antibodies leads

216 gocytosis by displaying inhibitory ligands ("don't eat me" signals).

217 transduces anti-phagocytic signals from the 'don't eat me' CD47 ligand through negative Shp1 signalin

218 The released anti-CD47 antibody blocks the 'don't eat me' signal in cancer cells, thereby increasing

219 In particular, cancer cells express CD47, a 'don't eat me' signal that interacts with signal regulato

220 ple, inhibition of the macrophage SIRPalpha 'don't eat me' signal with a SIRPalpha-CD45 RIPR molecule

221 ent neoplastic cells generate this dominant 'don't eat me' signal, we analyse the CD47 regulatory gen

222 tibodies that antagonize the interaction of 'don't eat me' signals with their macrophage-expressed re

223 of anti-phagocytic surface proteins called 'don't eat me' signals-including CD47(1), programmed cell

224 the presence of additional, as yet unknown 'don't eat me' signals.

225 that aged keratinocytes near the wound edge don't efficiently upregulate Skints or activate STAT3.

226 rds, how will we know what to look for if we don't first know what is stable and what is dynamic, and

227 may give us misleading results if the models don't fit the data but are blindly used.

228 st changes will very likely be those that we don't foresee, even now.

229 of new treatments is meaningless if patients don't have access to them.

230 e only available in a laboratory setting and don't have the portability to meet the needs in point-of

231 hnical improvement ("As you get older if you don't have the stimulation from surgery to get better or

232 h benefits, although most scientific reports don't include the analysis of both properties.

233 and Robert Shepherd, respectively, "Dennis, don't just be a test tube scientist, do something to hel

234 What consumers don't know about genetically modified food, and how that

235 lity, but researchers new to computing often don't know where to start.

236 n-where some is right, some is wrong, but we don't know which.

237 "Don't know" responses concerning clinical interventions

238 duce verbal acknowledgments of ignorance ("I don't know").

239 s of nature-based recreation and tourism, we don't know.

240 they used low-consensus terms or responded "don't know." However, even low-consensus color terms gro

241 larity and overall protein homology and thus don't mirror the complex multidomain architecture and th

242 that up to two-thirds at risk to inherit LS don't participate.

243 Most clustering algorithms don't properly account for ambiguity in the source data,

244 e already have 7 billion people on earth and don't really need more.

245 d rewards can be exploited if their partners don't reciprocate.

246 architecture-particularly if these elements don't repeat in periodic patterns or collectively occupy

247 Hoerl & McCormack claim that animals don't represent time.

248 l ability to manufacture cell therapies that don't require customization for each patient.

249 responses (e.g. optokinetic nystagmus) that don't require involvement of visual cortex and higher or

250 is dominated by the magnetic field yet they don't require large currents.

251 ing is that actually, a lot of times, people don't see the world accurately.

252 onment, though most contemporary biomonomers don't spontaneously oligomerize under mild conditions wi

253 found that about 10-20% of stage IB patients don't survive 5 years, whereas patients with advanced-st

254 But we don't think that the existing results provide strong sup

255 However, we don't understand the influence of obesity on susceptibil

256 , 0.24; 95% CI, 0.09-0.70), illuminated walk/don't walk signs (OR, 0.16; 95% CI, 0.03-0.92), painted

257 negations applied to action contents (e.g., "don't write the letter") impede the activation of motor

258 the literature, negations such as "not" or "don't" reduce the accessibility in memory of the concept

259 ed if binary patterns representing match and don't-care positions are used as a filter, such that onl

260 ry factors PD-L1, HLA-G, and the macrophage "don't-eat me" signal CD47 from the AAVS1 safe harbor loc

261 is composed of what has become known as the 'don't-eat me' signal CD47, which is a protein broadly ex

262 at-me signals on tumor cells is countered by don't-eat-me signals such as CD47, which binds macrophag

263 its an anti-phagocytic signal, known as the "don't-eat-me" signal, to macrophages upon engaging its r

264 prediction uncertainty outperform those that don't.

265 d suggest alternative regimens for those who don't.

266 almost infinite number of movements, but we don't: the motions executed to pick up the remote are hi

267 e number of visual stimulus patterns, but we don't: we distinguish two actors' faces with ease but tw

268 s tests that pass the filter from those that don't?

269 cation systems produced larger amount of LMW-DON than CAS.

270 found between phytoplankton biomass and LMW-DON than other N forms.

271 ant produced a significantly higher level of DON than the tri10 mutant.

272 that wheat bran and flour may contain hidden DON that may be enzymatically released during the breadm

273 significantly less reddish immediately upon donning the glasses.

274 ereas the tri6 pde2 mutant failed to produce DON, the tri10 pde2 double mutant produced a significant

275 considerable attention over the potential of DON to provoke oxidative stress.

276 f freshwater, 14-71 kg of DOC, and 1-4 kg of DON to the coastal ocean per km of shoreline per day dur

277 modulation with 6-diazo-5-oxo-L-norleucine (DON) to decrease or O-(2-acetamido-2-deoxy-D-glucopyrano

278 mine antagonist, 6-diazo-5-oxo-l-norleucine (DON), to target a metabolic program that underlies both

279 DON treatment for 7 days from the time of infection dela

280 Remarkably, within hours of DON treatment mice showed blood-brain barrier integrity,

281 Don) under 'normal' combinations of temperature and prec

282 This strategy may provide a path to DON utilization in glioblastoma multiforme patients.

283 Neuse River Basin showed that while >70% of DON was attributed to natural sources, nonpoint sources,

284 runoff to 40% in the pond outflow water and DON was less aromatic and had lower overall molecular we

285 DON was not detected in any of the analyzed samples wher

286 ctrometry based quantification revealed that DON was partially degraded to isoDON (1.3-3.9%), norDON

287 Although photoproduction of DON was prominent with natural dissolved organic matter

288 When in vivo treatment with DON was stopped, paralytic disease developed along with

289 DON was the most commonly found, being detected in 4 sam

290 incidence of HT-2 toxin and deoxynivalenol (DON) were found in 9.1% and 59.7% of total samples, resp

291 TA), aflatoxin B1 (AFB1) and deoxynivalenol (DON) which are strictly regulated food contaminants with

292 at response to the mycotoxin deoxynivalenol (DON), which is a virulence factor produced by the causal

293 ecenes on cereals, including deoxynivalenol (DON), which is harmful for humans and animals.

294 ow-molecular-weight dissolved organic N (LMW-DON), which is highly bioavailable and stimulates phytop

295 was decreased by reducing O-GlcNAc levels by DON, which also led to suppressed expression of the cyto

296 nal CT scans of 13 patients with and without DON who subsequently underwent orbital decompression wer

297 Don with that of conventional supercritical CO2 (SC-CO2)

298 Nursing homes led by DONs with Bachelor's degrees or higher plus certificatio

299 ulcer rates were higher in facilities led by DONs with more education.

300 d for three Fusarium toxins, deoxynivalenol (DON), zearalenone (ZEA) and T-2 toxin.