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1 s of capsaicin-responsive neurons in primate dorsal root ganglion.
2 a the release of substance P in the skin and dorsal root ganglion.
3 eferentially expressed in C fibers in lumbar dorsal root ganglions.
4 ressed in a subset of sensory neurons of the dorsal root ganglion and in cutaneous mechanoreceptors k
5 antly over time, a number of changes, in the dorsal root ganglion and in dorsal horn observed after t
6 NR2B receptor protein expression in both the dorsal root ganglion and spinal dorsal horn ipsilateral
7 mpairs channel slow inactivation within both dorsal root ganglion and superior cervical ganglion neur
8 the peripheral nervous system within sensory dorsal root ganglion and sympathetic ganglion neurons an
9 m channel is preferentially expressed within dorsal root ganglion and sympathetic ganglion neurons an
11 be responsible for pathology observed in the dorsal root ganglion and the sensory ganglionopathy docu
12 conditions originate in the periphery, where dorsal root ganglion and trigeminal ganglion neurons fee
13 s: cortical (embryonic rat), embryonic chick dorsal root ganglion, and P-19 (mouse embryonic carcinom
16 d force apparatus, we demonstrate that chick dorsal root ganglion axons exhibit a tension buffering o
18 siveness of thin fibre afferents not only at dorsal root ganglion, but also at muscle tissue levels.
19 ple host axons when co-cultured with primary dorsal root ganglion cells and formed myelin after trans
20 n the plasma membrane of pancreatic beta and dorsal root ganglion cells and link steroid hormone sign
23 contrast to its effect on the hair cell and dorsal root ganglion cells in culture; (iv) SLV recyclin
25 sensory terminals of primary mechanosensory dorsal root ganglion cells, so the presence of such a sy
27 tion of lymphoid cell kinase in Schwann cell-dorsal root ganglion cocultures and dorsal root ganglion
28 The administration of NO donors to primary dorsal root ganglion cultures prevents axonal degenerati
30 y of glial cells and reduced spinal cord and dorsal root ganglion cytokine levels without affecting p
32 y establishment and reactivation using human dorsal root ganglion-derived neuronal HD10.6 cells as an
33 hat CXCL12 and CXCR4 were upregulated in the dorsal root ganglion (DRG) after chronic compression of
34 injury causes down-regulation of MORs in the dorsal root ganglion (DRG) and diminishes the opioid eff
36 ed and manipulated MrgprA3(+) neurons in the dorsal root ganglion (DRG) and found that they exclusive
37 sensory neuronal preparations, such as whole dorsal root ganglion (DRG) and hindpaw tissues, revealed
38 titutive autophagosome biogenesis in primary dorsal root ganglion (DRG) and hippocampal cultures.
39 KChIP2, KChIP3, DPP6, and DPP10 in adult rat dorsal root ganglion (DRG) and spinal cord by immunohist
40 hannel NaV1.7 is preferentially expressed in dorsal root ganglion (DRG) and sympathetic ganglion neur
41 tion after axotomy was abolished in both the dorsal root ganglion (DRG) and the distal sciatic nerve.
42 ncreases in osmolality excite isolated mouse dorsal root ganglion (DRG) and trigeminal ganglion (TG)
44 port that stimulated membrane enlargement in dorsal root ganglion (DRG) axons is triggered by intra-a
45 tigate mRNA expression in colonic tissue and dorsal root ganglion (DRG) cells isolated from 3- and 24
47 at a dose of 100 mpk PO due to insufficient dorsal root ganglion (DRG) exposure attributed to poor m
48 ge-diameter (>30 mum) sensory neurons of the dorsal root ganglion (DRG) express distinct combinations
50 IL-6 in skin and phosphorylation of ERKs in dorsal root ganglion (DRG) in a dose-dependent manner.
51 pression and functional role of Panx1 in the dorsal root ganglion (DRG) in the development of chronic
52 ion of HIV infection and is characterized by dorsal root ganglion (DRG) inflammation and intraepiderm
54 gated potassium channel subunit Kcna2 in the dorsal root ganglion (DRG) is critical for DRG neuronal
55 Several recent papers have described a human dorsal root ganglion (DRG) neuron culture model and huma
56 nsgenic mice lacking Merkel cells had normal dorsal root ganglion (DRG) neuron numbers, but fewer DRG
58 hondrial trafficking plays a central role in dorsal root ganglion (DRG) neuronal cell survival and ne
60 nduced spontaneous pain and axonal injury of dorsal root ganglion (DRG) neurons and inhibited CCI-evo
61 um channel that is specifically expressed in dorsal root ganglion (DRG) neurons and peripheral nerve
62 ubcellular distribution of mammalian PATs in dorsal root ganglion (DRG) neurons and, strikingly, foun
63 satellite glial cells (SGCs) surrounding the dorsal root ganglion (DRG) neurons appears to play a rol
64 P1 shows remarkably decreased RNA binding in dorsal root ganglion (DRG) neurons compared with wild-ty
66 fast-inactivating Kv3.4 potassium current in dorsal root ganglion (DRG) neurons contributes to the hy
67 voltage-gated sodium channels (VGSC) on the dorsal root ganglion (DRG) neurons controlling electrica
70 v5 are expressed by developing TrkA-positive dorsal root ganglion (DRG) neurons during the period of
72 s axon growth from neurons; adult miR-155 KO dorsal root ganglion (DRG) neurons extend 44% longer neu
73 en implicated in the hyperexcitable state of dorsal root ganglion (DRG) neurons following direct inju
74 arization-evoked Ca2+ transient is larger in dorsal root ganglion (DRG) neurons from tumor-bearing mi
76 e firing frequency and spontaneous firing of dorsal root ganglion (DRG) neurons have recently been id
77 ng Na(V)1.7 mutation, which is known to make dorsal root ganglion (DRG) neurons hyperexcitable, but d
80 e find that chemogenetic activation of adult dorsal root ganglion (DRG) neurons improves axon growth
81 GKIalpha) that regulates axon bifurcation of dorsal root ganglion (DRG) neurons in the spinal cord.
83 gnitude and properties by voltage clamp from dorsal root ganglion (DRG) neurons in vivo, after classi
84 -expression network analysis, we categorized dorsal root ganglion (DRG) neurons into different subtyp
85 hat the activity of TRPM3 expressed in mouse dorsal root ganglion (DRG) neurons is inhibited by agoni
86 tion of G(alphaq)-coupled receptors in mouse dorsal root ganglion (DRG) neurons isolated from both se
87 nduced current with activation of ASIC(3) in dorsal root ganglion (DRG) neurons of control rats and r
88 es putative mechanosensitive channels in the dorsal root ganglion (DRG) neurons of these afferents.
89 mate transporter 3-lineage (Vglut3(lineage)) dorsal root ganglion (DRG) neurons play an important rol
90 uronal cell line (NG108-15) and with primary dorsal root ganglion (DRG) neurons resulted in significa
91 cell patch-clamp recordings from dissociated dorsal root ganglion (DRG) neurons revealed enhanced tet
92 rophysiological characterization of isolated dorsal root ganglion (DRG) neurons revealed that RPRFami
96 t IgG-IC directly excited a subpopulation of dorsal root ganglion (DRG) neurons through the neuronal
97 ciatic nerve allows the central processes of dorsal root ganglion (DRG) neurons to spontaneously rege
98 4-well format axon degeneration assay in rat dorsal root ganglion (DRG) neurons using a trophic facto
99 native background K(+) conductance of mouse dorsal root ganglion (DRG) neurons was examined by the w
100 Depolarization-induced calcium influx in dorsal root ganglion (DRG) neurons was inhibited by both
101 sly reported that sustained ASIC currents in dorsal root ganglion (DRG) neurons were enhanced by natu
103 hat voltage-gated sodium channels (VGSCs) in dorsal root ganglion (DRG) neurons were sensitized in a
104 that reduction of GRK2 or increased EPAC1 in dorsal root ganglion (DRG) neurons would promote the tra
105 uring the development and differentiation of dorsal root ganglion (DRG) neurons, and on adult DRG neu
106 ts were performed on small-diameter (<30 um) dorsal root ganglion (DRG) neurons, cultured from fentan
109 thereby producing hyperexcitability of small dorsal root ganglion (DRG) neurons, which include nocice
110 ury induces changes in gene transcription in dorsal root ganglion (DRG) neurons, which may contribute
125 with microglia BV-2 cells exposed to G-CSF, dorsal root ganglion (DRG) nociceptors become hyperexcit
126 melanoma and normal neural tissues including dorsal root ganglion (DRG) produce PD-L1 that can potent
128 ort functional expression of SHANK3 in mouse dorsal root ganglion (DRG) sensory neurons and spinal co
135 ta) might act directly on nociceptors in the dorsal root ganglion (DRG) to cause pain sensitization.
136 neurons infected in vivo was examined using dorsal root ganglion (DRG) xenografts maintained in mice
137 dent outgrowth and traction forces from PNS (dorsal root ganglion (DRG)) and CNS (hippocampal) neuron
138 set of nonpeptidergic nociceptors within the dorsal root ganglion (DRG), and knockdown of Kv4.3 selec
139 etermine the protein levels of P2X(3) in the dorsal root ganglion (DRG), and the whole cell patch cla
140 ceptive neurons in the adjacent uninjured L4 dorsal root ganglion (DRG), as revealed by both in vivo
141 reduction in K(+) channel expression in the dorsal root ganglion (DRG), but little is known about th
143 icular pain model, local inflammation of the dorsal root ganglion (DRG), we observed marked increases
144 in nociceptor (pain-sensing) neurons of the dorsal root ganglion (DRG), where they transmit the larg
145 agi-1 are largely unknown, but we found that dorsal root ganglion (DRG)-specific knockdown of Magi-1
151 vivo forepaw muscles/median and ulnar nerves/dorsal root ganglion (DRG)/spinal cord (SC) recording pr
152 n in capsaicin-sensitive nociceptors using a dorsal root ganglion (DRG)/spinal cord neuron co-culture
153 ar CFA injection and in the ipsilateral L4/5 dorsal root ganglions (DRGs) 1 and 3 days after CFA inje
154 ly by small-sized primary sensory neurons in dorsal root ganglions (DRGs) that coexpress the itch sig
155 efficacy could be correlated with the mouse dorsal root ganglion exposure and Na(V)1.7 potency assoc
157 ann cell-dorsal root ganglion cocultures and dorsal root ganglions from Lck(-/-) mice show a reductio
159 at the Ascl1 promoter, isolated from murine dorsal root ganglion (hypermethylated) and striated cell
160 permeant cAMP analog, 8-bromo cAMP, into the dorsal root ganglion induced mechanical hyperalgesia and
161 analysis (TCA), we focused on large-diameter dorsal-root ganglion (L-DRG) neurons with myelinated axo
162 that signals emanating from within the mouse dorsal root ganglion mediated partly by early-born neuro
163 imulated oligodendrocytes was validated in a dorsal root ganglion microfluidics chamber platform.
164 rsal column lesions and reduced in models of dorsal root ganglion neuron (DRGN) axon regeneration.
165 n of the effects of the Del-L955 mutation on dorsal root ganglion neuron hyperexcitability with those
166 ng a CD4-Na(v)1.2/L2 reporter protein in rat dorsal root ganglion neuron-Schwann cell myelinating coc
168 adapting, mechanically activated currents in dorsal root ganglion neuronal cultures are absent in Pie
169 voltage-gated sodium channel Nav1.7 underlie dorsal root ganglion neuronal hyperexcitability and pain
171 that TLRs 3, 7, and 9 are expressed by human dorsal root ganglion neurons (DRGNs) and in cultures of
173 , PKCdelta, and PKC was also observed in the dorsal root ganglion neurons after chronic treatment wit
174 receptor that increases cAMP, in a subset of dorsal root ganglion neurons and also within neurons of
176 ase ion transporter in both cultured primary dorsal root ganglion neurons and injured peripheral nerv
177 for PKCepsilon was found in the majority of dorsal root ganglion neurons and intensely labeled lamin
178 nnel isoform Na(V)1.7 is highly expressed in dorsal root ganglion neurons and is obligatory for nocic
179 illin(Cre/+) mice completely ablates MORs in dorsal root ganglion neurons and reduces the MOR express
180 t channels enhance resurgent currents within dorsal root ganglion neurons and show by current-clamp t
181 ch MOR expression is completely deleted from dorsal root ganglion neurons and substantially reduced i
182 to alter TTX-S Na+ current density in medium dorsal root ganglion neurons and, importantly, mechanica
184 s reduces desensitization of native TRPV1 in dorsal root ganglion neurons as well as of recombinant T
185 f3r gene expression could not be detected in dorsal root ganglion neurons by single-cell RT-PCR.
186 that in small PLCdelta4(-/-) TRPM8-positive dorsal root ganglion neurons cold, menthol and WS-12, a
187 n; and (5) electrophysiology recordings from dorsal root ganglion neurons collected during remission
188 tion of the alpha2delta-1 subunit in sensory dorsal root ganglion neurons contributes to the generati
190 iated inhibition of M current in nociceptive dorsal root ganglion neurons did not reduce the efficacy
192 Here, we show that only 43% of CQ-excited dorsal root ganglion neurons expressed TRPA1; as expecte
194 pic screen, we find that CAST/Ei mouse adult dorsal root ganglion neurons extend axons more on CNS my
195 tion of NFATc3 and NFATc4 in hippocampal and dorsal root ganglion neurons following electrically evok
196 cid Early 1 (RAE1), is re-expressed in adult dorsal root ganglion neurons following peripheral nerve
197 1.6 is involved in the functional changes of dorsal root ganglion neurons following vincristine treat
198 -S) and resistant (TTX-R) sodium currents in dorsal root ganglion neurons following vincristine treat
202 between TRPV1 and Pirt, and that dissociated dorsal root ganglion neurons from Pirt knock-out mice ha
203 ne also acts on endogenous TRPA1 in cultured dorsal root ganglion neurons from rats and in the entero
204 voltage-clamp recordings of small and medium dorsal root ganglion neurons from vincristine-treated an
206 erior cervical ganglion neurons, and renders dorsal root ganglion neurons hyperexcitable and superior
207 ons in sodium channel Na(V)1.7, which render dorsal root ganglion neurons hyperexcitable, are present
210 from synaptic vesicles along axons of mouse dorsal root ganglion neurons in culture promotes myelin
212 We now find that treatment of adult rat dorsal root ganglion neurons in vitro with LRP1 agonists
213 rocytes are functional and can myelinate rat dorsal root ganglion neurons in vitro, and form myelin i
214 c or optogenetic depolarization of GABAergic dorsal root ganglion neurons in vivo reduced acute and c
216 K-2 channels was also demonstrated in native dorsal root ganglion neurons indicating that heterodimer
217 We detected increased Trpm3 mRNA levels in dorsal root ganglion neurons innervating the inflamed pa
218 t spontaneous action potential generation in dorsal root ganglion neurons is associated with radicula
222 In addition, genetically deleting GluN1 in dorsal root ganglion neurons or alpha2delta-1 genetic KO
223 channels in rat endocrine GH(3) cells, mouse dorsal root ganglion neurons or cardiac myocytes, and re
224 affolds Na(V)1.8 and Slack K(Na) channels in dorsal root ganglion neurons regulating excitability and
226 mouse and human by a subpopulation of TRPV1+ dorsal root ganglion neurons specialized in detecting pa
227 .2/Kv7.3 heteromers and native M currents in dorsal root ganglion neurons suggest the following concl
228 at dorsal root injury model, transduction of dorsal root ganglion neurons to express kindlin-1 promot
230 onstellation pharmacology to investigate rat dorsal root ganglion neurons using two models of periphe
231 teers, sensitized TRPV1 in mouse nociceptive dorsal root ganglion neurons via HRH1; this effect could
232 ratching behavior and activation of cultured dorsal root ganglion neurons was dependent on Mrgprs rat
233 Retrograde labelling of bladder-projecting dorsal root ganglion neurons was used to investigate exp
235 two distinct types of cutaneous nociceptive dorsal root ganglion neurons were identified as respondi
238 -resistant sodium current, in small-diameter dorsal root ganglion neurons, an effect that was attenua
240 n prostate cells, PC3 prostate cancer cells, dorsal root ganglion neurons, and hippocampal neurons.
241 Ca(V)2 channels in mammalian cardiomyocytes, dorsal root ganglion neurons, and pancreatic beta cells.
242 calcitonin gene-related peptide (CGRP) from dorsal root ganglion neurons, and reduced inflammation i
243 eurons, chemotherapy-induced cytotoxicity of dorsal root ganglion neurons, and retinal ganglion cells
244 s hTRPM8, and rTRPM3, which are expressed in dorsal root ganglion neurons, are insensitive toward apo
245 1 x 10(12) vg of AAV-PHP.S transduced 82% of dorsal root ganglion neurons, as well as cardiac and ent
246 age-gated K(+) channel robustly expressed in dorsal root ganglion neurons, becomes dysfunctional upon
247 el, depolarize resting membrane potential of dorsal root ganglion neurons, enhance spontaneous firing
248 including altered ion channel expression in dorsal root ganglion neurons, enhanced dorsal horn gluta
251 ed sustained ASIC currents in both groups of dorsal root ganglion neurons, independent of mu opioid r
253 physiology and Ca(2+) imaging experiments on dorsal root ganglion neurons, NGF- and IL-6-induced incr
254 nic spinal commissural neurons, motoneurons, dorsal root ganglion neurons, retinal ganglion cells, an
255 ppressed spontaneous activity in dissociated dorsal root ganglion neurons, reversed hypersensitivity
256 g calcium imaging in cultured primary murine dorsal root ganglion neurons, the response of neurons af
257 een TLR4 and TRPV1 is shown in rat and human dorsal root ganglion neurons, TLR4/TRPV1-coexpressing HE
258 tion and inhibits neurite outgrowth in adult dorsal root ganglion neurons, validating Slit2 signaling
260 tion in both male and female embryonic mouse dorsal root ganglion neurons, we show that MAP4K4, MINK1
261 duced robust neurite outgrowth by cocultured dorsal root ganglion neurons, which was prevented by neu
283 currents recorded from small neurons in the dorsal root ganglion of normal rats are potentiated by e
285 ore variable across cultures than in primary dorsal root ganglion, particularly for genes related to
287 y processes characterized by spinal cord and dorsal root ganglion production of proinflammatory cytok
289 fer elevated intracellular calcium levels in dorsal root ganglion pruriceptors, and (iii) injection o
292 P that binds and regulates multiple mRNAs in dorsal root ganglion sensory neurons and thereby promote
293 ated virus (AAV) has been shown to transduce dorsal root ganglion sensory neurons following direct in
295 ta from an in vitro preparation of small rat dorsal root ganglion somata showing a reduction in the m
298 peripheral immune cell infiltration into the dorsal root ganglion, suggesting that adaptive immune re
299 We show here that synaptic contacts from dorsal root ganglions to a small number of dorsal column