戻る
「早戻しボタン」を押すと検索画面に戻ります。 [閉じる]

コーパス検索結果 (１語後でソート)

通し番号をクリックするとPubMedの該当ページを表示します
1 ation, little is known about the dynamics of double infection.
2 o examined the nature of cell-mediated HIV-1 double infection.
3 sequence types, raising the possibility of a double infection.
4 ot the only factor contributing to nonrandom double infection.
5 fered additional survival costs when hosting double infections.
6 nt types of JCV at the same time, indicating double infections.
7 ymorphism and infection type suggesting that double infections are the stable type, with singly infec
8                            Results show that double infections can reinforce interspecies reproductiv
9         We observed a decreased frequency of double infection compared with experiments in which DCs
10                    However, the frequency of double infection differed significantly, depending on th
11 lysed datasets of cell-free HIV-1 single and double infection experiments in cell culture.
12  of previously uninfected matrilines, (ii) a double infection in a matriline already bearing a differ
13 is is the first direct evidence of nonrandom double infection in HIV-1.
14 nfection line could be advantageous over the double-infection line in maximizing transmission efficie
15 ion line lost one dsRNA segment, whereas the double-infection line lost eight segments, including one
16 n line and at the embryo-larval stage in the double-infection line of OGV1 and OGV3; the RNA load of
17 ore, the duration of the larval stage of the double-infection line was found to be significantly long
18 ected larvae, we established a OGV1 and OGV3 double-infection line, in addition to a triple-infection
19  measured and were used to calculate whether double infection occurred at frequencies expected from r
20 r CXCR4-tropic HIV-1 Env and have found that double infection occurred more frequently than random ev
21 e found that regardless of the protein used, double infection occurred more frequently than random ev
22                   In primary CD4(+) T cells, double infection occurred most frequently when both viru
23                                We found that double infection occurred significantly more frequently
24   Therefore, our results indicate that HIV-1 double infection occurs more frequently than it would at
25 pe 1 (HIV-1) infection is nonrandom and that double infection occurs more frequently than predicted f
26 evious study, we also demonstrated nonrandom double infection via dendritic cell (DC)-mediated HIV-1
27                                      Using a double-infection viral vector technique, we demonstrate
28                                    Increased double infection was observed in all experiments regardl
29 from random distribution; increased rates of double infection were observed in both a T cell line and
30 asma cpn60 UT sequences, while identifying a double infection with SbGP/MPV and aster yellows (16SrI)
31                            Each species have double infections with a representative from both the A
32       Both triple (wNlonAwNlonB1wNlonB2) and double infections (wNlonAwNlonB1, wNlonAwNlonB2) have be