1 he IC and auditory cortex did not reveal any
double labeling.
2 d JNK proteins were localized by fluorescent
double labeling.
3 ionation, immunofluorescence, and immunogold
double labeling.
4 method and bisbenzamide/tyrosine hydroxylase
double labeling.
5 enalidomide treatment using CD138(+)/IRF4(+)
double labeling.
6 ected to both the AI and the LO as judged by
double-labeling.
7 Our
double-labeling analyses of intraneuronal Abeta and CD40
8 A quantitative
double labeling analysis in layers II and III revealed t
9 Finally,
double-labeling analysis of CTb + Fos showed that contex
10 Combining
double labeling and confocal microscopy analyses, we fou
11 ergic neurons, we performed BDNF/parvalbumin
double labeling and discovered that axons from cortical
12 Double labeling and fluorescence-activated cell sorter a
13 Fifteen of these were confirmed with
double labeling and microscopy, including the transcript
14 his study, Western blots, immunofluorescence
double labeling,
and the retrograde tracing method were
15 otope probing (Raman-SIP) using a single and
double labeling approach.
16 nstrated by cell lineage tracing and mineral
double-labeling approaches that murine PDL progenitor ce
17 To do this, we used
double-labeling combinations of Fos immunocytochemistry
18 Under the same
double-labeling conditions, however, cones in homozygous
19 Double labeling confirmed up-regulation of CCL2/CCR2 on
20 tochrome c from mitochondria, as revealed by
double-labeling confocal immunofluorescence microscopy.
21 gocytophilum entry into neutrophils by using
double-labeling confocal microscopy.
22 Extensive
double-labeling crossover studies have been conducted.
23 e mechanism was obtained through an isotopic
double-labeling crossover study.
24 Double labeling demonstrated coexpression of JCV T antig
25 Double labeling demonstrated that apoptotic cells includ
26 BrdU staining, providing a valuable tool of
double labeling DNA synthesis, e.g., for tracking the tw
27 Immunogold
double-labeling electron microscopy, using antisera agai
28 abundantly labeled with Fast blue (FB) in a
double-labeling experiment in which FB was co-injected i
29 A
double-labeling experiment with the dimeric model showed
30 ochemical methods to test the feasibility of
double labeling experiments during development.
31 Double labeling experiments indicate that the neurochemi
32 Double labeling experiments revealed that complexes of O
33 Double labeling experiments showed that the location of
34 Confocal scanning laser microscopy and
double labeling experiments with rhodamine 123 suggested
35 Double labeling experiments, by either in situ hybridiza
36 bone formation rates in in vivo fluorochrome
double labeling experiments.
37 Double-labeling experiments (biocytin backfill x seroton
38 Double-labeling experiments colocalizing the bradykinin
39 Double-labeling experiments confirm that NADPH-d, the mo
40 cell the "distal Dab1-immunoreactive cell."
Double-labeling experiments demonstrate that the distal
41 Double-labeling experiments demonstrated that CRN termin
42 ons affected by such lesions, we carried out
double-labeling experiments for the inhibitory neurotran
43 Double-labeling experiments further demonstrate that V1
44 c gut-associated lymphoid tissue at 14 days;
double-labeling experiments indicated that 93.5% of the
45 Double-labeling experiments reveal NMB is expressed in a
46 Double-labeling experiments revealed several possible li
47 Moreover,
double-labeling experiments revealed that activation of
48 Double-labeling experiments showed striking cellular col
49 Double-labeling experiments showed that most of the prin
50 s of vallate papillae and nasoincisor ducts,
double-labeling experiments showed that syntaxin-1, SNAP
51 Double-labeling experiments showed that the dendritic an
52 of GABA antagonists, uptake inhibitors, and
double-labeling experiments showing that Cr-Aint neurons
53 in the control of food intake, we performed
double-labeling experiments to evaluate the potential ov
54 tly labeled retinal terminals was counted in
double-labeling experiments using confocal microscopy an
55 Double-labeling experiments using probes for previously
56 Single- and
double-labeling experiments were performed to establish
57 Double-labeling experiments with a subset of modulators
58 Double-labeling experiments with ArINTs and ArINTstBu (T
59 etween caspase activation and NFT formation,
double-labeling experiments with fodrin CCP and PHF-1 we
60 When mAb-GlyRbeta was used in
double-labeling experiments with GlyRalpha1-, alpha2-, a
61 In
double-labeling experiments, antibodies to acetylated hi
62 Also, we performed
double-labeling experiments, injecting BDA in the CRNs a
63 in V1 or V2, and, consistently with previous
double-labeling experiments, some axons can branch to bo
64 t that we identified as the Golgi complex in
double-labeling experiments, while wild type caveolin-3
65 thalamic neurons were compared in retrograde
double-labeling experiments.
66 Double-labeling FISH showed colocalization of tyrosine h
67 pproach to evaluate this hypothesis, we used
double-labeling fluorescence immunohistochemistry to det
68 Double-labeling fluorescence immunohistochemistry using
69 Using
double labeling fluorescent histochemistry with confocal
70 amined using myeloperoxidase (MPO) assay and
double-labeling fluorescent immunohistochemical analysis
71 Combinations of
double-labeling fluorescent immunohistochemistry (D-FIHC
72 As expected,
double labeling for ATF-3, a marker of cell bodies with
73 t macaque monkeys, using immunohistochemical
double labeling for bromodeoxyuridine and cell-type-spec
74 Double labeling for GAD67 and GHRHR in vitro and in vivo
75 tion of the in situ hybridization signal and
double labeling for glial fibrillary acidic protein were
76 The current investigation used
double labeling for NADPHd and Fos-like immunoreactivity
77 Double labeling for NMDA NR1 and c-fos revealed marked a
78 Double labeling for Prox1 and cell-type-specific markers
79 nventional retinal synapses was confirmed by
double labeling for synapsin I, a marker for conventiona
80 bserved in dendritic profiles, verified with
double labeling for the dendrite-specific marker microtu
81 ze the phenotype of ER-beta-bearing cells by
double labeling for the GABAergic-associated calcium-bin
82 al cells were identified as keratinocytes by
double labeling for WNV antigen and keratin 10.
83 After
double-labeling for Cx36 and Cx43 by FRIL, neuronal gap
84 Double-labeling for flow cytometric analysis was employe
85 Double-labeling for GAD and synaptophysin confirmed that
86 Double labeling further reveals that the majority of Cck
87 Light and electron microscopic
double labeling further showed that the VGLUT1 subtype o
88 To address this question,
double-labeling histochemical studies were performed for
89 Double labeling identified significant co-localization o
90 and protein immunofluorescence (IF)-RNA FISH
double labeling (
IF/FISH).
91 Double labeling immunocytochemical studies confirmed tha
92 Double-labeling immunocytochemistry revealed m1 receptor
93 By
double-labeling immunocytochemistry, alpha-MSH-IR axon v
94 , cat, and monkey was studied by single- and
double-labeling immunocytochemistry.
95 Double labeling Immunofluorescence was performed using c
96 Using
double-labeling immunofluorescence analysis of intraneur
97 Double-labeling immunofluorescence experiments revealed
98 Double-labeling immunofluorescence histochemistry reveal
99 Double-labeling immunofluorescence microscopic analyses
100 Double-labeling immunofluorescence revealed that approxi
101 from stage 14 (S14) to adulthood by using a
double-labeling immunofluorescence technique.
102 mbinant, we have been able to demonstrate by
double-labeling immunofluorescence that matrix protein (
103 In
double-labeling immunofluorescent preparations, 61.1 +/-
104 With
double-labeling immunogold electron microscopy (EM), we
105 Double-labeling immunogold experiments showed that appro
106 Using
double labeling immunohistochemistry on mouse and rat sp
107 We demonstrate using
double labeling immunohistochemistry that Group II metab
108 Animals were sacrificed for single and
double labeling immunohistochemistry to identify which c
109 Double labeling immunohistochemistry was used to identif
110 Using
double-labeling immunohistochemistry and confocal micros
111 ne (BrdU) labeling of newly generated cells,
double-labeling immunohistochemistry and TUNEL labeling
112 Double-labeling immunohistochemistry indicated that PR-i
113 By using
double-labeling immunohistochemistry or immunohistochemi
114 Double-labeling immunohistochemistry revealed that all o
115 Double-labeling immunohistochemistry showed NPY-IR axon
116 Double-labeling immunohistochemistry was performed with
117 Double-labeling immunohistochemistry was used to determi
118 Light microscopic
double-labeling immunoperoxidase experiments demonstrate
119 For
double labeling,
immunoreactivity was visualized using i
120 Double-labeling immunostaining and co-immunoprecipitatio
121 ted into the pylorus of mice and fluorescent
double-labeling immunostaining for betaIII-tubulin or PG
122 Protein
double labeling in aqueous buffer at physiological pH, t
123 lateral lemniscus (VNLL and INLL), with some
double labeling in ipsilateral lateral and medial superi
124 ents to clarify the causes of BrdU-gammaH2AX
double labeling in mouse and human beta-cells.
125 tion neurons exhibited a higher incidence of
double labeling in the superficial dorsal horn.
126 Results from
double-labeling in situ hybridization suggest that in Tg
127 The purpose of this study was to use
double-labeling in situ hybridization to identify nAChR
128 By
double-labeling in situ hybridization, a subpopulation o
129 e projecting to accumbens shell, with higher
double-labeling in the ipsilateral projection than in th
130 In both species, lack of overt
double labeling indicated that the ChAT(+) and urocortin
131 verlap of retrograde labeling but negligible
double labeling,
indicating that ventral striatopallidum
132 oxidase staining and using immunofluorescent
double labeling,
infrequent TH-immunoreactive, transplan
133 The advantage of
double labeling is revealed in our demonstration of nove
134 d pre-oromotor neurons, but there was sparse
double-labeling (&
lt;10%).
135 ucleus (XII) motoneurons was studied using a
double labeling method of anterogradely biotinylated dex
136 ural level, we develop and apply a "two-tag"
double-labeling method to label LT11's dendrites and the
137 eparate populations of synapses, we employed
double-labeling methods.
138 a tripeptide tag CIS that allows facile N, S-
double labeling of a protein of interest with >90% yield
139 We hypothesized that the
double labeling of an adenovirus with fluorescent protei
140 Double labeling of CB2R and glutamine synthetase shows t
141 Double labeling of cells with 12-(N-methyl)-N-[(7-nitrob
142 4-5-day hatchling chicks by using single and
double labeling of fibers and terminals with biocytin co
143 al surgery activated brain CRF neurons using
double labeling of Fos/CRF in naive rats.
144 The
double labeling of ganglion neurons indicates their site
145 Double labeling of JO and ocellar afferents revealed tha
146 a coli OM B(12) transport protein, BtuB, the
double labeling of many cysteine pairs is not possible i
147 Indeed,
double labeling of miR156 showed a meristem-specific pat
148 Double labeling of mRNA and beta-galactosidase immunorea
149 Double labeling of myocilin with other ECM components wa
150 cally for overlap of retrograde labeling and
double labeling of neurons.
151 Transgenesis and
double labeling of NTS and HCRT neurons showed that NTS
152 Double-transgenic mice showed 55%
double labeling of periurethral neuroendocrine cells exp
153 ectly in blood and plasma, demonstrating the
double labeling of platelet EVs with CD61 and CD9, as we
154 rviving ganglion cells were quantified after
double labeling of retinal tissue with TUNEL and Brn3a.
155 incorporated at distinct sites, enabling the
double labeling of ScFv with distinct probes, through mu
156 Double labeling of sGC with neuronal nitric oxide syntha
157 Moreover, we demonstrate that the
double labeling of sites on the periplasmic-facing surfa
158 The C-7,19
double labeling of the A-ring enyne was achieved by the
159 Double labeling of the HCMV(CR208)-infected HF cells dem
160 Double labeling of the N- and C-terminally tagged peripl
161 g context-induced renewal tests by measuring
double labeling of the retrograde tracer cholera toxin s
162 Double labeling of tissue with two different hair cell m
163 Double labeling of TSH4 with the ramosa2, branched silkl
164 Although
double-labeling of microglia with Iba1 and ED1 revealed
165 Double-labeling of neurons in which GFP was driven by C3
166 First, we measured
double-labeling of the neuronal activity marker Fos with
167 process through crossover experiments using
double-labeling (
oxo and phosphine).
168 Double-labeling procedures showed that calretinin-positi
169 ed an iododeoxyuridine and bromodeoxyuridine
double labeling protocol for use in the developing embry
170 Double labeling revealed that 30.2% of nodose neurons ex
171 Double labeling revealed that a large percentage of thes
172 Double labeling revealed that GRIP and GluR2 were coloca
173 versely, m2/choline acetyltransferase (ChAT)
double labeling revealed that m2-positive neurons corres
174 Double labeling revealed that the compartment contained
175 Immunofluorescence
double-labeling revealed a punctate pattern for ENT1 clo
176 Double-labeling revealed that only two regions of the mo
177 Double labeling reveals that all PV+ cells also contain
178 By means of
double-labeling RNA in situ hybridization in mice, we sh
179 alitative abnormalities, with bone turnover (
double labeling)
seen in all specimens.
180 Double labeling showed that all protein kinase C-immunor
181 Immunofluorescent
double labeling showed that in aged vessels IL-1beta and
182 in regions (e.g. hypothalamus and amygdala),
double labeling showed that most CBP was not co-localize
183 Double labeling showed that neuronal nitric oxide syntha
184 Double labeling showed that the thalamic valop populatio
185 Double labeling showed that type 5b cone bipolar cells e
186 Double labeling showed these to be both astrocytes and o
187 r, immunofluorescence of BRCA1 and nucleolin
double-labeling showed colocalization in both nucleoli a
188 Double labeling shows coincidence of STIM1 and STIM2 wit
189 Immunofluorescence
double labeling shows that approximately 80% of the c-ne
190 Double labeling shows that primarily separate population
191 Immunofluorescence
double-labeling staining showed no significant differenc
192 Here, we used a
double-labeling strategy (varying both the distance betw
193 This
double-labeling strategy provides a simple method to rel
194 Immunofluorescent
double labeling studies additionally reveal the prominen
195 Double labeling studies confirmed that the alpha-syn::GF
196 Double labeling studies revealed (EGFP)hHsp27 and actin
197 Double labeling studies showed that nicotine induced c-f
198 Immunohistochemical
double labeling studies with YFP and serotonin antisera
199 Double-labeling studies and examination of preclinical c
200 Double-labeling studies demonstrated colocalization of T
201 Double-labeling studies demonstrated that NGF mRNA was e
202 Double-labeling studies demonstrated the localization of
203 Double-labeling studies examining CARTp with tyrosine hy
204 Double-labeling studies revealed that approximately 50%
205 Double-labeling studies revealed that early OL progenito
206 The
double-labeling studies revealed that mGluR5 immunoreact
207 Double-labeling studies show that lMAN(shell) contains t
208 Double-labeling studies showed colocalization of the two
209 Double-labeling studies showed that BrdU-labeled cells t
210 Double-labeling studies using confocal microscopy showed
211 In
double-labeling studies using confocal microscopy, fluor
212 Double-labeling studies with antibodies to phosphorylate
213 on of all the major septal receptor genes in
double-labeling studies.
214 he present results and those from our recent
double-labeling study suggest that following spinal cord
215 In this
double-labeling study, we placed a green axonal tracer i
216 d with the region that showed FluoroGold/Fos
double labeling,
suggesting reciprocal connections betwe
217 retrogradely labeled neurons and significant
double labeling,
suggesting that cortical projections sp
218 of markers were correlated, and an antibody
double labeling technique was employed.
219 Using a new
double-labeling technique we have colocalized antibodies
220 Immunohistochemical
double-labeling technique with Fos and markers for norad
221 nd thus the complete covalent structure, the
double-labeling technique, along with mass spectrometry,
222 Y-NB (Lucifer yellow-Neurobiotin) retrograde
double-labeling technique, in conjunction with specific
223 Immunohistochemistry performed using
double labeling techniques with several neuronal markers
224 by employing electron spin and nuclear spin
double-labeling techniques did not yield unambiguous cha
225 This was investigated with immunofluorescent
double-labeling techniques to coregister PV- and CB-expr
226 In these studies, we used
double-labeling techniques to examine whether Cal conten
227 d that intracellular flora by using the same
double-labeling techniques to identify Fusobacterium nuc
228 bers in the IPL, standard immunocytochemical
double-labeling techniques were applied, using antibodie
229 Accordingly, immunocytochemical
double-labeling techniques were used to identify GABA an
230 BA-type neurons using retrograde marking and
double-labeling techniques, are profoundly excited by mu
231 Using
double-labeling techniques, some presynaptic terminals w
232 Using
double-labeling techniques, we established that these in
233 Double labeling the brainstem sections revealed that irI
234 Double labeling the hypothalamic sections with NPB antis
235 Double-labeling the hypothalamic sections with guinea-pi
236 ons of the RVM and caudal pons and performed
double labeling to evaluate the expression of alpha-7 an
237 However,
double-labeling uncovered no coexpression of FRU(M) and
238 born neurons using doublecortin, as well as
double labeling using an additional antibody to glial fi
239 Fluorescent
double labeling using antibodies to bNOS and glutamic ac
240 En face
double labeling using Ki-67 and progenitor markers revea
241 ions were processed for immunohistochemistry/
double labeling using patient sera/cerebrospinal fluid a
242 myocilin with ECM components was examined by
double labeling,
using different-sized gold particles.
243 Double labeling was carried out using anti-CD3, anti-CD6
244 No
double labeling was found when using a fluorescent trace
245 la, and midbrain as CRH neurons, although no
double labeling was found.
246 Double labeling was observed in the cell bodies of Purki
247 c and any or all of the ipsilateral targets,
double labeling was rare, suggesting that contralateral
248 Immunofluorescence
double labeling was used to determine cellular localizat
249 Much less
double-labeling was associated with injections into eith
250 function (bone formation) after tetracycline
double-labeling was performed by fluorescence microscopy
251 The same pattern of
double-labeling was seen from injections at each spinal
252 Commissural divergence, assessed by
double labeling,
was less than 3% in each area.
253 abeling and with preembedding immunogold for
double labeling,
was localized in cell bodies with ultra
254 By using
double labeling,
we found no evidence for mGluR1alpha or
255 Double labeling with a caveolin antibody indicated that
256 Cell death was quantified by
double labeling with a membrane impermeable dye and 4',6
257 AII amacrine cells, positively identified by
double labeling with an antibody against calretinin, wer
258 Double labeling with anti-PECAM1 antibody and anti-place
259 Double labeling with anti-PECAM1 antibody and one of thr
260 TULP1-positive cells were identified by
double labeling with antibodies specific for cones, rods
261 Double labeling with antibodies to calretinin and gamma-
262 Double labeling with antibodies to each amino acid and t
263 ype of BrdU-positive cells was identified by
double labeling with antibodies to neuronal or glial mar
264 transgenic mice were confirmed as neurons by
double labeling with antineurofilament antibody.
265 Double labeling with BDA and (3)H-leucine signifies that
266 Double labeling with BrdU and the cell cycle marker Ki-6
267 Double labeling with c-FLIP antibody and terminal deoxyn
268 In
double labeling with calretinin and parvalbumin, few neu
269 Double labeling with ChAT and synapsin antibodies showed
270 Double labeling with confocal microscopy of DLB midbrain
271 We used
double labeling with fluorescent retrograde tracers to i
272 Moreover,
double labeling with Fluoro-Jade B and glial fibrillary
273 Double labeling with glial fibrillary acidic protein (as
274 Double labeling with isolectin B4 (IB4) showed that all
275 ricular nucleus of the hypothalamus (PVH) by
double labeling with markers expressed in viruses inject
276 Double labeling with mGluR4a antibodies and antibodies t
277 Double labeling with neuropeptide Y (NPY), a marker for
278 Double labeling with OMP and synaptophysin demonstrated
279 Double labeling with other molecular markers confirmed t
280 Double labeling with parvalbumin antibodies in monkey re
281 Double labeling with Plk3 and gamma-tubulin, the latter
282 By
double labeling with rhodopsin, we demonstrate that earl
283 Double labeling with several other markers for amacrine
284 Double labeling with SMI-32 and parvalbumin confirmed th
285 Double labeling with SNAP25 and calbindin antibodies dem
286 Cone cell death was analyzed by
double labeling with TdT-dUTP terminal nick-end labeling
287 al apoptosis was determined by histochemical
double labeling with terminal deoxynucleotidyl transfera
288 Double labeling with the 275 kD hair cell antigen (HCA)
289 Double labeling with the ganglion cell marker RBPMS demo
290 esence of small, clear synaptic vesicles and
double labeling with the presynaptic markers synaptophys
291 Double labeling with the vesicular glutamate transporter
292 o functional recovery, sequential retrograde
double labeling with two fluorescent dextran amines was
293 Double labeling with various glutamate receptor subunit
294 Double-labeling with a monoclonal antibody against micro
295 Double-labeling with anti-CD34 antibodies demonstrated t
296 Immunohistochemistry was performed by
double-labeling with anti-human MMP1 and collagen type I
297 rrested in the G2 phase of the cell cycle by
double-labeling with BrdU and a mitosis-specific marker;
298 Double-labeling with CTB and NPY was observed in the Arc
299 Also,
double-labeling with synaptotagmin showed that Kv1.2 col
300 Double labeling (
with NeuN and GFAP) immunohistochemistr