ライフサイエンスコーパス: dps

1 dps is differentially expressed in vivo- low in mice and

2 dps mRNA was induced over 500-fold by oxidative stress i

3 dps mutants exhibit increased levels of the G x C-->T x

4 iF(6))] (SIFSIX-dps-Cu, SIFSIX = SiF(6)(2-), dps = 4.4'-dipyridylsulfide, also termed as NCU-100) exh

5 eased by 1 mg/kg, but not 0.1 mg/kg LPS at 3 dps (that is, 6 days post-MCAO), as was brain atrophy at

6 iently increased by 0.1 and 1 mg/kg LPS at 3 dps.

7 ll infiltrates in ischemic brain tissue at 3 dps.

8 Animals were sacrificed at 15 and 36 dps.

9 ost-MCAO), as was brain atrophy at 28 and 56 dps.

10 330 cm(3) g(-1) (273 K) at 1 atm for CPM-733-dps (the Co(2) V-BDC form, BDC=1,4-benzenedicarboxylate)

11 CPM-733-dps is stable and shows no loss of C(2) H(2) adsorption

12 The peroxide response-inducible genes ahpCF, dps, and katB in the obligate anaerobe Bacteroides fragi

13 s indicate a role for H. influenzae arcA and dps in pre-emptive defence against transitions from grow

14 Comparing isogenic dps+ and dps::kan strains by flow cytometry and by quantitative p

15 Moreover, ftnB and dps are repressed by the Fe-responsive regulator Fur and

16 onstitutive response of the ahpCF, katB, and dps genes, respectively.

17 RpoS is a regulatory element for osmY and dps.

18 oxyR and dps were found to be divergently transcribed, with an ov

19 generated with mutations in hktE, pgdX, and dps.

20 Common OxyR-regulated genes such as dps and ahpFC were not positively regulated in P. gingiv

21 te pore size and specific binding sites, [Cu(dps)(2)(SiF(6))] (SIFSIX-dps-Cu, SIFSIX = SiF(6)(2-), dp

22 f in situ synthesized 4,4'-dipyridylsulfide (dps) ligands.

23 d with gene expression studies of duplicated dps genes shows that paralogous gene pairs are expressed

24 However, dps expression was induced approximately fourfold by oxy

25 an iron-sequestration protein, because Hpx- dps mutants exhibited sensitivity similar to that of the

26 Comparing isogenic dps+ and dps::kan strains by flow cytometry and by quant

27 lator OxyR controls several OSR genes (katB, dps, and ahpC), but there is little else known about oth

28 In silico analysis of dps promoter regions coupled with gene expression studie

29 Deletion of dps resulted in hydrogen peroxide sensitivity and comple

30 explanation that variation in the number of dps per genome among closely related Streptomyces can be

31 Moreover, the numbers of dps genes per bacterial genome is variable; even amongst

32 Reduced numbers of dps mutant bacteria in the livers and spleens of infecte

33 re evaluated over up to 56 days post-sepsis (dps) by behavioral tests, immunohistochemistry and flow

34 binding sites, [Cu(dps)(2)(SiF(6))] (SIFSIX-dps-Cu, SIFSIX = SiF(6)(2-), dps = 4.4'-dipyridylsulfide

35 t room temperature, the pore space of SIFSIX-dps-Cu significantly inhibits CO(2) molecules but takes

36 As a result, expression of sod, dps, and ahpC was also upregulated.

37 L) for 14 days beginning 1 day post surgery (dps).

38 ressed as a monocistronic 1-kb mRNA and that dps mRNA was approximately 500 bases in length.

39 bility under competitive conditions and that dps mutants have altered lag phases compared to wild-typ

40 This indicates that dps expression is also under the control of an oxygen-de

41 ated proteins, Fis and H-NS, can bind at the dps gene promoter and downregulate its activity.

42 Both Fis and H-NS selectively repress the dps promoter, preventing transcription initiation by RNA

43 Array data revealed that the dps gene, not previously assigned to the ArcA modulon in