ライフサイエンスコーパス: drinking

1 of dissolved arsenic in groundwater used for drinking.

2 t dogs would ingest copepods readily through drinking.

3 % prediction accuracy for >=50% reduction in drinking.

4 d vermis of youths who initiated substantial drinking.

5 e, but not in mice with a history of ethanol drinking.

6 protected against anxiety caused by alcohol drinking.

7 gets, which plays an important role in binge drinking.

8 sloped), using distinct paradigms to measure drinking.

9 in REM sleep when individuals with AUD stop drinking.

10 s in adolescents before and after initiating drinking.

11 responses to stress following heavy alcohol drinking.

12 ie the transition from moderate to excessive drinking.

13 1.70-2.76), those reporting moderate alcohol drinking 1.76 (1.21-2.57), and those with increased numb

14 The reduction in drinking after naltrexone was negatively associated with

15 Even in the absence of binge drinking, alcohol consumption during pregnancy can leave

16 hol dependence or a consequence of excessive drinking and alcohol exposure.

17 l nucleus (RMTg) decreased voluntary alcohol drinking and alcohol self-administration.

18 lective agonist pioglitazone reduces alcohol drinking and alcohol-seeking behavior in rats.

19 f naltrexone (50 mg/day) in reducing alcohol drinking and craving among FHP drinkers with beneficial

20 from the perspective of the roles of alcohol drinking and dietary factors in a rural population.

21 RCTs, we computed medication effects on any drinking and heavy drinking (k = 118 studies, 17 medicat

22 the effects of PPARgamma agonism on alcohol drinking and seeking in msP rats.

23 the effects of PPARgamma agonists on alcohol drinking and seeking.

24 y provides a foundation that shows how binge drinking and the oral microbiome dysbiosis lead to perme

25 havior relationships: one related to age and drinking and the other one related to depression.

26 Ozonation of drinking and wastewater relies on ozone (O(3)) and hydro

27 arious disinfection methods commonly used in drinking and wastewater treatment plants.

28 astating illness defined by periods of heavy drinking and withdrawal, often leading to a chronic rela

29 icidal ideation, suicide attempts, hazardous drinking, and opioid use.

30 physical inactivity, current smoking, heavy drinking, and oral estrogen use to assess independent as

31 Controlled-drinking approaches should be promoted and comorbidity m

32 ucleus is reinforcing, and increases ethanol drinking as well as consumption of sucrose and saccharin

33 Sustained male hazardous drinking (as measured by the AUDIT-C scale) was also ass

34 S) to examine the association between coffee drinking, as assessed by a semi-quantitative food freque

35 on paradigm followed by a compulsive ethanol drinking assay.

36 Current drinking at 19-21 weeks (RR 3.96 95% CI [1.04-15.05]) an

37 Drinking behavior and osmotic regulatory mechanisms exhi

38 trols metabolic organ homeostasis and eating/drinking behavior via FGF receptor 1/Klothobeta (FGFR1/K

39 s, current drinker, defined as any recurrent drinking behavior, and regular drinker, defined as the s

40 that these contribute to daily regulation of drinking behavior.

41 generated and how it subsequently motivates drinking behavior.

42 t from reward contribute to predicting risky drinking behaviors.

43 e, would have a greater influence on alcohol drinking behaviors.

44 ioid systems play important roles in alcohol drinking behaviors.

45 were developed to identify pig postures and drinking behaviours of group-housed pigs.

46 eding routine in standing, lateral lying and drinking behaviours.

47 l, E2) are associated with increased alcohol drinking by women and experimentally in rodents.

48 werfully drive later alcohol use in familiar drinking contexts, yet we know little about what patient

49 mpared with healthy controls (HCs) and heavy drinking controls (HDCs).

50 tes that rely upon nonpublic water wells for drinking, cooking, and other household uses.

51 ransferase (ALT), and higher number of heavy drinking days (all ps < 0.05).

52 bo had 35.58% heavy drinking days and 58.47% drinking days (heavy drinking days: odds ratio=0.14, 95%

53 al symptoms on prazosin reported 7.07% heavy drinking days and 27.46% drinking days, while those on p

54 ays, while those on placebo had 35.58% heavy drinking days and 58.47% drinking days (heavy drinking d

55 Primary outcomes were daily self-reported drinking days and heavy drinking days, and secondary out

56 relapse, and reduced the likelihood of heavy drinking days compared with midazolam.

57 Proportions of participants with any heavy drinking days decreased in both groups at 6 months but d

58 ence and also predicted greater number heavy drinking days during the subsequent 2 weeks of treatment

59 period (weeks 3-12) for drinking days, heavy drinking days, and average drinks/day.

60 daily self-reported drinking days and heavy drinking days, and secondary outcomes were average drink

61 full-dose treatment period (weeks 3-12) for drinking days, heavy drinking days, and average drinks/d

62 eported 7.07% heavy drinking days and 27.46% drinking days, while those on placebo had 35.58% heavy d

63 rinking days and 58.47% drinking days (heavy drinking days: odds ratio=0.14, 95% CI=0.058, 0.333; dri

64 days: odds ratio=0.14, 95% CI=0.058, 0.333; drinking days: odds ratio=0.265, 95% CI=0.146, 0.481).

65 Lastly, a history of alcohol drinking did not alter synaptic transmission in PDYN neu

66 rrent (weekly) drinkers, AAI <18.1 years and drinking duration >30.0 years were associated with 18% (

67 betes, compared with AAI 18.1-29.0 years and drinking duration <10.1 years, respectively.

68 used to estimate the association of AAI and drinking duration with type 2 diabetes.

69 Drinking durations of <10.1 years, 10.1-20.0 years, and

70 s disruption in turn predicted greater heavy drinking during early treatment.

71 abstinence and whether they influence heavy drinking during the early treatment phase.

72 ter amount consumed in the first half of the drinking episode.

73 ate the number of copepods ingested during a drinking event.

74 Memory for prior drinking experiences may powerfully drive later alcohol

75 .02 (95% CI: -0.05, 0.08)) and stable, heavy drinking (for Blacks, adjusted MD = 0.08 (95% CI: -0.34,

76 Stable, low to moderate drinking (for Blacks, adjusted mean difference (MD) = 0.

77 metabolic syndrome who fasted (no eating or drinking) from dawn to sunset for more than 14 h daily f

78 ch discourages the initiation of feeding and drinking (fully recapitulating the symptoms of gastric d

79 nificant in the never-smoker and non-alcohol drinking groups.

80 y lifestyle including current smoking, heavy drinking (> 30 g/day), and lack of regular exercise, and

81 Heavy smokers tended to have drinking habits, which was associated with increased BMI

82 The process of diagnosing hazardous alcohol drinking (HAD) is based on self-reported data and is the

83 Excessive alcohol drinking has been shown to modify brain circuitry to pre

84 In this population based setting, drinking high volumes of alcohol may contribute to the p

85 can ingest copepod intermediate hosts while drinking; however, low numbers were ingested at the dens

86 during imagined thirst relative to imagined drinking, implying functional connectivity between these

87 obability of suicidal ideation and hazardous drinking in adolescence and young adulthood as well as o

88 understanding the neural basis of compulsive drinking in alcohol use disorder.

89 nses wherein PDYN knockout decreased alcohol drinking in both male and female mice, whereas KOR knock

90 )-GPCR signaling in PFC astrocytes increased drinking in ethanol-naive mice, but not in mice with a h

91 The influence of ERs on binge drinking in female mice suggests that treatments for alc

92 ceptor in the VTA reduced binge-like ethanol drinking in female, but not male, mice.

93 nd NOV stress more effectively increased 2BC drinking in males and females, respectively.

94 female mice, whereas KOR knockout decreased drinking in males only.

95 table low, low to moderate, and stable heavy drinking in midlife are not associated with lesser and g

96 (p = 0.002) and higher subsequent (p = 0.01) drinking in patients with AUD.

97 ne infusion was found to improve measures of drinking in persons with alcohol dependence engaged in m

98 these genes and assessed the effects on the Drinking in the Dark (DID) and Intermittent Access (IA)

99 amounts of ethanol in the first days of the drinking in the dark protocol, as compared with WT mice.

100 an those traditionally associated with binge drinking in young adults.

101 mbining alcohol use disorder and problematic drinking, in 435,563 European-ancestry individuals.

102 kers have been administered to heavy alcohol drinking individuals.

103 age, sex, education, race/ethnicity, alcohol drinking intensity, cigarette smoking duration and inten

104 Binge drinking is associated with disease and death, and devel

105 medication effects on any drinking and heavy drinking (k = 118 studies, 17 medications).

106 D. medinensis, but due to the method of dog drinking (lapping) compared to humans (suction and/or re

107 t cerebellar structures affected by youthful drinking may be vulnerable to age-alcohol interactions i

108 NTX + MEM resulted in a further reduction in drinking (mean: -1.94; 95% CI: -2.6, -0.8, p = 0.0005).

109 ocampus and following a rat adolescent binge drinking model.

110 For men, drinking more than 21 units (approximately 168 grams) of

111 is did not support a causal effect of coffee drinking on IOP (P > 0.1).

112 o psychological decompensation and increased drinking or relapse.

113 ages combined with prospective assessment of drinking outcomes during early outpatient treatment, in

114 ta = 3.28, p < 0.001) in the laboratory, and drinking outcomes in RCTs, such that medications that re

115 in motivational enhancement therapy affects drinking outcomes.

116 were prospectively predictive of early heavy drinking outcomes.

117 Men who maintained a heavy volume of drinking over the three decades of observation, or who h

118 number of drinks consumed during an alcohol drinking paradigm (ADP) before and after 1 week of super

119 intake of alcohol without accounting for the drinking pattern.

120 intake in a two-bottle choice, intermittent drinking procedure without affecting saccharin intake, e

121 intermittent ethanol vapor two-bottle choice drinking procedure.

122 ce for alcohol in a 24 h intermittent access drinking procedure.

123 To assess which memories predicted drinking, real-world behavior was assessed in patients w

124 in preclinical models as a target to reduce drinking-related behaviors and cue-induced reinstatement

125 e neural representation of thirst contains a drinking-related component.

126 However, drinking-resilient males showed the highest G-CSF, IL-13

127 tracted withdrawal from intermittent alcohol drinking resulted in enhanced prefrontal cortex (PFC)-dr

128 ific mechanisms that drive excessive alcohol drinking.SIGNIFICANCE STATEMENT Estrogen has potent effe

129 Brain processes underlie risky drinking, so we examined whether neural and psychosocial

130 Two fMRI-based models predicted binge drinking status better than chance, corresponding to the

131 Smoking and alcohol-drinking status were not associated with unilateral or b

132 oms, number of cigarettes smoked per day and drinking status were related to suicide attempts in SCZ

133 cs, dietary intake, and detailed smoking and drinking status.

134 regions were also activated during imagined drinking, suggesting the neural representation of thirst

135 , and paraoxon, and without matrix effect in drinking, surface, and wastewater.

136 e analysis of nanoplastics of up to 1 mum in drinking, tap, and river water.

137 VTA had a more dramatic effect on binge-like drinking than reducing ERbeta, consistent with the abili

138 s method to establish the link between binge drinking, the oral microbiome and AD.

139 In Study 1 (N = 200), though total drinking time was equivalent, participants consumed a so

140 me in relation to alcohol misuse- from binge drinking to addiction.

141 but later stages of AUD are characterized by drinking to alleviate withdrawal-induced negative emotio

142 l use disorder (AUD) involves binge or heavy drinking to high levels of intoxication that leads to co

143 te of intense thirst and while they imagined drinking to satiate thirst.

144 observed for the stable former and "mostly" drinking trajectories with 15-year cognitive decline.

145 Stressed male rats, especially drinking-vulnerable individuals (>=0.8 g/kg average 2-h

146 rams) of alcohol per week, compared with not drinking, was associated with waking several times a nig

147 n in the galc gene were given fingolimod via drinking water (1 mg/kg/d).

148 compassing the current EPA limit for lead in drinking water (15 ppb).

149 tion of water isotopes has been monitored in drinking water (DW; deltaD = -36.59 +/- 10.64 per mille

150 tions relevant to chloramine disinfection of drinking water (pH 6-9 and carbonate-buffered) was devel

151 ceive chow diet, high fat diet with sugar in drinking water (Western diet- WD).

152 onmental interventions that foster effective drinking water access, a concept that encompasses key el

153 water quality is regulated through the Safe Drinking Water Act, there are no drinking water standard

154 ater system that is regulated under the Safe Drinking Water Act.

155 FAS accounted for about 50% of total PFAS in drinking water and 90% in serum.

156 ces, evaluated the microbiological safety of drinking water and associated health outcomes, and estim

157 dy, we aimed to describe the PFAS profile in drinking water and biological samples (paired serum and

158 ed organic matter (DOM) is ubiquitous in raw drinking water and can efficiently scavenge oxidants, su

159 lacement coupled with CCT for reducing Pb in drinking water and children's BLLs, and (2) in some age

160 Research has focused on PFAS exposure via drinking water and diet, and fewer studies have focused

161 a ubiquitous source of chemical exposure in drinking water and have been associated with serious hea

162 The presence of PFASs in drinking water and in the environment is an urgent globa

163 ) and household characteristics (ie, type of drinking water and sanitation facilities).

164 that poor housing, which includes inadequate drinking water and sanitation facility, is associated wi

165 rganisms is a low-cost approach to disinfect drinking water and wastewater.

166 vels in North Carolina school and child care drinking water by building age, (ii) evaluate the effect

167 Decreased contamination of drinking water by C. jejuni and S. enterica was also obs

168 ve indicates that they periodically obtained drinking water by using fire to melt cave ice, and sheds

169 local and residential water use conditions, drinking water can be the dominant exposure pathway.

170 umbing and generated distinctively different drinking water chemical and microbial quality profiles.

171 and chlorine radical decrease by 38-100% in drinking water compared to ultrapure water, which is pri

172 hat likely resulted from the impact of HM on drinking water composition.

173 Drinking water containing no supplement or the PDK (pyru

174 r exposure to lead, a high-profile regulated drinking water contaminant.

175 Drinking water contamination related to the use of aqueo

176 Fragmentation in responsibility for drinking water contributes to disparities in drought vul

177 by harboring and shedding microbes into the drinking water distribution system.

178 Microbial presence and regrowth in drinking water distribution systems (DWDSs) is routinely

179 sion in contact with residual disinfectants, drinking water distribution systems have become potentia

180 ggest that corticosterone, delivered through drinking water even 24 h after acute stress, is capable

181 t Americans and discuss strategies to reduce drinking water exposure to lead, a high-profile regulate

182 nistered either vehicle or IAP (100 U/ml) in drinking water for 14 days in C57BL/6 mice.

183 ice were exposed to 5 ppm ( ~ 65 muM) iAs in drinking water for 3 months.

184 ce were administered estradiol or vehicle in drinking water for 6 weeks.

185 We obtained data on microbial content of drinking water for all participants; 585 children were r

186 ffects households' access to clean, reliable drinking water for basic needs is through the organizati

187 Drinking water from tube wells, compared to other source

188 threatens the availability of safe and clean drinking water globally.

189 e 0.1-0.2 mg L(-1) World Health Organization drinking water guidelines in all regimes, and inorganic

190 ze groundwater As treatment to meet relevant drinking water guidelines, while considering the As upta

191 and quantitatively measuring aqueous lead in drinking water has been developed.

192 rs, environmental lead (Pb) exposure through drinking water has resulted in community public health c

193 and recognition of the health importance of drinking water in lieu of sugar-sweetened beverages, hav

194 pheric water capture (AWC) can provide clean drinking water in locations not connected to the central

195 nificantly with increasing bacterial load in drinking water in the first year of life (0.79 [0.70,0.8

196 anoparticles were an important form of Pb in drinking water in the Pequannock water quality zone of N

197 as well as several other divalent metals in drinking water including copper, zinc, iron, and mangane

198 Lack of access to safe drinking water is a global problem, and methods to relia

199 Access to clean and safe drinking water is a perpetual concern in Arctic communit

200 Access to clean drinking water is a recognized societal need that touche

201 until centralized, or decentralized, treated drinking water is available; displacing biomass use for

202 The data reveal that ClO(4)(-) pollution in drinking water is more dangerous than previously thought

203 ribe US regulations that seek to ensure that drinking water is safe to consume for most Americans and

204 ng need in view of increasingly stringent As drinking water limits in some US states and European cou

205 Treatment of drinking water may decrease microbial exposure.

206 ection Agency's lifetime health advisory for drinking water may or may not be protective of vegetable

207 The co-occurrence of contaminants in drinking water may pose enhanced risks to health beyond

208 High commensal bacterial content in drinking water may protect against allergic diseases.

209 of microbial exposure (bacterial load in the drinking water measured during the child's first year of

210 There was a dominant seasonal effect on the drinking water microbiomes at all three locations.

211 SCFA supplementation in the drinking water of male mice significantly improved recov

212 When we added indoxyl sulfate to the drinking water of rats fed an adenine-rich diet, we foun

213 Following a pH reduction in their drinking water over a span of more than 20 years, the Ci

214 ntial impacts on blood Pb levels (BLLs) from drinking water Pb reduction actions (i.e., combinations

215 ortant to OGW-impacted source waters because drinking water plants with high-bromide source waters ma

216 tic ecosystems and poses a major problem for drinking water production.

217 Research on the local political economy of drinking water provision reveals the constraints on comm

218 ent of the impact of Hurricane Maria (HM) on drinking water quality in Puerto Rico (PR) by integratin

219 terial communities in biofilters can improve drinking water quality through the biodegradation of dis

220 The results indicate that point of use drinking water quality was impacted by conditions in the

221 able regrowth conditions affecting the final drinking water quality.

222 bution to BLLs from ingestion of residential drinking water ranged from ~10 to 80%, with the highest

223 otensin receptor blocker losartan to mice in drinking water reduced both allodynia and muscle fibrosi

224 Inadequate access to safe drinking water remains a global health problem, particul

225 Germany's largest drinking water reservoir was sampled for 1 year, and DOM

226 om subsample of households, we tested stored drinking water samples for Escherichia coli, concurrentl

227 Drinking water samples were analyzed for F(-), and the r

228 The presence of bromide in drinking water significantly accelerated Cr(VI) release

229 Drinking water source and sanitation facility alone were

230 s (PFAS) to the Cape Fear River, the primary drinking water source for Wilmington, North Carolina, re

231 hese biofilms were grown from groundwater (a drinking water source), and this groundwater was amended

232 ns between children's blood Pb and household drinking water source.

233 gh the Safe Drinking Water Act, there are no drinking water standards for nonpublic water well qualit

234 regional groundwater uranium exceedances of drinking water standards, 30 mug L(-1), are dependent on

235 Household drinking water storage is commonly practiced in rural In

236 pplied for the quantification of BPA present drinking water stored in the plastic bottles.

237 Addition of inorganic salts to drinking water such as KH(2)PO(4) + NaCl+KNO(3) resulted

238 In low-income countries, monitoring all drinking water supplies is impractical because financial

239 ultiple locations in a decentralized trucked drinking water system in Nunavut, Canada, over the cours

240 assessments that inform decisions involving drinking water systems.

241 ons of PFHxA, PFHpA, and PFBS were higher in drinking water than in serum.

242 Classical transmission to humans occurs via drinking water that contains cyclopoid copepods infected

243 tural experiment whereby individuals receive drinking water through public mains supply or individual

244 ge with water from PND 14 to 21 and received drinking water till the time of sacrifice.

245 rocess (AOP) for micropollutant abatement in drinking water treatment and water reuse plants.

246 l generation for micropollutant abatement in drinking water treatment or potable water reuse.

247 Analysis of a full-scale drinking water treatment plant GAC filter influent, effl

248 the life cycle environmental performance of drinking water treatment using LFMs under likely design

249 w-dose individual ABX or ABX cocktail in the drinking water until the time of sacrifice.

250 5b in drinking water was given to mice expressing wild-type hu

251 Cumulative bacterial load in drinking water was higher (median [IQR]: 6390 [4190-9550

252 This multitool approach was applied to a drinking water well, where bentazon and dichlorprop cont

253 ifying the source and age of contaminants in drinking water wells by combining depth-specific samplin

254 In drinking water wells, where water is typically abstracte

255 l elephant food, soil from enclosure(s), and drinking water were also sampled.

256 s, wealth index, toilet types and sources of drinking water were the most significant contributors to

257 Mice were given drinking water with ampicillin or without (controls).

258 Germ-free mice were given drinking water with TLR2 agonist or without (controls).

259 itored to assess the biological stability of drinking water without a residual disinfectant, but the

260 ce treated with deferiprone (1 or 3 mg/mL in drinking water) for 26 weeks was reversed.

261 ne of the dominant genera in the distributed drinking water, already occurred in the clean water rese

262 minants, and increases lead contamination of drinking water, but its effects are not well integrated

263 uent antibiotic misuse; and (3) insufficient drinking water, drainage and sanitation infrastructure.

264 s of 5-bromo-2'-deoxyuridine (BrdU) in their drinking water, followed by chase periods without BrdU,

265 is study used data on fecal contamination of drinking water, food, soil, hands, and objects and secon

266 on comparing improved housing (with improved drinking water, improved sanitation, sufficient living a

267 Drinking water, in an attempt to release it, led to a to

268 ses ultimately depends on the access to safe drinking water, properly managed sanitation, and hygiene

269 costerone, given 1 day after acute stress in drinking water, reversed enhanced anxiety-like behavior

270 Ensuring urban areas have access to clean drinking water, safe food supply, and uncontaminated wat

271 We hypothesized that drinking water, sanitation, handwashing (WSH), and nutri

272 followed 24 h later by corticosterone in the drinking water, the surge in corticosterone was prevente

273 e presence of this cyanotoxin in freshwater, drinking water, water reservoir supplies and food (veget

274 potential to displace other sources of safe drinking water, which could in turn hamper efforts in Ch

275 F(-)) is one of the most harmful elements in drinking water.

276 quantitative measurement of aqueous lead in drinking water.

277 ally exposed to nicotine (100 mug/ml) in the drinking water.

278 ose of nitrate-depleted BRJ (BRJ-) or normal drinking water.

279 nganese) and lead and copper in point of use drinking water.

280 on by bacteria, but not bacterial growth, in drinking water.

281 y PFAS (i.e., PFOA, PFHxS, and PFOS) through drinking water.

282 cterize microplastics in both wastewater and drinking water.

283 dispersed insoluble and soluble toxins from drinking water.

284 ormal chow, or zero-fiber diets, or SCFAs in drinking water.

285 which have been reported as contaminants in drinking water.

286 kout mice overloaded with indoxyl sulfate in drinking water.

287 induced by administration of doxycycline in drinking water.

288 le soluble lead concentrations in the city's drinking water.

289 ericans depending on private wells for their drinking water.

290 levels in rats that received vehicle in the drinking water.

291 al indicator bacteria in groundwater-derived drinking water.

292 ation infrastructure to separate sewage from drinking water.

293 i/L (picocuries per liter), respectively, in drinking-water supplies may pose human-health concerns.

294 etermination of Al(3+) in real food samples, drinking waters and herbal teas, were employed.

295 0.11)) in midlife compared with stable never-drinking were not associated with 15-year decline in gen

296 e is fundamental to survival as it motivates drinking, which subsequently corrects the fluid deficit.

297 15-year cognitive decline, and stable, heavy drinking with greater 15-year cognitive decline.

298 alcohol consumption and duration of alcohol drinking with type 2 diabetes mellitus among Chinese adu

299 We hypothesized that stable, low to moderate drinking would be associated with lesser 15-year cogniti

300 s negatively associated with number of years drinking (YOD) in FH positive, but not FH negative, part