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1 that these contribute to daily regulation of drinking behavior.
2  generated and how it subsequently motivates drinking behavior.
3 rospective measurement of their "real world" drinking behavior.
4 ased myelination may contribute to escalated drinking behavior.
5 to binge alcohol drinking and limits further drinking behavior.
6 anges in the brain that promote pathological drinking behavior.
7 also been shown to play a role in thirst and drinking behavior.
8 eward circuitry is associated with return to drinking behavior.
9 s in network properties associated with high-drinking behavior.
10 ncreased diuresis and consequently stimulate drinking behavior.
11 idual vulnerability in predisposing to risky drinking behavior.
12 ore gene-gene interactions with rs671 behind drinking behavior.
13 which conceivably could regulate feeding vs. drinking behavior.
14 jections have a crucial role in coordinating drinking behavior.
15 te to brain function and behavior, including drinking behavior.
16 i (CSs), need to be dissociated from alcohol drinking behavior.
17 l function, blood pressure, food intake, and drinking behavior.
18 n COAs who have not demonstrated any problem drinking behavior.
19 ons that may be encountered when engaging in drinking behavior.
20 ng of genetic mechanisms influencing alcohol drinking behavior.
21 ereby 5-HT(1A) receptor activation modulates drinking behavior.
22 er that appears to play a role in eating and drinking behavior.
23 f 15-year-old adolescents assessed for risky drinking behavior.
24 contributes to mechanisms underlying alcohol drinking behavior.
25 lies high anxiety-like and excessive alcohol-drinking behavior.
26 r many individuals with harmful or hazardous drinking behavior.
27 tionally involved in, mediating high alcohol drinking behavior.
28 ed in alcohol reinforcement and high alcohol drinking behavior.
29 ontrols could help mitigate subsequent risky drinking behaviors.
30 revealed analogous effects on feeding versus drinking behaviors.
31  were categorized according to self-reported drinking behaviors.
32  and neuroadaptations that may contribute to drinking behaviors.
33 armful drinking based on their self-reported drinking behaviors.
34 to the DLS, which is associated with alcohol drinking behaviors.
35 dence for the involvement of FGF2 in alcohol-drinking behaviors.
36  as well as in mechanisms underlying alcohol-drinking behaviors.
37 tend to exhibit heterogeneous postdeployment drinking behaviors.
38 l circuit responsible for individual alcohol drinking behaviors.
39 3beta, that in turn drives excessive alcohol-drinking behaviors.
40 burst activity in HAD mice decreases alcohol drinking behaviors.
41 us Arf6 in the PFC to modulate human alcohol-drinking behaviors.
42 t from reward contribute to predicting risky drinking behaviors.
43 nship between core ADHD symptoms and smoking/drinking behaviors.
44 art of the brain (DMS) that controls alcohol-drinking behaviors.
45 e, would have a greater influence on alcohol drinking behaviors.
46 he development and/or maintenance of ethanol drinking behaviors.
47 al role of HDAC2 in anxiety-like and alcohol-drinking behaviors.
48 oadaptations that underlie excessive alcohol-drinking behaviors.
49 ioid systems play important roles in alcohol drinking behaviors.
50 ion is associated with healthier smoking and drinking behaviors.
51 r to molecular mechanisms underlying alcohol-drinking behaviors.
52 st the actions of GDNF in the VTA on ethanol-drinking behaviors.
53 dence and comorbidity of anxiety and alcohol-drinking behaviors.
54 ar mechanisms underlying anxiety and alcohol-drinking behaviors.
55 EB gene is associated with increased alcohol-drinking behaviors.
56 lateral ventricle, thereby suppressing water-drinking behavior after hyperosmotic shock, similar to S
57 l subset of alcoholic transplant recipients, drinking behavior after liver transplantation is associa
58 anic, and they lend support to the view that drinking behavior among those with panic disorder is rei
59 rate links between alcohol conversations and drinking behavior among young adults.
60                   They were also assessed on drinking behavior and AUD symptoms at numerous intervals
61               Since L-type channels modulate drinking behavior and contribute to neuronal hyperexcita
62  area (LHA) might be important in modulating drinking behavior and fluid balance has led to numerous
63 brain neuropeptide that controls feeding and drinking behavior and gastric emptying and elicits neuro
64                                              Drinking behavior and osmotic regulatory mechanisms exhi
65 docannabinoid anandamide, may play a role in drinking behavior and risk for alcohol use disorder.
66 ian preoptic nucleus, involved in initiating drinking behavior and salt appetite), neuroendocrine sys
67 ear mixed-effect models were used to compare drinking behavior and subjective responses (stimulation,
68 rtance of assessing multiple levels of binge drinking behavior and their predictors among youth to ta
69 he possible associations between smoking and drinking behavior and three COVID-19 outcomes: severe CO
70  quarterly assessments for 2 years examining drinking behaviors and alcohol diagnoses.
71 es over 6 years after the sessions to assess drinking behaviors and alcohol use disorder (AUD) sympto
72 lysis, we replicated prior associations with drinking behaviors and identified multiple novel phenome
73 s, current drinker, defined as any recurrent drinking behavior, and regular drinker, defined as the s
74 blood pressure, body mass index, smoking and drinking behaviors, and exercise were examined in an ong
75 minergic signaling to neuronal responses and drinking behavior are poorly understood.
76 al sessions was measured on both seeking and drinking behaviors, as well as on drinking in the 2-bott
77                       The reduction in binge drinking behavior at 3-month follow-up compared to usual
78                   Interestingly, spontaneous drinking behavior but not thirst drive reduction trigger
79 not significantly associated with a person's drinking behavior, but the behavior of relatives and fri
80 rgic neurons in the subfornical organ drives drinking behavior, but the brain targets that mediate th
81 ing promise in the prevention of problematic drinking behavior, but their effect on illicit drug use
82 ructures are key sites in the stimulation of drinking behavior by AngII.
83 s well as slightly increased risk of smoking/drinking behaviors by an indirect effect of depression.
84 ble of vlOFC-to-DLS neurons may gate alcohol drinking behaviors by attenuating habitual alcohol seeki
85 r findings suggest that GDNF reduces alcohol-drinking behaviors by reversing an alcohol-induced allos
86           Future guidelines aim at modifying drinking behaviors could help players maintain optimal h
87 reward seeking) and predicted the subsequent drinking behavior during a free-access paradigm.
88 dicate that the hPer1 gene regulates alcohol drinking behavior during stressful conditions and provid
89 eals, suggesting the importance of improving drinking behaviors, especially among people with higher
90                            To predict future drinking behavior from PIT activation patterns, we used
91 er1 were tested for association with alcohol drinking behavior in 273 adolescents and an adult case-c
92 pse in alcohol-dependent patients and future drinking behavior in adolescents.
93 c neurotransmission is implicated in alcohol-drinking behavior in animal models.
94 e a generalized prognostic marker for future drinking behavior in established alcohol use disorder an
95 esis inhibitor endostatin reduced relapse to drinking behavior in female CIE-ED rats without affectin
96 n of candidate genes associated with alcohol drinking behavior in human populations.
97 lacement of the gut hormone ghrelin restored drinking behavior in P rats following RYGB.
98 and facial skin, was used to study motivated drinking behavior in rats.
99 951-33 in attenuating the binge-like alcohol drinking behavior in the drinking-in-the-dark paradigm.
100 ion potentials in brain slices, and finally, drinking behavior in the mouse.
101  thereby increasing anxiety-like and alcohol-drinking behaviors in control rats.
102  The present investigation evaluated alcohol-drinking behaviors in mice that are haplodeficient in CR
103 cational attainment has been associated with drinking behaviors in observation studies.
104 pathway involved in anxiety-like and alcohol-drinking behaviors in rats.
105 ubstantial individual variability in alcohol drinking behaviors in the population, the neural circuit
106 her educational attainment causally affected drinking behaviors, including amount of alcohol intakes
107  Prevalence of current drinking and of risky drinking behaviors, including exceeding moderate drinkin
108  reports showing increased KOR regulation of drinking behaviors induced by ethanol exposure, the stro
109  the causal role of the CREB gene in alcohol-drinking behaviors is unknown.
110 e disorder (AUD); few have examined habitual drinking behaviors like maximum habitual alcohol intake
111 ence may contribute to the disparate alcohol drinking behavior of the P and NP rats.
112 ining the high anxiety and excessive alcohol-drinking behaviors of P rats.
113 DNF expression ablated the return to alcohol drinking behavior over a 12-month period of repeated abs
114             Participants self-reported their drinking behavior over the past 90 days at baseline and
115 bic dopamine system have been shown to alter drinking behavior, presumably because this dopaminergic
116 interneurons (PV:delta(-/-)) increased binge drinking behavior, reduced sensitivity to alcohol-induce
117                                Initiation of drinking behavior relies on both internal state and peri
118  for smoking cessation (primary outcome) and drinking behavior (secondary outcome) among smokers who
119 ssociated with the predisposition to alcohol-drinking behavior seen in Lewis rats.
120 enetic predisposition to anxiety and alcohol-drinking behaviors using alcohol-preferring (P) and -non
121 trols metabolic organ homeostasis and eating/drinking behavior via FGF receptor 1/Klothobeta (FGFR1/K
122  was 6 +/- 1, and the mean duration of binge drinking behavior was 4 +/- 0.6 years.
123 g an alcohol-related PIT paradigm, and their drinking behavior was assessed in a 12-month follow-up.
124                                              Drinking behavior was assessed using annual interviews c
125                                              Drinking behavior was best characterized by a linear tra
126                               However, their drinking behavior was not affected by acamprosate, an FD
127                                In adulthood, drinking behavior was tested under nondependent conditio
128 resonance imaging-based prediction of future drinking behavior, we applied the support vector machine
129 l drinking, alcohol-seeking, or relapse-like drinking behavior, we demonstrate that N-acetylhomotauri
130 rmine if brain TLR3 pathways are involved in drinking behavior, we used CRISPR/Cas9 genome editing to
131 ed using high-throughput RNA sequencing, for drinking behavior were dominated by neurophysiological t
132 uestionnaire and the Yale Craving Scale, and drinking behavior were recorded in each ADP.
133      Alcohol use disorder (AUD) symptoms and drinking behaviors were assessed in the interim follow-u
134  suggests that alcohol consumption and risky drinking behaviors were common among cancer survivors, e
135                     Anxiety-like and alcohol-drinking behaviors were measured after infusion of BDNF
136 m of ADHD had a robust impact on smoking and drinking behaviors, while being mediated by anxiety and
137 ng mechanism in the establishment of alcohol drinking behavior with changing the DA-5-HT balance as a
138 , and 12) and 13 suggestive loci for ethanol-drinking behaviors with narrow confidence intervals (1-4

 
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