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1 er drugs, a phenomenon termed 'incubation of drug craving'.
2 ch can at times be maladaptive (for example, drug cravings).
3 ies in brain structures involved in food and drug craving.
4 praisal as associated with lower cue-induced drug craving.
5 (vmPFC) activity positively correlated with drug craving.
6 statement of drug seeking, and incubation of drug craving.
7 ensembles in this new form of incubation of drug craving.
8 exposure to drug-associated cues that induce drug craving.
9 ned despite adverse consequences and intense drug craving.
10 ous opioids in the neural systems underlying drug craving.
11 entral striatum were associated with intense drug craving.
12 sue that is frequently caused by cue-induced drug craving.
13 sity to engage in drug-seeking behavior, and drug craving.
14 th the conditioned place preference model of drug craving.
15 cocaine and whether this is associated with drug craving.
16 ion, including those that mediate reward and drug craving.
17 ssociated drug craving, termed incubation of drug craving.
18 rence for partner odors over cocaine buffers drug craving.
19 be important for understanding the basis of drug craving, a key factor in the maintenance of substan
20 use disorder (HUD) have been associated with drug craving, a reliable predictor of treatment outcomes
21 duced an animal model to study incubation of drug craving after prolonged voluntary abstinence, mimic
22 here may have implications for cue-elicited drug craving after repeated exposure to drugs of abuse.
24 ues that are associated with self-reports of drug craving and appear to be of a greater magnitude tha
25 e findings support the use of stress-induced drug craving and associated hypothalamic-pituitary-adren
27 h stress and drug cue exposure each increase drug craving and contribute to relapse in cocaine depend
28 a novel animal model to study incubation of drug craving and cue-induced drug seeking after prolonge
30 ng the neurobiological processes that incite drug craving and drive relapse has the potential to help
37 e use produces neuroadaptations that support drug craving and relapse in substance use disorders (SUD
41 rly long-lived aspects of addiction, such as drug craving and relapse, and to identify specific genes
43 o develop medications that effectively treat drug craving and relapse, the core features of addictive
44 the neurobiological factors responsible for drug craving and relapse, with promising therapeutic imp
53 ated with previous drug exposure can trigger drug craving and seeking, and form a substantial obstacl
55 ceptor agonists in decreasing stress-induced drug craving and stress-induced initial heroin lapse.
57 s like BP 897 could be used for reducing the drug craving and vulnerability to relapse that are elici
60 potential adjunctive treatment for reducing drug cravings and use in individuals with severe opioid
61 ncrease the likelihood of relapse by evoking drug craving, and brain-imaging studies have identified
62 dels in addition to baseline and cue-induced drug craving, and other clinical outcome variables (mood
64 hlight sex-specific effects of guanfacine on drug craving, anxiety, and negative mood with significan
65 brain regions involved in the incubation of drug craving, as well as evidence for the underlying cel
66 ere no differences between the two groups in drug craving, depressed mood, anxiety, or Clinical Globa
67 nd induced relief of withdrawal symptoms and drug cravings, despite acting on the same opioid recepto
68 ental procedures used to study incubation of drug craving do not incorporate negative consequences of
69 the medial prefrontal cortex associated with drug craving, effective treatments are still elusive.
71 eloped for mouse brains, while incubation of drug craving has primarily been studied in rats, and inc
72 gaine results in a long-lasting reduction of drug craving (humans) and drug and alcohol intake (roden
73 in synaptic activity related to cue-induced drug craving in 8 crack cocaine-dependent African Americ
74 f regions previously shown to be involved in drug craving in addicted subjects (orbitofrontal cortex,
75 It was suggested in 1986 that cue-induced drug craving in cocaine addicts progressively increases
77 anding the neurobiology of the incubation of drug craving in rodents is likely to have significant im
82 d animal models and associated with enhanced drug craving, is eliminated in flies mutant for period,
83 itization is proposed to model the increased drug craving observed in human psychostimulant abusers.
84 s was associated with self-ratings of robust drug craving or anger, and comparable alterations in hea
87 ics vs whites: OR, 0.48; 95% CI, 0.24-0.95), drug craving (OR, 2.78; 95% CI, 1.54-5.02), chronic illn
88 drug action and thereby partially alleviate drug craving, or that affect the addiction process per s
90 ked by pathological drug seeking and intense drug craving, particularly in response to drug-related s
93 ther relapse models, including incubation of drug craving, reacquisition and resurgence models, and p
95 luence of ibudilast on subjective (including drug craving), reinforcing, and analgesic effects of oxy
98 addiction retain some degree of control over drug craving that correlates with neural activity in the
99 homotor sensitization and the development of drug craving-that are associated with chronic use of add
100 ce from the drug and tests for incubation of drug craving (the time-dependent increase in drug seekin
101 reinforced responding, and the incubation of drug craving (the time-dependent increase in drug seekin
102 cocaine use disorders is the "incubation of drug craving" - the drive for the drug increases the lon
103 Here, we used a rat model of incubation of drug craving, the neuronal activity marker Fos, and the
104 ed frontal lobe contributions to cue-induced drug craving under different intertemporal drug availabi
106 l imagery control condition, imagery-induced drug craving was associated with bilateral (right hemisp
107 nger control condition, internally generated drug craving was associated with bilateral activation of