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1 hour, consistent with rapid distribution and drug elimination.
2 escence and accelerated growth recovery post drug elimination.
3 ustained therapeutic actions arise following drug elimination.
4 s hepatic expression and function in hepatic drug elimination.
5 ember (OATP) 1B3 is a facilitator of hepatic drug elimination, although much is unknown of how OATP1B
6 ysis revealed a three-compartmental model of drug elimination and a long terminal half-life (154 +/-
7 s detailed pharmacokinetic phenomena such as drug elimination and accumulation over multiple doses.
8 ressant, maintains symptom relief days after drug elimination, and how repeated doses sustain longer-
11 h ketamine and CP-101,606 augment AEPs after drug elimination, consistent with synaptic potentiation
12 secretion is the primary mechanism of kidney drug elimination, current kidney drug dosing strategies
13 ic self-resistance mechanisms, which include drug elimination, drug modification, target modification
14 wer volume of distribution (17.4mL/kg).Total drug elimination from the circulation after the administ
16 ivity analyses identified trade-offs between drug elimination half-life, maximum parasite killing eff
17 clearance of these drugs, a measure of liver drug elimination, is highly comparable to the clearance
18 therapy through skin transplants that elicit drug elimination may offer a therapeutic option to addre
19 s have identified variants in genes encoding drug elimination or drug target pathways that in some ca
20 mors versus the intestinal mucosa during the drug elimination period, compared with the ratio during
22 r filtration and a major mechanism for renal drug elimination, suggesting important clinical conseque
25 ar secretory clearance for predicting kidney drug elimination, we evaluated stable outpatients with e
26 association of oxidative-stress defense and drug elimination with treatment failure in DLBCL and ide