ライフサイエンスコーパス: dual infection

1 y (ADCC) in HIV-1 and HIV-2 monoinfection or dual infection.

2 EE, along with 11 cases of VEE and 1 case of dual infection.

3 ners as independent risk factors for HBV-HCV dual infection.

4 th the initial isolate failed to result in a dual infection.

5 l burdens, manifestations of disease, and/or dual infection.

6 might contribute to control of HIV-1 during dual infection.

7 pe B, 4.3% B/CRF01_AE recombinants, and 0.5% dual infections.

8 mpeting viruses in a single infection versus dual infections.

9 by Zika or chikungunya mono-infection and by dual infections.

10 dial infection, 3 trichomoniasis), and 4 had dual infections.

11 high-risk sexual behavior was the source of dual infections.

12 ition for infection and enhanced severity of dual infections.

13 95% CI, 9.7-28.7), HCV (16.7; 6.9-40.1), and dual infection (35.3; 3.9-323).

14 nificantly more common in those with HBV-HCV dual infection (84.6% versus 29.9%; P < 0.001).

15 /=1:8) was higher in cases of yaws (63%) and dual infections (92%) than in H ducreyi infections (29%;

16 types along with samples from three cases of dual infection (A(2254)+G(2254)) were correctly identifi

17 Among these, KSHV/EBV dual-infection allows for the priming of CD8(+) T cells

18 mary quarters there were naturally occurring dual infections, although this was identified in only 0.

19 terval [CI], 75 to 133) in participants with dual infection and 68 months (95% CI, 60 to 76) in parti

20 strate a window period for susceptibility to dual infection and indicate that protection from retrovi

21 ISRs, originating from recent dual infections and limited transmission events, partly

22 OBAS-positive specimens (82%), including six dual infections, and identified N. gonorrhoeae in 102 (5

23 er, frequency and pathogenic consequences of dual infection are unknown.

24 nked to these viruses, and the importance of dual infection, are not known fully.

25 present in the lymph nodes of patients with dual infection as compared with lymph nodes from HIV(+)

26 Moreover, we developed a dual infection assay with the wild-type Candid1 JUNV and

27 f HLA-B*57/B*5801 escape, a highly sensitive dual-infection assay that uses synonymous nucleotide seq

28 rompt larger studies to assess the effect of dual infection at the population level.

29 Dual infection by hMPV and hRSV is associated with sever

30 association between severe bronchiolitis and dual infection by human metapneumovirus (hMPV) and human

31 were significantly steeper for patients with dual infection compared with patients with single infect

32 o introduce the novel DP Q727R mutation, and dual infection-competition assays were performed to asse

33 hermore, we estimate that 47% of symptomatic dual infections contain sibling strains likely to have b

34 that the higher intensities in rabbits with dual infections could be explained by a weakened or low

35 gions either from the FRP system or from the dual infection culture, and very few from the HIV epidem

36 on breakpoints from either the FRP system or dual infection cultures.

37 HIV-1 dual infection (DI) and CXCR4 (X4) coreceptor usage are

38 Human immunodeficiency virus type 1 (HIV-1) dual infection (DI) has been associated with decreased C

39 Although KSHV-EBV dual infection does not increase eBL risk, EBV appears t

40 el for KSHV infection and find that EBV/KSHV dual infection enhanced KSHV persistence and tumorigenes

41 hanced ability of the Abbott assay to detect dual infections, especially in the presence of large amo

42 In dual infection experiments, the gag chimeric LV failed t

43 ggest a potential novel hypothesis involving dual infections for the adaptation of heterologous rotav

44 Most of the piglets in the PRRSV-S. suis dual-infection group developed suppurative meningitis.

45 l, 20 out of 22 piglets in the PRRSV-S. suis dual-infection group died within 1 week after challenge

46 fecting arbovirus, and whether patients with dual infection had a different disease spectrum or sever

47 7%) of 46 patients with Zika and chikungunya dual infection had a stroke or transient ischaemic attac

48 -Barre syndrome who had Zika and chikungunya dual infection had more aggressive disease, requiring in

49 Following dual infection, HIV-infected cells were localized to sit

50 During dual infection, IFN-gamma receptor expression on macroph

51 isk for HCC among patients with HBV, HCV, or dual infection in a dose-dependent manner.

52 Dual infection in a husband, his wife, and their child w

53 cies raises concern about the possibility of dual infection in humans.

54 ession was most evident in participants with dual infection in whom HIV-2 infection preceded HIV-1 in

55 Interestingly, the multiplex assay detected dual infections in 16/280 (5.7%) samples tested.

56 Dual infection induced severe damage to the airway epith

57 ited by concomitant HIV-2 infection and that dual infection is associated with slower disease progres

58 e complex and that the observable outcome of dual infection is dependent on the target cell type.

59 llagen I in the adjacent interstitium in the dual infection may facilitate dissemination of H. somni

60 ngs, HIV-1 recombinants generated from these dual infections may be used as a model for in vivo inter

61 ute cerebrovascular disease in patients with dual infection merits further investigation.

62 infection), whereas 50 (25%) had evidence of dual infection, mostly with Zika and chikungunya viruses

63 ; n=19), or coinfection with both organisms (dual infection; n=12).

64 HBV-HCV viral interactions in patients with dual infection necessitates careful but aggressive clini

65 sitive cohort in Cameroon exhibits a rate of dual infection of 11% per year, signifying that these in

66 uential plasma obtained before and after the dual infection of 4 subjects were compared to those of m

67 biologic analysis revealed the presence of a dual infection of CMV and specific bacterial plaque.

68 Little is known regarding the effect of dual infections on host immunity, despite the fact that

69 follow-up were characterized with regard to dual infection or single infection and to coreceptor use

70 ted with CD4(+) T-cell decline over time was dual infection (P = .001).

71 HDV-hepatitis B virus (HBV) dual infection progresses rapidly, has more complication

72 ased at a lower rate among participants with dual infection, reflecting slower disease progression.

73 Simulations also indicated that rabbits with dual infections shed more free-living stages that surviv

74 was significantly lower in participants with dual infections than in those with HIV-1 infection alone

75 gs, we hypothesized that among patients with dual infection the control of HIV-1 is associated with t

76 The impact of dual infection upon the clinical course of LD is not kno

77 The increase in bacterial burden during dual infection was associated with enhanced acquisition

78 ients, including 26 patient samples in which dual infection was found.

79 Dual infection was uncommon, but the patterns of risk ap

80 ions in patients with yaws and in those with dual infection were larger than those in patients infect

81 The lesions in patients with yaws and dual infection were more circular in shape (79% and 67%)

82 Dual infections were established in macaques simultaneou

83 Five cases with dual infections were further investigated using a sensit

84 noviral types, primarily AdB/AdC and Ad3/Ad7 dual infections, were detected.

85 All five patients with HBV-HCV dual infection who had undetectable HBV DNA levels had H

86 We investigated the influence of dual infection with B. burgdorferi and the HGE agent on

87 lous atrophy (jejunum and/or ileum), whereas dual infection with both viruses induced lesions through

88 f mortality, was found to be associated with dual infection with EPEC and L. intracellularis.

89 .8% (95% confidence interval, 4.5%-7.1%) had dual infection with HBV (hepatitis B surface antigen-pos

90 We assessed 64 patients for dual infection with heteroduplex mobility assay, viral s

91 Five of these with AIDS endpoints had dual infection with HIV-1: four were cases of coinfectio

92 Participants with dual infection with HIV-2 infection preceding HIV-1 infe

93 Dual infection with hMPV and hRSV confers a 10-fold incr

94 lacked the profound potentiation found with dual infection with M. hyopneumoniae and PRRSV.

95 an: the blood-stage population dynamics of a dual infection with Plasmodium malariae and Plasmodium f

96 Dual infection with these two pathogens in rabbits has n

97 Dual infections with both pathogens have been noted fair

98 nts had type 1, eight had type 2, and 10 had dual infections with both types.

99 otoxigenic Escherichia coli, indicating that dual infections with this leading bacterial cause of TD

100 variant outcompeted Omicron B.1.1.529 after dual infection within and between hosts in naive, vaccin