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1 n expression of Duox, an H(2)O(2) generating dual oxidase.
2 zed that CS exposure modulates expression of Dual oxidase 1 (Duox-1), a NADPH oxidases known to gener
3 hlighted the importance of the NADPH oxidase dual oxidase 1 (DUOX1) in acute airway epithelial IL-33
4 indicating involvement of the NADPH oxidase dual oxidase 1 (DUOX1) in epithelial wound responses, we
7 itical for hBD-3 induction, while ADAM10 and dual oxidase 1 (Duox1) were not required for hBD-3 induc
11 metry, we identified the local production of dual oxidase 1 (Duox1)-derived H(2)O(2) by Tnfa- and Tnf
14 a novel TLR-4-->protein kinase C alphabeta-->dual oxidase 1-->reactive oxygen species-->TACE-->TGF-al
19 SDR significantly upregulated expression of Dual oxidase 2 (Duox2), Dual oxidase maturation factor 2
22 r function) and rs269868-C/Ser1067 in DUOX2 (dual oxidase 2, related to thyroid function and innate i
25 ditis elegans (Ce), and are termed Duox for "dual oxidase" because they have both a peroxidase homolo
27 o light new processes and mediators, such as dual oxidases, defense against radiation injuries, and n
29 idence supports a role for the NADPH oxidase dual oxidase (Duox) as an important source for regulated
31 tor of Chagas disease, we show that an ovary dual oxidase (Duox), a NADPH oxidase, is the source of t
32 on, we show that this gradient is created by dual oxidase (Duox), and that it is required for rapid r
33 dityrosine network (DTN) is dependent upon a dual oxidase (Duox), which is a member of the NADPH oxid
35 ore of this enzyme, were described recently; dual oxidase (Duox)1/thyroid oxidase 1 and Duox2/thyroid
42 uggests a potentially conserved role for the dual oxidase family in hydrogen peroxide production and
44 ecreted by the Anopheles gambiae midgut, and dual oxidase form a dityrosine network that decreases gu
48 2 pathway in resistant cells is sustained by dual oxidase maturation factor 1 (DUOXA1)-mediated produ
49 ulated expression of Dual oxidase 2 (Duox2), Dual oxidase maturation factor 2 (Duoxa2), and inducible
50 ination with BLI-3, an H2O2-generating NADPH dual oxidase, MLT-7 is essential for post-embryonic deve