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1 s a treatment for eosinophilic gastritis and duodenitis.
2 between patients with and without histologic duodenitis.
3 struction and biopsy showed acute on chronic duodenitis.
4 erticulum could be a predisposing factor for duodenitis.
6 stinal infectious diseases (26%), gastritis/ duodenitis (21%), diseases of the esophagus (15%), non-i
8 In patients with eosinophilic gastritis or duodenitis, AK002 reduced gastrointestinal eosinophils a
12 AOR, 1.31; 95% CI, 1.10-1.55), gastritis and duodenitis (AOR, 1.35; 95% CI, 1.08-1.70), patella disor
14 psy proven H. pylori-associated gastritis or duodenitis as compared with 33.9% of asymptomatic patien
15 of upper GI examination for mild and severe duodenitis combined was 46% with a specificity of 98%, w
17 verticulitis, cholecystitis, duodenal ulcer, duodenitis, enteritis, or adnexal or testis pathologies.
20 nd an H. pylori-independent pangastritis and duodenitis (gastroduodenitis) associated with increased
21 sed moderately severe gastritis and proximal duodenitis in 3X mice that were more severe than the gas
23 coeliac disease in nine (35%), non-specific duodenitis in ten (38%), and no lesion in seven (26%) in
24 us TRAEs included one count each of pyrexia, duodenitis, increased transaminases and hyperthyroidism.
25 5% CI, 9.25-121.02]), APC with gastritis and duodenitis (OR, 4.66 [95% CI, 2.61-8.33]), and PTEN with
26 tomatic eosinophilic gastritis, eosinophilic duodenitis, or both conditions in a 1:1:1 ratio to recei
27 r vomiting (n=5); diarrhea (n=8); gastritis, duodenitis, or esophagitis (n=4); and ulcers (n=2)] or b
28 ctivity measures nor the presence of gastro- duodenitis per VCE, suggesting it might be part of proxi
33 re chart review, only 1 diagnosis, gastritis/duodenitis, was more likely to occur in the 14 days afte