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1 PVR and PA pressure-flow response [DeltaPQ] during exercise).
2 hemodynamics, blood gases, and gas exchange during exercise.
3 Skeletal muscle generates ROS at rest and during exercise.
4 by impaired coronary blood flow at rest and during exercise.
5 relative differences of 10% at rest and 16% during exercise.
6 are not obligatory to observe sympatholysis during exercise.
7 racy to estimate pulmonary arterial pressure during exercise.
8 for abnormal pulmonary hemodynamic response during exercise.
9 glucose release from the liver are increased during exercise.
10 iance and elastance and central hemodynamics during exercise.
11 l variability in the capacity to oxidize fat during exercise.
12 and prompted a reduction in arterial glucose during exercise.
13 F and EF >/=40%, IASD treatment reduces PCWP during exercise.
14 s, electrolyte balance, and electrolyte loss during exercise.
15 t was the pulmonary capillary wedge pressure during exercise.
16 ng men relied more on endogenous fatty acids during exercise.
17 ssion was explored by administering caffeine during exercise.
18 d peak CBF augmentation and increased demand during exercise.
19 activity and lessen oxidative perturbations during exercise.
20 tterns were, however, more likely to persist during exercise.
21 nd radionuclide ventriculography at rest and during exercise.
22 s and facilitate breathing rate acceleration during exercise.
23 e potential contribution of its upregulation during exercise.
24 termined by the magnitude of fatigue accrued during exercise.
25 hmias, or attenuated blood pressure response during exercise.
26 is common in patients with ePVH at rest and during exercise.
27 r-maximum levels of neural respiratory drive during exercise.
28 patients with COPD than in control subjects during exercise.
29 bserved phosphocreatine, pH and vOX kinetics during exercise.
30 ular oscillations in minute ventilation (VE) during exercise.
31 reduction of glucagon and cortisol responses during exercise.
32 own to improve energy metabolism at rest and during exercise.
33 anterior leaflet (AL) tethering at rest and during exercise.
34 are characterized by an impaired O2 kinetic during exercise.
35 ing despite increased right-to-left shunting during exercise.
36 l muscle blood flow and vascular conductance during exercise.
37 ave key roles in governing lipid homeostasis during exercise.
38 nce of the brain in the control of breathing during exercise.
39 which have now been recorded from in humans during exercise.
40 d-diastolic volume index (P=0.001) decreased during exercise.
41 phic measurements were performed at rest and during exercise.
42 isplaying a marginally prolonged QT interval during exercise.
43 and right heart catheterization at rest and during exercise.
44 tory parameters, and peripheral oxygen usage during exercise.
45 A oxidation but not glucose uptake in muscle during exercise.
46 and exaggerated increases in blood pressure during exercise.
47 ess changes in hemodynamics and gas exchange during exercise.
48 t for the increase in blood-muscle O(2) flux during exercise.
49 vasodilatation with muscle fibre recruitment during exercise.
50 ular oxygen delivery-to-utilization matching during exercise.
51 essel and showed excellent correlation, even during exercise.
52 ygen delivery to contracting skeletal muscle during exercise.
53 sion via local antagonism of these receptors during exercise.
54 no reduction in Qs following IASD at rest or during exercise.
55 y lesser deoxygenation of the knee extensors during exercise.
56 organs, and with nitrate being reduced to NO during exercise.
57 end point was pulmonary vascular resistance during exercise.
58 ial implications for blood flow distribution during exercise.
59 in response to insulin and also into muscle during exercise.
60 e intake and may contribute to NO generation during exercise.
61 er to assist with upper-body posture control during exercising.
62 ,721 vs. 9,707 mm Hg/min(-1); p = 0.003) and during exercise (27,467 vs. 20,841 mm Hg/min(-1); p = 0.
63 t (16.8% high affinity vs. 21.7% normal) and during exercise (55.8% high affinity vs. 72.2% normal).
64 too sensor coupled to a wireless transceiver during exercise activity demonstrated its ability to con
65 ood flow (QIPAVA ) is either increased, e.g. during exercise, acute normobaric hypoxia, and the intra
68 cerebral and femoral circulation at rest and during exercise, an ideal model system characterized by
69 s responsible for glycogen dephosphorylation during exercise and acts during the cytosolic degradatio
70 a greater decrease in muscle glycogen stores during exercise and elevated circulating free fatty acid
72 iological evaluation can be safely performed during exercise and hyperemia in patients with severe ao
73 hors describe coronary physiological changes during exercise and hyperemia in the healthy heart and i
75 SIT and ET both increase net IMTG breakdown during exercise and increase in PLIN2 and PLIN5 protein
77 amine whether lactate, a metabolite produced during exercise and known to reproduce specific brain ex
79 s = glucose + (1/2)lactate) would be similar during exercise and recovery at HA and sea level (SL).
83 did not observe associations of QT dynamics during exercise and recovery with cardiovascular events.
86 ardiovascular consequences of these reflexes during exercise and revealed various modes of interactio
89 would predict greater dead space ventilation during exercise and that this would lead to impairment i
90 , but is reduced in older (>50 years) adults during exercise and with alveolar hypoxia, suggesting po
91 bic capacities) and IPAQ score were obtained during exercises and it was used to construction of four
92 tains cellular homeostasis(2)-is upregulated during exercise, and a core autophagy protein, beclin 1,
93 gy, perceived exertion and running endurance during exercise, and changed satiety physiology and perc
94 ems to be safe, reduces left atrial pressure during exercise, and could be a new strategy for the man
95 hway is also active in human skeletal muscle during exercise, and if it is sensitive to local nitrate
96 pid rates of high-energy phosphate depletion during exercise, and impaired maximal oxidative capacity
97 ntains beclin 1 and UVRAG-in skeletal muscle during exercise, and knockout of beclin 1 or UVRAG inhib
98 plications for physiological fuel management during exercise, and relevance to pathophysiological con
100 propose that the new SR-TT junctions formed during exercise, and that contain STIM1 and Orai1, funct
101 ndidate genes for combating oxidative damage during exercise, and within the "Straight Egyptian" subg
102 04); however, Ascorbate did not modulate CVC during exercise ( approximately 60% CVCmax ; both P > 0.
105 tory adaptations, in which calories expended during exercise are counteracted by decreases in other a
107 age-related reductions in cerebral perfusion during exercise are partly associated with a lower P aC
109 o assess the role of RV measures at rest and during exercise as predictors of prognosis in asymptomat
110 f echocardiographic measurements at rest and during exercise as predictors of valve surgery in asympt
113 uscle AMPK activation and glucose metabolism during exercise, as well as unexpected crosstalk between
118 ygen consumption can be achieved at rest and during exercise at the assumed cardiac output levels, wi
119 to augment endothelium-dependent signalling during exercise attenuated alpha(1) -adrenergic vasocons
120 imultaneous expired gas analysis at rest and during exercise before and after treatment with inhaled
121 imultaneous expired gas analysis at rest and during exercise, before and 15 min after treatment with
122 dynamic process, with increasing AL opening during exercise being associated with higher exercise EO
123 1 +/- 3% vs.; P < 0.05) and remained blunted during exercise (blockade: -15 +/- 5 vs. CONTROL: -14 +/
126 ally-mediated sweating alterations in humans during exercise brought about by warm and cool fluid ing
127 availability by increasing glucose synthesis during exercise but rather adapted by altering whole bod
128 AC5 phosphorylation in mouse skeletal muscle during exercise, but resulted in a compensatory increase
129 ct relative carbohydrate and fat utilization during exercise, but the older men had higher uptake of
130 participates in cardiopulmonary performance during exercise by accessing an enormous amount of contr
131 ng IL-6 is thought to maintain energy status during exercise by acting as an energy sensor for contra
132 The metabolite lactate, which is released during exercise by the muscles, crosses the blood-brain
133 ion for the age-related reduction in P aC O2 during exercise by the provision of supplementary CO2 is
134 uptake ( max) and cardiac output at rest and during exercise (C2H2 rebreathing) were measured at the
141 t would have higher rates of gluconeogenesis during exercise compared to those who follow a mixed mac
142 ker significantly reduced ventricular ectopy during exercise compared with placebo plus beta-blocker
143 showed a 37% increment in the tricuspid area during exercise, compared with 4% in patients with PEX (
144 ies and redefine the role of AMPK activation during exercise/contraction as being important for maint
145 Here we demonstrate that glucose uptake during exercise/contraction was not compromised in AMPK-
146 suggested to regulate muscle glucose uptake during exercise/contraction, but findings from studies o
151 essure (PCWP) develop in patients with HFpEF during exercise coupled with impaired nitric oxide (NO)
152 as an endogenous metabolite that is produced during exercise, crosses the blood-brain barrier and pro
153 exercise-induced arterial hypoxaemia (EIAH) during exercise decreases the severity of quadriceps fat
156 symptoms were difficulties in walking, pain during exercise, delayed motor milestones and learning d
157 those who developed the greatest hypoxaemia during exercise demonstrated the most attenuation of qua
159 ed with wheeze in the past 12 months, wheeze during exercise, doctor and/or emergency room visits for
163 ed in rates of GNG between groups at rest or during exercise (Exercise: LCHF, 2.8 +/- 0.4 mg kg(-1) m
165 a1 -adrenergic vasoconstriction is augmented during exercise following inhibition of inwardly rectify
169 tify abnormal pulmonary hemodynamic response during exercise (>3.0 mm Hg/L per minute increase), with
170 d influence of temperature on CBF regulation during exercise has not been investigated The present st
174 utrient uptake and catabolism into myofibers during exercise in an osteocalcin-dependent manner.
177 r blockade increased FBF and FVC at rest and during exercise in both groups, although the increase in
178 ntilation were higher (P < 0.05) at rest and during exercise in both patients with ILD and patients w
179 oral contraceptive pill (OCP) (>=12 months) during exercise in dry and humid heat, across their acti
180 a role in regulating blood flow and pressure during exercise in health, little is known about the rol
181 f pulmonary artery (PA) function at rest and during exercise in HF patients without reducing systemic
188 report that an endogenous molecule produced during exercise in male mice induces the Mus musculus Bd
190 increasing endothelium-dependent signalling during exercise in older adults can improve sympatholysi
193 hat sildenafil would reduce filling pressure during exercise in patients with diastolic dysfunction a
194 buterol would improve pulmonary vasodilation during exercise in patients with HFpEF, without increasi
197 did not decrease filling pressure at rest or during exercise in post-myocardial infarction patients w
198 increases rates of whole-body fat oxidation during exercise in race walkers over a range of exercise
200 oves pulmonary vascular function at rest and during exercise in selected patients with HF and EF >=40
201 take (V'O2) slope were significantly greater during exercise in subjects with asthma or misdiagnosed
202 ed indices of arterial stiffness at rest and during exercise in subjects with HFpEF and hypertensive
203 ies were prospectively conducted at rest and during exercise in subjects with invasively proven HFpEF
204 ot reduce muscle glucose uptake or oxidation during exercise in vivo, excluding a general impairment
206 nary arteriovenous anastomoses are recruited during exercise, in hypoxia, and when cardiac output is
207 ic changes in the respiratory exchange ratio during exercise indicated that LFABP(-/-) mice use more
208 al mechanisms governing cutaneous blood flow during exercise-induced heat stress and provide directio
209 de a cascade of pathophysiological responses during exercise-induced ischemia and reperfusion at rest
211 nce, oxygen delivery, and oxygen consumption during exercise; interestingly, these changes occurred i
213 portance of this size variation to diffusion during exercise is reinforced by functional links betwee
214 Elevated left atrial pressure, particularly during exercise, is a key contributor to morbidity and m
215 o an increased perception of dyspnea, which, during exercise, is mainly associated with systemic infl
217 y manifests in vascular beds highly perfused during exercise, it has been postulated that increased b
218 in the dynamic deterioration of secondary MR during exercise, its functional and prognostic impact, a
220 is indicative of high dead space ventilation during exercise, leading to excessive and inefficient ve
223 Compensatory increases in minute ventilation during exercise maintained alveolar ventilation and arte
224 d is closely related to RV systolic function during exercise, maximal exercise capacity, and survival
225 lustrate how ROS released from muscle fibres during exercise may help maintain the integrity of axons
226 vel methods which increase muscle activation during exercise may improve responses of mechanically ve
228 ther the cellular origin of circulating IL-6 during exercise nor the means by which this cytokine enh
230 ng men demonstrated net leg glycerol release during exercise, older men showed net glycerol uptake.
231 could enable monitoring of electrolyte loss during exercise or for individuals working in extreme en
232 leep or at rest were more common than deaths during exercise or with emotional stress: 82% versus 16%
233 the majority of circulating IL-6 detectable during exercise originates from muscle and that to incre
236 ced increases observed in femoral blood flow during exercise (P<0.05 versus rest) in proportion to th
237 xia (P<0.05 versus normoxia), and especially during exercise (P<0.05 versus rest), with the most pron
239 In response to skeletal muscle contraction during exercise, paracrine factors coordinate tissue rem
240 5.5 mm Hg to 30.2 +/- 14.3 mm Hg, p < 0.001) during exercise (paradoxical response to exercise [PRE])
241 eous vasodilatation is reportedly diminished during exercise performed at a high (700 W) relative to
242 We propose that greater oxygen availability during exercise permits females to sustain a higher rela
244 quired to eliminate carbon dioxide, VE/VCO2) during exercise potently predicts outcomes in advanced h
245 l and elevates skeletal muscle O(2) delivery during exercise predominantly in fast-twitch type II mus
246 ot enhance alpha1 -mediated vasoconstriction during exercise (Protocol 1: -27 +/- 3%; P = 0.2 vs. con
250 he consumption of whole eggs with egg whites during exercise recovery in young men.In crossover trial
251 Recent evidence suggests muscle contractions during exercise release factors into the blood which cro
252 in the open probability of the CFTR channel during exercise, resulting in a decrease in reabsorption
254 ed venous o2 content difference, [C(a-v)o2]) during exercise significantly contributes to impaired ex
255 increasing endothelium-dependent signalling during exercise significantly enhanced the ability of co
256 breakdown of IMTG in type I fibres occurred during exercise (SIT 27 +/- 13%, ET 43 +/- 6%; P < 0.05)
257 bit elevated rates of carbohydrate oxidation during exercise, taking advantage of its high ATP yield
258 on peak metabolic equivalents (MET) achieved during exercise test and eight categories based on fitne
259 s, and these abnormalities are more apparent during exercise testing, with little relationship at res
261 lumes and arterial pressure both at rest and during exercise than HCM patients in whom the gradient i
263 anxiety were more likely to exhibit ischemia during exercise than women without anxiety (odds ratio,
264 erature-mediated elevations in posterior CBF during exercise that are independent of changes in PCO2
269 athways are not obligatory for sympatholysis during exercise; therefore, we tested the hypothesis tha
272 ics, exposed in this cohort by exercise (TMR during exercise, TMR(ex)) and recovery from exercise (TM
275 rified fatty acid, and glucose were measured during exercise treadmill test in C57/BL6 mice fed eithe
278 on influences oxygen consumption at rest and during exercise via alterations in cardiac output and mi
280 (-1) , cardiac output = 5.70 L min(-1) ) and during exercise ( VO2 = 2.75 L min(-1) , cardiac output
281 in the endogenous rate of glucose appearance during exercise was blunted in the KO mice because of a
282 primary end point of ventricular arrhythmias during exercise was compared between the flecainide and
286 nsor for real-time monitoring of individuals during exercise, we recorded and analyzed the sweat prof
287 tty acids) and endogenous glucose production during exercise were also reduced, and glucose infusion
290 our oxygen, and those with desaturation only during exercise were prescribed oxygen during exercise a
293 t times of greater need for NO signaling, as during exercise when left ventricular filling pressures
294 w that K(IR) channels augment vasodilatation during exercise which demands greater muscle fibre recru
295 related to the magnitude of fatigue accrued during exercise, which may explain the reported consiste
296 Maximal pressures increased significantly during exercise, while end-diastolic volumes were essent
297 patients, sildenafil improved cardiac index during exercise with a decrease in total pulmonary resis
298 ears +/- 1 [standard deviation]) at rest and during exercise with an MRI-compatible exercise stepper
299 ers diagnosed with hypertension were studied during exercise with either saline or BQ-123 (ET(A) rece
300 s in the maximal rate of fat oxidation (MFO) during exercise with potential implications for metaboli