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1  PVR and PA pressure-flow response [DeltaPQ] during exercise).
2  hemodynamics, blood gases, and gas exchange during exercise.
3    Skeletal muscle generates ROS at rest and during exercise.
4  by impaired coronary blood flow at rest and during exercise.
5  relative differences of 10% at rest and 16% during exercise.
6  are not obligatory to observe sympatholysis during exercise.
7 racy to estimate pulmonary arterial pressure during exercise.
8  for abnormal pulmonary hemodynamic response during exercise.
9 glucose release from the liver are increased during exercise.
10 iance and elastance and central hemodynamics during exercise.
11 l variability in the capacity to oxidize fat during exercise.
12 and prompted a reduction in arterial glucose during exercise.
13 F and EF >/=40%, IASD treatment reduces PCWP during exercise.
14 s, electrolyte balance, and electrolyte loss during exercise.
15 t was the pulmonary capillary wedge pressure during exercise.
16 ng men relied more on endogenous fatty acids during exercise.
17 ssion was explored by administering caffeine during exercise.
18 d peak CBF augmentation and increased demand during exercise.
19  activity and lessen oxidative perturbations during exercise.
20 tterns were, however, more likely to persist during exercise.
21 nd radionuclide ventriculography at rest and during exercise.
22 s and facilitate breathing rate acceleration during exercise.
23 e potential contribution of its upregulation during exercise.
24 termined by the magnitude of fatigue accrued during exercise.
25 hmias, or attenuated blood pressure response during exercise.
26  is common in patients with ePVH at rest and during exercise.
27 r-maximum levels of neural respiratory drive during exercise.
28  patients with COPD than in control subjects during exercise.
29 bserved phosphocreatine, pH and vOX kinetics during exercise.
30 ular oscillations in minute ventilation (VE) during exercise.
31 reduction of glucagon and cortisol responses during exercise.
32 own to improve energy metabolism at rest and during exercise.
33  anterior leaflet (AL) tethering at rest and during exercise.
34  are characterized by an impaired O2 kinetic during exercise.
35 ing despite increased right-to-left shunting during exercise.
36 l muscle blood flow and vascular conductance during exercise.
37 ave key roles in governing lipid homeostasis during exercise.
38 nce of the brain in the control of breathing during exercise.
39  which have now been recorded from in humans during exercise.
40 d-diastolic volume index (P=0.001) decreased during exercise.
41 phic measurements were performed at rest and during exercise.
42 isplaying a marginally prolonged QT interval during exercise.
43  and right heart catheterization at rest and during exercise.
44 tory parameters, and peripheral oxygen usage during exercise.
45 A oxidation but not glucose uptake in muscle during exercise.
46  and exaggerated increases in blood pressure during exercise.
47 ess changes in hemodynamics and gas exchange during exercise.
48 t for the increase in blood-muscle O(2) flux during exercise.
49 vasodilatation with muscle fibre recruitment during exercise.
50 ular oxygen delivery-to-utilization matching during exercise.
51 essel and showed excellent correlation, even during exercise.
52 ygen delivery to contracting skeletal muscle during exercise.
53 sion via local antagonism of these receptors during exercise.
54 no reduction in Qs following IASD at rest or during exercise.
55 y lesser deoxygenation of the knee extensors during exercise.
56 organs, and with nitrate being reduced to NO during exercise.
57  end point was pulmonary vascular resistance during exercise.
58 ial implications for blood flow distribution during exercise.
59  in response to insulin and also into muscle during exercise.
60 e intake and may contribute to NO generation during exercise.
61 er to assist with upper-body posture control during exercising.
62 ,721 vs. 9,707 mm Hg/min(-1); p = 0.003) and during exercise (27,467 vs. 20,841 mm Hg/min(-1); p = 0.
63 t (16.8% high affinity vs. 21.7% normal) and during exercise (55.8% high affinity vs. 72.2% normal).
64 too sensor coupled to a wireless transceiver during exercise activity demonstrated its ability to con
65 ood flow (QIPAVA ) is either increased, e.g. during exercise, acute normobaric hypoxia, and the intra
66  and pulmonary arterial pressure at rest and during exercise after 9 weeks.
67 tricle actively contributes to venous return during exercise, along with the muscle pump.
68 cerebral and femoral circulation at rest and during exercise, an ideal model system characterized by
69 s responsible for glycogen dephosphorylation during exercise and acts during the cytosolic degradatio
70 a greater decrease in muscle glycogen stores during exercise and elevated circulating free fatty acid
71 e electrocardiographic recording at rest and during exercise and genetic analyses.
72 iological evaluation can be safely performed during exercise and hyperemia in patients with severe ao
73 hors describe coronary physiological changes during exercise and hyperemia in the healthy heart and i
74 irculating interleukin 6 (IL-6) levels surge during exercise and IL-6 favors exercise capacity.
75  SIT and ET both increase net IMTG breakdown during exercise and increase in PLIN2 and PLIN5 protein
76 t compromised in muscle of patients with T2D during exercise and insulin stimulation.
77 amine whether lactate, a metabolite produced during exercise and known to reproduce specific brain ex
78                         QT interval dynamics during exercise and recovery are heritable markers but d
79 s = glucose + (1/2)lactate) would be similar during exercise and recovery at HA and sea level (SL).
80        In conclusion, the elevations in gCBF during exercise and recovery at HA serve to maintain CDO
81                  Heritability of QT dynamics during exercise and recovery were 10.7% and 5.4%, respec
82                                  QT dynamics during exercise and recovery were derived in 56 643 indi
83  did not observe associations of QT dynamics during exercise and recovery with cardiovascular events.
84 appears to increase non-oxidative metabolism during exercise and recovery.
85       We tested this sensor on 20 volunteers during exercise and rest and measured their individual b
86 ardiovascular consequences of these reflexes during exercise and revealed various modes of interactio
87  only during exercise were prescribed oxygen during exercise and sleep.
88      Identifying molecules that are produced during exercise and that mediate hippocampal Bdnf expres
89 would predict greater dead space ventilation during exercise and that this would lead to impairment i
90 , but is reduced in older (>50 years) adults during exercise and with alveolar hypoxia, suggesting po
91 bic capacities) and IPAQ score were obtained during exercises and it was used to construction of four
92 tains cellular homeostasis(2)-is upregulated during exercise, and a core autophagy protein, beclin 1,
93 gy, perceived exertion and running endurance during exercise, and changed satiety physiology and perc
94 ems to be safe, reduces left atrial pressure during exercise, and could be a new strategy for the man
95 hway is also active in human skeletal muscle during exercise, and if it is sensitive to local nitrate
96 pid rates of high-energy phosphate depletion during exercise, and impaired maximal oxidative capacity
97 ntains beclin 1 and UVRAG-in skeletal muscle during exercise, and knockout of beclin 1 or UVRAG inhib
98 plications for physiological fuel management during exercise, and relevance to pathophysiological con
99                 Echocardiography at rest and during exercise, and selected biomarkers were assessed i
100  propose that the new SR-TT junctions formed during exercise, and that contain STIM1 and Orai1, funct
101 ndidate genes for combating oxidative damage during exercise, and within the "Straight Egyptian" subg
102 04); however, Ascorbate did not modulate CVC during exercise ( approximately 60% CVCmax ; both P > 0.
103 long-duration spaceflight on CBT at rest and during exercise are clearly lacking.
104 ity at rest and excessively high ventilation during exercise are common in heart failure.
105 tory adaptations, in which calories expended during exercise are counteracted by decreases in other a
106 skeletal muscle oxygen consumption (V(o)(2)) during exercise are not well understood.
107 age-related reductions in cerebral perfusion during exercise are partly associated with a lower P aC
108 P fusion and associated myonuclear accretion during exercise are unclear.
109 o assess the role of RV measures at rest and during exercise as predictors of prognosis in asymptomat
110 f echocardiographic measurements at rest and during exercise as predictors of valve surgery in asympt
111 ddress the dynamic component of secondary MR during exercise as well.
112 d skin conductance were evaluated before and during exercise, as well as during recovery.
113 uscle AMPK activation and glucose metabolism during exercise, as well as unexpected crosstalk between
114                                              During exercise at a given absolute intensity or minute
115 l variant that enhances O(2) delivery or use during exercise at altitude in Peruvian Quechua.
116 ed research on the topic of oxidative stress during exercise at altitude.
117 went right-heart catheterization at rest and during exercise at baseline and 12-week follow-up.
118 ygen consumption can be achieved at rest and during exercise at the assumed cardiac output levels, wi
119  to augment endothelium-dependent signalling during exercise attenuated alpha(1) -adrenergic vasocons
120 imultaneous expired gas analysis at rest and during exercise before and after treatment with inhaled
121 imultaneous expired gas analysis at rest and during exercise, before and 15 min after treatment with
122  dynamic process, with increasing AL opening during exercise being associated with higher exercise EO
123 1 +/- 3% vs.; P < 0.05) and remained blunted during exercise (blockade: -15 +/- 5 vs. CONTROL: -14 +/
124                                              During exercise, blood flow to working skeletal muscle i
125                                              During exercise breathing cold and dry air, PFO+ subject
126 ally-mediated sweating alterations in humans during exercise brought about by warm and cool fluid ing
127 availability by increasing glucose synthesis during exercise but rather adapted by altering whole bod
128 AC5 phosphorylation in mouse skeletal muscle during exercise, but resulted in a compensatory increase
129 ct relative carbohydrate and fat utilization during exercise, but the older men had higher uptake of
130  participates in cardiopulmonary performance during exercise by accessing an enormous amount of contr
131 ng IL-6 is thought to maintain energy status during exercise by acting as an energy sensor for contra
132    The metabolite lactate, which is released during exercise by the muscles, crosses the blood-brain
133 ion for the age-related reduction in P aC O2 during exercise by the provision of supplementary CO2 is
134 uptake ( max) and cardiac output at rest and during exercise (C2H2 rebreathing) were measured at the
135                                  RV dilation during exercise can predict adverse ventricular-vascular
136                                              During exercise, cardiac output and leg blood flow ( QL
137                                              During exercise, cardiac output, leg blood flow and radi
138                            We here show that during exercise CBT rises higher and faster in space tha
139                                              During exercise, central command and reflexes from exerc
140                                              During exercise, CFIp declined within 1 minute from 0.56
141 t would have higher rates of gluconeogenesis during exercise compared to those who follow a mixed mac
142 ker significantly reduced ventricular ectopy during exercise compared with placebo plus beta-blocker
143 showed a 37% increment in the tricuspid area during exercise, compared with 4% in patients with PEX (
144 ies and redefine the role of AMPK activation during exercise/contraction as being important for maint
145      Here we demonstrate that glucose uptake during exercise/contraction was not compromised in AMPK-
146  suggested to regulate muscle glucose uptake during exercise/contraction, but findings from studies o
147 te period after exercise/contraction but not during exercise/contraction.
148 skeletal muscle in the period after, but not during, exercise/contraction.
149                        Stroke volume reserve during exercise correlates with exercise training respon
150                     Measurement of EMGdi%max during exercise could prove useful in identifying patien
151 essure (PCWP) develop in patients with HFpEF during exercise coupled with impaired nitric oxide (NO)
152 as an endogenous metabolite that is produced during exercise, crosses the blood-brain barrier and pro
153  exercise-induced arterial hypoxaemia (EIAH) during exercise decreases the severity of quadriceps fat
154        Women have a higher work of breathing during exercise, dedicate a greater fraction of whole-bo
155       Women have a greater work of breathing during exercise, dedicate a greater fraction of whole-bo
156  symptoms were difficulties in walking, pain during exercise, delayed motor milestones and learning d
157  those who developed the greatest hypoxaemia during exercise demonstrated the most attenuation of qua
158               In RMA patients, EOA increases during exercise despite fixed annular size.
159 ed with wheeze in the past 12 months, wheeze during exercise, doctor and/or emergency room visits for
160          Increased exposure to air pollution during exercise does not outweigh beneficial effects of
161 or quiet at rest that, when activated, e.g., during exercise, drives active expiration.
162  differences with respect to chest tightness during exercise, dyspnoea and gender.
163 ed in rates of GNG between groups at rest or during exercise (Exercise: LCHF, 2.8 +/- 0.4 mg kg(-1) m
164                           Endurance capacity during exercise exhaustion test was impaired in apoA-I k
165 a1 -adrenergic vasoconstriction is augmented during exercise following inhibition of inwardly rectify
166                                              During exercise, GLI reduced V O(2) max (71.5 +/- 3.1 vs
167                                              During exercise, glycolytic intermediates, TCA cycle int
168 illary wedge pressure at rest (>15 mm Hg) or during exercise (&gt;25 mm Hg).
169 tify abnormal pulmonary hemodynamic response during exercise (&gt;3.0 mm Hg/L per minute increase), with
170 d influence of temperature on CBF regulation during exercise has not been investigated The present st
171                                              During exercise (high heart rate), there is elevated ste
172 right-to-left shunting through PFO increases during exercise impairing exercise performance.
173 n in resting skeletal muscle that is blunted during exercise in an intensity-dependent manner.
174 utrient uptake and catabolism into myofibers during exercise in an osteocalcin-dependent manner.
175               We characterized QT adaptation during exercise in anorexia.
176                Rates of GLY and GNG increase during exercise in athletes following a mixed macronutri
177 r blockade increased FBF and FVC at rest and during exercise in both groups, although the increase in
178 ntilation were higher (P < 0.05) at rest and during exercise in both patients with ILD and patients w
179  oral contraceptive pill (OCP) (>=12 months) during exercise in dry and humid heat, across their acti
180 a role in regulating blood flow and pressure during exercise in health, little is known about the rol
181 f pulmonary artery (PA) function at rest and during exercise in HF patients without reducing systemic
182 s and pulmonary artery pressures at rest and during exercise in HFpEF.
183 f intravascular ATP in blood flow regulation during exercise in humans.
184 a therapeutic approach to improve blood flow during exercise in hypertension.
185 e therapeutic approach to improve blood flow during exercise in hypertension.
186 derangements of cardiac failure that develop during exercise in individuals with HFpEF.
187 luding a general impairment of substrate use during exercise in LKB1 MKO mice.
188  report that an endogenous molecule produced during exercise in male mice induces the Mus musculus Bd
189 was a shift to a higher and more stable CHOx during exercise in men but not in women.
190  increasing endothelium-dependent signalling during exercise in older adults can improve sympatholysi
191 L in both OL and OO, SSL area only increased during exercise in OO.
192             Shunting significantly increased during exercise in patients with COPD with PFO.
193 hat sildenafil would reduce filling pressure during exercise in patients with diastolic dysfunction a
194 buterol would improve pulmonary vasodilation during exercise in patients with HFpEF, without increasi
195 ng the prognostic value of B-line assessment during exercise in patients with HFpEF.
196                  PCWP was higher at rest and during exercise in patients with LA volume index >/=35 m
197 did not decrease filling pressure at rest or during exercise in post-myocardial infarction patients w
198  increases rates of whole-body fat oxidation during exercise in race walkers over a range of exercise
199 xis is necessary to increase muscle function during exercise in rodents and humans.
200 oves pulmonary vascular function at rest and during exercise in selected patients with HF and EF >=40
201 take (V'O2) slope were significantly greater during exercise in subjects with asthma or misdiagnosed
202 ed indices of arterial stiffness at rest and during exercise in subjects with HFpEF and hypertensive
203 ies were prospectively conducted at rest and during exercise in subjects with invasively proven HFpEF
204 ot reduce muscle glucose uptake or oxidation during exercise in vivo, excluding a general impairment
205 nd uptake of lactate and glucose are similar during exercise in young and older individuals.
206 nary arteriovenous anastomoses are recruited during exercise, in hypoxia, and when cardiac output is
207 ic changes in the respiratory exchange ratio during exercise indicated that LFABP(-/-) mice use more
208 al mechanisms governing cutaneous blood flow during exercise-induced heat stress and provide directio
209 de a cascade of pathophysiological responses during exercise-induced ischemia and reperfusion at rest
210 ance, and degradation of individual proteins during exercise-induced muscle adaptation.
211 nce, oxygen delivery, and oxygen consumption during exercise; interestingly, these changes occurred i
212 istribution of blood flow to skeletal muscle during exercise is altered with advancing age.
213 portance of this size variation to diffusion during exercise is reinforced by functional links betwee
214  Elevated left atrial pressure, particularly during exercise, is a key contributor to morbidity and m
215 o an increased perception of dyspnea, which, during exercise, is mainly associated with systemic infl
216 hanisms, in particular the dynamic component during exercise, is of timely importance.
217 y manifests in vascular beds highly perfused during exercise, it has been postulated that increased b
218 in the dynamic deterioration of secondary MR during exercise, its functional and prognostic impact, a
219           Limited heart rate (HR) rise (HRR) during exercise, known as chronotropic incompetence (CI)
220 is indicative of high dead space ventilation during exercise, leading to excessive and inefficient ve
221 to compensate for the further increase in Ea during exercise led to deterioration in Ees/Ea.
222                                              During exercise, leg substrate utilization (respiratory
223 Compensatory increases in minute ventilation during exercise maintained alveolar ventilation and arte
224 d is closely related to RV systolic function during exercise, maximal exercise capacity, and survival
225 lustrate how ROS released from muscle fibres during exercise may help maintain the integrity of axons
226 vel methods which increase muscle activation during exercise may improve responses of mechanically ve
227 ysis) is critical for maintaining blood flow during exercise-mediated sympathoexcitation.
228 ther the cellular origin of circulating IL-6 during exercise nor the means by which this cytokine enh
229            Abnormal pulsatile aortic loading during exercise occurs in HFpEF independent of hypertens
230 ng men demonstrated net leg glycerol release during exercise, older men showed net glycerol uptake.
231  could enable monitoring of electrolyte loss during exercise or for individuals working in extreme en
232 leep or at rest were more common than deaths during exercise or with emotional stress: 82% versus 16%
233  the majority of circulating IL-6 detectable during exercise originates from muscle and that to incre
234 systemic vascular resistance index decreased during exercise (P<0.0001).
235 lines, E/e', and BNP significantly increased during exercise (P<0.001 for all).
236 ced increases observed in femoral blood flow during exercise (P<0.05 versus rest) in proportion to th
237 xia (P<0.05 versus normoxia), and especially during exercise (P<0.05 versus rest), with the most pron
238 ndex was reduced at rest and reduced further during exercise (P=0.008-interaction P=0.029).
239   In response to skeletal muscle contraction during exercise, paracrine factors coordinate tissue rem
240 5.5 mm Hg to 30.2 +/- 14.3 mm Hg, p < 0.001) during exercise (paradoxical response to exercise [PRE])
241 eous vasodilatation is reportedly diminished during exercise performed at a high (700 W) relative to
242  We propose that greater oxygen availability during exercise permits females to sustain a higher rela
243                                              During exercise, postprandial EE was lower after EGCG th
244 quired to eliminate carbon dioxide, VE/VCO2) during exercise potently predicts outcomes in advanced h
245 l and elevates skeletal muscle O(2) delivery during exercise predominantly in fast-twitch type II mus
246 ot enhance alpha1 -mediated vasoconstriction during exercise (Protocol 1: -27 +/- 3%; P = 0.2 vs. con
247 ndependent predictors of the Borg-V'O2 slope during exercise (r(2) = 0.853, P < .001).
248  related to elevated systemic blood pressure during exercise (r=0.49, P=0.0005).
249 edge pressure at rest (r=0.63, P<0.0001) and during exercise (r=0.57, P<0.0001).
250 he consumption of whole eggs with egg whites during exercise recovery in young men.In crossover trial
251 Recent evidence suggests muscle contractions during exercise release factors into the blood which cro
252  in the open probability of the CFTR channel during exercise, resulting in a decrease in reabsorption
253 f the DCA muscle leads to arytenoid collapse during exercise, resulting in poor performance.
254 ed venous o2 content difference, [C(a-v)o2]) during exercise significantly contributes to impaired ex
255  increasing endothelium-dependent signalling during exercise significantly enhanced the ability of co
256  breakdown of IMTG in type I fibres occurred during exercise (SIT 27 +/- 13%, ET 43 +/- 6%; P < 0.05)
257 bit elevated rates of carbohydrate oxidation during exercise, taking advantage of its high ATP yield
258 on peak metabolic equivalents (MET) achieved during exercise test and eight categories based on fitne
259 s, and these abnormalities are more apparent during exercise testing, with little relationship at res
260 d >85% of their maximum-predicted heart rate during exercise testing.
261 lumes and arterial pressure both at rest and during exercise than HCM patients in whom the gradient i
262 ests) showed a slower increase in heart rate during exercise than in the placebo-treated group.
263 anxiety were more likely to exhibit ischemia during exercise than women without anxiety (odds ratio,
264 erature-mediated elevations in posterior CBF during exercise that are independent of changes in PCO2
265 eft ventricular (LV) end-diastolic pressures during exercise that contribute to dyspnea.
266                                              During exercise, the blood pressure (BP) response is exa
267                                              During exercise, the structural group had a higher systo
268                                              During exercise there is a balance between vasoactive fa
269 athways are not obligatory for sympatholysis during exercise; therefore, we tested the hypothesis tha
270                                              During exercise, this results in ventricular-pulmonary v
271                                              During exercise, this sensor reveals clearly the onset a
272 ics, exposed in this cohort by exercise (TMR during exercise, TMR(ex)) and recovery from exercise (TM
273                                 Qp increased during exercise to a greater extent following IASD compa
274 ic response of individual proteins in humans during exercise training.
275 rified fatty acid, and glucose were measured during exercise treadmill test in C57/BL6 mice fed eithe
276 nce as interactions at distinct body changes during exercise, using complex network models.
277                                              During exercise ventilation increased by 6.64 +/- 0.84 i
278 on influences oxygen consumption at rest and during exercise via alterations in cardiac output and mi
279         Despite having no direct supervision during exercise, virtually supervised Home-HIT resulted
280 (-1) , cardiac output = 5.70 L min(-1) ) and during exercise ( VO2 = 2.75 L min(-1) , cardiac output
281 in the endogenous rate of glucose appearance during exercise was blunted in the KO mice because of a
282 primary end point of ventricular arrhythmias during exercise was compared between the flecainide and
283                                         PCWP during exercise was compared between treatment groups us
284      The median ventricular arrhythmia score during exercise was significantly reduced by flecainide
285 etween dyspnea intensity and EMGdi/EMGdi,max during exercise was similar in all three groups.
286 nsor for real-time monitoring of individuals during exercise, we recorded and analyzed the sweat prof
287 tty acids) and endogenous glucose production during exercise were also reduced, and glucose infusion
288           Significant changes in flow and KE during exercise were identified by using t tests.
289                   Changes in the QT interval during exercise were measured, and QT/RR-interval slopes
290 our oxygen, and those with desaturation only during exercise were prescribed oxygen during exercise a
291        Rates of EGP and GLY both at rest and during exercise were significantly lower in the LCHF gro
292         Work rate and mean arterial pressure during exercise were similar in controls and SIPE-suscep
293 t times of greater need for NO signaling, as during exercise when left ventricular filling pressures
294 w that K(IR) channels augment vasodilatation during exercise which demands greater muscle fibre recru
295  related to the magnitude of fatigue accrued during exercise, which may explain the reported consiste
296    Maximal pressures increased significantly during exercise, while end-diastolic volumes were essent
297  patients, sildenafil improved cardiac index during exercise with a decrease in total pulmonary resis
298 ears +/- 1 [standard deviation]) at rest and during exercise with an MRI-compatible exercise stepper
299 ers diagnosed with hypertension were studied during exercise with either saline or BQ-123 (ET(A) rece
300 s in the maximal rate of fat oxidation (MFO) during exercise with potential implications for metaboli

 
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