ライフサイエンスコーパス: dysarthria

1 l known to induce auditory disorders such as dysarthria.

2 h impaired gait coordination, dysmetria, and dysarthria.

3 irments except for limb motor impairment and dysarthria.

4 ing that is distinguishable from aphasia and dysarthria.

5 gait ataxia, upper limb incoordination, and dysarthria.

6 a, cerebral atrophy and speech delay/apraxia/dysarthria.

7 toxicity evidenced by ataxia, confusion, and dysarthria.

8 , mild ataxia, mild cognitive deficiency and dysarthria.

9 ith amyotrophic lateral sclerosis and severe dysarthria.

10 differed from that for rigidity, tremor, or dysarthria.

11 ism; (ii) prominent AOS; and (iii) prominent dysarthria].

12 antly more frequent and permanent cerebellar dysarthria (12% vs 54%; p=0.009) significantly less freq

13 pathy, 53%; cerebellar ataxia, dysmetria, or dysarthria, 38%; and encephalopathy, 27%.

14 ted signs and symptoms were dysphagia (53%), dysarthria (39%), and generalized weakness (37%).

15 turbance (99%), pyramidal involvement (95%), dysarthria (90%), vision disturbances (82%), with all bu

16 axia, sensory loss, reduced tendon reflexes, dysarthria, absent lower limb reflexes, and loss of posi

17 In a person with ALS and severe dysarthria, an intracortical speech neuroprosthesis reac

18 ; and (ii) that, within the left hemisphere, dysarthria and AoS map onto dorsally versus ventrally lo

19 assessed motor speech features indicative of dysarthria and apraxia of speech (AOS).

20 rred from an outside facility with worsening dysarthria and confusion after having presented 4 weeks

21 ed 2 Omani families with HSP, short stature, dysarthria and developmental delay-core features of Troy

22 Dysarthria and disequilibrium were common in patients wi

23 of somatosensory sensations, motor symptoms, dysarthria and multimodal responses were significantly a

24 The sensory ataxic neuropathy with dysarthria and ophthalmoparesis (SANDO) syndrome is a su

25 Alpers syndrome, sensory ataxia, neuropathy, dysarthria and ophthalmoparesis (SANDO), Parkinsonism, a

26 ome to adult-onset sensory ataxic neuropathy dysarthria and ophthalmoparesis.

27 In addition, many patients exhibit dysarthria and/or receptive agrammatism.

28 and communicative, but with choreoathetosis, dysarthria, and an unsteady gait.

29 s a subacute encephalopathy, with confusion, dysarthria, and dysphagia, and that progresses to severe

30 al speech therapy can lead to improvement of dysarthria, and intensive programmes have had substantia

31 cally characterized by gait and limb ataxia, dysarthria, and nystagmus.

32 t movement disorder characterized by ataxia, dysarthria, and optic atrophy.

33 t over hours to days of dystonia, dysphagia, dysarthria, and parkinsonism.

34 had an atypical clinical triad of epilepsy, dysarthria, and paroxysmal kinesigenic dystonia, and a h

35 temic manifestations, including ataxic gait, dysarthria, and upper and lower limb weakness.

36 parate occasions developed transient ataxia, dysarthria, and weakness within 3 days of returning from

37 ic paraplegia, intellectual disability (ID), dysarthria, and white matter abnormalities.

38 right eyelid with narrow palpebral fissure, dysarthria, anisocoria (narrower pupil on the right side

39 (sensory/motor) or negative (speech arrest, dysarthria, anomia, phonological and semantic paraphasia

40 events are rare, but adverse events such as dysarthria are common and probably under-reported.

41 Blurred vision, dysphagia, and dysarthria are common initial complaints.

42 cognitive decline, dysphagia, optic atrophy, dysarthria, as well as urinary and bowel incontinence.

43 She had dysarthria but was able to speak in full sentences.

44 AoS versus dysarthria, highlighting (i) that dysarthria, but not AoS, is significantly influenced by

45 ter small focal lesions (ataxic hemiparesis, dysarthria-clumsy hand syndrome, dysarthria-dysmetria an

46 auses focal seizures, generalized hypotonia, dysarthria, congenital ataxia and, in one case, cerebral

47 t accuracies of 98.74% and 99.86% for binary dysarthria detection, and 95.69% and 97.91% for multi-cl

48 ed brainstem symptoms (eg, isolated vertigo, dysarthria, diplopia) are not consistently classified as

49 of directionality often improves dyskinesia, dysarthria, dysesthesia and pyramidal side effects.

50 remor severity correlated with the degree of dysarthria, dysmetria and dysdiadochokinesia but not wit

51 On examination, there was slight dysarthria, dysmetria on a finger-to-nose test, slowing

52 Physical examination revealed dysarthria, dysmetria, and ataxia.

53 ads to moderate disability with gait ataxia, dysarthria, dysmetria, mild oculomotor abnormalities, an

54 emiparesis, dysarthria-clumsy hand syndrome, dysarthria-dysmetria and dysarthria-facial paresis).

55 onset, ID, progressive spastic paraparesis, dysarthria, dysmorphisms, and white matter abnormalities

56 had a rapid progression with disequilibrium, dysarthria, dysphagia, and central hypoventilation, and

57 was preceded by gait instability followed by dysarthria, dysphagia, ataxia, or chorea.

58 pastic tetraparesis and truncal instability, dysarthria, dysphagia, cerebellar ataxia, and cognitive

59 ributed to other anatomical regions, such as dysarthria, dysphagia, pseudobulbar affect, eye movement

60 Patients had dystonia, chorea, parkinsonism, dysarthria, dysphagia, seizures, cognitive abnormalities

61 s associated with AS (ataxia, action tremor, dysarthria, dysphagia, sialorrhea and excessive chewing/

62 lessly progressive choreoathetoid movements, dysarthria, dysphagia, spastic paralysis, and behavioral

63 Both patients exhibited dysarthria, dysphagia, tongue atrophy, neck extensor wea

64 h prominent bulbar palsies such as diplopia, dysarthria, dysphonia, and dysphagia that would typicall

65 characterized by gait abnormalities, ataxia, dysarthria, dystonia, vertical gaze palsy, and cognitive

66 ted detection and severity classification of dysarthria enables timely intervention and tailored clin

67 ability in the presence and severity of AoS, dysarthria, expressive agrammatism or receptive agrammat

68 umsy hand syndrome, dysarthria-dysmetria and dysarthria-facial paresis).

69 the subthalamic nucleus was associated with dysarthria, fatigue, paraesthesias, and oedema, whereas

70 on after having presented 4 weeks prior with dysarthria, gait ataxia, and bilateral upper extremity w

71 vents in the active-treatment group included dysarthria, gait disturbance, loss of taste, visual dist

72 ond presented at 3.5 years with gait ataxia, dysarthria, gross motor regression, hypotonia, ptosis an

73 uroanatomical distinction between AoS versus dysarthria, highlighting (i) that dysarthria, but not Ao

74 ctive reports of a worsening in pre-existing dysarthria, hypophonia and hypersalivation/drooling foll

75 Bulbar signs and symptoms, including dysarthria in 10 and dysphagia in 8 patients, also were

76 Ocular signs were observed in 84% and dysarthria in 66%.

77 Dysarthria in Parkinson's disease can be characterised b

78 ), cognitive impairment (8/9), ataxia (6/9), dysarthria in probands with verbal ability (6/9), hypoto

79 Dysarthria (in four patients assigned to neurostimulatio

80 Treatment studies for poststroke dysarthria indicate that speech supplementation strategi

81 Dysarthria is a neurological speech disorder characteriz

82 ehabilitation and strategies for aphasia and dysarthria is scarce.

83 ffects on parkinsonian speech, management of dysarthria is still challenging for the clinician and sh

84 Common accompanying features included dysarthria, labile emotions, and unsteady gait.

85 gnitive dysexecutive slowing, forgetfulness, dysarthria, mood changes, urinary symptoms, and short-st

86 At presentation, dysarthria (Motor Speech Examination median [IQR] score:

87 her symptoms include joint hyperflexibility, dysarthria, myopia, and growth delay.

88 ): hemiparesis, vertigo/dizziness, diplopia, dysarthria, nystagmus, nausea/vomiting, head pain, hemia

89 rd of developing disabling symptoms, such as dysarthria or dysphagia (smaller allele HR = 3.40, P < 0

90 nvolvement by weakness, wasting, spasticity, dysarthria or dysphagia of one central nervous system re

91 in movement "vigor," leading to "hypokinetic dysarthria" or "hypophonia." This is an additional examp

92 tients, the initial symptoms included falls, dysarthria, or clumsiness followed by a complete cerebel

93 0%), vision disturbances (82%), with all but dysarthria presenting early in the disease course.

94 nt of our hospital with a 2-month history of dysarthria, progressively worsening vertigo, and difficu

95 enting with nfvPPA, presence of early severe dysarthria, relatively selective white matter atrophy at

96 phic lateral sclerosis--including dysphagia, dysarthria, respiratory distress, pain, and psychologica

97 Higher dysarthria severity, reduced words per minute and expres

98 ment was correlated with overall symptom and dysarthria severity.

99 networks associated with stimulation-induced dysarthria (SID) and to predict stimulation-induced wors

100 Stimulation-induced dysarthria (SID) is a troublesome and potentially therap

101 anguage sites); the left vPMC (speech arrest/dysarthria sites) and the right SMA (negative motor resp

102 inspiratory sighs, severe dysphonia, severe dysarthria, snoring, cold hands and feet, pathological l

103 sclerosis (ALS) with tetraparesis and severe dysarthria underwent surgical implantation of four micro

104 disorder, characterized by spells of ataxia, dysarthria, vertigo, and migraines, associated with muta

105 al negative symptoms (eg, isolated diplopia, dysarthria, vertigo, ataxia, sensory loss, and bilateral

106 sory symptoms, oscillopsia, dysautonomia and dysarthria were also variably associated.

107 Dysphagia and dysarthria were the most commonly reported cranial nerve

108 Imbalance, sensory disturbances and dysarthria were the most frequent acute adverse events.

109 re variably effective for the alleviation of dysarthria, whereas bilateral procedures typically lead

110 ogic illness defined initially by ataxia and dysarthria, which fluctuated in relation to each subsequ

111 ospital with severe non-systemic vertigo and dysarthria, which had lasted for a couple of weeks.

112 esented with seizure, right hemiparesis, and dysarthria with positive findings for severe acute respi

113 g to acquired aphasia, apraxia of speech and dysarthria with special attention to clinically signific

114 non-fluent speech, executive dysfunction and dysarthria without apraxia of speech or frank agrammatis