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1                           Here, we show that dysbindin-1, a protein decreased in the brain of individ
2                                              Dysbindin-1, a protein that regulates aspects of early a
3 ctional roles of the three major isoforms of dysbindin-1, (A, B, and C) remain unknown, we generated
4 an hippocampal formations revealed that both dysbindin-1 and snapin are concentrated in tissue enrich
5 trophysiological deficits related to reduced dysbindin-1 and the potential role of PV cells, we exami
6                           Further studies of dysbindin-1 and the proteins with which it interacts can
7 ucleotide polymorphisms in the gene encoding dysbindin-1 at 6p22.3.
8                               We explored in dysbindin-1-deficient mice (dys-/-) (1) schizophrenia-re
9                                              Dysbindin-1 disruption altered dopamine-related behavior
10 tions in the gene encoding the novel protein dysbindin-1 (DTNBP1) are among the most commonly reporte
11                                 The gene for dysbindin-1 (DTNBP1) is located at 6p and has also been
12 ne-protein phosphatase-alpha (RPTPalpha) and dysbindin-1, each of which reduces Src activity via prot
13                            The gene encoding dysbindin-1 has recently been implicated in susceptibili
14 ion of this gene and of its encoded protein, dysbindin-1, have been reported in the brains of schizop
15 ric diagnoses, suggesting a general role for dysbindin-1 in cognition.
16                   In contrast to the role of dysbindin-1 in glutamatergic transmission, gamma-band ab
17                        We found that reduced dysbindin-1 in mice yielded deficits in auditory-evoked
18                              The function of dysbindin-1 in presynaptic, postsynaptic and microtubule
19 ate here that snapin is a binding partner of dysbindin-1 in vitro and in the brain.
20                                              Dysbindin-1 is best known as dystrobrevin-binding protei
21 nding, immunoelectron microscopy showed that dysbindin-1 is located in (i) synaptic vesicles of axosp
22  et al. show that, contrary to expectations, dysbindin-1 is located presynaptically in glutamatergic
23 oded protein dystrobrevin-binding protein 1 (dysbindin-1) is often reduced in excitatory cortical lim
24 ound between case-control differences in any dysbindin-1 isoform and the case-control differences in
25 ver, if the protein reductions encompass all dysbindin-1 isoforms or if they are associated with decr
26                                          How dysbindin-1 might affect glutamate release nevertheless
27 and novel object recognition are impaired in dysbindin-1 null mice.
28                                   We studied dysbindin-1 null mutant mice (Dys(-/-)) to shed light on
29 axon terminal loss or neuroleptic effects on dysbindin-1 or VGluT-1.
30 s of the DTBPN1 gene associated with reduced dysbindin-1 protein (Dys) expression negatively impact c
31 izophrenia populations displayed presynaptic dysbindin-1 reductions averaging 18-42% (P = 0.027-0.000
32 bindin-1C and known post-synaptic effects of dysbindin-1 reductions in the rodent equivalent of the D
33       Our findings indicate that presynaptic dysbindin-1 reductions independent of the dystrophin gly
34                                              Dysbindin-1 regulates D2-receptor trafficking and is imp
35       The dominant circuit impact of reduced dysbindin-1 was impaired inhibition, and PV cell immunor