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1 diction and chronic pain, but are limited by dysphoric and aversive side effects.
2 e increases in negative emotional states and dysphoric and stress-like responses in the withdrawal/ne
3         One was characterized by symptoms of dysphoric arousal (anhedonia, anxiety, hypervigilance) w
4 ences with others, for example by undergoing dysphoric collective rituals together, can lead to "iden
5 ppa opioid receptor (KOR) system encodes the dysphoric component of the stress response and controls
6                                 Premenstrual dysphoric disorder (PMDD) affects over 5% of women, with
7  allopregnanolone in women with premenstrual dysphoric disorder (PMDD) and in asymptomatic control wo
8 in healthy women and those with premenstrual dysphoric disorder (PMDD) and that a menstrual cycle pha
9 te evidence for the validity of premenstrual dysphoric disorder (PMDD) and the inclusion of the disor
10                                 Premenstrual dysphoric disorder (PMDD) disrupts the lives of millions
11 tedly experienced by women with Premenstrual Dysphoric Disorder (PMDD) during the late luteal phase o
12 is substantial information that premenstrual dysphoric disorder (PMDD) is a clinically significant di
13                                 Premenstrual dysphoric disorder (PMDD) is a common mood disorder, cha
14                                 Premenstrual dysphoric disorder (PMDD) is characterized by affective,
15                                 Premenstrual Dysphoric Disorder (PMDD) is characterized by debilitati
16 tric illnesses tested, although premenstrual dysphoric disorder (PMDD) may be an exception.
17 y demonstrated that symptoms of premenstrual dysphoric disorder (PMDD) remit during ovarian hormone s
18                                 Premenstrual dysphoric disorder (PMDD) symptoms are eliminated by ova
19  are efficacious treatments for premenstrual dysphoric disorder (PMDD) when given daily or for half o
20 mood and behavioral symptoms in premenstrual dysphoric disorder (PMDD), a common, recently recognized
21 premenstrual syndrome (PMS) and premenstrual dysphoric disorder (PMDD), are associated with significa
22 e distinguishable in women with premenstrual dysphoric disorder (PMDD).
23 se women also meet criteria for premenstrual dysphoric disorder (PMDD).
24 epilepsy and enhance anxiety in premenstrual dysphoric disorder (PMDD).
25 sychiatric disorders, including premenstrual dysphoric disorder (PMDD).
26 errecognized conditions such as Premenstrual Dysphoric Disorder (PMDD).
27                   Patients with premenstrual dysphoric disorder (whose symptoms had remitted during t
28 led protocol to nine women with premenstrual dysphoric disorder and 11 healthy female volunteers in t
29  were measured in 27 women with premenstrual dysphoric disorder and 21 comparison women during the th
30                      Women with premenstrual dysphoric disorder and a past history of major depressiv
31 ere particularly pronounced for premenstrual dysphoric disorder and for PMDs with symptom onset befor
32 h major depression, and 10 with premenstrual dysphoric disorder and in 34 normal comparison subjects.
33 ts to examine the literature on premenstrual dysphoric disorder and provide recommendations regarding
34 icipation, 243 met criteria for premenstrual dysphoric disorder and were randomized; 200 women comple
35 h, including menstruation (with premenstrual dysphoric disorder appearing for the first time in recen
36                                 Premenstrual dysphoric disorder appears to be associated with seroton
37 anic disorder and patients with premenstrual dysphoric disorder are highly susceptible to CO(2)-induc
38                   In women with premenstrual dysphoric disorder but no past major depressive disorder
39 s of serotonergic deficiency in premenstrual dysphoric disorder by measuring the prolactin response t
40               The patients with premenstrual dysphoric disorder experienced a return of symptoms 24 h
41 normal subjects, the women with premenstrual dysphoric disorder had a significantly blunted prolactin
42 icacy for premenstrual syndrome/premenstrual dysphoric disorder is a general or more serotonergic eff
43                                 Premenstrual dysphoric disorder is an important cause of symptoms and
44                                 Premenstrual dysphoric disorder is often associated with major depres
45 d a state-dependent decrease in premenstrual dysphoric disorder might imply a possible continuum betw
46 he panic rate for patients with premenstrual dysphoric disorder was similar to that for panic disorde
47 to determine whether women with premenstrual dysphoric disorder with or without prior major depressiv
48 anxiety, postpartum depression, premenstrual dysphoric disorder, and schizophrenia and (2) whether an
49  posttraumatic stress disorder, premenstrual dysphoric disorder, and social phobia.
50 uding premenstrual syndrome and premenstrual dysphoric disorder, have symptom onset during the teen y
51 or depression and patients with premenstrual dysphoric disorder, respectively).
52 he menstrual cycle, as in DSM-5 premenstrual dysphoric disorder, symptom changes of similar magnitude
53                                 Premenstrual dysphoric disorder, which affects 2%-5% of premenopausal
54 ch as postpartum depression and premenstrual dysphoric disorder.
55  efficacy of SSRI treatment for premenstrual dysphoric disorder.
56 nhibitors (SSRIs) in women with premenstrual dysphoric disorder.
57 evere premenstrual syndrome and premenstrual dysphoric disorder.
58 involved in the pathogenesis of premenstrual dysphoric disorder.
59 uch as acne, endometriosis, and premenstrual dysphoric disorder.
60 th the observation of an association between dysphoric disorders of epilepsy and POE described nearly
61 ween subtypes of depressive and premenstrual dysphoric disorders.
62 nd to be an important site of action for the dysphoric effects of dynorphin-kappa-opioid receptor sys
63  signaling results in analgesia, whereas the dysphoric effects of KOR agonists are mediated by a diff
64  this endogenous opioid peptide mediates the dysphoric effects of marijuana.
65 eptor (KOR) system in the brain mediates the dysphoric effects of stress, and KOR antagonists may hav
66 y been revived by studies showing that their dysphoric effects require arrestin recruitment, whereas
67 one+dronabinol 10 mg produced a high rate of dysphoric effects, and hydromorphone+dronabinol 5 mg and
68 ignaling may be effective analgesics lacking dysphoric effects.
69 ns analysis of participants who had a single dysphoric episode, and they were replicated in an indepe
70 ted that no adverse life events caused their dysphoric episodes reported fatigue, appetite gain, and
71       Practically, our account of how shared dysphoric experiences produce identity fusion helps us b
72                                              Dysphoric features are statistically salient in patients
73  pain thresholds, anxiety-like behavior, and dysphoric-like responses.
74 e that varenicline and cytisine diminish the dysphoric-like state associated with nicotine withdrawal
75                                In this view, dysphoric mania is associated with paranoid-destructive
76              Effective treatment of acute or dysphoric mania is provided by modern antipsychotics, so
77 s and may be a clinical marker for recurrent dysphoric mania.
78 tidepressants during bipolar mixed states or dysphoric manias.
79 pressive mixed states (mixed depression) and dysphoric (mixed) hypomania (opposite polarity symptoms
80                                Depressive or dysphoric-mixed episodes were more prevalent in pregnant
81 ater total depressive symptoms, specifically dysphoric mood and somatic complaints.
82 st that persistent mood symptoms and overall dysphoric mood are associated with the early perimenopau
83 crease a woman's vulnerability to an overall dysphoric mood during the early perimenopausal period.
84  the changing hormonal milieu contributes to dysphoric mood during transition to menopause.
85  17.0% of the association between stress and dysphoric mood for 27 months after imaging (beta = .04,
86 ponse in terms of perceptual alterations and dysphoric mood relative to those without such a family h
87 ore symptoms of hypothyroidism (P<0.001) and dysphoric mood states (P<0.001) after withdrawal of thyr
88 anning is associated with fewer symptoms and dysphoric mood states.
89 ect of being early perimenopausal on overall dysphoric mood was greatest among women with an educatio
90            Depressive symptoms, particularly dysphoric mood, presage future cognitive losses among el
91        Five independent factors representing dysphoric mood, psychomotor pressure, psychosis, increas
92  features of mania and 5 features related to dysphoric mood.
93 such as early alcohol or nicotine use, early dysphoric or anhedonic mood, conduct disorder, and child
94 ssified as classic (predominately euphoric), dysphoric, or depressed.
95 y is often beneficial for gender-diverse or -dysphoric patients.
96 y of patients best characterized as having a dysphoric personality constellation.
97    Kappa-opioid receptor (KOR) agonists have dysphoric properties in humans and are aversive in roden
98                     Here, we report that the dysphoric properties of chronic stress are encoded by th
99 ime elapses after trauma, fear circuitry and dysphoric PTSD symptoms appear to emerge as connected ne
100      Elevations in ICSS thresholds reflect a dysphoric state and a lowering of thresholds is indicati
101  melancholia report a distinct and intrusive dysphoric state during internally generated thought.
102  opioid system contributes to depressive and dysphoric states, but whether this system contributes to
103 isorder, especially for early depressive and dysphoric states.
104 iated transcription within the NAc regulates dysphoric states.
105 ink between cocaine-cue responses and normal dysphoric states.
106 s on medication also had fewer fixations for dysphoric stimuli compared with depressed participants n
107 healthy volunteers and resolution of chronic dysphoric symptoms in depressed patients were examined w
108  different mechanisms underlie cognitive and dysphoric symptoms in nondemented patients with Parkinso
109 prove quality of life, and further alleviate dysphoric symptoms in transgender women.
110                                              Dysphoric symptoms, such as sadness and anhedonia, respo
111 sed transdiagnostic treatments for loss (ie, dysphoric) symptoms.
112 d with scores on mnemonic, visuospatial, and dysphoric tests.
113  assessing selective attention for positive, dysphoric, threatening, and neutral stimuli in addition

 
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