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1 cochlear nucleus following occlusion of the ear canal.
2 h functions comparable to stimulation in the ear canal.
3 of the middle ear cavity and opening of the ear canal.
4 at the stapes, and as sound pressure in the ear canal.
5 e exposure, and increased after ligating the ear canal.
6 measures of speech recorded in the external ear canal.
7 survey organisms present in the human outer ear canal.
8 ransfer function (HRTF) from sound source to ear canal.
9 ion and music privately without blocking the ear canal.
10 lar surface formed by the dorsal wall of the ear canal.
11 citation at the entrance of the guinea pig's ear canal.
12 issions (SOAEs) can often be measured in the ear canal.
13 rms from nearby speakers mix together in our ear canals.
14 dge practically eliminates the effect of the ear-canal air volume interposed between the tympanic mem
17 values were lower for otoscopy (72% for the ear canal and 86% for the tympanic membrane), throat and
21 sked to capture pictures and videos of their ear canals and oropharynx with digital videoscopes and t
22 e, lies at a steep angle with respect to the ear canal, and has organized radial and circumferential
23 to developing tumors in the skin, the inner ear canal, and the oral epithelium after 1 year of age.
24 hearing aids generate amplified sound in the ear canal, and they are the standard of care for patient
25 stortion-products that are detectable in the ear canal as distortion-product otoacoustic emissions (D
29 anes in each ear and internally via a narrow ear canal (EC) derived from the respiratory tracheal sys
31 middle ear effusion (MEE) samples, external ear canal (EEC) lavages, and nasopharynx (NPH) samples f
33 We found an optimal configuration using an ear canal electrode and low-frequency (<300 Hz) sinusoid
34 6/Adgrg6 regulates Schwann cell myelination, ear canal formation, and heart development; and GPR126 m
37 d sound delivery devices and receiver-in-the-ear-canal hearing aid configuration) to reduce the occlu
38 of 10,000 FE simulations of stapes velocity, ear-canal input impedance, and absorbance, paired with s
41 r results suggest, therefore, that the outer ear canal may serve as a reservoir for normally commensa
43 n animals 7 to 14 d prior to eye-opening and ear canal opening, spontaneous activity in both sensory
44 h then slows until PND11, around the time of ear canal opening; subsequently, MGv accelerates growth
46 stnatal day (P)7-P9) and after (P14-P20) the ear canal opens and when circuits are mature (P60-P80).
47 imple as an otoscope to better visualize the ear canal or as complex as a wireless capsule endoscope
48 amic afferents coincides with the opening of ear canal (~P11 in mice) and precedes the later critical
49 ough the air, and internally via a narrowing ear canal running through the leg from an acoustic spira
52 , middle and inner ear (e.g., short external ear canal, small tympanic membrane, large oval window).
53 and amplitude growth were calculated for an ear canal speaker versus the intracochlear actuator for
54 s (EMREOs), pressure changes recorded in the ear canal that occur in conjunction with simultaneous ey
55 greater than 70% (ranging from 74.5% for the ear canal to 99.7% for hemorrhagic suffusion) for all va
58 ed from microphones placed in each subject's ear canals, which preserved the interaural time and leve