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1 d in draining lymph nodes nor did it prevent ear swelling.
2 oduction, and neutrophil influx and prevents ear swelling.
3 R4KO mice showed a 1.3-fold increase in mean ear swelling, a 2-fold increase in epidermal thickness,
6 n of MC903 in obese animals led to increased ear swelling and a pronounced Th17-biased inflammatory r
7 king gammadelta T cells) exhibited decreased ear swelling and downregulated expression of IL-22 and I
12 mice and characterized by significantly less ear swelling and inflammatory cell infiltration than in
14 ation effectively decreased contact allergic ear swelling and myeloid immune cell infiltration not on
15 n Bid KO mice and observed markedly enhanced ear swelling and proliferation responses compared with w
16 itivity response, characterized by decreased ear swelling and reduced inflammatory cell infiltrates.
17 antibody to IL-1alpha significantly reduced ear swelling and suppressed the levels of MIP-2, MIP-1al
18 s for in vitro efficacy, and mouse models of ear swelling and systemic anaphylaxis responses for in v
19 ion of PF6-miR-146a nanocomplexes attenuated ear-swelling and reduced the expression of pro-inflammat
22 nal expression of beta gal and inhibited the ear-swelling assay for delayed type hypersensitivity.
24 r hapten challenge but resulted in increased ear swelling at 48 hours and delayed resolution of the i
25 athelicidin demonstrated a large increase in ear swelling, cell infiltration, and MIP-2 expression co
29 ment conditions and mouse genotypes measured ear swelling, epidermal thickness, and cytokine expressi
30 ficient (MyD88(-/-)) C57BL/6 mice had intact ear swelling, exaggerated inflammation, and higher level
31 Tolerance was measured from inhibition of ear swelling in a delayed-type hypersensitivity reaction
33 After hapten sensitization and re-challenge, ear swelling in CCR6-/- animals was reduced 80% as compa
35 adin-specific IgG/IgG2c (in models 1 and 2), ear swelling (in model 1), gluten-dependent enteropathy
36 was orally efficacious in an MMP-12 induced ear-swelling inflammation model in the mouse with a good
37 uces CHS responses, as measured by decreased ear swelling, inhibition of local DETC activation, and a
38 to DNFB in wild-type mice, and DNFB-induced ear swelling is reduced by approximately 50% in TCRdelta
39 yl ester considerably attenuated TPA-induced ear swelling, leukocyte infiltration, epidermal cell pro
40 skin inflammation in both the IL-23-induced ear swelling model and the topical imiquimod model, and
44 ctions into IL-17A(-/-) mice produced little ear swelling (p < 0.001, versus IL-23-injected WT mice)
45 L-22(-/-) mice resulted in relatively little ear swelling (p < 0.09) and epidermal hyperplasia (p < 0
46 e mice, ODCER transgenic mice showed reduced ear swelling, reduced neutrophil infiltration, and decre
48 esponded to DNFB challenge with a pronounced ear swelling response without previous sensitization to
49 expression in basophils was required for the ear swelling response, and basophils promoted the expres
50 deficient DeltadblGATA mice showed only weak ear swelling response, which could be enhanced by eosino
52 dingly, Myo9b(-/-) mice showed an attenuated ear-swelling response in a model of contact hypersensiti
53 igh concentrations of anti-IgE Ab induced an ear-swelling response in these strains, implying some ca
55 let-B irradiation showed markedly suppressed ear swelling responses to dinitrofluorobenzene challenge
56 E and Ag-specific IgG1, and generated strong ear-swelling responses to intradermal administration of
58 t of the inflammatory response, and examined ear swelling, SK activity, vascular permeability, leukoc
59 BPZE1 nasal pretreatment markedly inhibited ear swelling, skin inflammation, and production of pro-i
60 positive allosteric modulator AEA061 reduced ear swelling, skin thickness, erythema, scale formation,
61 y (CHS) response to oxazolone with increased ear swelling, T-cell infiltration, and expression of Ifn
62 utaneous inflammation was evaluated by mouse ear swelling test (MEST), histology, serum IgE levels, a
63 and tested for immune responsiveness by the ear-swelling test for delayed-type hypersensitivity (DTH