ライフサイエンスコーパス: eating disorder

1 piramate reduced weight in adults with binge-eating disorder.

2 patients-in particular, depression and binge eating disorder.

3 racteristic symptomatology observed in binge-eating disorder.

4 otential pharmacological treatment for binge-eating disorder.

5 limia nervosa, and adding BED as a specified eating disorder.

6 l diagnosis when evaluating a patient for an eating disorder.

7 and nutritional complaints suggestive of an eating disorder.

8 ion towards pharmacologically treating binge eating disorder.

9 Binge-eating disorder is the most common eating disorder.

10 strate for observed sex differences in binge-eating disorder.

11 intake of high-fat food (HFF) seen in binge eating disorder.

12 cacy in adults with moderate to severe binge-eating disorder.

13 of therapeutic targets for the treatment of eating disorders.

14 all conditions categorized under feeding and eating disorders.

15 cide attempts are common in individuals with eating disorders.

16 a potential therapeutic for alcohol use and eating disorders.

17 y, obsessive compulsive disorder, autism and eating disorders.

18 e after adjusting for the index individuals' eating disorders.

19 t deposition may be particularly relevant to eating disorders.

20 0 million of people in the world suffer from eating disorders.

21 its associated with addiction and binge-type eating disorders.

22 t are associated with increased incidence of eating disorders.

23 d-habit system, which could differ among the eating disorders.

24 ising therapeutic strategies for obesity and eating disorders.

25 ned more sex-neutral diagnostic criteria for eating disorders.

26 ent strategies tailored for older women with eating disorders.

27 adolescent girls at high risk of obesity and eating disorders.

28 osis, and treatment for males suffering from eating disorders.

29 armacological treatment for bingeing-related eating disorders.

30 athology, including depression, anxiety, and eating disorders.

31 s significant to the cause and expression of eating disorders.

32 ic disorders, alcohol or substance abuse, or eating disorders.

33 RXFP3 as a therapeutic target for binge-like eating disorders.

34 e related to food avoidance commonly seen in eating disorders.

35 al (GI) symptoms are common in patients with eating disorders.

36 elopment of compulsive eating in obesity and eating disorders.

37 gical findings on the cause and treatment of eating disorders.

38 ent evidence on psychological treatments for eating disorders.

39 cess to care, thereby reducing the burden of eating disorders.

40 ion and low interoceptive awareness, such as eating disorders.

41 nd treatment of both compulsive behavior and eating disorders.

42 the upper quintile were classified as having eating disorders.

43 tality in a long-term study of patients with eating disorders.

44 ent implications of cognitive flexibility in eating disorders.

45 pears to be dysregulated in individuals with eating disorders.

46 y disrupt homeostatic mechanisms and lead to eating disorders.

47 (CBT) has shown efficacy in the treatment of eating disorders.

48 ion has been recently proposed as pivotal to eating disorders.

49 a key role in the origin and maintenance of eating disorders.

50 t and current weight are at greatest risk of eating disorders.

51 on, as in the putative role of mass media in eating disorders.

52 of the associations among the conditions and eating disorders.

53 pect of serious clinical conditions, such as eating disorders.

54 tric conditions such as forms of obesity and eating disorders.

55 a notoriously difficult-to-treat symptom of eating disorders.

56 on contributes to failed diets, obesity, and eating disorders.

57 rgets for the treatment of cachexia or other eating disorders.

58 dition (2.3%; 95% CI, 1.7% to 2.9%), and any eating disorder (0%).

59 For eating disorders, 0.8% had partial- or full-criteria bul

60 depression (19% [95% CI, 14%-25%]) and binge eating disorder (17% [95% CI, 13%-21%]).

61 ) and 991 males (0.09% of all males) had any eating disorder, 7680 females (0.70%) and 453 males (0.0

62 And with regards to eating disorders, a closer examination of both full diag

63 e of the shared risk factors for obesity and eating disorders, a targeted prevention of both conditio

64 culture in both the emergence and spread of eating disorders across the globe.

65 Eating disorders affect 13% of females and contribute to

66 the prevalence of concerns with physique and eating disorders among males and their relation to subse

67 mia nervosa and more likely to exhibit binge eating disorder and eating disorder not otherwise specif

68 and obesity, emphasizing binge eating, binge eating disorder and food addiction as useful conceptual

69 ondary outcomes included OBEs at follow-ups, eating disorder and general psychopathologic findings, b

70 ted in a specialist inpatient unit and their eating disorder and psychological distress features.

71 tory processes may contribute to symptoms in eating disorders and addictive disorders, but little is

72 PT may also be effective in the treatment of eating disorders and anxiety disorders and has shown pro

73 We examined the association between eating disorders and death by suicide separately, but th

74 velop much needed new therapeutics to tackle eating disorders and obesity.

75 evelop better pharmacological treatments for eating disorders and obesity.

76 havior may inspire new treatment options for eating disorders and obesity.

77 iology, features, and potential treatment of eating disorders and related body-image concerns in midd

78 Eating disorders and schizophrenia are both moderately t

79 ility contributes to the association between eating disorders and schizophrenia.

80 whether a shared neurobiology contributes to eating disorders and substance abuse, this review focuse

81 Prevalence and correlates of eating disorders and subthreshold conditions.

82 l cortex, insula, and striatum is altered in eating disorders and suggests altered brain circuitry th

83 milial liability for the association between eating disorders and suicide.

84 uicide attempts in individuals with lifetime eating disorders and their relatives.

85 rich opportunity to sharpen animal models of eating disorders and to identify neural mechanisms that

86 0.80 to 0.84), and the highest was 3.62 for eating disorders and urogenital conditions (95% CI, 3.11

87 f BN and PD but not onset of AN, BED, or any eating disorder, and baseline current weight was inverse

88 ating disorders (OED: bulimia nervosa, binge-eating disorder, and eating disorder not otherwise speci

89 sociated with posttraumatic stress disorder, eating disorders, and anxiety disorders other than speci

90 luding substance and behavioural addictions, eating disorders, and attention deficit/hyperactivity di

91 me, obsessive-compulsive spectrum disorders, eating disorders, and autism.

92 role, such as mood, substance-use disorders, eating disorders, and obesity.

93 uding post-traumatic stress disorder [PTSD], eating disorders, and psychoses).

94 disorders were captured by 3 variables (any eating disorder, anorexia nervosa, and bulimia nervosa)

95 rity of individuals seeking treatment for an eating disorder are classified as eating disorder not ot

96 The best treatment options for binge-eating disorder are unclear.

97 ected by stress-related emotional states and eating disorders are comorbid with psychiatric symptoms

98 Eating disorders are complex heritable conditions influe

99 Anorexia nervosa (AN) and related eating disorders are complex, multifactorial neuropsychi

100 Eating disorders are disabling, deadly, and costly menta

101 Incidence rates of eating disorders are higher in schools characterised by

102 and mental disorders, but findings regarding eating disorders are inconsistent and inconclusive.

103 strialized Western Europe and North America, eating disorders are increasingly documented in diverse

104 Eating disorders are lethal and heritable; however, the

105 Eating disorders are life-interrupting psychiatric condi

106 Six main feeding and eating disorders are now recognised in diagnostic system

107 review first identifies diseases with which eating disorders are often confused and then explores fe

108 Eating disorders are one of the most common chronic cond

109 Eating disorders are serious psychiatric conditions requ

110 that pathological eating behaviors and frank eating disorders are surprisingly common in older women,

111 As eating disorders are under-researched, there is a great

112 bulimia nervosa; and the inclusion of binge eating disorder as a formal diagnosis.

113 esearch also supports the inclusion of binge eating disorder as a formal diagnosis.

114 a core feature of some forms of obesity and eating disorders, as well as of the recently proposed di

115 or anxiety, somatoform, neurocognitive, and eating disorders, as well as prolonged grief and autism.

116 Outcome was any diagnosed eating disorder at 16-20 years, as defined by an ICD (9

117 n, attention-deficit/hyperactivity disorder, eating disorders, autism spectrum disorder, substance us

118 ms: anorexia nervosa, bulimia nervosa, binge eating disorder, avoidant-restrictive food intake disord

119 rls deemed at high risk of adult obesity and eating disorders because of a body mass index (BMI) betw

120 Binge-eating disorder (BED) is characterized by recurring epis

121 ey, the average lifetime prevalence of binge eating disorder (BED) was 2%.

122 etamine dimesylate (LDX) vs placebo in binge eating disorder (BED) was evaluated in two multicenter,

123 bese individuals seeking treatment for binge eating disorder (BED) were compared with 19 non-BED obes

124 cts with (n = 30) and without (n = 30) binge eating disorder (BED) were compared with matched healthy

125 Binge-eating disorder (BED), a public health problem associate

126 ia nervosa (AN), bulimia nervosa (BN), binge eating disorder (BED), and purging disorder (PD).

127 he criterion standard for treatment of binge eating disorder (BED), most individuals do not have acce

128 ood, in alcohol use disorders (AUD) or binge-eating disorder (BED), suggest a disturbance in explore-

129 e United States on the epidemiology of binge eating disorder (BED).

130 otypes: pathological gambling (PG) and binge eating disorder (BED).

131 Finally, some eating disordered behaviors, which have until now receiv

132 A 4.4% variation in incidence of eating disorders between schools was seen; after taking

133 and 2.9% had partial or full-criteria binge eating disorder but no association with the outcomes of

134 IPT had significant effects on eating disorders, but the effects are probably slightly

135 The changing face of eating disorders calls for a new conceptualization of cu

136 However, an eating disorder can coexist with another chronic disease

137 ng disorders that appear in the 'Feeding and Eating Disorders' chapter of the Diagnostic and Statisti

138 exia nervosa (AN) is a complex and heritable eating disorder characterized by dangerously low body we

139 Anorexia nervosa (AN) is an eating disorder characterized by extreme hypophagia, hyp

140 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation and ext

141 Anorexia nervosa (AN) is a serious eating disorder characterized by self-starvation, extrem

142 inferred from an appointment at a specialist eating disorder clinic.

143 ention deficit hyperactivity disorder, binge eating disorder, cocaine addiction, obesity, and type 2

144 ns in symptoms of binge eating, purging, and eating disorders compared with standard care.

145 isorders, including gambling disorder, binge eating disorder, compulsive sexual behaviour, and compul

146 ine weight suppression to onset risk of each eating disorder controlling for age, dietary restraint,

147 Eligible adults met DSM-IV-R binge-eating disorder criteria and had moderate to severe bing

148 numerous clinical conditions such as autism, eating disorders, depression, and chronic pain.

149 atives was compared with individuals without eating disorder diagnoses and their relatives.

150 Treatments have been evaluated within each eating disorder diagnosis and across diagnoses.

151 pecified' (EDNOS) was the most common DSM-IV eating disorder diagnosis in both clinical and community

152 , 1,019 males and 15,395 females received an eating disorder diagnosis.

153 he recent publication of revised feeding and eating disorder diagnostic criteria in DSM-5.

154 e, 6 wk, 6 mo, 12 mo, and 24 mo by using the Eating Disorder Diagnostic Interview, the Multidimension

155 lar affective disorder, depressive disorder, eating disorder, drug dependency, or alcohol dependency,

156 To summarize the recent literature examining eating disorders, eating behavior, and body image in mid

157 People with eating disorders (ED) frequently present with inflexible

158 Eating disorders (EDs) are severe, life-threatening ment

159 While eating disorders (EDs) are thought to result from a comb

160 on-deficit/hyperactivity disorder (ADHD) and eating disorders (EDs) frequently co-occur, little is kn

161 ion between impulsivity, dietary intake, and eating disorders (EDs) in a general population.

162 direction of the association between BMI and eating disorders (EDs) in adults via a two-sample MR app

163 Eating disorders (EDs) lead to multiple psychiatric and

164 sa and bulimia nervosa are common and severe eating disorders (EDs) of unknown etiology.

165 atus (SES) is associated with higher risk of eating disorders (EDs).

166 s have confirmed that the DSM-5 criteria for eating disorders effectively reduce the proportion of ED

167 characteristics are associated with rates of eating disorders, even after accounting for characterist

168 The primary outcome measure was the Eating Disorder Examination (EDE) Global score.

169 re and after weight restoration by using the Eating Disorder Examination interview and the Global Sev

170 The main outcome measure was the Eating Disorder Examination interview, which was adminis

171 asures were bulimic symptoms assessed by the Eating Disorder Examination-Questionnaire.

172 both treatments, substantial improvements in eating disorder features and general psychopathology wer

173 rapy and was generally faster in alleviating eating disorder features and general psychopathology.

174 This issue provides a clinical overview of eating disorders focusing on prevention, diagnosis, trea

175 tion of both full diagnostic and less common eating disorders following bariatric surgery would be pr

176 irls than boys had an increased incidence of eating disorders: for each 10% increase in the proportio

177 The eating disorder groups also showed reduced white matter

178 All eating disorder groups exhibited increased gray matter v

179 predicted sensitivity to reward in all three eating disorder groups.

180 er criteria and had moderate to severe binge eating disorder (>/=3 binge-eating days per week for 14

181 Individuals with any eating disorder had an increased risk (reported as odds

182 uals (index) who had a full sibling with any eating disorder had an increased risk of suicide attempt

183 The conceptual framework of the cause of eating disorders has undergone great changes in the past

184 rweight and higher BMIs at various stages of eating disorders have been confirmed repeatedly.

185 Eating disorders have been increasing over the past 50 y

186 aracteristics, assessment, and mortality for eating disorders have been reported independently for ma

187 The risk was attenuated for any eating disorder in more-distant relatives (maternal half

188 findings support a distinct subtype of binge eating disorder in obesity with similarities in risk-tak

189 We aimed to investigate whether rates of eating disorders in 16-20-year-old girls vary between up

190 out the epidemiology, course, and outcome of eating disorders in accordance with the fifth edition of

191 This Review describes what is known about eating disorders in adolescents with chronic gastrointes

192 orthern European groups; increasing rates of eating disorders in Asia; increasing rates of eating dis

193 The area of feeding and eating disorders in children remains relatively under-re

194 in journal articles relating to feeding and eating disorders in children, making a succinct overview

195 ure about recent trends in the occurrence of eating disorders in different cultures.

196 m genomic and neuroimaging investigations of eating disorders in humans presents a rich opportunity t

197 Research in eating disorders in males has been active lately compare

198 arental education showed no association with eating disorders in males, but twin or triplet status an

199 verview of the recently published studies of eating disorders in males.

200 hasize the need for further investigation of eating disorders in military service members.

201 ating disorders in Asia; increasing rates of eating disorders in the Arab region; and increasing rate

202 high prevalence and incidence of obesity and eating disorders in US adolescent girls are serious heal

203 that most commonly mimic the presentation of eating disorders including Crohn disease (CrD), celiac d

204 disease-specific roles in the development of eating disorders, including via perinatal variation with

205 The Internet and Binge Eating Disorder (INTERBED) study is a prospective, multi

206 The majority of epigenetic analyses of eating disorders investigated methylation at candidate g

207 Binge eating disorder is an addiction-like disorder characteri

208 Binge-eating disorder is characterized by excessive, uncontrol

209 Identifying risk factors specific to each eating disorder is critical for advancing etiologic know

210 nderstanding the underpinning biology of the eating disorder is important, as well as potential co-oc

211 eating behavior.SIGNIFICANCE STATEMENT Binge-eating disorder is the most common eating disorder world

212 Binge-eating disorder is the most common eating disorder.

213 mproved understanding of the neural basis of eating disorders is a timely challenge because these dis

214 of the timing and predictors of mortality in eating disorders is limited.

215 ted of 5 factors: internalizing (anxiety and eating disorders, major depressive episode, and cluster

216 Clinically diagnosed eating disorders may have adverse cardiometabolic conseq

217 The presentation form of eating disorders might vary for men versus women, for ex

218 (n = 13), bulimia nervosa (n = 6), and binge-eating disorder (n = 1), published between January 2003

219 153), substance use disorders (N = 131), and eating disorders (N = 14)-who failed to increase their p

220 A broader range of eating disorders needs to be defined to diagnose these i

221 ent for an eating disorder are classified as eating disorder not otherwise specified based on DSM-IV

222 m for anorexia nervosa, bulimia nervosa, and eating disorder not otherwise specified until the end of

223 The residual category 'eating disorder not otherwise specified' (EDNOS) was the

224 bulimia nervosa, binge-eating disorder, and eating disorder not otherwise specified) for probands an

225 gned to the heterogeneous residual category, eating disorder not otherwise specified, which provides

226 likely to exhibit binge eating disorder and eating disorder not otherwise specified.

227 considerable potential for the treatment of eating disorders, obesity, and/or diabetes.

228 Anorexia nervosa (AN) is an eating disorder observed predominantly in women and girl

229 iagnoses for anorexia nervosa (AN) and other eating disorders (OED: bulimia nervosa, binge-eating dis

230 xt Revision (DSM-IV-TR) section 'Feeding and Eating Disorders of Infancy or Early Childhood'; clarifi

231 ood consumption is fundamental for life, and eating disorders often result in devastating or life-thr

232 s index >=18.5, met criteria for DSM-5 binge eating disorder or bulimia nervosa, had 12 months of con

233 tion to maladaptive feeding behavior seen in eating disorders or obesity may arise from dysregulation

234 No studies were identified on eating disorders or puerperal psychosis.

235 , PD (OR: 1.46; 95% CI: 1.23, 1.74), and any eating disorder (OR: 1.32; 95% CI: 1.12, 1.56), but not

236 t may be useful targets for sleep disorders, eating disorders, or addictive behavior.

237 al well-being, lower risk of mental illness, eating disorders, overweight or obesity and marijuana us

238 uits and neurotransmitters involved in binge-eating disorder pathology and identify RXFP3 as a therap

239 ether these GI symptom factors (clusters) in eating disorder patients hold true to the Rome II classi

240 The GI symptoms common in eating disorder patients very likely represent the same

241 onal constipation, however, are prominent in eating disorder patients.

242 oss the genome of anorexia nervosa and binge-eating disorder patients.

243 in high status clothes) or non-aspirational (eating disordered patients in grey leotards), or to comb

244 on-affective psychosis, affective psychosis, eating disorders, personality disorders, alcohol misuse

245 ers, schizophrenia, mood disorders, anxiety, eating disorders, personality disorders, mental retardat

246 he number of OBE days, abstinence rates, and eating disorder psychopathologic findings and may be a b

247 Reductions in eating disorder psychopathologic findings were significa

248 Eating disorder psychopathology and psychological distre

249 vosa (AN), its influence in women with AN on eating disorder psychopathology and psychological distre

250 shown between body-composition variables and eating disorder psychopathology in the AN group, and the

251 does not, however, seem to influence either eating disorder psychopathology or psychological distres

252 s of touch was not modulated by subthreshold eating disorder psychopathology.

253 The changes to eating disorders, recommended by the Eating Disorders Wo

254 However, eating disorders remain understudied in military samples

255 The field of epigenetics in eating disorders remains in its infancy.

256 Binge eating disorder represents a public health problem at le

257 ered eating and body image, older women with eating disorder resemble younger women with similar cond

258 He started Pica eating disorder (sand and sponge) due to anemia from 5 y

259 related and addictive disorders, feeding and eating disorders, schizophrenia, anxiety disorder, OCD,

260 th conditions, including substance abuse and eating disorders, seem to be exacerbated or triggered in

261 anticipation of rewards, subjects with binge eating disorder show greater risk-taking, similar to sub

262 c, stress-related, and somatoform disorders; eating disorders; specific personality disorders; and a

263 eflect the changing landscape of culture and eating disorders: stabilization of the incidence of anor

264 These results may have implications for eating disorders such as anorexia nervosa (AN), in which

265 as a therapeutic target for the treatment of eating disorders such as anorexia nervosa.

266 Yet, males tended to score lower on eating disorder symptom measures than females.

267 ed with 56.8%; number needed to treat=5.11), eating disorder symptoms (body shape, weight, eating con

268 cues) rBN subjects had a greater increase in eating disorder symptoms during CD compared with healthy

269 associated with vulnerability for developing eating disorder symptoms in response to reduced catechol

270 reatment groups for the primary outcome, for eating disorder symptoms or comorbid psychiatric disorde

271 ry to earlier suggestions, no differences in eating disorder symptoms such as binging, vomiting, or l

272 ian of 9.5 years to obtain weekly ratings of eating disorder symptoms, comorbidity, treatment partici

273 This article reviews the modifications to eating disorders that appear in the 'Feeding and Eating

274 is a highly heritable trait associated with eating disorders that is comorbid with mood and substanc

275 ith a lack of control over eating, and binge eating disorder, the Diagnostic and Statistical Manual-5

276 nt body of evidence on epigenetic factors in eating disorders to inform future directions in this are

277 of topics relevant to childhood feeding and eating disorders, to include: presentation, diagnosis an

278 in standard care, none of whom received any eating disorder treatment during the intervention period

279 However, widespread access to effective eating disorder treatments remains limited.

280 zes, and to broaden the focus to include all eating disorder types.

281 Inpatients in a specialised eating disorder unit completed the Rome II questionnaire

282 first study to investigate whether rates of eating disorders vary between schools; however, use of r

283 Clinical impression is that rates of eating disorders vary between schools; we are not aware

284 Neither height nor history of an eating disorder was associated with menopausal age.

285 Neither depression nor binge eating disorder was consistently associated with differe

286 ion of girls at a school, the odds ratio for eating disorders was 1.07 (95% CI 1.01-1.13, p=0.017).

287 post-secondary education, the odds ratio for eating disorders was 1.14 (1.09-1.19, p<0.0001).

288 The fourth pattern, seen in eating disorders, was directly opposite of that observed

289 motor-disturbances, night-time behavior and eating disorders were also worse in the institutionalize

290 Eating disorders were captured by 3 variables (any eatin

291 While historically, eating disorders were conceptualized as primarily afflic

292 ly controlled research of brain structure in eating disorders, which will ultimately help predict the

293 Anorexia nervosa is a complex eating disorder with genetic, metabolic, and psychosocia

294 uding drug abuse, behavioral addictions, and eating disorders with binge features.

295 hed treatments for bulimia nervosa and binge eating disorder, with stepped-care approaches showing pr

296 nges to eating disorders, recommended by the Eating Disorders Work Group, aim to clarify existing cri

297 vosa Genetics Initiative (ANGI)(8,9) and the Eating Disorders Working Group of the Psychiatric Genomi

298 ENT Binge-eating disorder is the most common eating disorder worldwide, affecting women twice as freq

299 l and social/interpersonal issues underlying eating disorders would increase treatment efficacy.

300 sive eating characterizes many binge-related eating disorders, yet its neurobiological basis is poorl